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1. Full Text Human papillomavirus DNA prevalence and type distribution in anal carcinomas worldwide
by Alemany, Laia and Saunier, Maëlle and Alvarado‐Cabrero, Isabel and Quirós, Beatriz and Salmeron, Jorge and Shin, Hai‐Rim and Pirog, Edyta C and Guimerà, Núria and Hernandez‐Suarez, Gustavo and Felix, Ana and Clavero, Omar and Lloveras, Belen and Kasamatsu, Elena and Goodman, Marc T and Hernandez, Brenda Y and Laco, Jan and Tinoco, Leopoldo and Geraets, Daan T and Lynch, Charles F and Mandys, Vaclav and Poljak, Mario and Jach, Robert and Verge, Josep and Clavel, Christine and Ndiaye, Cathy and Klaustermeier, JoEllen and Cubilla, Antonio and Castellsagué, Xavier and Bravo, Ignacio G and Pawlita, Michael and Quint, William G and Muñoz, Nubia and Bosch, Francesc X and Sanjosé, Silvia and HPV VVAP Study Grp and HPV VVAP Study Group
International Journal of Cancer, ISSN 0020-7136, 01/2015, Volume 136, Issue 1, pp. 98 - 107
Knowledge about human papillomaviruses (HPV) types involved in anal cancers in some world regions is scanty. Here, we describe the HPV DNA prevalence and type... 
anal cancer | vaccine | human papillomavirus | CELLS | ASSAY | ONCOLOGY | GENOTYPE ATTRIBUTION | RISK | INFECTION | INTRAEPITHELIAL NEOPLASIA | CANCER | LESIONS | Prevalence | Cross-Sectional Studies | Anus Neoplasms - metabolism | Carcinoma, Squamous Cell - epidemiology | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - virology | Humans | Middle Aged | Papillomavirus Infections - epidemiology | Anus Neoplasms - virology | Male | Papillomavirus Infections - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Female | Papillomavirus Infections - virology | Poisson Distribution | Aged | Retrospective Studies | Anus Neoplasms - epidemiology | DNA, Viral - genetics | Human papillomavirus 16 - genetics | Papillomaviruses | Squamous cell carcinoma | Analysis | DNA | Genetic aspects | Papillomavirus infections | Formaldehyde | Prevalence studies (Epidemiology) | Cancer | Genital cancers | Human papillomavirus | Deoxyribonucleic acid--DNA | Index Medicus
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2. Full Text PD-1-expressing tumor-infiltrating T cells are a favorable prognostic biomarker in HPV-Associated head and neck cancer
by Badoual, Cécile and Hans, Stéphane and Merillon, Nathalie and Van Ryswick, Cordéli and Ravel, Patrice and Benhamouda, Nadine and Levionnois, Emeline and Nizard, Mevyn and Si-Mohamed, Ali and Besnier, Nicolas and Gey, Alain and Rotem-Yehudar, Rinat and Pere, Hél̀ene and Tran, Thi and Guerin, Coralie L and Chauvat, Anne and Dransart, Estelle and Alanio, Cécile and Albert, Sebastien and Barry, Beatrix and Sandoval, Federico and Quintin-Colonna, Françoise and Bruneval, Patrick and Fridman, Wolf H and Lemoine, Francois M and Oudard, Stephane and Johannes, Ludger and Olive, Daniel and Brasnu, Daniel and Tartour, Eric
Cancer Research, ISSN 0008-5472, 01/2013, Volume 73, Issue 1, pp. 128 - 138
Head and neck cancers positive for human papillomavirus (HPV) have a more favorable clinical outcome than HPV-negative cancers, but it is unknown why this is... 
