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1. Full Text Proposed Morphologic Classification of Prostate Cancer With Neuroendocrine Differentiation
by Epstein, Jonathan I and Amin, Mahul B and Beltran, Himisha and Lotan, Tamara L and Mosquera, Juan-Miguel and Reuter, Victor E and Robinson, Brian D and Troncoso, Patricia and Rubin, Mark A
The American journal of surgical pathology, ISSN 0147-5185, 06/2014, Volume 38, Issue 6, pp. 756 - 767
Prostate adenocarcinonma | Carcinoid | Paneth cell like | Small cell carcinoma | Large cell neuroendocrine carcinoma | Pathology | Life Sciences & Biomedicine | Surgery | Science & Technology | Immunohistochemistry | Predictive Value of Tests | Prostatic Neoplasms - chemistry | Adenocarcinoma - pathology | Prognosis | Neuroendocrine Tumors - chemistry | Adenocarcinoma - classification | Carcinoma, Large Cell - pathology | Humans | Carcinoma, Acinar Cell - pathology | Paneth Cells - pathology | Male | Prostatic Neoplasms - therapy | Carcinoma, Small Cell - classification | Neoplasms, Complex and Mixed - classification | Cell Differentiation | Adenocarcinoma - chemistry | Neuroendocrine Tumors - pathology | Prostatic Neoplasms - pathology | Biomarkers, Tumor - analysis | Terminology as Topic | Carcinoma, Acinar Cell - classification | Carcinoid Tumor - pathology | Neuroendocrine Tumors - classification | Adenocarcinoma - therapy | Neoplasms, Complex and Mixed - pathology | Prostatic Neoplasms - classification | Carcinoma, Large Cell - classification | Neuroendocrine Tumors - therapy | Paneth Cells - classification | Carcinoma, Small Cell - pathology | Carcinoid Tumor - classification | Index Medicus | prostate adenocarcinonma | small cell carcinoma | carcinoid | large cell neuroendocrine carcinoma
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2. Full Text Lineage Tracing Reveals Lgr5+Stem Cell Activity in Mouse Intestinal Adenomas
by Arnout G. Schepers and Hugo J. Snippert and Daniel E. Stange and Maaike van den Born and Johan H. van Es and Marc van de Wetering and Hans Clevers
Science (American Association for the Advancement of Science), ISSN 0036-8075, 8/2012, Volume 337, Issue 6095, pp. 730 - 735
Datasets | Architecture | Crypts | REPORTS | Stem cells | Alleles | Colorectal cancer | Cultured cells | Adenoma | Daughter cells | Tumors | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Biological and medical sciences | Medical sciences | Animal tumors. Experimental tumors | Experimental digestive system and abdominal tumors | Tumor Stem Cell Assay | Intestinal Mucosa - metabolism | Receptors, G-Protein-Coupled - analysis | Stem Cell Niche | Adenoma - genetics | Biomarkers - analysis | Paneth Cells - pathology | Gene Expression Profiling | Adenoma - metabolism | Gene Knock-In Techniques | Intestinal Neoplasms - pathology | Cell Lineage | Multipotent Stem Cells - pathology | Animals | Cell Transformation, Neoplastic | Tamoxifen - pharmacology | Adenoma - pathology | Neoplastic Stem Cells - pathology | Mice | Multipotent Stem Cells - physiology | Intestinal Neoplasms - genetics | Genes, Reporter | Neoplastic Stem Cells - physiology | Intestinal Mucosa - pathology | Genotype & phenotype | Gene expression | Rodents | Index Medicus | Receptors | Genes | Markers | Monitors | Online | Cancer
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3. Full Text Interleukin-22 promotes intestinal-stem-cell-mediated epithelial regeneration
by Lindemans, C.A and Calafiore, Marco and Mertelsmann, Anna M and O'Connor, Margaret H and Dudakov, Jarrod A and Jenq, Robert R and Velardi, Enrico and Young, Lauren F and Smith, Odette M and Lawrence, Gillian and Ivanov, Juliet A and Fu, Ya-Yuan and Takashima, Shuichiro and Hua, Guoqiang and Martin, Maria L and O'Rourke, Kevin P and Lo, Yuan-Hung and Mokry, Michal and Romera Hernández, Mónica and Cupedo, Tom and Dow, Lukas E and Nieuwenhuis, Edward and Shroyer, Noah F and Liu, Chen and Kolesnick, Richard and Van Den Brink, Marcel R. M and Hanash, Alan M
Nature (London), ISSN 0028-0836, 12/2015, Volume 528, Issue 7583, pp. 560 - 564
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Phosphorylation | Intestine, Small - pathology | Humans | Immunity, Mucosal | Stem Cells - cytology | Stem Cells - metabolism | Intestinal Mucosa - cytology | Intestinal Mucosa - immunology | Interleukins - immunology | Female | Epithelial Cells - cytology | Intestine, Small - immunology | STAT3 Transcription Factor - metabolism | Stem Cell Niche | Signal Transduction | Paneth Cells - cytology | Organoids - cytology | Epithelial Cells - pathology | Graft vs Host Disease - pathology | Regeneration | Animals | Intestine, Small - cytology | Organoids - growth & development | Epithelial Cells - immunology | Interleukins - deficiency | Mice | Organoids - immunology | Intestinal Mucosa - pathology | Physiological aspects | Regeneration (Biology) | Interleukins | Epithelial cells | Health aspects | Epidermal growth factor | Transplants & implants | Cytokines | Large intestine | Rodents | Cell cycle | Gene expression | Small intestine | Immune system | Index Medicus
