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1. Full Text Current concepts in the diagnosis and management of cytokine release syndrome
by Lee, Daniel W and Gardner, Rebecca and Porter, David L and Louis, Chrystal U and Ahmed, Nabil and Jensen, Michael and Grupp, Stephan A and Mackall, Crystal L
Blood, ISSN 0006-4971, 07/2014, Volume 124, Issue 2, pp. 188 - 195
As immune-based therapies for cancer become potent, more effective, and more widely available, optimal management of their unique toxicities becomes... 
C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | ADVERSE EVENT | MACROPHAGE ACTIVATION SYNDROME | ACUTE LYMPHOBLASTIC-LEUKEMIA | CHRONIC LYMPHOCYTIC-LEUKEMIA | MONOCLONAL-ANTIBODY | TNF-ALPHA | HEMATOLOGY | ANTI-INTERLEUKIN-6 RECEPTOR ANTIBODY | T-CELLS | Paraneoplastic Syndromes - therapy | Cytokines - metabolism | Humans | Leukemia - therapy | Paraneoplastic Syndromes - diagnosis | Leukemia - metabolism | Immune System Diseases - diagnosis | Immune System Diseases - metabolism | Young Adult | Leukemia - immunology | Paraneoplastic Syndromes - metabolism | Leukemia - diagnosis | Female | Immune System Diseases - therapy | Child | How I Treat
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2. Full Text Paraneoplastic endocrine syndromes
by Dimitriadis, Georgios K and Angelousi, Anna and Weickert, Martin O and Randeva, Harpal S and Kaltsas, Gregory and Grossman, Ashley
Endocrine-Related Cancer, ISSN 1351-0088, 06/2017, Volume 24, Issue 6, pp. R173 - R190
The majority of neoplasms are responsible for symptoms caused by mass effects to surrounding tissues and/or through the development of metastases. However,... 
Hypoglycaemia | Hypercalcaemia | Acromegaly | Cushing's syndrome | Paraneoplastic syndromes | Endocrine tumours | Siadh | hypoglycaemia | ECTOPIC SECRETION | ANTI-DIURETIC HORMONE | HUMAN CHORIONIC-GONADOTROPIN | hypercalcaemia | CELL LUNG-CANCER | CUSHINGS-SYNDROME | HORMONE-RELEASING-HORMONE | ONCOLOGY | BRONCHIAL CARCINOID-TUMOR | ENDOCRINOLOGY & METABOLISM | endocrine tumours | paraneoplastic syndromes | PARATHYROID-HORMONE | acromegaly | GROWTH-FACTOR | SIADH | PANCREATIC NEUROENDOCRINE TUMOR | Neoplasms - secretion | Animals | Paraneoplastic Endocrine Syndromes - etiology | Humans | Paraneoplastic Endocrine Syndromes - metabolism | Paraneoplastic Endocrine Syndromes - diagnosis | Peptide Hormones - secretion
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3. Full Text Pancreatic cancer-derived exosomes cause paraneoplastic β-cell dysfunction
by Javeed, Naureen and Sagar, Gunisha and Dutta, Shamit K and Smyrk, Thomas C and Lau, Julie S and Bhattacharya, Santanu and Truty, Mark and Petersen, Gloria M and Kaufman, Randal J and Chari, Suresh T and Mukhopadhyay, Debabrata
Clinical Cancer Research, ISSN 1078-0432, 04/2015, Volume 21, Issue 7, pp. 1722 - 1733
Purpose: Pancreatic cancer frequently causes diabetes. We recently proposed adrenomedullin as a candidate mediator of pancreatic beta-cell dysfunction in... 
