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Nature (London), ISSN 0028-0836, 01/2018, Volume 553, Issue 7689, pp. 501 - 505
Journal Article
Nature (London), ISSN 1476-4687, 2018, Volume 553, Issue 7689, pp. 461 - 466
.... Dimerization of the stabilized ternary complexes and receptor activation remain dependent on the binding of heparan sulfate, a mandatory cofactor of paracrine FGF signalling... 
BETA-KLOTHO | TRANSCRIPTS ENCODING MEMBRANE | FIBROBLAST-GROWTH-FACTOR-1 FGF1 | STRUCTURAL BASIS | CRYSTAL-STRUCTURE | MULTIDISCIPLINARY SCIENCES | METABOLIC-ACTIVITY | RECEPTOR | FIBROBLAST-GROWTH-FACTOR | C-TERMINAL TAIL | HEPARIN | Humans | Protein Multimerization | Glucuronidase - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Male | Multiprotein Complexes - genetics | Heparitin Sulfate - metabolism | Fibroblast Growth Factors - chemistry | Fibroblast Growth Factors - metabolism | Multiprotein Complexes - metabolism | Protein Domains | Female | Paracrine Communication | Signal Transduction | Solubility | Models, Molecular | Glucuronidase - chemistry | Binding Sites - genetics | Multiprotein Complexes - chemistry | Animals | Glucuronidase - genetics | Body Fluids - metabolism | Protein Binding | Ligands | Mice | Receptor, Fibroblast Growth Factor, Type 1 - chemistry | Mutation | Binding | Heparan sulfate | Fibroblast growth factor | Fibroblast growth factor 23 | Homeostasis | Paracrine signalling | Metabolism | Proteins | Vitamin D | Klotho protein | Fibroblasts | Aging | Tethers | Receptor mechanisms | Sulfate | Atomic structure | Dimerization | Growth factors | Crystal structure | Fibroblast growth factor receptors | X-ray crystallography | BASIC BIOLOGICAL SCIENCES | Ageing
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2012, Volume 123, Issue 1, pp. 189 - 205
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 2017, Volume 312, Issue 6, pp. H1163 - H1175
.... The immediate response to high glucose and the secretion of endothelial heparanase is the release of this surface-bound VEGFB, which triggers signaling pathways and gene... 
Heparanase | Endothelial cell | Streptozotocin diabetes | Vascular endothelial growth factor B | CARDIAC-FUNCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | GROWTH-FACTOR-B | endothelial cell | KINASE | heparanase | DILATED CARDIOMYOPATHY | LIPOPROTEIN-LIPASE | ENDOTHELIAL HEPARANASE SECRETION | SULFATE PROTEOGLYCANS | streptozotocin diabetes | FATTY-ACID | DISEASE | PERIPHERAL VASCULAR DISEASE | RAT-HEART | vascular endothelial growth factor B | Autocrine Communication | Diabetic Cardiomyopathies - metabolism | Paracrine Communication | Rats, Wistar | Signal Transduction | Streptozocin | Cells, Cultured | Glucuronidase - metabolism | Male | Myocardium - pathology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Apoptosis Regulatory Proteins - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Vascular Endothelial Growth Factor B - metabolism | Myocardium - metabolism | Apoptosis Regulatory Proteins - genetics | Diabetic Cardiomyopathies - pathology | Vascular Endothelial Growth Factor B - genetics | Diabetes Mellitus, Experimental - chemically induced | Diabetic Cardiomyopathies - chemically induced | Endothelial Cells - enzymology | Apoptosis | Vascular Endothelial Growth Factor Receptor-1 - genetics | Physiological aspects | Cell death | Vascular endothelial growth factor | Endothelium | Hyperglycemia | Analysis | Muscles | Development and progression | Biochemistry | Gene expression
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2011, Volume 141, Issue 4, pp. 1486 - 1497.e14
Background & Aims Patients with pancreatic ductal adenocarcinoma are deficient in vitamin A, resulting in activation of pancreatic stellate cells (PSCs). We... 
Gastroenterology and Hepatology | 9RA | Therapy | Secreted Frizzled-Related Protein 4 | 13RA | Mouse Model | ACTIVATION | CANCER CELLS | TRANSCRIPTOME | ADENOCARCINOMA | STABILIZATION | MODEL | CULTURE | IN-VITRO | GASTROENTEROLOGY & HEPATOLOGY | IMMORTALIZATION | EXPRESSION | Transcription, Genetic - drug effects | Pancreatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Pancreatic Stellate Cells - drug effects | Carcinoma, Pancreatic Ductal - metabolism | Cellular Senescence - drug effects | Wnt Proteins - metabolism | Carcinoma, Pancreatic Ductal - genetics | Dose-Response Relationship, Drug | Pancreatic Neoplasms - drug therapy | RNA Interference | Time Factors | Mice, Mutant Strains | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Pancreatic Stellate Cells - pathology | Disease Models, Animal | Tretinoin - pharmacology | Proto-Oncogene Proteins - metabolism | Isotretinoin - pharmacology | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | beta Catenin - metabolism | Disease Progression | Carcinoma, Pancreatic Ductal - drug therapy | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Paracrine Communication - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Pancreatic Stellate Cells - metabolism
Journal Article