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Journal Article
Diabetes, Obesity and Metabolism, ISSN 1462-8902, 09/2017, Volume 19, Issue Suppl 1, pp. 124 - 136
The progressive loss of pancreatic β‐cell mass that occurs in both type 1 and type 2 diabetes is a primary factor driving efforts to identify strategies for... 
beta‐cell | islets | beta-cell | Glucagon-Secreting Cells - immunology | Cell Proliferation | Species Specificity | Humans | Somatostatin-Secreting Cells - cytology | Extracellular Matrix - immunology | Diabetes Mellitus, Type 1 - metabolism | Extracellular Matrix - metabolism | Islets of Langerhans - innervation | Diabetes Mellitus, Type 2 - metabolism | Pancreatic Polypeptide-Secreting Cells - metabolism | Pancreatic Polypeptide-Secreting Cells - cytology | Insulin-Secreting Cells - metabolism | Glucagon-Secreting Cells - metabolism | Islets of Langerhans - cytology | Pancreatic Polypeptide-Secreting Cells - immunology | Somatostatin-Secreting Cells - immunology | Insulin-Secreting Cells - cytology | Pancreatic Polypeptide-Secreting Cells - pathology | Macrophages - immunology | Glucagon-Secreting Cells - cytology | Islets of Langerhans - blood supply | Somatostatin-Secreting Cells - pathology | Islets of Langerhans - pathology | Macrophages - pathology | Glucagon-Secreting Cells - pathology | Diabetes Mellitus, Type 1 - pathology | Cell Communication | Cellular Microenvironment | Macrophages - cytology | Insulin-Secreting Cells - immunology | Macrophages - metabolism | Animals | Models, Biological | Diabetes Mellitus, Type 2 - pathology | Insulin-Secreting Cells - pathology | Extracellular Matrix - pathology | Somatostatin-Secreting Cells - metabolism | Apoptosis | beta cell
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2015, Volume 10, Issue 12, p. e0144597
The transcription factor Pax6 is an important regulator of development and cell differentiation in various organs. Thus, Pax6 was shown to promote neural... 
IDENTITY | ALPHA-CELLS | MOUSE PANCREAS | MULTIDISCIPLINARY SCIENCES | BETA-CELLS | INSULIN-SECRETION | 7B2 | MICE | ENZYMATIC-ACTIVITY | DIFFERENTIATION | EXPRESSION | Paired Box Transcription Factors - genetics | Homeodomain Proteins - metabolism | Somatostatin-Secreting Cells - cytology | Male | Cell Lineage - drug effects | Pancreatic Polypeptide-Secreting Cells - metabolism | Pancreatic Polypeptide-Secreting Cells - cytology | Glucagon-Secreting Cells - drug effects | Insulin-Secreting Cells - metabolism | Gene Expression Regulation, Developmental | Glucagon-Secreting Cells - metabolism | Somatostatin-Secreting Cells - drug effects | Female | Integrases - metabolism | Cell Differentiation | Insulin-Secreting Cells - cytology | Eye Proteins - genetics | Insulin - genetics | Cell Lineage - genetics | Genes, Reporter | Repressor Proteins - metabolism | Glucagon-Secreting Cells - cytology | Signal Transduction | Bacterial Proteins - genetics | Ghrelin - genetics | Repressor Proteins - genetics | PAX6 Transcription Factor | Pancreatic Polypeptide-Secreting Cells - drug effects | Homeodomain Proteins - genetics | Mice, Knockout | Ghrelin - metabolism | Insulin - metabolism | Animals | Eye Proteins - metabolism | Insulin-Secreting Cells - drug effects | Tamoxifen - pharmacology | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | Integrases - genetics | Somatostatin-Secreting Cells - metabolism | Paired Box Transcription Factors - metabolism | Crosses, Genetic | Luminescent Proteins - metabolism | Care and treatment | Research | Glucagon | Pancreas | Ghrelin | Risk factors | Spinal cord | Cerebral cortex | Transcription factors | Deactivation | Maturation | Developmental biology | Genes | Cortex | Organs | Glucose | Kinases | Cell differentiation | Inactivation | Insulin | Proteins | Pax6 protein | Rodents | Diabetes | Alzheimers disease | Life Sciences | Development Biology
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, p. e0157138
Journal Article
Diabetes, Obesity and Metabolism, ISSN 1462-8902, 09/2016, Volume 18, Issue S1, pp. 10 - 22
During embryonic development, endocrine cells of the pancreas are specified from multipotent progenitors. The transcription factor Neurogenin 3 (NEUROG3) is... 