DIAGNOSIS | HYPOTENSIVE PEPTIDE | ONCOLOGY | GLUCOSE-METABOLISM | INSULIN-RESISTANCE | INCREASE | ISLET AMYLOID POLYPEPTIDE | PREVALENCE | SECRETION | ADRENOMEDULLIN | Exosomes - metabolism | Pancreatic Neoplasms - metabolism | Humans | Pancreatic Neoplasms - pathology | Adrenomedullin - metabolism | Paraneoplastic Syndromes - etiology | Blotting, Western | Microscopy, Confocal | CA-19-9 Antigen - metabolism | Insulin-Secreting Cells - metabolism | Paraneoplastic Syndromes - metabolism | Diabetes Mellitus - etiology | Pancreatic Neoplasms - complications | Real-Time Polymerase Chain Reaction
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4. Full Text Metabolomics and transcriptomics identify pathway differences between visceral and subcutaneous adipose tissue in colorectal cancer patients: The ColoCare study
by Liesenfeld, David B and Grapov, Dmitry and Fahrmann, Johannes F and Salou, Mariam and Scherer, Dominique and Toth, Reka and Habermann, Nina and Böhm, Jürgen and Schrotz-King, Petra and Gigic, Biljana and Schneider, Martin and Ulrich, Alexis and Herpel, Esther and Schirmacher, Peter and Fiehn, Oliver and Lampe, Johanna W and Ulrich, Cornelia M
American Journal of Clinical Nutrition, ISSN 0002-9165, 08/2015, Volume 102, Issue 2, pp. 433 - 443
Background: Metabolic and transcriptomic differences between visceral adipose tissue (VAT) and subcutaneous adipose tissue (SAT) compartments, particularly in... 
Metabolomics | Obesity | Inflammation | Adipose tissue | Colorectal cancer | Visceral adiposity | EXTRACTION | NUTRITION & DIETETICS | colorectal cancer | ACID | metabolomics | inflammation | ETHER | adipose tissue | visceral adiposity | ASSOCIATION | EXPRESSION | obesity | Colorectal Neoplasms - surgery | Prospective Studies | Oligonucleotide Array Sequence Analysis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Gene Expression Profiling | Subcutaneous Fat, Abdominal - immunology | Female | Panniculitis - immunology | Paraneoplastic Syndromes - immunology | Carcinoma - pathology | Colorectal Neoplasms - metabolism | Biomarkers - metabolism | Intra-Abdominal Fat - immunology | Panniculitis - etiology | Carcinoma - surgery | Lipid Metabolism | Biomarkers - blood | Intra-Abdominal Fat - metabolism | Paraneoplastic Syndromes - etiology | Metabolomics - methods | Carcinoma - physiopathology | Colorectal Neoplasms - physiopathology | Paraneoplastic Syndromes - pathology | Subcutaneous Fat, Abdominal - pathology | Paraneoplastic Syndromes - metabolism | Aged | Carcinoma - metabolism | Intra-Abdominal Fat - pathology | Panniculitis - pathology | Colorectal Neoplasms - pathology | Neoplasm Staging | Subcutaneous Fat, Abdominal - metabolism | Panniculitis - metabolism | Cohort Studies
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5. Full Text X-Linked Hypophosphatemia and FGF23-Related Hypophosphatemic Diseases: Prospect for New Treatment
by Kinoshita, Yuka and Fukumoto, Seiji
Endocrine Reviews, ISSN 0163-769X, 06/2018, Volume 39, Issue 3, pp. 274 - 291
Phosphate plays essential roles in many biological processes, and the serum phosphate level is tightly controlled. Chronic hypophosphatemia causes impaired... 
