Blood, ISSN 0006-4971, 07/2014, Volume 124, Issue 2, pp. 188 - 195
As immune-based therapies for cancer become potent, more effective, and more widely available, optimal management of their unique toxicities becomes...
C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | ADVERSE EVENT | MACROPHAGE ACTIVATION SYNDROME | ACUTE LYMPHOBLASTIC-LEUKEMIA | CHRONIC LYMPHOCYTIC-LEUKEMIA | MONOCLONAL-ANTIBODY | TNF-ALPHA | HEMATOLOGY | ANTI-INTERLEUKIN-6 RECEPTOR ANTIBODY | T-CELLS | Paraneoplastic Syndromes - therapy | Cytokines - metabolism | Humans | Leukemia - therapy | Paraneoplastic Syndromes - diagnosis | Leukemia - metabolism | Immune System Diseases - diagnosis | Immune System Diseases - metabolism | Young Adult | Leukemia - immunology | Paraneoplastic Syndromes - metabolism | Leukemia - diagnosis | Female | Immune System Diseases - therapy | Child | How I Treat
C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | ADVERSE EVENT | MACROPHAGE ACTIVATION SYNDROME | ACUTE LYMPHOBLASTIC-LEUKEMIA | CHRONIC LYMPHOCYTIC-LEUKEMIA | MONOCLONAL-ANTIBODY | TNF-ALPHA | HEMATOLOGY | ANTI-INTERLEUKIN-6 RECEPTOR ANTIBODY | T-CELLS | Paraneoplastic Syndromes - therapy | Cytokines - metabolism | Humans | Leukemia - therapy | Paraneoplastic Syndromes - diagnosis | Leukemia - metabolism | Immune System Diseases - diagnosis | Immune System Diseases - metabolism | Young Adult | Leukemia - immunology | Paraneoplastic Syndromes - metabolism | Leukemia - diagnosis | Female | Immune System Diseases - therapy | Child | How I Treat
Journal Article
Endocrine-Related Cancer, ISSN 1351-0088, 06/2017, Volume 24, Issue 6, pp. R173 - R190
The majority of neoplasms are responsible for symptoms caused by mass effects to surrounding tissues and/or through the development of metastases. However,...
Hypoglycaemia | Hypercalcaemia | Acromegaly | Cushing's syndrome | Paraneoplastic syndromes | Endocrine tumours | Siadh | hypoglycaemia | ECTOPIC SECRETION | ANTI-DIURETIC HORMONE | HUMAN CHORIONIC-GONADOTROPIN | hypercalcaemia | CELL LUNG-CANCER | CUSHINGS-SYNDROME | HORMONE-RELEASING-HORMONE | ONCOLOGY | BRONCHIAL CARCINOID-TUMOR | ENDOCRINOLOGY & METABOLISM | endocrine tumours | paraneoplastic syndromes | PARATHYROID-HORMONE | acromegaly | GROWTH-FACTOR | SIADH | PANCREATIC NEUROENDOCRINE TUMOR | Neoplasms - secretion | Animals | Paraneoplastic Endocrine Syndromes - etiology | Humans | Paraneoplastic Endocrine Syndromes - metabolism | Paraneoplastic Endocrine Syndromes - diagnosis | Peptide Hormones - secretion
Hypoglycaemia | Hypercalcaemia | Acromegaly | Cushing's syndrome | Paraneoplastic syndromes | Endocrine tumours | Siadh | hypoglycaemia | ECTOPIC SECRETION | ANTI-DIURETIC HORMONE | HUMAN CHORIONIC-GONADOTROPIN | hypercalcaemia | CELL LUNG-CANCER | CUSHINGS-SYNDROME | HORMONE-RELEASING-HORMONE | ONCOLOGY | BRONCHIAL CARCINOID-TUMOR | ENDOCRINOLOGY & METABOLISM | endocrine tumours | paraneoplastic syndromes | PARATHYROID-HORMONE | acromegaly | GROWTH-FACTOR | SIADH | PANCREATIC NEUROENDOCRINE TUMOR | Neoplasms - secretion | Animals | Paraneoplastic Endocrine Syndromes - etiology | Humans | Paraneoplastic Endocrine Syndromes - metabolism | Paraneoplastic Endocrine Syndromes - diagnosis | Peptide Hormones - secretion
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 04/2015, Volume 21, Issue 7, pp. 1722 - 1733
Purpose: Pancreatic cancer frequently causes diabetes. We recently proposed adrenomedullin as a candidate mediator of pancreatic beta-cell dysfunction in...