HIGH NUMBERS | ANTI-PD-1 ANTIBODY | ONCOLOGY | OROPHARYNGEAL CANCER | FOLLICULAR LYMPHOMA | HUMAN-PAPILLOMAVIRUS | ANTITUMOR IMMUNITY | UP-REGULATION | CARCINOMA | PD-1 EXPRESSION | LYMPHOCYTES | T-Lymphocyte Subsets - immunology | Head and Neck Neoplasms - virology | Prognosis | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - virology | Humans | Head and Neck Neoplasms - metabolism | Lymphocyte Activation - immunology | Flow Cytometry | Programmed Cell Death 1 Receptor - biosynthesis | Biomarkers, Tumor - metabolism | Papillomavirus Infections - immunology | Female | Lymphocytes, Tumor-Infiltrating - metabolism | Papillomavirus Infections - complications | Mice, Inbred C57BL | Carcinoma, Squamous Cell - immunology | Tumor Microenvironment - immunology | Head and Neck Neoplasms - immunology | Animals | Papillomavirus Infections - metabolism | Fluorescent Antibody Technique | T-Lymphocytes - immunology | Mice | Programmed Cell Death 1 Receptor - immunology | Lymphocytes, Tumor-Infiltrating - immunology | Index Medicus
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3. Full Text Molecular evidence of viral DNA in non-small cell lung cancer and non-neoplastic lung
by Robinson, Lary A and Jaing, Crystal J and Pierce Campbell, Christine and Magliocco, Anthony and Xiong, Yin and Magliocco, Genevra and Thissen, James B and Antonia, Scott and Lawrence Livermore National Lab. (LLNL), Livermore, CA (United States)
British Journal of Cancer, ISSN 0007-0920, 08/2016, Volume 115, Issue 4, pp. 497 - 504
Background: Although similar to 20% of human cancers are caused by microorganisms, only suspicion exists for a microbial cause of lung cancer. Potential... 
non-small cell lung cancer | retrovirus | DETECTION MICROARRAY | NSCLC | HUMAN-PAPILLOMAVIRUS | virus | MICROBIAL DETECTION | HPV | microarray | carcinogenesis | ONCOLOGY | human papillomavirus | SARCOMA | INFECTION | SAMPLES | HEPATITIS-B | ASSOCIATION | CARCINOMA | Retroviridae Infections - virology | Retroviridae Infections - complications | Tumor Virus Infections - virology | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - virology | Humans | Lung Neoplasms - metabolism | Middle Aged | DNA, Viral - analysis | Male | Papillomaviridae - genetics | Lung - virology | Receptor, Epidermal Growth Factor - metabolism | Adenocarcinoma - metabolism | Retroviridae - genetics | Polymerase Chain Reaction | Aged, 80 and over | Female | Papillomavirus Infections - virology | Lung - metabolism | Carcinoma, Non-Small-Cell Lung - virology | Papillomavirus Infections - complications | Tumor Virus Infections - metabolism | Carcinoma, Non-Small-Cell Lung - metabolism | Genotype | Retroviridae Infections - metabolism | Lung Neoplasms - virology | Adenocarcinoma - complications | Adenocarcinoma - virology | Lung Neoplasms - complications | Tumor Virus Infections - complications | Carcinoma, Non-Small-Cell Lung - complications | Papillomavirus Infections - metabolism | Carcinoma, Squamous Cell - complications | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Oncogenic Viruses - genetics | Aged | Index Medicus | Virus | Retrovirus | Human papillomavirus | BASIC BIOLOGICAL SCIENCES | Non-small cell lung cancer | Microarray | Carcinogenesis | Molecular Diagnostics
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4. Full Text Entry of human papillomavirus type 16 by actin-dependent, clathrin- and lipid raft-independent endocytosis
by Schelhaas, Mario and Shah, Bhavin and Holzer, Michael and Blattmann, Peter and Kühling, Lena and Day, Patricia M and Schiller, John T and Helenius, Ari
PLoS Pathogens, ISSN 1553-7366, 04/2012, Volume 8, Issue 4, pp. e1002657 - e1002657
Infectious endocytosis of incoming human papillomavirus type 16 (HPV-16), the main etiological agent of cervical cancer, is poorly characterized in terms of... 
CAPSID PROTEIN | HOST-CELLS | PHOSPHOLIPASE-C | MICROBIOLOGY | SEMLIKI-FOREST-VIRUS | HUMAN KERATINOCYTES | VIROLOGY | PATHWAY | SURFACE HEPARAN-SULFATE | VIRUS-LIKE PARTICLES | CALMODULIN ANTAGONISTS | INFECTIOUS ENTRY | PARASITOLOGY | Human papillomavirus 16 - physiology | Signal Transduction | Membrane Microdomains - metabolism | Humans | Actins - metabolism | Caveolins - metabolism | p21-Activated Kinases - metabolism | Clathrin - metabolism | Virus Internalization | Papillomavirus Infections - genetics | Sodium-Hydrogen Exchangers - metabolism | Endocytosis | Papillomavirus Infections - metabolism | Protein Kinase C - metabolism | HeLa Cells | Viral genetics | Physiological aspects | Development and progression | Genetic aspects | Virulence (Microbiology) | Research | Gene expression | Papillomavirus infections | Heparan sulfate | Automation | Viruses | Lipids | Infections | Cholesterol | Proteins | Human papillomavirus | Microscopy | Plasmids | Influenza | Comparative analysis | Cervical cancer | Index Medicus
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5. Full Text Effect of HPV-Associated p16INK4A Expression on Response to Radiotherapy and Survival in Squamous Cell Carcinoma of the Head and Neck
by Pernille Lassen and Jesper G. Eriksen and Stephen Hamilton-Dutoit and Trine Tramm and Jan Alsner and Jens Overgaard
Journal of Clinical Oncology, ISSN 0732-183X, 04/2009, Volume 27, Issue 12, pp. 1992 - 1998
A subset of head and neck cancers is associated with the human papillomavirus (HPV). Viral infection is closely correlated with expression of p16(INK4A) in... 
