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Journal Article
Journal Article
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 06/2012, Volume 36, Issue 6, pp. 799 - 807
Journal Article
Neoplasia, ISSN 1476-5586, 2014, Volume 16, Issue 11, pp. 961 - 971
Abstract PURPOSE: Foxhead box M1 (FOXM1) expression has been shown to be linked with human papillomavirus (HPV) 16/18–infected cervical cancer. However, the... 
Oncology | CELL LUNG-CANCER | TRANSFORMATION | TARGET | PROTEIN | VIRUS | ONCOLOGY | ADENOCARCINOMA | HUMAN-PAPILLOMAVIRUS-16/18 INFECTION | TRANSCRIPTION FACTOR | EXPRESSION | PROGRESSION | Up-Regulation | Alphapapillomavirus - physiology | Humans | Lung Neoplasms - metabolism | Forkhead Transcription Factors - metabolism | Mouth Neoplasms - genetics | Repressor Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - metabolism | Repressor Proteins - genetics | Mouth Neoplasms - virology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Human papillomavirus 16 - metabolism | Host-Pathogen Interactions | Alphapapillomavirus - metabolism | Mice, Nude | Survival Analysis | Cell Line, Tumor | Mice, Inbred BALB C | Kruppel-Like Transcription Factors - genetics | Neoplasms - metabolism | Thyroid Nuclear Factor 1 | Human papillomavirus 16 - physiology | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - virology | Mouth Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Neoplasms - virology | Neoplasms - genetics | Kruppel-Like Transcription Factors - metabolism | Human papillomavirus 18 - metabolism | Female | Oncogene Proteins, Viral - genetics | Carcinoma, Non-Small-Cell Lung - virology | Human papillomavirus 18 - genetics | Nuclear Proteins - genetics | Human papillomavirus 18 - physiology | Oncogene Proteins, Viral - metabolism | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Nuclear Proteins - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Forkhead Transcription Factors - genetics | Lung Neoplasms - virology | Transcription Factors - metabolism | Alphapapillomavirus - genetics | Animals | Forkhead Box Protein M1 | Human papillomavirus 16 - genetics
Journal Article
Oncogene, ISSN 0950-9232, 03/2017, Volume 36, Issue 9, pp. 1245 - 1255
Liver kinase B1 (LKB1) is mutationally inactivated in Peutz-Jeghers syndrome and in a variety of cancers including human papillomavirus (HPV)-caused cervical... 
PATHWAYS | ONCOPROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CERVICAL-CANCER | HUMAN-PAPILLOMAVIRUS | IDENTIFICATION | HEXOKINASE II | CELL BIOLOGY | LUNG-CANCER | ONCOLOGY | GROWTH | ROS | GENETICS & HEREDITY | TUMOR INITIATION | Cell Proliferation | Human papillomavirus 16 - physiology | Prognosis | Follow-Up Studies | Humans | Uterine Cervical Neoplasms - pathology | Case-Control Studies | Papillomavirus E7 Proteins - genetics | Papillomavirus Infections - pathology | Glycolysis - physiology | Uterine Cervical Neoplasms - metabolism | Biomarkers, Tumor - metabolism | Female | Oncogene Proteins, Viral - genetics | Papillomavirus Infections - virology | Tumor Cells, Cultured | Hexokinase - genetics | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Papillomavirus E7 Proteins - metabolism | Animals | Papillomavirus Infections - metabolism | Glucose - metabolism | Uterine Cervical Neoplasms - virology | Biomarkers, Tumor - genetics | Mice | Cell Transformation, Neoplastic - pathology | Hexokinase - metabolism | Neoplasm Staging | Apoptosis | Complications and side effects | Care and treatment | Cell metabolism | Development and progression | Genetic aspects | Phosphotransferases | Health aspects | Papillomavirus infections | Cervical cancer | Liver cancer | Human papillomavirus | Mutation | Metabolism | Kinases | human papillomavirus (HPV) | liver kinase B1 (LKB1) | c-MYC | cervical cancer | hexokinase-ll (HK-II) | glycolysis
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e49047
Inflammatory conditions may promote tumor progression and aggressiveness. In previous reports, we found a group of breast cancer tumors characterized by... 