DIAGNOSIS | HYPOTENSIVE PEPTIDE | ONCOLOGY | GLUCOSE-METABOLISM | INSULIN-RESISTANCE | INCREASE | ISLET AMYLOID POLYPEPTIDE | PREVALENCE | SECRETION | ADRENOMEDULLIN | Exosomes - metabolism | Pancreatic Neoplasms - metabolism | Humans | Pancreatic Neoplasms - pathology | Adrenomedullin - metabolism | Paraneoplastic Syndromes - etiology | Blotting, Western | Microscopy, Confocal | CA-19-9 Antigen - metabolism | Insulin-Secreting Cells - metabolism | Paraneoplastic Syndromes - metabolism | Diabetes Mellitus - etiology | Pancreatic Neoplasms - complications | Real-Time Polymerase Chain Reaction
DIAGNOSIS | HYPOTENSIVE PEPTIDE | ONCOLOGY | GLUCOSE-METABOLISM | INSULIN-RESISTANCE | INCREASE | ISLET AMYLOID POLYPEPTIDE | PREVALENCE | SECRETION | ADRENOMEDULLIN | Exosomes - metabolism | Pancreatic Neoplasms - metabolism | Humans | Pancreatic Neoplasms - pathology | Adrenomedullin - metabolism | Paraneoplastic Syndromes - etiology | Blotting, Western | Microscopy, Confocal | CA-19-9 Antigen - metabolism | Insulin-Secreting Cells - metabolism | Paraneoplastic Syndromes - metabolism | Diabetes Mellitus - etiology | Pancreatic Neoplasms - complications | Real-Time Polymerase Chain Reaction
Journal Article
American Journal of Clinical Nutrition, ISSN 0002-9165, 08/2015, Volume 102, Issue 2, pp. 433 - 443
Background: Metabolic and transcriptomic differences between visceral adipose tissue (VAT) and subcutaneous adipose tissue (SAT) compartments, particularly in...
Metabolomics | Obesity | Inflammation | Adipose tissue | Colorectal cancer | Visceral adiposity | EXTRACTION | NUTRITION & DIETETICS | colorectal cancer | ACID | metabolomics | inflammation | ETHER | adipose tissue | visceral adiposity | ASSOCIATION | EXPRESSION | obesity | Colorectal Neoplasms - surgery | Prospective Studies | Oligonucleotide Array Sequence Analysis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Gene Expression Profiling | Subcutaneous Fat, Abdominal - immunology | Female | Panniculitis - immunology | Paraneoplastic Syndromes - immunology | Carcinoma - pathology | Colorectal Neoplasms - metabolism | Biomarkers - metabolism | Intra-Abdominal Fat - immunology | Panniculitis - etiology | Carcinoma - surgery | Lipid Metabolism | Biomarkers - blood | Intra-Abdominal Fat - metabolism | Paraneoplastic Syndromes - etiology | Metabolomics - methods | Carcinoma - physiopathology | Colorectal Neoplasms - physiopathology | Paraneoplastic Syndromes - pathology | Subcutaneous Fat, Abdominal - pathology | Paraneoplastic Syndromes - metabolism | Aged | Carcinoma - metabolism | Intra-Abdominal Fat - pathology | Panniculitis - pathology | Colorectal Neoplasms - pathology | Neoplasm Staging | Subcutaneous Fat, Abdominal - metabolism | Panniculitis - metabolism | Cohort Studies
Metabolomics | Obesity | Inflammation | Adipose tissue | Colorectal cancer | Visceral adiposity | EXTRACTION | NUTRITION & DIETETICS | colorectal cancer | ACID | metabolomics | inflammation | ETHER | adipose tissue | visceral adiposity | ASSOCIATION | EXPRESSION | obesity | Colorectal Neoplasms - surgery | Prospective Studies | Oligonucleotide Array Sequence Analysis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Gene Expression Profiling | Subcutaneous Fat, Abdominal - immunology | Female | Panniculitis - immunology | Paraneoplastic Syndromes - immunology | Carcinoma - pathology | Colorectal Neoplasms - metabolism | Biomarkers - metabolism | Intra-Abdominal Fat - immunology | Panniculitis - etiology | Carcinoma - surgery | Lipid Metabolism | Biomarkers - blood | Intra-Abdominal Fat - metabolism | Paraneoplastic Syndromes - etiology | Metabolomics - methods | Carcinoma - physiopathology | Colorectal Neoplasms - physiopathology | Paraneoplastic Syndromes - pathology | Subcutaneous Fat, Abdominal - pathology | Paraneoplastic Syndromes - metabolism | Aged | Carcinoma - metabolism | Intra-Abdominal Fat - pathology | Panniculitis - pathology | Colorectal Neoplasms - pathology | Neoplasm Staging | Subcutaneous Fat, Abdominal - metabolism | Panniculitis - metabolism | Cohort Studies
Journal Article
Endocrine Reviews, ISSN 0163-769X, 06/2018, Volume 39, Issue 3, pp. 274 - 291
Phosphate plays essential roles in many biological processes, and the serum phosphate level is tightly controlled. Chronic hypophosphatemia causes impaired...