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4. Full Text RSPO3 expands intestinal stem cell and niche compartments and drives tumorigenesis
by Hilkens, John and Timmer, Nikki C and Boer, Mandy and Ikink, Gerjon J and Schewe, Matthias and Sacchetti, Andrea and Koppens, Martijn A J and Song, Ji-Ying and Bakker, Elvira R M
Gut, ISSN 0017-5749, 06/2017, Volume 66, Issue 6, pp. 1095 - 1105
INTESTINAL STEM CELL | CANCER | SIGNALING | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Adenocarcinoma - pathology | Thrombospondins - genetics | Adenoma - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Paneth Cells - pathology | Intestines - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Wnt Proteins - metabolism | Cell Movement - genetics | Hyperplasia - genetics | Adenocarcinoma - genetics | Intestinal Neoplasms - genetics | Wnt Signaling Pathway | Paneth Cells - metabolism | Cell Proliferation - genetics | Gene Expression | Intestines - pathology | Organoids - pathology | Receptors, G-Protein-Coupled - analysis | Carcinogenesis - genetics | Mice, Transgenic | Cell Enlargement | Stem Cells - chemistry | beta Catenin - metabolism | Intestinal Neoplasms - pathology | Hyperplasia - pathology | Animals | Adenoma - pathology | Stem Cells - pathology | Mice | Mutation | Thrombospondins - metabolism | Research | Gene expression | Gastrointestinal cancer | Carcinogenesis | Stem cells | Animal models | Wnt protein | Cross-breeding | Genes | Colorectal carcinoma | Colorectal cancer | Transgenic mice | Stem cell transplantation | Paneth cells | Polyposis coli | Adenomatous polyposis coli | Genotype & phenotype | β-catenin | Intestine | Gastroenterology | Xenografts | Tumorigenesis | Tumors | Index Medicus | Abridged Index Medicus | GI cancer | 1506
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5. Full Text RIPK1 maintains epithelial homeostasis by inhibiting apoptosis and necroptosis
by Dannappel, Marius and Vlantis, Katerina and Kumari, Snehlata and Polykratis, Apostolos and Kim, Chun and Wachsmuth, Laurens and Eftychi, Christina and Lin, Juan and Corona, Teresa and Hermance, Nicole and Zelic, Matija and Kirsch, Petra and Basic, Marijana and Bleich, Andre and Kelliher, Michelle and Pasparakis, Manolis
Nature (London), ISSN 0028-0836, 2014, Volume 513, Issue 7516, pp. 90 - 94
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Inflammation - pathology | Skin - cytology | Epithelial Cells - metabolism | Skin - metabolism | Caspase 8 - metabolism | Homeostasis | Paneth Cells - pathology | Male | Intestines - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Necrosis | Receptors, Tumor Necrosis Factor, Type I - deficiency | Inflammation - metabolism | Female | Epithelial Cells - cytology | Skin - pathology | Paneth Cells - metabolism | Intestines - pathology | Cell Survival | Fas-Associated Death Domain Protein - metabolism | Fas-Associated Death Domain Protein - deficiency | Epithelial Cells - pathology | Mice, Knockout | Keratinocytes - pathology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Keratinocytes - metabolism | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Myeloid Differentiation Factor 88 - metabolism | Apoptosis | Intestines - cytology | Physiological aspects | Physiological research | Inflammation | Research | Protein kinases | Pathology | Antibiotics | Rodents | Skin | Kinases | Index Medicus
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6. Full Text Intestinal label-retaining cells are secretory precursors expressing Lgr5
by Buczacki, Simon J. A and Zecchini, Heather Ireland and Nicholson, Anna M and Russell, Roslin and Vermeulen, Louis and Kemp, Richard and Winton, Douglas J
Nature (London), ISSN 0028-0836, 2013, Volume 495, Issue 7439, pp. 65 - 69
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Biomarkers - metabolism | Paneth Cells - metabolism | Clone Cells - cytology | Intestines - pathology | Stem Cell Niche | Multipotent Stem Cells - secretion | Receptors, G-Protein-Coupled - metabolism | Cell Separation | Multipotent Stem Cells - metabolism | Paneth Cells - cytology | Biomarkers - analysis | Intestines - injuries | Clone Cells - metabolism | Intestinal Neoplasms - pathology | Cell Lineage | Regeneration | Animals | Multipotent Stem Cells - cytology | Cell Division | Staining and Labeling | Cell Differentiation | Mice | Intestines - cytology | Codon | Epithelial cells | Stem cells | Research | Leucine | Properties | Gene expression | Principal components analysis | Microscopy | Rodents | Methods | Cell cycle | Index Medicus