OSTEOSCLEROTIC BONE DYSPLASIA | INDUCED VASCULAR CALCIFICATION | POSTERIOR LONGITUDINAL LIGAMENT | ENDOCRINOLOGY & METABOLISM | GROWTH-FACTOR 23 | TUMOR-INDUCED OSTEOMALACIA | MUSCLE-CELL CALCIFICATION | CHRONIC KIDNEY-DISEASE | PHOSPHATURIC MESENCHYMAL TUMOR | LEFT-VENTRICULAR HYPERTROPHY | ANTI-FGF23 ANTIBODY KRN23 | Familial Hypophosphatemic Rickets - drug therapy | Humans | Familial Hypophosphatemic Rickets - metabolism | Hypophosphatemia - metabolism | Antibodies, Monoclonal | Fibroblast Growth Factors - immunology | Fibroblast Growth Factors - metabolism | Osteomalacia - drug therapy | Paraneoplastic Syndromes - metabolism | Paraneoplastic Syndromes - drug therapy | Female | Osteomalacia - metabolism | Hypophosphatemia - drug therapy | Phosphates | Therapy | Fibroblast growth factor | Salts | Fibroblast growth factor 23 | Pathogenesis | Medical services | Clinical trials | Homology | Proximal tubules | Biomedical materials | Rickets | Vitamin D | Mineralization | Intestine | Phosphate | Hypophosphatemia | Biocompatibility | Osteomalacia | Bone matrix | Medical research | Medical treatment | Tubules | Reabsorption | Metabolism | Patients | Biological activity | Diseases | Endopeptidase | Monoclonal antibodies | Paraneoplastic syndrome | Bone | Mutation | Tumors
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6. Full Text Selectin-mucin interactions as a probable molecular explanation for the association of Trousseau syndrome with mucinous adenocarcinomas
by Wahrenbrock, Mark and Borsig, Lubor and Le, Dzung and Varki, Nissi and Varki, Ajit
Journal of Clinical Investigation, ISSN 0021-9738, 09/2003, Volume 112, Issue 6, pp. 853 - 862
Trousseau described spontaneous, recurrent superficial migratory thrombophlebitis associated with occult cancers, and this was later correlated with... 
THROMBOEMBOLIC DISEASE | MEDICINE, RESEARCH & EXPERIMENTAL | HUMAN NEUTROPHILS | OVARIAN-CANCER ANTIGEN | TYROSINE PHOSPHORYLATION | PANCREATIC-CANCER | P-SELECTIN | LEUKOCYTE INTERACTION | TISSUE FACTOR | COLON-CARCINOMA | GLYCOPROTEIN LIGAND-1 | Antifibrinolytic Agents - therapeutic use | Neoplasm Transplantation | P-Selectin - metabolism | Humans | Heparin - therapeutic use | Transplantation, Heterologous | Thrombophlebitis - drug therapy | L-Selectin - genetics | Lung - cytology | L-Selectin - metabolism | Fibrinolytic Agents - therapeutic use | Mucins - metabolism | Adenocarcinoma, Mucinous - chemistry | Lung - metabolism | Tumor Cells, Cultured | Vitamin K | Comorbidity | Mice, Inbred C57BL | P-Selectin - genetics | Syndrome | Thrombosis - metabolism | Animals | Blood Platelets - metabolism | Platelet Activation | Paraneoplastic Syndromes - metabolism | Adenocarcinoma, Mucinous - metabolism | Thrombophlebitis - metabolism | Mice | Thrombin - metabolism | Mucins - isolation & purification
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7. Full Text Onco-Nephrology: The Pathophysiology and Treatment of Malignancy-Associated Hypercalcemia
by Mitchell H. Rosner and Alan C. Dalkin
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 10/2012, Volume 7, Issue 10, pp. 1722 - 1729
Hypercalcemia complicates the course of 10%-30% of all patients with malignancies and can be a sign of very poor prognosis and advanced malignancy. Prompt... 