OSTEOSCLEROTIC BONE DYSPLASIA | INDUCED VASCULAR CALCIFICATION | POSTERIOR LONGITUDINAL LIGAMENT | ENDOCRINOLOGY & METABOLISM | GROWTH-FACTOR 23 | TUMOR-INDUCED OSTEOMALACIA | MUSCLE-CELL CALCIFICATION | CHRONIC KIDNEY-DISEASE | PHOSPHATURIC MESENCHYMAL TUMOR | LEFT-VENTRICULAR HYPERTROPHY | ANTI-FGF23 ANTIBODY KRN23 | Familial Hypophosphatemic Rickets - drug therapy | Humans | Familial Hypophosphatemic Rickets - metabolism | Hypophosphatemia - metabolism | Antibodies, Monoclonal | Fibroblast Growth Factors - immunology | Fibroblast Growth Factors - metabolism | Osteomalacia - drug therapy | Paraneoplastic Syndromes - metabolism | Paraneoplastic Syndromes - drug therapy | Female | Osteomalacia - metabolism | Hypophosphatemia - drug therapy | Phosphates | Therapy | Fibroblast growth factor | Salts | Fibroblast growth factor 23 | Pathogenesis | Medical services | Clinical trials | Homology | Proximal tubules | Biomedical materials | Rickets | Vitamin D | Mineralization | Intestine | Phosphate | Hypophosphatemia | Biocompatibility | Osteomalacia | Bone matrix | Medical research | Medical treatment | Tubules | Reabsorption | Metabolism | Patients | Biological activity | Diseases | Endopeptidase | Monoclonal antibodies | Paraneoplastic syndrome | Bone | Mutation | Tumors
OSTEOSCLEROTIC BONE DYSPLASIA | INDUCED VASCULAR CALCIFICATION | POSTERIOR LONGITUDINAL LIGAMENT | ENDOCRINOLOGY & METABOLISM | GROWTH-FACTOR 23 | TUMOR-INDUCED OSTEOMALACIA | MUSCLE-CELL CALCIFICATION | CHRONIC KIDNEY-DISEASE | PHOSPHATURIC MESENCHYMAL TUMOR | LEFT-VENTRICULAR HYPERTROPHY | ANTI-FGF23 ANTIBODY KRN23 | Familial Hypophosphatemic Rickets - drug therapy | Humans | Familial Hypophosphatemic Rickets - metabolism | Hypophosphatemia - metabolism | Antibodies, Monoclonal | Fibroblast Growth Factors - immunology | Fibroblast Growth Factors - metabolism | Osteomalacia - drug therapy | Paraneoplastic Syndromes - metabolism | Paraneoplastic Syndromes - drug therapy | Female | Osteomalacia - metabolism | Hypophosphatemia - drug therapy | Phosphates | Therapy | Fibroblast growth factor | Salts | Fibroblast growth factor 23 | Pathogenesis | Medical services | Clinical trials | Homology | Proximal tubules | Biomedical materials | Rickets | Vitamin D | Mineralization | Intestine | Phosphate | Hypophosphatemia | Biocompatibility | Osteomalacia | Bone matrix | Medical research | Medical treatment | Tubules | Reabsorption | Metabolism | Patients | Biological activity | Diseases | Endopeptidase | Monoclonal antibodies | Paraneoplastic syndrome | Bone | Mutation | Tumors
Journal Article
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Full Text
Onco-Nephrology: The Pathophysiology and Treatment of Malignancy-Associated Hypercalcemia
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 10/2012, Volume 7, Issue 10, pp. 1722 - 1729
Hypercalcemia complicates the course of 10%-30% of all patients with malignancies and can be a sign of very poor prognosis and advanced malignancy. Prompt...