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7. Full Text FADD prevents RIP3-mediated epithelial cell necrosis and chronic intestinal inflammation
by Welz, Patrick-Simon and Wullaert, Andy and Vlantis, Katerina and Kondylis, Vangelis and Fernández-Majada, Vanesa and Ermolaeva, Maria and Kirsch, Petra and Sterner-Kock, Anja and Van Loo, Geert and Pasparakis, Manolis
Nature (London), ISSN 0028-0836, 09/2011, Volume 477, Issue 7364, pp. 330 - 334
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Gastroenterology. Liver. Pancreas. Abdomen | Stomach. Duodenum. Small intestine. Colon. Rectum. Anus | Biological and medical sciences | Medical sciences | Other diseases. Semiology | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Colon - enzymology | Epithelial Cells - metabolism | Paneth Cells - pathology | Colitis - pathology | Intracellular Signaling Peptides and Proteins - metabolism | Inflammatory Bowel Diseases - metabolism | Metagenome - physiology | Necrosis | Intracellular Signaling Peptides and Proteins - deficiency | Cysteine Endopeptidases - metabolism | Inflammatory Bowel Diseases - enzymology | Inflammatory Bowel Diseases - pathology | Enteritis - metabolism | Signal Transduction | Colon - pathology | Fas-Associated Death Domain Protein - metabolism | Fas-Associated Death Domain Protein - deficiency | Epithelial Cells - pathology | Enteritis - pathology | Tumor Necrosis Factors - deficiency | Colon - metabolism | Animals | Colitis - enzymology | Myeloid Differentiation Factor 88 - deficiency | Enteritis - enzymology | Colitis - metabolism | Epithelial Cells - enzymology | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Chronic Disease | Myeloid Differentiation Factor 88 - metabolism | Apoptosis | Proteins | Inflammatory bowel disease | Pathogenesis | Colleges & universities | Homeostasis | Colon | Index Medicus
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8. Full Text Yap-dependent reprogramming of Lgr5+ stem cells drives intestinal regeneration and cancer
by Gregorieff, Alex and Liu, Yu and Inanlou, Mohammad R and Khomchuk, Yuliya and Wrana, Jeffrey L
Nature (London), ISSN 0028-0836, 10/2015, Volume 526, Issue 7575, pp. 715 - 718
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Intestinal Mucosa - metabolism | Neoplastic Stem Cells - cytology | Receptors, G-Protein-Coupled - metabolism | Intestines - radiation effects | Male | Stem Cells - cytology | Phosphoproteins - metabolism | Stem Cells - metabolism | Colonic Neoplasms - metabolism | Regeneration - radiation effects | Adenoma - metabolism | Neoplastic Stem Cells - metabolism | Organoids - metabolism | Neoplastic Stem Cells - pathology | Female | Epiregulin - metabolism | Protein-Serine-Threonine Kinases - metabolism | Wnt Signaling Pathway | Disease Models, Animal | Radiation, Ionizing | Cell Differentiation - radiation effects | ErbB Receptors - metabolism | Paneth Cells - cytology | Stem Cells - radiation effects | Homeostasis - radiation effects | Cell Survival - radiation effects | Animals | Cell Transformation, Neoplastic | Adaptor Proteins, Signal Transducing - deficiency | Colonic Neoplasms - pathology | Phosphoproteins - deficiency | Adenoma - pathology | Mice | Paneth Cells - radiation effects | Adaptor Proteins, Signal Transducing - metabolism | Cell Cycle Proteins | Intestines - cytology | Index Medicus
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9. Full Text Replacement of Lost Lgr5-Positive Stem Cells through Plasticity of Their Enterocyte-Lineage Daughters
by Tetteh, Paul W and Basak, Onur and Farin, Henner F and Wiebrands, Kay and Kretzschmar, Kai and Begthel, Harry and van den Born, Maaike and Korving, Jeroen and de Sauvage, Frederic and van Es, Johan H and van Oudenaarden, Alexander and Clevers, Hans
Cell stem cell, ISSN 1934-5909, 02/2016, Volume 18, Issue 2, pp. 203 - 213
Cell & Tissue Engineering | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Biomarkers - metabolism | Paneth Cells - metabolism | Cell Line | Cell Proliferation | Receptors, G-Protein-Coupled - metabolism | Enterocytes - metabolism | Alkaline Phosphatase - metabolism | Cell Dedifferentiation | Paneth Cells - pathology | Mutation - genetics | Stem Cells - metabolism | Intestinal Neoplasms - pathology | Cell Lineage | Animals | Organoids | Regeneration - genetics | beta-Galactosidase - metabolism | Integrases - metabolism | Enterocytes - pathology | Mice | Single-Cell Analysis | Index Medicus
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