antibodies | diabetes | endocrine progenitors | pancreas | Neurogenin 3 | PROGENITOR CELLS | HUMAN PANCREAS DEVELOPMENT | FATE SPECIFICATION | CONGENITAL MALABSORPTIVE DIARRHEA | MUTANT NEUROGENIN-3 | BETA-LIKE CELLS | ISLET TRANSPLANTATION | ENDOCRINE PANCREAS | IN-VITRO | EMBRYONIC STEM-CELLS | ENDOCRINOLOGY & METABOLISM | Immunohistochemistry | Glucagon-Secreting Cells - cytology | Basic Helix-Loop-Helix Transcription Factors - physiology | Nerve Tissue Proteins - physiology | Basic Helix-Loop-Helix Transcription Factors - genetics | Humans | Somatostatin-Secreting Cells - cytology | Gene Expression Regulation, Developmental - genetics | Antibodies, Monoclonal | Nerve Tissue Proteins - genetics | Pancreatic Polypeptide-Secreting Cells - metabolism | Cellular Reprogramming | Pancreatic Polypeptide-Secreting Cells - cytology | Nerve Tissue Proteins - metabolism | Cell Differentiation - genetics | Insulin-Secreting Cells - metabolism | Animals | Basic Helix-Loop-Helix Transcription Factors - metabolism | Islets of Langerhans - metabolism | Glucagon-Secreting Cells - metabolism | Islets of Langerhans - cytology | Insulin-Secreting Cells - cytology | Mice | Somatostatin-Secreting Cells - metabolism | Monoclonal antibodies | Embryonic development | Cytochemistry | Stem cells | Antigenic determinants | Pancreas | Rodents
Journal Article
Pancreas, ISSN 0885-3177, 2017, Volume 46, Issue 6, pp. 820 - 824
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e72213
Mutations in the human homolog of the Vhlh gene [encoding the von-Hippel Lindau (VHL) protein] lead to tumor development. In mice, depletion of Vhlh in... 
GLUCAGON-SECRETION | REGULATOR | ALPHA-CELLS | GLUCOSE | STABILIZATION | MULTIDISCIPLINARY SCIENCES | BETA-CELLS | IN-VIVO | INSULIN-SECRETION | GENE-EXPRESSION | HYPOXIA | Embryo, Mammalian | Hypoxia-Inducible Factor 1 - metabolism | Pancreatic Polypeptide-Secreting Cells - metabolism | Hypoxia - metabolism | Hypoxia-Inducible Factor 1 - genetics | Insulin-Secreting Cells - metabolism | Gene Expression Regulation, Developmental | Gene Deletion | Glucagon-Secreting Cells - metabolism | Endocrine System - metabolism | Pancreatic Polypeptide-Secreting Cells - pathology | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Somatostatin-Secreting Cells - pathology | Signal Transduction | Glucagon-Secreting Cells - pathology | Cells, Cultured | Endocrine System - pathology | Mice, Knockout | Hypoxia - genetics | Insulin - metabolism | Animals | Hypoxia - pathology | Glucose - metabolism | Glucagon - metabolism | Mice | Insulin-Secreting Cells - pathology | Somatostatin-Secreting Cells - metabolism | Homeostasis - genetics | Glucose metabolism | Glucagon | Physiological aspects | Homeostasis | Glucose | Insulin | Dextrose | Cell culture | Homology | Lethality | Defects | Rodents | Gastroenterology | Standard deviation | Pancreas | Secretion | Hypoglycemia | Metabolism | Gene expression | Epithelium | Embryos | Depletion | Hypoxia | VHL protein | Islets of Langerhans | Diabetes | Mutation | Neoplasia | Endocrinology
Journal Article
Journal Article