Papillomavirus Infections - radiotherapy | Head and Neck Neoplasms - virology | Prognosis | Prospective Studies | Follow-Up Studies | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - virology | Humans | Middle Aged | Male | Carcinoma, Squamous Cell - radiotherapy | Head and Neck Neoplasms - metabolism | Young Adult | Carcinoma, Squamous Cell - mortality | In Situ Hybridization | Aged, 80 and over | Biomarkers, Tumor - metabolism | Placebos | Adult | Female | Papillomavirus Infections - virology | Survival Rate | Treatment Outcome | Papillomaviridae - isolation & purification | Papillomavirus Infections - mortality | Head and Neck Neoplasms - radiotherapy | Papillomavirus Infections - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Aged | Head and Neck Neoplasms - mortality | Neoplasm Staging | Cohort Studies | Index Medicus
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6. Full Text Oncogenic HPV infection interrupts the expression of tumor-suppressive miR-34a through viral oncoprotein E6
by Wang, Xiaohong and Wang, Hsu-Kun and Mccoy, J. Philip and Banerjee, Nilam S and Rader, Janet S and Broker, Thomas R and Meyers, Craig and Chow, Louise T and Zheng, Zhi-Ming
RNA, ISSN 1355-8382, 04/2009, Volume 15, Issue 4, pp. 637 - 647
MicroRNAs (miRNA) play pivotal roles in controlling cell proliferation and differentiation. Aberrant miRNA expression in human is becoming recognized as a new... 
Mir-34a | Human papillomavirus | Oncoprotein | P53 | CARCINOMA CELL-LINES | HUMAN-PAPILLOMAVIRUS TYPE-16 | miR-34a | BIOCHEMISTRY & MOLECULAR BIOLOGY | SARCOMA-ASSOCIATED HERPESVIRUS | DIFFERENTIATED KERATINOCYTES | CERVICAL-CANCER | p53 | MESSENGER-RNAS | RAFT CULTURES | GROWTH ARREST | E7 ONCOPROTEIN | human papillomavirus | GENE-EXPRESSION | oncoprotein | Humans | Middle Aged | MicroRNAs - metabolism | Human papillomavirus 16 - metabolism | Gene Knockdown Techniques | DNA-Binding Proteins - metabolism | Papillomavirus Infections - metabolism | Human papillomavirus 18 - metabolism | Cell Line, Tumor | Adult | Female | Papillomavirus Infections - virology | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Index Medicus
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7. Full Text The T Antigen Locus of Merkel Cell Polyomavirus Downregulates Human Toll-Like Receptor 9 Expression
by Naveed Shahzad and Masahiro Shuda and Tarik Gheit and Hyun Jin Kwun and Iris Cornet and Djamel Saidj and Claudia Zannetti and Uzma Hasan and Yuan Chang and Patrick S. Moore and Rosita Accardi and Massimo Tommasino
Journal of Virology, ISSN 0022-538X, 12/2013, Volume 87, Issue 23, pp. 13009 - 13019
Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
TRANSCRIPTION FACTORS | C/EBP-ALPHA | VIROLOGY | HUMAN-PAPILLOMAVIRUS TYPE-16 | SARCOMA-ASSOCIATED HERPESVIRUS | P53 FUNCTIONS | GENE-EXPRESSION | TLR9 | PROLIFERATION | CARCINOMA | EPSTEIN-BARR-VIRUS | Merkel Cells - virology | Tumor Virus Infections - genetics | Promoter Regions, Genetic | Tumor Virus Infections - metabolism | Tumor Virus Infections - virology | Down-Regulation | Humans | Carcinoma, Merkel Cell - genetics | Toll-Like Receptor 9 - genetics | Merkel cell polyomavirus - metabolism | Merkel Cells - metabolism | Polyomavirus Infections - metabolism | Antigens, Viral, Tumor - metabolism | Host-Pathogen Interactions | Polyomavirus Infections - virology | Polyomavirus Infections - genetics | Carcinoma, Merkel Cell - metabolism | Merkel cell polyomavirus - genetics | Carcinoma, Merkel Cell - virology | Antigens, Viral, Tumor - genetics | Toll-Like Receptor 9 - metabolism | Index Medicus | Virus-Cell Interactions
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8. Full Text The role of NF-κB and miRNA in oral cancer and cancer stem cells with or without HPV16 infection
by Bano, Nasreen and Yadav, Manisha and Mohania, Dheeraj and Das, Bhudev C
PLoS ONE, ISSN 1932-6203, 10/2018, Volume 13, Issue 10, pp. e0205518 - e0205518
A small subpopulation of cancer stem-like cells (CSCs) present in almost all tumors is responsible for drug resistance and tumor recurrence. The role of... 