TUMOR-INDUCED HYPERCALCEMIA | ZOLEDRONIC ACID | BREAST-CANCER | PHASE-II TRIAL | HORMONE-RELATED PROTEIN | DOUBLE-BLIND | UROLOGY & NEPHROLOGY | PARATHYROID-HORMONE | CANCER-RELATED HYPERCALCEMIA | HIGH-DOSE PAMIDRONATE | BONE-RESORPTION | Neoplasms - metabolism | Paraneoplastic Syndromes - therapy | Humans | Hypercalcemia - therapy | Risk Factors | Osteolysis - physiopathology | Paraneoplastic Syndromes - physiopathology | Hypercalcemia - metabolism | Osteolysis - etiology | Paraneoplastic Syndromes - diagnosis | Treatment Outcome | Paraneoplastic Syndromes - etiology | Nephrology - trends | Neoplasms - complications | Neoplasms - therapy | Osteolysis - therapy | Hypercalcemia - physiopathology | Hypercalcemia - diagnosis | Hypercalcemia - etiology | Medical Oncology - trends | Paraneoplastic Syndromes - metabolism | Neoplasms - pathology
TUMOR-INDUCED HYPERCALCEMIA | ZOLEDRONIC ACID | BREAST-CANCER | PHASE-II TRIAL | HORMONE-RELATED PROTEIN | DOUBLE-BLIND | UROLOGY & NEPHROLOGY | PARATHYROID-HORMONE | CANCER-RELATED HYPERCALCEMIA | HIGH-DOSE PAMIDRONATE | BONE-RESORPTION | Neoplasms - metabolism | Paraneoplastic Syndromes - therapy | Humans | Hypercalcemia - therapy | Risk Factors | Osteolysis - physiopathology | Paraneoplastic Syndromes - physiopathology | Hypercalcemia - metabolism | Osteolysis - etiology | Paraneoplastic Syndromes - diagnosis | Treatment Outcome | Paraneoplastic Syndromes - etiology | Nephrology - trends | Neoplasms - complications | Neoplasms - therapy | Osteolysis - therapy | Hypercalcemia - physiopathology | Hypercalcemia - diagnosis | Hypercalcemia - etiology | Medical Oncology - trends | Paraneoplastic Syndromes - metabolism | Neoplasms - pathology
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2003, Volume 112, Issue 6, pp. 853 - 862
Trousseau described spontaneous, recurrent superficial migratory thrombophlebitis associated with occult cancers, and this was later correlated with...
