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Circulation Research, ISSN 0009-7330, 09/2015, Volume 117, Issue 8, pp. 720 - 730
RATIONALE:Tissue engineering approaches may improve survival and functional benefits from human embryonic stem cell–derived cardiomyocyte transplantation,... 
Myocardial infarction | Cell survival | Cardiac function tests | Tissue engineering | Myocardial ischemia | Transplantation | Cardiac MRI | tissue engineering | cell survival | cardiac MRI | myocardial infarction | transplantation | myocardial ischemia | cardiac function tests | CARDIAC & CARDIOVASCULAR SYSTEMS | TISSUE | CARDIOMYOPATHY | CELL-DERIVED CARDIOMYOCYTES | COCULTURE | SHEETS | CARDIAC RHABDOMYOMA | RAT HEARTS | REPAIR | EMBRYONIC STEM-CELLS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Embryonic Stem Cells - metabolism | Myocytes, Cardiac - immunology | Humans | Male | Papillary Muscles - transplantation | Rats, Nude | Myocardial Infarction - immunology | Heterografts | Transfection | Time Factors | Myocardial Infarction - pathology | Myocardial Infarction - physiopathology | Papillary Muscles - metabolism | Cell Differentiation | Embryonic Stem Cells - immunology | Immunosuppressive Agents - pharmacology | Disease Models, Animal | Biomarkers - metabolism | Myocardial Contraction | Myocardial Infarction - surgery | Cell Line | Tissue Engineering - methods | Connexin 43 - metabolism | Papillary Muscles - immunology | Cell Survival | Heart Transplantation - adverse effects | Graft Survival | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Stroke Volume | Myocytes, Cardiac - pathology | Myocytes, Cardiac - transplantation | Papillary Muscles - physiopathology | Animals | Heart Transplantation - methods | Embryonic Stem Cells - transplantation | Myocytes, Cardiac - metabolism | Papillary Muscles - pathology | Index Medicus | Human embryonic stem cells | engineered heart muscle | cardiac magnetic resonance imaging | cardiac function
Journal Article
Circulation, ISSN 0009-7322, 08/2015, Volume 132, Issue 7, pp. 556 - 566
Journal Article
The Annals of Thoracic Surgery, ISSN 0003-4975, 09/2017, Volume 104, Issue 3, pp. e247 - e249
Abnormal papillary muscle is a rare cause of midventricular obstruction. In this case report, hypertrophied abnormal papillary muscles and abnormal tissue... 
SURGERY | RESPIRATORY SYSTEM | CARDIAC & CARDIOVASCULAR SYSTEMS | Humans | Hypertrophy, Left Ventricular - complications | Adult | Female | Papillary Muscles - pathology | Ventricular Outflow Obstruction - etiology | Ventricular Septum - pathology
Journal Article
Circulation: Cardiovascular Imaging, ISSN 1941-9651, 05/2015, Volume 8, Issue 7, p. e003132
Background-In patients with hypertrophic cardiomyopathy and left ventricular outflow tract (LVOT) obstruction, but without basal septal hypertrophy, we sought... 
Mitral valve | Echocardiography | Magnetic resonance imaging | Papillary muscles | Cardiomyopathy | Hypertrophic | Surgery | Multimodal imaging | papillary muscles | magnetic resonance imaging | CARDIAC & CARDIOVASCULAR SYSTEMS | cardiomyopathy, hypertrophic | echocardiography | mitral valve | multimodal imaging | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | surgery | Multivariate Analysis | Predictive Value of Tests | Cardiomyopathy, Hypertrophic - surgery | Ventricular Function, Left | Humans | Middle Aged | Male | Mitral Valve - surgery | Echocardiography, Doppler, Color | Ventricular Outflow Obstruction - diagnosis | Mitral Valve - physiopathology | Papillary Muscles - surgery | Papillary Muscles - diagnostic imaging | Ventricular Outflow Obstruction - pathology | Adult | Female | Registries | Retrospective Studies | Ventricular Outflow Obstruction - diagnostic imaging | Cardiomyopathy, Hypertrophic - diagnostic imaging | Ventricular Outflow Obstruction - surgery | Risk Factors | Linear Models | Treatment Outcome | Magnetic Resonance Imaging, Cine | Chi-Square Distribution | Cardiomyopathy, Hypertrophic - diagnosis | Stroke Volume | Papillary Muscles - physiopathology | Ventricular Outflow Obstruction - physiopathology | Mitral Valve - diagnostic imaging | Cardiomyopathy, Hypertrophic - physiopathology | Aged | Mitral Valve - pathology | Ohio | Papillary Muscles - pathology | Cardiomyopathy, Hypertrophic - pathology
Journal Article
Journal Article
European Urology, ISSN 0302-2838, 2016, Volume 71, Issue 3, pp. 447 - 461
Journal Article
Circulation Research, ISSN 0009-7330, 09/2017, Volume 121, Issue 8, pp. 923 - 929
RATIONALE:Duchenne muscular dystrophy is a severe inherited form of muscular dystrophy caused by mutations in the reading frame of the dystrophin gene... 
