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1. Full Text Serenoa repens and N‐acetyl glucosamine/milk proteins complex differentially affect the paracrine communication between endothelial and follicle dermal papilla cells
by Bassino, Eleonora and Gasparri, Franco and Munaron, Luca
Journal of cellular physiology, ISSN 0021-9541, 05/2019, Volume 234, Issue 5, pp. 7320 - 7329
N‐acetyl glucosamine | follicle dermal papilla cells | endothelial cells | Serenoa repens | lactoglobulin | N-acetyl glucosamine | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neovascularization, Physiologic - drug effects | Plant Extracts - pharmacology | Coculture Techniques | Humans | Vascular Endothelial Growth Factor A - metabolism | Interleukin-1beta - metabolism | Lipid Peroxidation - drug effects | Angiogenesis Inducing Agents - pharmacology | Tight Junctions - drug effects | Tight Junctions - metabolism | Serenoa - chemistry | Signal Transduction | Endothelial Cells - metabolism | Cells, Cultured | Acetylglucosamine - pharmacology | Plant Extracts - isolation & purification | Permeability | Hair Follicle - drug effects | Cell Movement - drug effects | Hair Follicle - cytology | Hair Follicle - metabolism | Paracrine Communication - drug effects | Milk Proteins - pharmacology | Cell Proliferation - drug effects | Endothelial Cells - drug effects | Hair | Glucosamine | Interleukin | Lipid peroxidation | Paracrine signalling | Lipids | Regrowth | Modulators | Endothelial cells | Endothelium | Vascularization | Proteins | Skin | Microvasculature | Hair loss | Vascular endothelial growth factor | Growth factors | Nutraceuticals | Peroxidation | Reductase | Index Medicus
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2. Full Text Autocrine/paracrine activation of the GABA A receptor inhibits the proliferation of neurogenic polysialylated neural cell adhesion molecule-positive (PSA-NCAM + ) precursor cells from postnatal striatum
by Nguyen, Laurent and Malgrange, Brigitte and Breuskin, Ingrid and Bettendorff, Lucien and Moonen, Gustave and Belachew, Shibeshih and Rigo, Jean-Michel
The Journal of neuroscience, ISSN 0270-6474, 04/2003, Volume 23, Issue 8, pp. 3278 - 3294
Newborn rat striata | receptors | Immunocytochemistry | RT-PCR | GABA | Whole-cell patch-clamp | Proliferation | PSA-NCAM | HPLC | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Glutamate Decarboxylase - biosynthesis | MAP Kinase Signaling System - physiology | Calcium Channels - metabolism | Rats, Wistar | Calcium - metabolism | gamma-Aminobutyric Acid - metabolism | Neurons - cytology | Autocrine Communication - drug effects | Stem Cells - cytology | Stem Cells - metabolism | Corpus Striatum - cytology | Paracrine Communication - physiology | Receptors, GABA-A - drug effects | Spheroids, Cellular | Cell Differentiation - physiology | Animals, Newborn | Cell Separation | Autocrine Communication - physiology | Neural Cell Adhesion Molecule L1 - biosynthesis | Cells, Cultured | Rats | gamma-Aminobutyric Acid - pharmacology | Sialic Acids - biosynthesis | Cell Division - physiology | Patch-Clamp Techniques | Animals | MAP Kinase Signaling System - drug effects | Signal Transduction - drug effects | Cell Differentiation - drug effects | Paracrine Communication - drug effects | Stem Cells - drug effects | Signal Transduction - physiology | Receptors, GABA-A - metabolism | Epidermal Growth Factor - pharmacology | Cell Cycle - drug effects | Isoenzymes - biosynthesis | Index Medicus
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3. Full Text Composite scaffolds of nano-hydroxyapatite and silk fibroin enhance mesenchymal stem cell-based bone regeneration via the interleukin 1 alpha autocrine/paracrine signaling loop
by Liu, Hua and Xu, Guo Wei and Wang, Ya Fei and Zhao, Hong Shi and Xiong, Si and Wu, Yan and Heng, Boon Chin and An, Cheng Rui and Zhu, Gang Hua and Xie, Ding Hua
Biomaterials, ISSN 0142-9612, 2015, Volume 49, pp. 103 - 112