SURVIVAL | HEAD | POPULATION | CONSTITUTIVE ACTIVATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | SELF-RENEWAL | CERVICAL-CANCER | C-REL | HUMAN-PAPILLOMAVIRUS | CARCINOMA | Mouth Neoplasms - complications | Papillomavirus Infections - complications | Carcinoma, Squamous Cell - metabolism | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Mouth Neoplasms - metabolism | NF-kappa B - metabolism | Human papillomavirus 16 | Papillomavirus E7 Proteins - metabolism | Neoplastic Stem Cells - metabolism | Papillomavirus Infections - metabolism | Carcinoma, Squamous Cell - complications | Biomarkers, Tumor - metabolism | Cell Line, Tumor | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Index Medicus
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9. Full Text Human papillomavirus 16 infection induces VAP-dependent endosomal tubulation
by Siddiqa, Abida and Massimi, Paola and Pim, David and Broniarczyk, Justyna and Banks, Lawrence
Journal of Virology, ISSN 0022-538X, 03/2018, Volume 92, Issue 6
Human papillomavirus (HPV) infection involves complex interactions with the endocytic transport machinery, which ultimately facilitates the entry of the... 
Infection | Human papillomavirus | Tubulating endosome | MICAL-L1 | Trafficking | infection | COMPLEX | ENTRY | CAPSID PROTEIN L1 | RETROMER | tubulating endosome | VIROLOGY | human papillomavirus | REQUIRES | HEPARAN-SULFATE | trafficking | BINDING | Membrane Glycoproteins - metabolism | Cytoskeletal Proteins - genetics | Vesicular Transport Proteins - metabolism | Humans | Virion - genetics | Biological Transport, Active - genetics | LIM Domain Proteins - metabolism | Endosomes - metabolism | Virus Internalization | Papillomavirus Infections - pathology | Endocytosis | trans-Golgi Network - metabolism | trans-Golgi Network - virology | Cytoskeletal Proteins - metabolism | Endosomes - virology | Endosomes - genetics | Vesicular Transport Proteins - genetics | Virion - metabolism | trans-Golgi Network - genetics | Human papillomavirus 16 - metabolism | Membrane Glycoproteins - genetics | Papillomavirus Infections - genetics | Papillomavirus Infections - metabolism | Adaptor Proteins, Signal Transducing - genetics | LIM Domain Proteins - genetics | HeLa Cells | Adaptor Proteins, Signal Transducing - metabolism | Human papillomavirus 16 - genetics | Index Medicus
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10. Full Text TRIP12 and UBR5 Suppress Spreading of Chromatin Ubiquitylation at Damaged Chromosomes
by Gudjonsson, Thorkell and Altmeyer, Matthias and Savic, Velibor and Toledo, Luis and Dinant, Christoffel and Grøfte, Merete and Bartkova, Jirina and Poulsen, Maria and Oka, Yasuyoshi and Bekker-Jensen, Simon and Mailand, Niels and Neumann, Beate and Heriche, Jean-Karim and Shearer, Robert and Saunders, Darren and Bartek, Jiri and Lukas, Jiri and Lukas, Claudia
Cell, ISSN 0092-8674, 08/2012, Volume 150, Issue 4, pp. 697 - 709
Histone ubiquitylation is a prominent response to DNA double-strand breaks (DSBs), but how these modifications are confined to DNA lesions is not understood.... 
