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Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 2783 - 14
The importance of the tumor-associated stroma in cancer progression is clear. However, it remains uncertain whether early events in the stroma are capable of... 
BREAST-CANCER | NUCLEAR PTEN | CONTRALATERAL BREAST | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | FOLLOW-UP | DNA-REPAIR | TUMOR MICROENVIRONMENT | MOUSE MODELS | FIBROBLASTS | RADIATION-THERAPY | Epithelial Cells - metabolism | Gamma Rays - adverse effects | Epithelial Cells - drug effects | Humans | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | Mammary Glands, Human - metabolism | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Mammary Neoplasms, Experimental - pathology | Stromal Cells - drug effects | Genomic Instability - drug effects | Female | Antineoplastic Agents - pharmacology | Mammary Glands, Human - radiation effects | Mammary Glands, Human - drug effects | Genomic Instability - radiation effects | PTEN Phosphohydrolase - genetics | Epithelial Cells - radiation effects | PTEN Phosphohydrolase - deficiency | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Signal Transduction | Stromal Cells - metabolism | Radiation Tolerance - genetics | Mammary Neoplasms, Experimental - radiotherapy | Mammary Glands, Animal - drug effects | Mammary Glands, Animal - radiation effects | Breast Neoplasms - radiotherapy | Animals | Breast Neoplasms - genetics | Mammary Glands, Animal - metabolism | Breast Neoplasms - pathology | Cell Transformation, Neoplastic | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Cell Proliferation - radiation effects | Stromal Cells - radiation effects | Hyperplasia | Radiation | Paracrine signalling | Homology | Genomic instability | Epidermal growth factor | Clonal deletion | Deletion | Tumorigenesis | Inhibition | Mammary gland | Tensin | Radiation effects | Stability | Epidermal growth factor receptors | Stroma | Risk reduction | Mammary glands | Breast cancer | Radiation therapy | Epithelium | Patients | Radiation dosage | Breast | Diagnostic systems | PTEN protein | Cancer | Tumors
Journal Article
Cell Stem Cell, ISSN 1934-5909, 2009, Volume 5, Issue 3, pp. 298 - 309
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2011, Volume 6, Issue 10, p. e25900
Sclerostin is a product of mature osteocytes embedded in mineralised bone and is a negative regulator of bone mass and osteoblast differentiation. While... 
MESSENGER-RNA | BONE MORPHOGENETIC PROTEIN | MULTIDISCIPLINARY SCIENCES | HUMAN PRIMARY OSTEOBLASTS | RECEPTOR ACTIVATOR | MECHANICAL STIMULATION | PARATHYROID-HORMONE | CELL-DIFFERENTIATION | WNT | EXPRESSION | KAPPA-B LIGAND | Osteocytes - drug effects | RANK Ligand - metabolism | Apoptosis - drug effects | Humans | Tartrate-Resistant Acid Phosphatase | Osteoclasts - cytology | Cell Differentiation - genetics | Acid Phosphatase - metabolism | Isoenzymes - metabolism | Adult | Osteoblasts - cytology | Bone Morphogenetic Proteins - pharmacology | Osteogenesis - genetics | Cell Survival - drug effects | Calcification, Physiologic - drug effects | Osteocytes - metabolism | Osteoblasts - drug effects | Osteocytes - cytology | Osteogenesis - drug effects | Cells, Cultured | Genetic Markers | Recombinant Proteins - pharmacology | Osteoclasts - metabolism | Gene Expression Regulation - drug effects | RANK Ligand - genetics | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Mice | Osteoblasts - metabolism | Osteoclasts - drug effects | Bones | RNA | Gene expression | Density | Genes | Homeostasis | Osteocytes | Leukocytes (mononuclear) | SOST protein | Biology | Kinases | Osteoprotegerin | Monoculture | Ethics | Genotype & phenotype | Splenocytes | Peripheral blood mononuclear cells | Tumor necrosis factor-TNF | Biocompatibility | TRANCE protein | Discipline | Recombinant | Cytokines | Caspase | Cultures | Bone mass | Osteoblastogenesis | Low density lipoprotein receptors | Osteoclasts | Acid phosphatase (tartrate-resistant) | Bone | Apoptosis
Journal Article
Bone, ISSN 8756-3282, 2015, Volume 74, pp. 95 - 105
Abstract Bisphosphonate-related osteonecrosis of the jaw (BRONJ) is defined as an exposed necrotic bone in the oral cavity that does not heal after appropriate... 