Advanced Basic Science | Dentistry | Silk | Hydroxyapatite | Bone regeneration | Stem cell | Interleukin | Osteogenesis | Engineering | Materials Science | Technology | Engineering, Biomedical | Materials Science, Biomaterials | Science & Technology | Receptors, Interleukin-1 Type II - metabolism | Nanoparticles - chemistry | Interleukin-1alpha - metabolism | Male | Autocrine Communication - drug effects | Tissue Scaffolds - chemistry | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Skull - pathology | Bone Morphogenetic Protein 2 - metabolism | Durapatite - pharmacology | Female | Skull - drug effects | Disease Models, Animal | Bone Regeneration - drug effects | Mesenchymal Stromal Cells - drug effects | Bone Marrow Cells - cytology | Osteogenesis - drug effects | Nanoparticles - ultrastructure | Fibroins - pharmacology | Biocompatible Materials - pharmacology | Rats, Sprague-Dawley | Cell Shape - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Paracrine Communication - drug effects | Cluster Analysis | Biological products | Interleukins | Tissue engineering | Analysis | Stem cells | Bone morphogenetic proteins | Gene expression | Index Medicus | Surgical implants | Regeneration | Biomedical materials | Biocompatibility | Bones | Nanostructure | Scaffolds | Biomaterials
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4. Full Text Human TH9 cells are skin-tropic and have autocrine and paracrine proinflammatory capacity
by Schlapbach, Christoph and Gehad, Ahmed and Yang, Chao and Watanabe, Rei and Guenova, Emmanuella and Teague, Jessica E and Campbell, Laura and Yawalkar, Nikhil and Kupper, Thomas S and Clark, Rachael A
Science translational medicine, ISSN 1946-6234, 01/2014, Volume 6, Issue 219, pp. 219ra8 - 219ra8
T-Lymphocytes, Helper-Inducer - microbiology | Inflammation - pathology | Humans | Autocrine Communication - drug effects | Paracrine Communication - immunology | T-Lymphocytes, Helper-Inducer - drug effects | Lymphocyte Activation - immunology | Candida albicans - drug effects | Autocrine Communication - immunology | Psoriasis - pathology | T-Lymphocytes, Helper-Inducer - immunology | Dermatitis, Atopic - immunology | Interleukin-9 - metabolism | Candida albicans - physiology | Skin - pathology | Skin - immunology | Psoriasis - immunology | Dermatitis, Atopic - pathology | Inflammation - immunology | Transforming Growth Factor beta - pharmacology | Animals | Lymphocyte Activation - drug effects | Paracrine Communication - drug effects | Interleukin-2 - pharmacology | Mice | Skin - drug effects | Index Medicus
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5. Full Text Systemic peripheral artery relaxation by KCNQ channel openers and hydrogen sulfide
by Schleifenbaum, Johanna and Köhn, Carolin and Voblova, Nadezda and Dubrovska, Galyna and Zavarirskaya, Olga and Gloe, Torsten and Crean, Christopher S and Luft, Friedrich C and Huang, Yu and Schubert, Rudolf and Gollasch, Maik
Journal of hypertension, ISSN 0263-6352, 09/2010, Volume 28, Issue 9, pp. 1875 - 1882
perivascular fat | ADRF | KCNQ channels | obesity hypertension | adipocyte-derived relaxing factor | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Hormones. Endocrine system | Cardiology. Vascular system | Arterial hypertension. Arterial hypotension | Pharmacology. Drug treatments | Biological and medical sciences | Medical sciences | Blood and lymphatic vessels | Hydrogen Sulfide - metabolism | Mesenteric Arteries - physiology | Adipose Tissue - physiology | Isometric Contraction - drug effects | Male | Mesenteric Arteries - drug effects | KCNQ Potassium Channels - antagonists & inhibitors | Paracrine Communication - physiology | Arteries - physiology | Vasodilation - physiology | Isometric Contraction - physiology | Carbamates - pharmacology | Aorta - drug effects | Mice, Inbred C57BL | KCNQ Potassium Channels - agonists | Rats | Arteries - drug effects | Rats, Sprague-Dawley | Anthracenes - pharmacology | Animals | Paracrine Communication - drug effects | Phenylenediamines - pharmacology | KCNQ Potassium Channels - physiology | Mice | Vasodilation - drug effects | In Vitro Techniques | Aorta - physiology | Index Medicus