GENOMIC INSTABILITY | REPAIR PROTEINS | CLASS SWITCH RECOMBINATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | BRCA1 DEFICIENCY | DOUBLE-STRAND BREAKS | END RULE PATHWAY | HOMOLOGOUS RECOMBINATION | REPLICATION STRESS | E3 UBIQUITIN LIGASE | CELL BIOLOGY | Neoplasms - metabolism | Cell Line | Chromatin - metabolism | Humans | Gene Silencing | Ubiquitin-Protein Ligases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | DNA Breaks, Double-Stranded | Alphapapillomavirus | Papillomavirus Infections - pathology | Neoplasms - virology | Ubiquitination | Carrier Proteins - metabolism | Papillomavirus Infections - metabolism | DNA Repair | Cell Line, Tumor | Transcription, Genetic | Tumor Suppressor p53-Binding Protein 1 | Neoplasms - pathology | Ubiquitin | Chromatin | Ligases | Oncology, Experimental | DNA | Histones | Genetic research | Research | Universities and colleges | DNA binding proteins | Chromosomes | Cancer | Index Medicus
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11. Full Text LKB1 inhibits HPV-associated cancer progression by targeting cellular metabolism
by Zeng, Q and Chen, J and Li, Y and Werle, K.D and Zhao, R.-X and Quan, C.-S and Wang, Y.-S and Zhai, Y.-X and Wang, J.-W and Youssef, M and Cui, R and Liang, J and Genovese, N and Chow, L.T and Li, Y.-L and Xu, Z.-X
Oncogene, ISSN 0950-9232, 03/2017, Volume 36, Issue 9, pp. 1245 - 1255
Liver kinase B1 (LKB1) is mutationally inactivated in Peutz-Jeghers syndrome and in a variety of cancers including human papillomavirus (HPV)-caused cervical... 
PATHWAYS | ONCOPROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CERVICAL-CANCER | HUMAN-PAPILLOMAVIRUS | IDENTIFICATION | HEXOKINASE II | CELL BIOLOGY | LUNG-CANCER | ONCOLOGY | GROWTH | ROS | GENETICS & HEREDITY | TUMOR INITIATION | Cell Proliferation | Human papillomavirus 16 - physiology | Prognosis | Follow-Up Studies | Humans | Uterine Cervical Neoplasms - pathology | Case-Control Studies | Papillomavirus E7 Proteins - genetics | Papillomavirus Infections - pathology | Glycolysis - physiology | Uterine Cervical Neoplasms - metabolism | Biomarkers, Tumor - metabolism | Female | Oncogene Proteins, Viral - genetics | Papillomavirus Infections - virology | Tumor Cells, Cultured | Hexokinase - genetics | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Papillomavirus E7 Proteins - metabolism | Animals | Papillomavirus Infections - metabolism | Glucose - metabolism | Uterine Cervical Neoplasms - virology | Biomarkers, Tumor - genetics | Mice | Cell Transformation, Neoplastic - pathology | Hexokinase - metabolism | Neoplasm Staging | Apoptosis | Complications and side effects | Care and treatment | Cell metabolism | Development and progression | Genetic aspects | Phosphotransferases | Health aspects | Papillomavirus infections | Cervical cancer | Liver cancer | Human papillomavirus | Mutation | Metabolism | Kinases | Index Medicus | human papillomavirus (HPV) | liver kinase B1 (LKB1) | c-MYC | cervical cancer | hexokinase-ll (HK-II) | glycolysis
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12. Full Text NF-κB-induced KIAA1199 promotes survival through EGFR signalling
by Shostak, Kateryna and Zhang, Xin and Hubert, Pascale and Göktuna, Serkan Ismail and Jiang, Zheshen and Klevernic, Iva and Hildebrand, Julien and Roncarati, Patrick and Hennuy, Benoit and Ladang, Aurélie and Somja, Joan and Gothot, André and Close, Pierre and Delvenne, Philippe and Chariot, Alain
Nature Communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, pp. 5232 - 5232
Constitutive activation of EGFR- and NF-kappa B-dependent pathways is a hallmark of cancer, yet signalling proteins that connect both oncogenic cascades are... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | GROWTH-FACTOR RECEPTOR | KINASE COMPLEX | HUMAN-PAPILLOMAVIRUS TYPE-16 | MULTIDISCIPLINARY SCIENCES | SQUAMOUS-CELL CARCINOMA | ANAPLASTIC LARGE-CELL | UP-REGULATION | CERVICAL INTRAEPITHELIAL NEOPLASIA | RISK HUMAN-PAPILLOMAVIRUS | Proto-Oncogene Proteins - metabolism | Cell Survival | Humans | Epidermal Growth Factor - metabolism | Semaphorin-3A - metabolism | Proteins - genetics | Transcription Factors - metabolism | Receptor, Epidermal Growth Factor - metabolism | Lysosomes - metabolism | Proteins - metabolism | MCF-7 Cells | Transcription Factor RelA - metabolism | Keratinocytes - metabolism | Papillomavirus Infections - metabolism | Epithelial-Mesenchymal Transition | Cervical Intraepithelial Neoplasia - metabolism | HeLa Cells | Index Medicus
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