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Orphanet Journal of Rare Diseases, ISSN 1750-1172, 11/2014, Volume 9, Issue 1, p. 160
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2016, Volume 126, Issue 4, pp. 1311 - 1322
Journal Article
Circulation Research, ISSN 0009-7330, 09/2010, Volume 107, Issue 5, pp. 650 - 658
Journal Article
British Journal of Haematology, ISSN 0007-1048, 05/2018, Volume 181, Issue 3, pp. 406 - 410
Journal Article
Mediators of Inflammation, ISSN 0962-9351, 2010, Volume 2010, pp. 173498 - 9
Potential links between metabolic derangements and adipose tissue (AT) inflammation in patients with chronic obstructive pulmonary disease (COPD) are... 
SYSTEMIC INFLAMMATION | SKELETAL-MUSCLE | CANCER CACHEXIA | OBESITY | INSULIN-RESISTANCE | SENSITIVITY | NECROSIS-FACTOR-ALPHA | BODY-COMPOSITION | TNF-ALPHA | IMMUNOLOGY | COPD PATIENTS | CELL BIOLOGY | Cachexia - complications | Inflammation - pathology | Overweight - complications | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Male | Metabolic Syndrome - metabolism | Pulmonary Disease, Chronic Obstructive - pathology | Panniculitis - complications | Antigens, CD - genetics | Obesity - genetics | Panniculitis - genetics | Overweight - metabolism | Insulin Resistance - physiology | Caspase 3 - genetics | Inflammation Mediators - metabolism | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Cachexia - metabolism | bcl-2-Associated X Protein - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Gene Expression | Overweight - pathology | Cachexia - genetics | Interleukin-6 - genetics | Obesity - complications | Adipose Tissue - pathology | Overweight - genetics | Pulmonary Disease, Chronic Obstructive - complications | Receptors, Tumor Necrosis Factor, Type I - genetics | Antigens, Differentiation, Myelomonocytic - genetics | Metabolic Syndrome - complications | Obesity - metabolism | Obesity - pathology | Phenotype | Metabolic Syndrome - genetics | Cachexia - pathology | Aged | Panniculitis - pathology | Panniculitis - metabolism
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2016, Volume 113, Issue 36, pp. 10127 - 10132
Systemic autoinflammatory diseases are caused by mutations in genes that function in innate immunity. Here, we report an autoinflammatory disease caused by... 
Autoinflammatory disease | Cytokines | Nf-κb pathway | Linear deubiquitinase | OTULIN | ACTIVATION | AMYLOPECTINOSIS | NF-kappa B pathway | UBIQUITIN | MULTIDISCIPLINARY SCIENCES | linear deubiquitinase | ASSEMBLY COMPLEX | CELL-DEATH | HOIP | SHARPIN | autoinflammatory disease | INFLAMMATION | LUBAC DEFICIENCY | cytokines | IMMUNODEFICIENCY | Fibroblasts - enzymology | Humans | NF-kappa B - immunology | Child, Preschool | Male | Fever - physiopathology | Leukocytes, Mononuclear - immunology | HEK293 Cells | Female | Child | Cytokines - genetics | Failure to Thrive - physiopathology | Hereditary Autoinflammatory Diseases - pathology | Cytokines - immunology | Dermatitis - physiopathology | Signal Transduction | Hereditary Autoinflammatory Diseases - enzymology | Gene Expression Regulation | Ubiquitin - genetics | Endopeptidases - deficiency | Hereditary Autoinflammatory Diseases - diagnosis | Fibroblasts - pathology | Endopeptidases - immunology | Leukocytes, Mononuclear - pathology | Endopeptidases - genetics | NF-kappa B - genetics | Pedigree | Age of Onset | Alleles | Fibroblasts - immunology | Leukocytes, Mononuclear - enzymology | Panniculitis - physiopathology | Consanguinity | Mutation | Ubiquitin - immunology | Hereditary Autoinflammatory Diseases - genetics | Allelomorphism | Genetic aspects | Gene mutations | Autoimmune diseases | Health aspects | Biological Sciences | NF-κB pathway
Journal Article
Journal Article
Journal Article
Journal of Lipid Research, ISSN 0022-2275, 01/2016, Volume 57, Issue 1, pp. 56 - 65
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2012, Volume 109, Issue 18, pp. 7049 - 7054
Type 2 diabetes (T2D) is a complex, polygenic disease affecting nearly 300 million people worldwide. T2D is primarily characterized by insulin resistance, and... 
Adipose tissues | Obesity | Receptors | Insulin resistance | Genetic loci | Inflammation | Type 2 diabetes mellitus | Diabetes | Macrophages | Insulin | Hyperglycemia | Integration | Bioinformatics | Meta-analysis | LEUKOCYTE RECRUITMENT | MULTIDISCIPLINARY SCIENCES | meta-analysis | SUSCEPTIBILITY LOCI | CLASSIFICATION | MELLITUS | IDENTIFICATION | hyperglycemia | INSULIN-RESISTANCE | INFLAMMATION | GLUCOSE | bioinformatics | integration | GENE-EXPRESSION | MICE | obesity | Oligonucleotide Array Sequence Analysis | Obesity - immunology | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Male | Adipose Tissue - immunology | Panniculitis - complications | Obesity - genetics | Case-Control Studies | Panniculitis - genetics | Diabetes Mellitus, Type 2 - immunology | Dietary Fats - administration & dosage | Hyaluronan Receptors - metabolism | Diabetes Mellitus, Type 2 - etiology | Female | Panniculitis - immunology | Macrophages - immunology | Gene Expression | Genome-Wide Association Study | Macrophages - pathology | Obesity - complications | Mice, Inbred C57BL | Adipose Tissue - pathology | Hyaluronan Receptors - genetics | Mice, Knockout | Animals | Insulin Resistance - immunology | Aged | Mice | Panniculitis - pathology | Blood Glucose - metabolism | Type 2 diabetes | Genomics | Physiological aspects | Development and progression | Genetic aspects | Research | Gene expression | Health aspects | Biological Sciences
Journal Article