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6. Full Text Retinoic Acid–Induced Pancreatic Stellate Cell Quiescence Reduces Paracrine Wnt–β-Catenin Signaling to Slow Tumor Progression
by Froeling, Fieke E.M and Feig, Christine and Chelala, Claude and Dobson, Richard and Mein, Charles E and Tuveson, David A and Clevers, Hans and Hart, Ian R and Kocher, Hemant M
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2011, Volume 141, Issue 4, pp. 1486 - 1497.e14
Gastroenterology and Hepatology | 9RA | Therapy | Secreted Frizzled-Related Protein 4 | 13RA | Mouse Model | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Transcription, Genetic - drug effects | Pancreatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Pancreatic Stellate Cells - drug effects | Carcinoma, Pancreatic Ductal - metabolism | Cellular Senescence - drug effects | Wnt Proteins - metabolism | Carcinoma, Pancreatic Ductal - genetics | Dose-Response Relationship, Drug | Pancreatic Neoplasms - drug therapy | RNA Interference | Time Factors | Mice, Mutant Strains | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Pancreatic Stellate Cells - pathology | Disease Models, Animal | Tretinoin - pharmacology | Proto-Oncogene Proteins - metabolism | Isotretinoin - pharmacology | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | beta Catenin - metabolism | Disease Progression | Carcinoma, Pancreatic Ductal - drug therapy | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Paracrine Communication - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Pancreatic Stellate Cells - metabolism | Index Medicus | Abridged Index Medicus
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7. Full Text Autocrine and Paracrine Angiopoietin 1/Tie-2 Signaling Promotes Muscle Satellite Cell Self-Renewal
by Abou-Khalil, Rana and Le Grand, Fabien and Pallafacchina, Giorgia and Valable, Samuel and Authier, François-Jérôme and Rudnicki, Michael A and Gherardi, Romain K and Germain, Stéphane and Chretien, Fabrice and Sotiropoulos, Athanassia and Lafuste, Peggy and Montarras, Didier and Chazaud, Bénédicte
Cell stem cell, ISSN 1934-5909, 2009, Volume 5, Issue 3, pp. 298 - 309
STEMCELL | Cell & Tissue Engineering | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Cell physiology | Molecular and cellular biology | Cell differentiation, maturation, development, hematopoiesis | Gene Silencing - drug effects | Apoptosis - drug effects | Cell Count | Humans | Autocrine Communication - drug effects | PAX7 Transcription Factor - metabolism | Stem Cells - cytology | Angiopoietin-1 - metabolism | Stem Cells - metabolism | Protein Binding - drug effects | Receptor, TIE-2 - metabolism | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Angiopoietin-1 - pharmacology | Fibroblasts - metabolism | Satellite Cells, Skeletal Muscle - cytology | Cells, Cultured | Satellite Cells, Skeletal Muscle - metabolism | Satellite Cells, Skeletal Muscle - drug effects | Satellite Cells, Skeletal Muscle - enzymology | Animals | Muscle Development - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Fibroblasts - drug effects | Paracrine Communication - drug effects | Stem Cells - drug effects | Cell Proliferation - drug effects | Mice | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
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8. Full Text Inhibition of CXCL12/CXCR4 autocrine/paracrine loop reduces viability of human glioblastoma stem-like cells affecting self-renewal activity
by Gatti, Monica and Pattarozzi, Alessandra and Bajetto, Adriana and Würth, Roberto and Daga, Antonio and Fiaschi, Pietro and Zona, Gianluigi and Florio, Tullio and Barbieri, Federica
Toxicology (Amsterdam), ISSN 0300-483X, 2013, Volume 314, Issue 2, pp. 209 - 220