TUMOR-INDUCED HYPERCALCEMIA | ZOLEDRONIC ACID | BREAST-CANCER | PHASE-II TRIAL | HORMONE-RELATED PROTEIN | DOUBLE-BLIND | UROLOGY & NEPHROLOGY | PARATHYROID-HORMONE | CANCER-RELATED HYPERCALCEMIA | HIGH-DOSE PAMIDRONATE | BONE-RESORPTION | Neoplasms - metabolism | Paraneoplastic Syndromes - therapy | Humans | Hypercalcemia - therapy | Risk Factors | Osteolysis - physiopathology | Paraneoplastic Syndromes - physiopathology | Hypercalcemia - metabolism | Osteolysis - etiology | Paraneoplastic Syndromes - diagnosis | Treatment Outcome | Paraneoplastic Syndromes - etiology | Nephrology - trends | Neoplasms - complications | Neoplasms - therapy | Osteolysis - therapy | Hypercalcemia - physiopathology | Hypercalcemia - diagnosis | Hypercalcemia - etiology | Medical Oncology - trends | Paraneoplastic Syndromes - metabolism | Neoplasms - pathology
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8. Full Text Epiplakin Is a Paraneoplastic Pemphigus Autoantigen and Related to Bronchiolitis Obliterans in Japanese Patients
by Tsuchisaka, Atsunari and Numata, Sanae and Teye, Kwesi and Natsuaki, Yohei and Kawakami, Tamihiro and Takeda, Yoshito and Wang, Wenqing and Ishikawa, Kazushi and Goto, Mizuki and Koga, Hiroshi and Sogame, Ryosuke and Ishii, Norito and Takamori, Shinzo and Hoshino, Tomoaki and Brandt, Oliver and Pas, Hendri H and Fujiwara, Sakuhei and Hashimoto, Takashi
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2016, Volume 136, Issue 2, pp. 399 - 408
All plakin family proteins are known to be autoantigens in paraneoplastic pemphigus (PNP). In this study, we first examined whether PNP sera also react with... 
Autoantigens - metabolism | Immunoprecipitation | Autoantibodies - blood | Humans | Middle Aged | Male | Reference Values | Bronchiolitis Obliterans - metabolism | Sampling Studies | Pemphigus - metabolism | Statistics, Nonparametric | Female | Paraneoplastic Syndromes - immunology | Paraneoplastic Syndromes - ethnology | Cells, Cultured | Bronchiolitis Obliterans - immunology | Rats | Biomarkers - blood | Keratinocytes - immunology | Pemphigus - ethnology | Animals | Bronchiolitis Obliterans - ethnology | Keratinocytes - metabolism | Autoantigens - immunology | Paraneoplastic Syndromes - metabolism | Fluorescent Antibody Technique | Pemphigus - immunology | Aged | Mice | Asian Continental Ancestry Group - statistics & numerical data | AUTOANTIBODIES | IN-VIVO | PERIPLAKIN | AUTOIMMUNE MULTIORGAN SYNDROME | HUMAN EPIDERMAL AUTOANTIGEN | DESMOGLEIN 3 | VULGARIS | IDENTIFICATION | PLAKIN FAMILY | MEMBER | DERMATOLOGY
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9. Full Text Onco-Nephrology: Glomerular Diseases with Cancer
by Jean-François Cambier and Pierre Ronco
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 10/2012, Volume 7, Issue 10, pp. 1701 - 1712
Glomerular diseases occurring in the course of malignancies remain rare. Diverse glomerular lesions can be observed in a variety of neoplasms and involve... 
NODULAR GLOMERULOSCLEROSIS SECONDARY | HENOCH-SCHONLEIN PURPURA | MONOCLONAL IGG DEPOSITS | UROLOGY & NEPHROLOGY | CHRONIC LYMPHOCYTIC-LEUKEMIA | IMMUNOGLOBULIN LIGHT-CHAINS | CHANGE NEPHROTIC SYNDROME | OF-THE-LITERATURE | CHAIN DEPOSITION DISEASE | IDIOPATHIC MEMBRANOUS NEPHROPATHY | RENAL-CELL CARCINOMA | Hematologic Neoplasms - therapy | Neoplasms - metabolism | Paraneoplastic Syndromes - therapy | Hematologic Neoplasms - diagnosis | Glomerulonephritis - therapy | Prognosis | Biomarkers, Tumor - analysis | Humans | Paraneoplastic Syndromes - epidemiology | Glomerulonephritis - diagnosis | Paraneoplastic Syndromes - diagnosis | Neoplasms - diagnosis | Hematologic Neoplasms - epidemiology | Kidney Glomerulus - pathology | Glomerulonephritis - metabolism | Nephrology - trends | Neoplasms - therapy | Medical Oncology - trends | Paraneoplastic Syndromes - metabolism | Kidney Glomerulus - metabolism | Glomerulonephritis - epidemiology | Hematologic Neoplasms - metabolism | Neoplasms - epidemiology | Glomerulonephritis | Nephrology | Tumor Markers, Biological | Hematologic Neoplasms | Biochemistry, Molecular Biology | Genomics | Neoplasms | Life Sciences | Medical Oncology | Paraneoplastic Syndromes | Kidney Glomerulus
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10. Full Text Cancer Cachexia Syndrome: Pathogenesis, Diagnosis, and New Therapeutic Options
by Mondello, Patrizia and Mian, Michael and Aloisi, Carmela and Famà, Fausto and Mondello, Stefania and Pitini, Vincenzo
Nutrition and Cancer, ISSN 0163-5581, 01/2015, Volume 67, Issue 1, pp. 12 - 26
Cancer anorexia-cachexia syndrome (CACS) is the most frequent paraneoplastic syndrome occurring in half of all oncologic patients and is considered as a poor... 