MATRIX METALLOPROTEINASES | CELLS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | HUMAN PAPILLOMA-VIRUS | GROWTH-FACTOR | INHIBITORS | TUMOR-GROWTH | CANCER | EXPRESSION | PROMOTES ANGIOGENESIS | Inflammation - pathology | Interleukin-1 Receptor-Associated Kinases - metabolism | Leukocytes, Mononuclear - metabolism | Interleukin-1 Receptor-Associated Kinases - genetics | Follow-Up Studies | Humans | NF-kappa B - metabolism | Interleukins - metabolism | Breast Neoplasms - metabolism | Interleukins - genetics | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Inflammation - metabolism | Neoplasm Metastasis | Chemokine CCL3 - genetics | Claudin-3 - genetics | Interferon-beta - genetics | Chemokine CCL3 - metabolism | Claudin-3 - metabolism | Female | Carcinoma - pathology | Matrix Metalloproteinase 1 - genetics | Myeloid Differentiation Factor 88 - genetics | Matrix Metalloproteinase 11 - metabolism | Up-Regulation - genetics | Disease Progression | Leukocytes, Mononuclear - pathology | Tissue Inhibitor of Metalloproteinase-1 - genetics | ADAM Proteins - metabolism | Annexin A2 - genetics | Breast Neoplasms - genetics | Interferon-beta - metabolism | NF-kappa B - genetics | Breast Neoplasms - pathology | Matrix Metalloproteinase 11 - genetics | Inflammation - genetics | Carcinoma - genetics | Carcinoma - metabolism | ADAM Proteins - genetics | Matrix Metalloproteinase 1 - metabolism | Annexin A2 - metabolism | Myeloid Differentiation Factor 88 - metabolism | Breast cancer | Inflammation | Prognosis | B cells | Annexins | Analysis | Gene regulation | Metastasis | Matrix metalloproteinase | Tissue inhibitor of metalloproteinase 1 | Metastases | Interleukin 6 | Proteins | Ethics | Angiogenesis | Breast carcinoma | Interleukin 5 | Interleukin 1 | Metalloproteinase | Growth factors | Interstitial collagenase | IRAK protein | ADAMTS-1 protein | Cytokines | Gene expression | Interleukin 17 | Womens health | Medical prognosis | MyD88 protein | Breast | Chemokines | Tumors | Cancer
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2007, Volume 282, Issue 18, pp. 13141 - 13145
Brd4 is a double bromodomain-containing protein that binds preferentially to acetylated chromatin. It belongs to the BET ( b romodomains and e xtra t erminal)... 
RNA-POLYMERASE-II | RECRUITMENT | ACTIVATION | COMPLEX | MITOTIC CHROMOSOMES | RECOGNITION | VIRAL-DNA | INTERACTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PAPILLOMAVIRUS E2 PROTEINS | P-TEFB | Neoplasms - metabolism | tat Gene Products, Human Immunodeficiency Virus | Chromatin - metabolism | Gene Products, tat - genetics | Papillomaviridae - metabolism | Translocation, Genetic | Protein Binding - genetics | Mitosis | Genome, Viral - genetics | Humans | Positive Transcriptional Elongation Factor B - metabolism | Drosophila Proteins - metabolism | Positive Transcriptional Elongation Factor B - genetics | Papillomaviridae - genetics | Tumor Suppressor Protein p53 - genetics | Chromosomes, Human, Pair 19 - genetics | Gene Products, tat - metabolism | Neoplasms - genetics | Transcription, Genetic | Nuclear Proteins - genetics | HIV-1 - metabolism | Drosophila | Retinoblastoma Protein - metabolism | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Saccharomyces cerevisiae Proteins - genetics | Transcription Factors - genetics | Chromosomes, Human, Pair 15 - genetics | HIV-1 - genetics | Sequence Homology, Amino Acid | Transcription Factors - metabolism | Animals | Retinoblastoma Protein - genetics | Saccharomyces cerevisiae Proteins - metabolism | Drosophila Proteins - genetics | Chromatin - genetics
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 04/2012, Volume 18, Issue 7, pp. 1925 - 1935
Purpose: Respiratory papillomas, caused by human papillomaviruses types 6 and 11 (HPV6/11), are premalignant lesions with potential for malignant conversion.... 