Orthopedics | Paracrine effects | Osteoclasts | Secretome | Conditioned media | Osteogenesis | Bisphosphonate-related osteonecrosis | MULTIPLE-MYELOMA | PAMIDRONATE | REGENERATION | CANCER | ZOLEDRONIC ACID | IN-VITRO | ENDOTHELIAL-CELLS | GROWTH | ENDOCRINOLOGY & METABOLISM | BONE | MARROW STROMAL CELLS | Neovascularization, Physiologic - drug effects | Rats, Wistar | Apoptosis - drug effects | Humans | Culture Media, Conditioned - pharmacology | Male | Dexamethasone - pharmacology | Bisphosphonate-Associated Osteonecrosis of the Jaw - diagnostic imaging | Diphosphonates - adverse effects | Real-Time Polymerase Chain Reaction | Osteogenesis - genetics | Disease Models, Animal | Biomarkers - metabolism | Cell Survival - drug effects | Mesenchymal Stromal Cells - drug effects | Osteoclasts - pathology | Neovascularization, Physiologic - genetics | Imidazoles - adverse effects | Osteogenesis - drug effects | Mesenchymal Stromal Cells - metabolism | Osteoclasts - metabolism | Gene Expression Regulation - drug effects | Radiography | Injections | Animals | Bisphosphonate-Associated Osteonecrosis of the Jaw - pathology | Cell Differentiation - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | Bisphosphonate-Associated Osteonecrosis of the Jaw - drug therapy | Osteoclasts - drug effects | Chemotherapy | Dexamethasone | Stem cells | Bones | Neovascularization | Health aspects | Necrosis | Cancer
Journal Article
Biomaterials, ISSN 0142-9612, 2015, Volume 49, pp. 103 - 112
Abstract Composite scaffolds of nano-hydroxyapatite (nHAp) and silk fibroin (SF) have been reported to promote bone regeneration mainly through signaling... 
Advanced Basic Science | Dentistry | Silk | Hydroxyapatite | Bone regeneration | Stem cell | Interleukin | Osteogenesis | ACTIVATION | MATERIALS SCIENCE, BIOMATERIALS | PARTICLES | ENGINEERING, BIOMEDICAL | OSTEOGENIC DIFFERENTIATION | ADIPOGENESIS | IN-VITRO | GENE-EXPRESSION | PPAR-GAMMA | FABRICATION | STROMAL CELLS | OSTEOINDUCTIVITY | Receptors, Interleukin-1 Type II - metabolism | Nanoparticles - chemistry | Interleukin-1alpha - metabolism | Male | Autocrine Communication - drug effects | Tissue Scaffolds - chemistry | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Skull - pathology | Bone Morphogenetic Protein 2 - metabolism | Durapatite - pharmacology | Female | Skull - drug effects | Disease Models, Animal | Bone Regeneration - drug effects | Mesenchymal Stromal Cells - drug effects | Bone Marrow Cells - cytology | Osteogenesis - drug effects | Nanoparticles - ultrastructure | Fibroins - pharmacology | Biocompatible Materials - pharmacology | Rats, Sprague-Dawley | Cell Shape - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Paracrine Communication - drug effects | Cluster Analysis | Biological products | Interleukins | Tissue engineering | Analysis | Stem cells | Bone morphogenetic proteins | Gene expression | Surgical implants | Regeneration | Biomedical materials | Biocompatibility | Bones | Nanostructure | Scaffolds | Biomaterials
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 4, pp. 1486 - 1497.e14
Background & Aims Patients with pancreatic ductal adenocarcinoma are deficient in vitamin A, resulting in activation of pancreatic stellate cells (PSCs). We... 
Gastroenterology and Hepatology | 9RA | Therapy | Secreted Frizzled-Related Protein 4 | 13RA | Mouse Model | ACTIVATION | CANCER CELLS | TRANSCRIPTOME | ADENOCARCINOMA | STABILIZATION | MODEL | CULTURE | IN-VITRO | GASTROENTEROLOGY & HEPATOLOGY | IMMORTALIZATION | EXPRESSION | Transcription, Genetic - drug effects | Pancreatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Pancreatic Stellate Cells - drug effects | Carcinoma, Pancreatic Ductal - metabolism | Cellular Senescence - drug effects | Wnt Proteins - metabolism | Carcinoma, Pancreatic Ductal - genetics | Dose-Response Relationship, Drug | Pancreatic Neoplasms - drug therapy | RNA Interference | Time Factors | Mice, Mutant Strains | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Pancreatic Stellate Cells - pathology | Disease Models, Animal | Tretinoin - pharmacology | Proto-Oncogene Proteins - metabolism | Isotretinoin - pharmacology | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | beta Catenin - metabolism | Disease Progression | Carcinoma, Pancreatic Ductal - drug therapy | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Paracrine Communication - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Pancreatic Stellate Cells - metabolism
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2018, Volume 233, Issue 11, pp. 8418 - 8428
Journal Article
Journal of Hypertension, ISSN 0263-6352, 09/2010, Volume 28, Issue 9, pp. 1875 - 1882
Journal Article
Cytokine, ISSN 1043-4666, 06/2015, Volume 73, Issue 2, pp. 184 - 197
Journal Article