THROMBOEMBOLIC DISEASE | MEDICINE, RESEARCH & EXPERIMENTAL | HUMAN NEUTROPHILS | OVARIAN-CANCER ANTIGEN | TYROSINE PHOSPHORYLATION | PANCREATIC-CANCER | P-SELECTIN | LEUKOCYTE INTERACTION | TISSUE FACTOR | COLON-CARCINOMA | GLYCOPROTEIN LIGAND-1 | Antifibrinolytic Agents - therapeutic use | Neoplasm Transplantation | P-Selectin - metabolism | Humans | Heparin - therapeutic use | Transplantation, Heterologous | Thrombophlebitis - drug therapy | L-Selectin - genetics | Lung - cytology | L-Selectin - metabolism | Fibrinolytic Agents - therapeutic use | Mucins - metabolism | Adenocarcinoma, Mucinous - chemistry | Lung - metabolism | Tumor Cells, Cultured | Vitamin K | Comorbidity | Mice, Inbred C57BL | P-Selectin - genetics | Syndrome | Thrombosis - metabolism | Animals | Blood Platelets - metabolism | Platelet Activation | Paraneoplastic Syndromes - metabolism | Adenocarcinoma, Mucinous - metabolism | Thrombophlebitis - metabolism | Mice | Thrombin - metabolism | Mucins - isolation & purification
THROMBOEMBOLIC DISEASE | MEDICINE, RESEARCH & EXPERIMENTAL | HUMAN NEUTROPHILS | OVARIAN-CANCER ANTIGEN | TYROSINE PHOSPHORYLATION | PANCREATIC-CANCER | P-SELECTIN | LEUKOCYTE INTERACTION | TISSUE FACTOR | COLON-CARCINOMA | GLYCOPROTEIN LIGAND-1 | Antifibrinolytic Agents - therapeutic use | Neoplasm Transplantation | P-Selectin - metabolism | Humans | Heparin - therapeutic use | Transplantation, Heterologous | Thrombophlebitis - drug therapy | L-Selectin - genetics | Lung - cytology | L-Selectin - metabolism | Fibrinolytic Agents - therapeutic use | Mucins - metabolism | Adenocarcinoma, Mucinous - chemistry | Lung - metabolism | Tumor Cells, Cultured | Vitamin K | Comorbidity | Mice, Inbred C57BL | P-Selectin - genetics | Syndrome | Thrombosis - metabolism | Animals | Blood Platelets - metabolism | Platelet Activation | Paraneoplastic Syndromes - metabolism | Adenocarcinoma, Mucinous - metabolism | Thrombophlebitis - metabolism | Mice | Thrombin - metabolism | Mucins - isolation & purification
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2016, Volume 136, Issue 2, pp. 399 - 408
All plakin family proteins are known to be autoantigens in paraneoplastic pemphigus (PNP). In this study, we first examined whether PNP sera also react with...
AUTOANTIBODIES | IN-VIVO | PERIPLAKIN | AUTOIMMUNE MULTIORGAN SYNDROME | HUMAN EPIDERMAL AUTOANTIGEN | DESMOGLEIN 3 | VULGARIS | IDENTIFICATION | PLAKIN FAMILY | MEMBER | Autoantigens - metabolism | Immunoprecipitation | Autoantibodies - blood | Humans | Middle Aged | Male | Reference Values | Bronchiolitis Obliterans - metabolism | Sampling Studies | Pemphigus - metabolism | Statistics, Nonparametric | Female | Paraneoplastic Syndromes - immunology | Paraneoplastic Syndromes - ethnology | Cells, Cultured | Bronchiolitis Obliterans - immunology | Rats | Biomarkers - blood | Keratinocytes - immunology | Pemphigus - ethnology | Animals | Bronchiolitis Obliterans - ethnology | Keratinocytes - metabolism | Autoantigens - immunology | Paraneoplastic Syndromes - metabolism | Fluorescent Antibody Technique | Pemphigus - immunology | Aged | Mice | Asian Continental Ancestry Group - statistics & numerical data | DERMATOLOGY
AUTOANTIBODIES | IN-VIVO | PERIPLAKIN | AUTOIMMUNE MULTIORGAN SYNDROME | HUMAN EPIDERMAL AUTOANTIGEN | DESMOGLEIN 3 | VULGARIS | IDENTIFICATION | PLAKIN FAMILY | MEMBER | Autoantigens - metabolism | Immunoprecipitation | Autoantibodies - blood | Humans | Middle Aged | Male | Reference Values | Bronchiolitis Obliterans - metabolism | Sampling Studies | Pemphigus - metabolism | Statistics, Nonparametric | Female | Paraneoplastic Syndromes - immunology | Paraneoplastic Syndromes - ethnology | Cells, Cultured | Bronchiolitis Obliterans - immunology | Rats | Biomarkers - blood | Keratinocytes - immunology | Pemphigus - ethnology | Animals | Bronchiolitis Obliterans - ethnology | Keratinocytes - metabolism | Autoantigens - immunology | Paraneoplastic Syndromes - metabolism | Fluorescent Antibody Technique | Pemphigus - immunology | Aged | Mice | Asian Continental Ancestry Group - statistics & numerical data | DERMATOLOGY
Journal Article
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 10/2012, Volume 7, Issue 10, pp. 1701 - 1712
Glomerular diseases occurring in the course of malignancies remain rare. Diverse glomerular lesions can be observed in a variety of neoplasms and involve...