5 DIFFERENT ARMS | ACUTE-PHASE RESPONSE | SKELETAL-MUSCLE | NUTRITION & DIETETICS | LEAN BODY-MASS | MUSCLE PROTEIN-SYNTHESIS | ONCOLOGY | ANOREXIA-CACHEXIA | DOUBLE-BLIND | III CLINICAL-TRIAL | QUALITY-OF-LIFE | PLACEBO-CONTROLLED TRIAL | Paraneoplastic Syndromes - therapy | Prognosis | Cachexia - diagnosis | Humans | Paraneoplastic Syndromes - diagnosis | Combined Modality Therapy | Paraneoplastic Syndromes - etiology | Animals | Cachexia - therapy | Paraneoplastic Syndromes - metabolism | Cachexia - etiology | Cachexia - metabolism | Disease Models, Animal
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11. Full Text Phosphaturic mesenchymal tumors show positive staining for somatostatin receptor 2A (SSTR2A)
by Houang, Michelle, MD and Clarkson, Adele, BSc and Sioson, Loretta, BSc and Elston, Marianne S., PhD and Clifton-Bligh, Roderick J., PhD and Dray, Michael, FRCPA and Ranchere-Vince, Dominique, MD and Decouvelaere, Anne-Valerie, PhD and de la Fouchardiere, Arnaud, PhD and Gill, Anthony J., FRCPA
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 12, pp. 2711 - 2718
Summary Tumor-induced osteomalacia (TIO) is a paraneoplastic syndrome associated with tumors that secrete phosphaturic hormones, most notably fibroblast growth... 
Pathology | Tumor-induced osteomalacia | FGF 23 | Gallium-68 DOTATATE PET/CT | Phosphaturic mesenchymal tumor | SSTR2A | FGF-23 | FIBROBLAST-GROWTH-FACTOR-23 | PATHOLOGY | ONCOGENIC OSTEOMALACIA | FGF23 | EXPRESSION | Mesenchymoma - metabolism | Neoplasms, Connective Tissue - diagnosis | Drugs, Chinese Herbal | Mesenchymoma - pathology | Humans | Middle Aged | Hypophosphatemia, Familial - metabolism | Male | Paraneoplastic Syndromes - diagnosis | Hypophosphatemia, Familial - diagnosis | Neoplasms, Connective Tissue - metabolism | Osteomalacia - pathology | Osteomalacia - diagnosis | Hypophosphatemia, Familial - pathology | Paraneoplastic Syndromes - pathology | Mesenchymoma - diagnosis | Paraneoplastic Syndromes - metabolism | Adult | Female | Osteomalacia - metabolism | Receptors, Somatostatin - metabolism | Neoplasms, Connective Tissue - pathology | Immunohistochemistry | Fibroblast growth factors | Sarcoma | Stem cells | Tomography | Bone cancer | Medical imaging | Cysts | Tumors
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