Cardiomyopathies | Mice | Dystrophin | Gene editing | Muscular dystrophy | FIBROSIS | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIOMYOPATHY | dystrophin | gene editing | mice | DELIVERY | NUCLEASES | muscular dystrophy | THERAPY | MOUSE MODEL | MUSCLE FUNCTION | PERIPHERAL VASCULAR DISEASE | DUCHENNE MUSCULAR-DYSTROPHY | CAS9 | HEMATOLOGY | cardiomyopathies | Dependovirus - genetics | RNA, Guide - genetics | Exons | RNA, Guide - metabolism | Utrophin - genetics | Recovery of Function | Cardiomyopathies - genetics | Cardiomyopathies - physiopathology | Muscular Dystrophy, Duchenne - physiopathology | Clustered Regularly Interspaced Short Palindromic Repeats | Mice, Inbred mdx | Papillary Muscles - metabolism | Muscular Dystrophy, Duchenne - therapy | Dystrophin - metabolism | Disease Models, Animal | Myocardial Contraction | Genetic Predisposition to Disease | CRISPR-Associated Proteins - genetics | Gene Expression Regulation | Cardiomyopathies - therapy | Papillary Muscles - physiopathology | Phenotype | Animals | Dystrophin - genetics | CRISPR-Associated Proteins - metabolism | Cardiomyopathies - metabolism | CRISPR-Cas Systems | Fibrosis | Gene Editing - methods | High-Throughput Nucleotide Sequencing | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Mutation | Genetic Vectors | Papillary Muscles - pathology | Genetic Therapy - methods | Neonates | CRISPR | Cardiac muscle | Cardiomyopathy | Contractility | Genomes | Ribonucleic acid--RNA | Muscle contraction | Proteins | Rodents | Duchenne's muscular dystrophy | Protein expression | Dystrophy | Immunofluorescence | Heart diseases | genome editing | AAV | gene therapy | Duchenne muscular dystrophy cardiomyopathy | cardiomyopathy
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3670 - 3678
Patients with ischaemic heart disease or chronic heart failure show altered levels of obestatin, suggesting a role for this peptide in human heart function. We... 
cardioprotection | obestatin | ischaemia/reperfusion | coronary flow | myocardial contractility | post‐conditioning | post-conditioning | SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | ISOLATED RAT-HEART | INJURY | DES-ACYL GHRELIN | PEPTIDE | CELL BIOLOGY | ischaemia | HORMONE | PROTEIN-COUPLED RECEPTOR | ENDOTHELIAL-CELLS | reperfusion | Endothelin-1 - antagonists & inhibitors | Myocardial Ischemia - genetics | Myocardial Infarction - genetics | Myocardial Ischemia - metabolism | Peptide Hormones - pharmacology | Rats, Wistar | Soluble Guanylyl Cyclase - metabolism | Cardiotonic Agents - metabolism | Endothelin-1 - pharmacology | Myocardial Contraction - drug effects | Male | Phosphatidylinositol 3-Kinases - metabolism | Peptide Hormones - genetics | Cyclic GMP-Dependent Protein Kinases - metabolism | Cyclic GMP-Dependent Protein Kinases - genetics | Proto-Oncogene Proteins c-akt - genetics | Peptide Hormones - metabolism | Potassium Channels - metabolism | Myocardial Reperfusion Injury - pathology | Soluble Guanylyl Cyclase - genetics | Myocardial Infarction - pathology | Papillary Muscles - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Myocardial Reperfusion Injury - genetics | Heart Ventricles - pathology | Organ Culture Techniques | Signal Transduction | Gene Expression Regulation | Myocardial Ischemia - prevention & control | Rats | Cardiotonic Agents - chemistry | Cardiotonic Agents - pharmacology | Myocardial Infarction - metabolism | Myocardial Ischemia - pathology | Potassium Channels - genetics | Phosphatidylinositol 3-Kinases - genetics | Myocardial Reperfusion Injury - metabolism | Animals | Heart Ventricles - metabolism | Myocardial Infarction - prevention & control | Nitric Oxide - metabolism | Papillary Muscles - drug effects | Papillary Muscles - pathology | Myocardial Reperfusion Injury - prevention & control | Heart Ventricles - drug effects | Endothelin | Heart diseases | Vasodilators | Nitric oxide | Reactive oxygen species | Protein kinase C | Peptides | AKT protein | Activation | Heart function | Guanylate cyclase | Signal transduction | Mitochondria | Reperfusion | Ischemia | Ghrelin | Cyclic GMP | Inotropism | Heart failure | Stresses | Cardiovascular system | Cardiac conditioning | Muscles | Pharmacology | Endothelin 1 | Potassium channels | Muscle contraction | Coronary artery disease | Stress | 1-Phosphatidylinositol 3-kinase | Signaling | Cell death | Myocardium | Growth hormone | ATP | Potassium | Original
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2014, Volume 64, Issue 18, pp. 1867 - 1879
Journal Article