NODULAR GLOMERULOSCLEROSIS SECONDARY | HENOCH-SCHONLEIN PURPURA | MONOCLONAL IGG DEPOSITS | UROLOGY & NEPHROLOGY | CHRONIC LYMPHOCYTIC-LEUKEMIA | IMMUNOGLOBULIN LIGHT-CHAINS | CHANGE NEPHROTIC SYNDROME | OF-THE-LITERATURE | CHAIN DEPOSITION DISEASE | IDIOPATHIC MEMBRANOUS NEPHROPATHY | RENAL-CELL CARCINOMA | Hematologic Neoplasms - therapy | Neoplasms - metabolism | Paraneoplastic Syndromes - therapy | Hematologic Neoplasms - diagnosis | Glomerulonephritis - therapy | Prognosis | Biomarkers, Tumor - analysis | Humans | Paraneoplastic Syndromes - epidemiology | Glomerulonephritis - diagnosis | Paraneoplastic Syndromes - diagnosis | Neoplasms - diagnosis | Hematologic Neoplasms - epidemiology | Kidney Glomerulus - pathology | Glomerulonephritis - metabolism | Nephrology - trends | Neoplasms - therapy | Medical Oncology - trends | Paraneoplastic Syndromes - metabolism | Kidney Glomerulus - metabolism | Glomerulonephritis - epidemiology | Hematologic Neoplasms - metabolism | Neoplasms - epidemiology | Glomerulonephritis | Nephrology | Tumor Markers, Biological | Hematologic Neoplasms | Biochemistry, Molecular Biology | Genomics | Neoplasms | Life Sciences | Medical Oncology | Paraneoplastic Syndromes | Kidney Glomerulus
NODULAR GLOMERULOSCLEROSIS SECONDARY | HENOCH-SCHONLEIN PURPURA | MONOCLONAL IGG DEPOSITS | UROLOGY & NEPHROLOGY | CHRONIC LYMPHOCYTIC-LEUKEMIA | IMMUNOGLOBULIN LIGHT-CHAINS | CHANGE NEPHROTIC SYNDROME | OF-THE-LITERATURE | CHAIN DEPOSITION DISEASE | IDIOPATHIC MEMBRANOUS NEPHROPATHY | RENAL-CELL CARCINOMA | Hematologic Neoplasms - therapy | Neoplasms - metabolism | Paraneoplastic Syndromes - therapy | Hematologic Neoplasms - diagnosis | Glomerulonephritis - therapy | Prognosis | Biomarkers, Tumor - analysis | Humans | Paraneoplastic Syndromes - epidemiology | Glomerulonephritis - diagnosis | Paraneoplastic Syndromes - diagnosis | Neoplasms - diagnosis | Hematologic Neoplasms - epidemiology | Kidney Glomerulus - pathology | Glomerulonephritis - metabolism | Nephrology - trends | Neoplasms - therapy | Medical Oncology - trends | Paraneoplastic Syndromes - metabolism | Kidney Glomerulus - metabolism | Glomerulonephritis - epidemiology | Hematologic Neoplasms - metabolism | Neoplasms - epidemiology | Glomerulonephritis | Nephrology | Tumor Markers, Biological | Hematologic Neoplasms | Biochemistry, Molecular Biology | Genomics | Neoplasms | Life Sciences | Medical Oncology | Paraneoplastic Syndromes | Kidney Glomerulus
Journal Article
Nutrition and Cancer, ISSN 0163-5581, 01/2015, Volume 67, Issue 1, pp. 12 - 26
Cancer anorexia-cachexia syndrome (CACS) is the most frequent paraneoplastic syndrome occurring in half of all oncologic patients and is considered as a poor...