Emergency | CXCR4 | CXCL12 | Cancer stem cells | Intracellular signaling | Glioblastoma | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Cell Survival - drug effects | Neoplastic Stem Cells - drug effects | Receptors, CXCR4 - antagonists & inhibitors | Autocrine Communication - physiology | Humans | Heterocyclic Compounds - pharmacology | Autocrine Communication - drug effects | Down-Regulation - drug effects | Mice, SCID | Chemokine CXCL12 - antagonists & inhibitors | Down-Regulation - physiology | Paracrine Communication - physiology | Animals | Neoplastic Stem Cells - metabolism | Receptors, CXCR4 - biosynthesis | Glioblastoma - pathology | Paracrine Communication - drug effects | Cell Line, Tumor | Chemokine CXCL12 - biosynthesis | Glioblastoma - metabolism | Cell Proliferation - drug effects | Glioblastoma - drug therapy | Cell Survival - physiology | Drug resistance | Glioblastoma multiforme | Stem cells | Index Medicus
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9. Full Text Exosomal Wnt-induced dedifferentiation of colorectal cancer cells contributes to chemotherapy resistance
by Hu, Y.-B and Yan, C and Mu, L and Mi, Y.L and Zhao, H and Hu, H and Li, X.-L and Tao, D.-D and Wu, Y.-Q and Gong, J.-P and Qin, J.-C
Oncogene, ISSN 0950-9232, 03/2019, Volume 38, Issue 11, pp. 1951 - 1965
Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Exosomes - drug effects | Exosomes - metabolism | Fibroblasts - physiology | Neoplastic Stem Cells - drug effects | Humans | Wnt Signaling Pathway - physiology | Exosomes - pathology | Colorectal Neoplasms - drug therapy | Neoplastic Stem Cells - pathology | Female | Antineoplastic Agents - pharmacology | Colorectal Neoplasms - metabolism | Neoplastic Stem Cells - physiology | Cell Dedifferentiation - drug effects | Cells, Cultured | Fibroblasts - pathology | Mice, SCID | HT29 Cells | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Oxaliplatin - pharmacology | Animals | Wnt Signaling Pathway - drug effects | Paracrine Communication - drug effects | Mice, Inbred NOD | Cell Proliferation - drug effects | Fluorouracil - pharmacology | Mice | Pyridines - pharmacology | Colorectal Neoplasms - pathology | Pyrazines - pharmacology | Cell Dedifferentiation - genetics | Drug Resistance, Neoplasm - drug effects | Chemotherapy | Cancer cells | Stem cells | Colorectal cancer | Drug resistance | Health aspects | Cancer | Wnt protein | Colorectal carcinoma | Chemoresistance | Fibroblasts | Cell interactions | Exosomes | Tumors | Index Medicus | Cell biology | Cancer therapeutic resistance | Cancer stem cells | Cancer microenvironment
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10. Full Text Differential Regulation of Vitamin D Receptor and Its Ligand in Human Monocyte-Derived Dendritic Cells
by Hewison, Martin and Freeman, Lisa and Hughes, Susan V and Evans, Katie N and Bland, Rosemary and Eliopoulos, Aristides G and Kilby, Mark D and Moss, Paul A. H and Chakraverty, Ronjon
The Journal of immunology (1950), ISSN 0022-1767, 06/2003, Volume 170, Issue 11, pp. 5382 - 5390
Life Sciences & Biomedicine | Immunology | Science & Technology | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - biosynthesis | Monocytes - cytology | Humans | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - genetics | Calcitriol - physiology | Monocytes - metabolism | Autocrine Communication - drug effects | Paracrine Communication - immunology | Receptors, Calcitriol - biosynthesis | Autocrine Communication - immunology | Enzyme Induction - drug effects | Dendritic Cells - drug effects | Growth Inhibitors - biosynthesis | Calcitriol - metabolism | Growth Inhibitors - pharmacology | Dendritic Cells - metabolism | Receptors, Calcitriol - antagonists & inhibitors | Gene Expression Regulation - immunology | Cells, Cultured | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - metabolism | Receptors, Calcitriol - metabolism | Down-Regulation - drug effects | Growth Inhibitors - metabolism | Gene Expression Regulation - drug effects | Monocytes - drug effects | Up-Regulation - drug effects | Cell Differentiation - immunology | Monocytes - enzymology | Enzyme Induction - genetics | Up-Regulation - immunology | Cell Differentiation - drug effects | Dendritic Cells - enzymology | Down-Regulation - immunology | Calcitriol - biosynthesis | Calcitriol - pharmacology | Paracrine Communication - drug effects | Growth Inhibitors - physiology | Ligands | Dendritic Cells - cytology | Index Medicus | Abridged Index Medicus
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