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Nature, ISSN 0028-0836, 01/2018, Volume 553, Issue 7689, pp. 501 - 505
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 189 - 205
Journal Article
2005, ISBN 1588295257, xi, 221
A comprehensive survey by leading basic and clinical researchers of the signal transduction mechanisms responsible for lung inflammation, including vascular... 
Vasculitis | pathology | Paracrine Communication | Mediators | Intercellular Signaling Peptides and Proteins | physiology | Cellular signal transduction | Inflammation | Surgery | Medicine & Public Health | Vascular Surgery
Book
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2017, Volume 312, Issue 6, pp. H1163 - H1175
Vascular endothelial growth factor B (VEGFB) is highly expressed in metabolically active tissues, such as the heart and skeletal muscle, suggesting a function... 
Heparanase | Endothelial cell | Streptozotocin diabetes | Vascular endothelial growth factor B | CARDIAC-FUNCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | GROWTH-FACTOR-B | endothelial cell | KINASE | heparanase | DILATED CARDIOMYOPATHY | LIPOPROTEIN-LIPASE | ENDOTHELIAL HEPARANASE SECRETION | SULFATE PROTEOGLYCANS | streptozotocin diabetes | FATTY-ACID | DISEASE | PERIPHERAL VASCULAR DISEASE | RAT-HEART | vascular endothelial growth factor B | Autocrine Communication | Diabetic Cardiomyopathies - metabolism | Paracrine Communication | Rats, Wistar | Signal Transduction | Streptozocin | Cells, Cultured | Glucuronidase - metabolism | Male | Myocardium - pathology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Apoptosis Regulatory Proteins - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Vascular Endothelial Growth Factor B - metabolism | Myocardium - metabolism | Apoptosis Regulatory Proteins - genetics | Diabetic Cardiomyopathies - pathology | Vascular Endothelial Growth Factor B - genetics | Diabetes Mellitus, Experimental - chemically induced | Diabetic Cardiomyopathies - chemically induced | Endothelial Cells - enzymology | Apoptosis | Vascular Endothelial Growth Factor Receptor-1 - genetics | Hyperglycemia | Cell death | Analysis | Muscles | Development and progression | Biochemistry | Gene expression | Vascular endothelial growth factor | Endothelium | Physiological aspects | Heart | Signal transduction | Diabetes | Glucose | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 4, pp. 1486 - 1497.e14
Background & Aims Patients with pancreatic ductal adenocarcinoma are deficient in vitamin A, resulting in activation of pancreatic stellate cells (PSCs). We... 
Gastroenterology and Hepatology | 9RA | Therapy | Secreted Frizzled-Related Protein 4 | 13RA | Mouse Model | ACTIVATION | CANCER CELLS | TRANSCRIPTOME | ADENOCARCINOMA | STABILIZATION | MODEL | CULTURE | IN-VITRO | GASTROENTEROLOGY & HEPATOLOGY | IMMORTALIZATION | EXPRESSION | Transcription, Genetic - drug effects | Pancreatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Pancreatic Stellate Cells - drug effects | Carcinoma, Pancreatic Ductal - metabolism | Cellular Senescence - drug effects | Wnt Proteins - metabolism | Carcinoma, Pancreatic Ductal - genetics | Dose-Response Relationship, Drug | Pancreatic Neoplasms - drug therapy | RNA Interference | Time Factors | Mice, Mutant Strains | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Pancreatic Stellate Cells - pathology | Disease Models, Animal | Tretinoin - pharmacology | Proto-Oncogene Proteins - metabolism | Isotretinoin - pharmacology | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | beta Catenin - metabolism | Disease Progression | Carcinoma, Pancreatic Ductal - drug therapy | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Paracrine Communication - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Pancreatic Stellate Cells - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Cancer Science, ISSN 1347-9032, 12/2018, Volume 109, Issue 12, pp. 3874 - 3882
Autocrine and paracrine factors, including glutamate and epidermal growth factor ( EGF ), are potent inducers of brain tumor cell invasion, a pathological... 
xCT | glioma | epidermal growth factor receptor | GluN2B | glutamate | MIGRATION | CELLS | TYROSINE PHOSPHORYLATION | NMDA RECEPTORS | EGFR | INVASION | ONCOLOGY | BIOLOGY | DEFICIENT | EXPRESSION | MODULATION | Neoplasm Transplantation | Phosphorylation | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Sulfasalazine - pharmacology | Brain Neoplasms - metabolism | Glioma - metabolism | Amino Acid Transport System y+ - metabolism | Receptors, N-Methyl-D-Aspartate - chemistry | Dizocilpine Maleate - administration & dosage | Protein Domains | Cell Survival - drug effects | ErbB Receptors - metabolism | Brain Neoplasms - drug therapy | Disease Progression | Drug Synergism | Sulfasalazine - administration & dosage | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Glutamic Acid - metabolism | Mice | Epidermal Growth Factor - pharmacology | Dizocilpine Maleate - pharmacology | Glioma - drug therapy | Methyl aspartate | Epidermal growth factor | Gliomas | Brain tumors | Development and progression | Aspartate | Glutamate | Immunoglobulins | Cell survival | Epidermal growth factor receptors | Paracrine signalling | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Kinases | Cell adhesion & migration | Glioma cells | Conflicts of interest | Sulfasalazine | Autocrine signalling | Cell migration | Tumors | Index Medicus | Original
Journal Article