5 DIFFERENT ARMS | ACUTE-PHASE RESPONSE | SKELETAL-MUSCLE | NUTRITION & DIETETICS | LEAN BODY-MASS | MUSCLE PROTEIN-SYNTHESIS | ONCOLOGY | ANOREXIA-CACHEXIA | DOUBLE-BLIND | III CLINICAL-TRIAL | QUALITY-OF-LIFE | PLACEBO-CONTROLLED TRIAL | Paraneoplastic Syndromes - therapy | Prognosis | Cachexia - diagnosis | Humans | Paraneoplastic Syndromes - diagnosis | Combined Modality Therapy | Paraneoplastic Syndromes - etiology | Animals | Cachexia - therapy | Paraneoplastic Syndromes - metabolism | Cachexia - etiology | Cachexia - metabolism | Disease Models, Animal
5 DIFFERENT ARMS | ACUTE-PHASE RESPONSE | SKELETAL-MUSCLE | NUTRITION & DIETETICS | LEAN BODY-MASS | MUSCLE PROTEIN-SYNTHESIS | ONCOLOGY | ANOREXIA-CACHEXIA | DOUBLE-BLIND | III CLINICAL-TRIAL | QUALITY-OF-LIFE | PLACEBO-CONTROLLED TRIAL | Paraneoplastic Syndromes - therapy | Prognosis | Cachexia - diagnosis | Humans | Paraneoplastic Syndromes - diagnosis | Combined Modality Therapy | Paraneoplastic Syndromes - etiology | Animals | Cachexia - therapy | Paraneoplastic Syndromes - metabolism | Cachexia - etiology | Cachexia - metabolism | Disease Models, Animal
Journal Article
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 12, pp. 2711 - 2718
Summary Tumor-induced osteomalacia (TIO) is a paraneoplastic syndrome associated with tumors that secrete phosphaturic hormones, most notably fibroblast growth...
Pathology | Tumor-induced osteomalacia | FGF 23 | Gallium-68 DOTATATE PET/CT | Phosphaturic mesenchymal tumor | SSTR2A | FGF-23 | FIBROBLAST-GROWTH-FACTOR-23 | PATHOLOGY | ONCOGENIC OSTEOMALACIA | FGF23 | EXPRESSION | Mesenchymoma - metabolism | Neoplasms, Connective Tissue - diagnosis | Drugs, Chinese Herbal | Mesenchymoma - pathology | Humans | Middle Aged | Hypophosphatemia, Familial - metabolism | Male | Paraneoplastic Syndromes - diagnosis | Hypophosphatemia, Familial - diagnosis | Neoplasms, Connective Tissue - metabolism | Osteomalacia - pathology | Osteomalacia - diagnosis | Hypophosphatemia, Familial - pathology | Paraneoplastic Syndromes - pathology | Mesenchymoma - diagnosis | Paraneoplastic Syndromes - metabolism | Adult | Female | Osteomalacia - metabolism | Receptors, Somatostatin - metabolism | Neoplasms, Connective Tissue - pathology | Immunohistochemistry | Fibroblast growth factors | Sarcoma | Stem cells | Tomography | Bone cancer | Medical imaging | Cysts | Tumors
Pathology | Tumor-induced osteomalacia | FGF 23 | Gallium-68 DOTATATE PET/CT | Phosphaturic mesenchymal tumor | SSTR2A | FGF-23 | FIBROBLAST-GROWTH-FACTOR-23 | PATHOLOGY | ONCOGENIC OSTEOMALACIA | FGF23 | EXPRESSION | Mesenchymoma - metabolism | Neoplasms, Connective Tissue - diagnosis | Drugs, Chinese Herbal | Mesenchymoma - pathology | Humans | Middle Aged | Hypophosphatemia, Familial - metabolism | Male | Paraneoplastic Syndromes - diagnosis | Hypophosphatemia, Familial - diagnosis | Neoplasms, Connective Tissue - metabolism | Osteomalacia - pathology | Osteomalacia - diagnosis | Hypophosphatemia, Familial - pathology | Paraneoplastic Syndromes - pathology | Mesenchymoma - diagnosis | Paraneoplastic Syndromes - metabolism | Adult | Female | Osteomalacia - metabolism | Receptors, Somatostatin - metabolism | Neoplasms, Connective Tissue - pathology | Immunohistochemistry | Fibroblast growth factors | Sarcoma | Stem cells | Tomography | Bone cancer | Medical imaging | Cysts | Tumors
Journal Article