MYCOBACTERIUM-TUBERCULOSIS | RECRUITMENT | POPULATION | RESPONSIVENESS | HOMEOSTASIS | ONCOLOGY | TRANSCRIPTION FACTOR FOXP3 | PD-1 | LINEAGE | VIRAL-INFECTION | LYMPHOCYTES | Forkhead Transcription Factors - immunology | Leukocytes, Mononuclear - metabolism | T-Lymphocytes, Regulatory - metabolism | Cell Proliferation | Human papillomavirus 6 - immunology | Humans | Gene Expression Regulation, Neoplastic | Programmed Cell Death 1 Ligand 2 Protein - metabolism | Chemokine CCL17 - genetics | Male | Precancerous Conditions - metabolism | Chemokine CCL22 - metabolism | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Chemokine CCL22 - immunology | Flow Cytometry | Forkhead Transcription Factors - metabolism | Leukocytes, Mononuclear - immunology | Chemokine CCL17 - immunology | Papillomavirus Infections - immunology | Female | Human papillomavirus 11 - immunology | Papilloma - metabolism | Papilloma - genetics | Respiratory Tract Neoplasms - immunology | Respiratory Tract Neoplasms - genetics | Programmed Cell Death 1 Ligand 2 Protein - genetics | CD4-Positive T-Lymphocytes - metabolism | Programmed Cell Death 1 Receptor - metabolism | Reverse Transcriptase Polymerase Chain Reaction | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Precancerous Conditions - genetics | Respiratory Tract Neoplasms - metabolism | Papillomavirus Infections - genetics | Programmed Cell Death 1 Ligand 2 Protein - immunology | Tumor Microenvironment - immunology | B7-H1 Antigen - metabolism | Chemokine CCL22 - genetics | Precancerous Conditions - immunology | Papillomavirus Infections - metabolism | Papilloma - immunology | Programmed Cell Death 1 Receptor - immunology | Chemokine CCL17 - metabolism | Programmed Cell Death 1 Receptor - genetics | PD-L1 | PD-L2 | immune suppression | CCL17 | Tregs | Recurrent Respiratory Papillomatosis
Journal Article
The FEBS Journal, ISSN 1742-464X, 10/2017, Volume 284, Issue 19, pp. 3171 - 3201
Protein ubiquitination and its reverse reaction, deubiquitination, regulate protein stability, protein binding activity, and their subcellular localization.... 
protein complementation assay | ubiquitination | HPV | interactomic | TARGET | COMPLEX | BIND | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | MEDIATED DEGRADATION | CANCER | HUMAN-PAPILLOMAVIRUS ONCOPROTEIN | TUMOR-SUPPRESSOR | COMPLEMENTATION ASSAY | LIGASE | Ubiquitin-Specific Proteases - genetics | Luciferases - metabolism | Humans | Ubiquitin - metabolism | Ubiquitin-Protein Ligase Complexes - genetics | Peptide Library | Luciferases - genetics | Papillomavirus E7 Proteins - genetics | Ubiquitination | Oncogene Proteins, Viral - genetics | Ubiquitin-Specific Proteases - metabolism | Genes, Reporter | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Molecular Sequence Annotation | Computational Biology | Gene Expression Regulation | Ubiquitin-Protein Ligases - metabolism | Repressor Proteins - genetics | Ubiquitin - genetics | Human papillomavirus 16 - metabolism | Protein Interaction Mapping | Host-Pathogen Interactions | Proteins - genetics | Carrier Proteins - genetics | Papillomavirus E7 Proteins - metabolism | Carrier Proteins - metabolism | Proteins - metabolism | Ubiquitin-Protein Ligase Complexes - metabolism | Virus Replication | Protein Binding | Proteasome Endopeptidase Complex - metabolism | Ubiquitin-Protein Ligases - genetics | Human papillomavirus 16 - genetics | Apoptosis | Ubiquitin | Proteases | Ligases | Papillomavirus infections | Protein binding | Binding | Immune response | Risk groups | Mucosa | Viruses | Chemical reactions | Homology | Oncology | Innate immunity | Adaptors | E7 protein | Immunity | Cullin | Proteins | TRAF2 protein | Human papillomavirus | Proteasomes | Complementation | Localization | Immune system | Life Sciences | Microbiology and Parasitology | Virology
Journal Article