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1. Full Text Genetic mimics of cerebral palsy
by Pearson, Toni S and Pons, Roser and Ghaoui, Roula and Sue, Carolyn M
Movement disorders, ISSN 0885-3185, 05/2019, Volume 34, Issue 5, pp. 625 - 636
cerebral palsy | inborn errors of metabolism | spasticity | ataxia | dystonia | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Brain - diagnostic imaging | Monosaccharide Transport Proteins - deficiency | Lesch-Nyhan Syndrome - therapy | Spastic Paraplegia, Hereditary - diagnosis | Humans | Movement Disorders - diagnosis | Folic Acid Deficiency - therapy | Lesch-Nyhan Syndrome - genetics | Cerebral Palsy - diagnosis | Multiple Carboxylase Deficiency - genetics | Carbohydrate Metabolism, Inborn Errors - therapy | Brain Diseases, Metabolic, Inborn - genetics | Dyskinesias - genetics | Folic Acid Deficiency - genetics | Multiple Carboxylase Deficiency - physiopathology | Carbohydrate Metabolism, Inborn Errors - diagnosis | Monosaccharide Transport Proteins - genetics | Chorea - physiopathology | Creatine - genetics | Hyperargininemia - physiopathology | Dyskinesias - physiopathology | Dystonia - physiopathology | Pelizaeus-Merzbacher Disease - diagnosis | Plasma Membrane Neurotransmitter Transport Proteins - genetics | Carbohydrate Metabolism, Inborn Errors - physiopathology | Magnetic Resonance Imaging | Hyperargininemia - diagnosis | Movement Disorders - physiopathology | Dyskinesias - diagnosis | Hyperargininemia - genetics | Ataxia Telangiectasia - genetics | Lesch-Nyhan Syndrome - diagnosis | Brain Diseases, Metabolic, Inborn - therapy | Muscle Spasticity - physiopathology | Movement Disorders - genetics | Multiple Carboxylase Deficiency - diagnosis | Multiple Carboxylase Deficiency - therapy | GTP-Binding Protein alpha Subunits, Gi-Go - genetics | Spastic Paraplegia, Hereditary - genetics | Cerebral Palsy - physiopathology | Lesch-Nyhan Syndrome - physiopathology | Mental Retardation, X-Linked - physiopathology | Carbohydrate Metabolism, Inborn Errors - genetics | Brain Diseases, Metabolic, Inborn - diagnosis | Mental Retardation, X-Linked - genetics | Mental Retardation, X-Linked - diagnosis | Hyperargininemia - therapy | Creatine - deficiency | Thyroid Nuclear Factor 1 - genetics | Pelizaeus-Merzbacher Disease - therapy | Spastic Paraplegia, Hereditary - physiopathology | Mental Retardation, X-Linked - therapy | Ataxia Telangiectasia - diagnosis | Ataxia Telangiectasia - physiopathology | Plasma Membrane Neurotransmitter Transport Proteins - deficiency | Ataxia - physiopathology | Ataxia Telangiectasia - therapy | Diagnosis, Differential | Pelizaeus-Merzbacher Disease - genetics | Spastic Paraplegia, Hereditary - therapy | Brain Diseases, Metabolic, Inborn - physiopathology | Folic Acid Deficiency - physiopathology | Dyskinesias - therapy | Movement Disorders - therapy | Pelizaeus-Merzbacher Disease - physiopathology | Folic Acid Deficiency - diagnosis | Adenylyl Cyclases - genetics | Cerebral palsy | Development and progression | Medical genetics | Neuroimaging | Genetic counseling | Etiology | Chorea | Spasticity | Ataxia | Dystonia | Children | Paralysis | Neurodevelopmental disorders | Metabolic disorders | Index Medicus
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2. Full Text Paraplegia Following Image‐Guided Transforaminal Lumbar Spine Epidural Steroid Injection: Two Case Reports
by Kennedy, David J and Dreyfuss, Paul and Aprill, Charles N and Bogduk, Nikolai
Pain medicine (Malden, Mass.), ISSN 1526-2375, 11/2009, Volume 10, Issue 8, pp. 1389 - 1394
Epidural (Injection Space) | Steroids | Medicine, General & Internal | Anesthesiology | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Steroids - administration & dosage | Postoperative Complications - etiology | Humans | Postoperative Complications - prevention & control | Arteries - injuries | Fluoroscopy - methods | Male | Methylprednisolone - adverse effects | Iatrogenic Disease - prevention & control | Urinary Bladder, Neurogenic - chemically induced | Infarction - physiopathology | Embolism - physiopathology | Spinal Cord Injuries - pathology | Urinary Bladder, Neurogenic - physiopathology | Surgery, Computer-Assisted - adverse effects | Lumbar Vertebrae - surgery | Injections, Epidural - adverse effects | Low Back Pain - physiopathology | Radiculopathy - drug therapy | Paraplegia - physiopathology | Spinal Cord Injuries - chemically induced | Zygapophyseal Joint - physiopathology | Aged, 80 and over | Radiculopathy - physiopathology | Female | Paraplegia - chemically induced | Embolism - chemically induced | Infarction - chemically induced | Zygapophyseal Joint - drug effects | Infarction - pathology | Steroids - adverse effects | Steroids - chemistry | Postoperative Complications - physiopathology | Lumbar Vertebrae - diagnostic imaging | Embolism - pathology | Surgery, Computer-Assisted - methods | Arteries - pathology | Injections, Epidural - methods | Low Back Pain - drug therapy | Leg - innervation | Magnetic Resonance Imaging | Arteries - physiopathology | Aged | Lumbar Vertebrae - pathology | Spinal Cord Injuries - physiopathology | Leg - physiopathology | Methylprednisolone - administration & dosage | Complications and side effects | Care and treatment | Corticosteroids | Pain | Air pollution | Paralysis | Methylprednisolone | Air quality management | Index Medicus
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3. Full Text Disrupted mitochondrial function in the Opa3L122P mouse model for Costeff Syndrome impairs skeletal integrity
by Navein, Alice E and Cooke, Esther J and Davies, Jennifer R and Smith, Terence G and Wells, Lois H M and Ohazama, Atsushi and Healy, Christopher and Sharpe, Paul T and Evans, Sam L and Evans, Bronwen A J and Votruba, Marcela and Wells, Timothy
Human molecular genetics, ISSN 0964-6906, 06/2016, Volume 25, Issue 12, pp. 2404 - 2416
Spastic Paraplegia, Hereditary - genetics | Retina - growth & development | Humans | Mandible - growth & development | Tooth - growth & development | Brain - growth & development | Mutation, Missense | Skeleton - physiopathology | Mitochondria - genetics | Mandible - physiopathology | Spastic Paraplegia, Hereditary - physiopathology | Disease Models, Animal | Optic Atrophy - genetics | Chorea - physiopathology | Brain - physiopathology | Retina - physiopathology | Skeleton - growth & development | Optic Atrophy - physiopathology | Head - growth & development | Head - physiopathology | Mitochondria - pathology | Metabolism, Inborn Errors - genetics | Proteins - genetics | Animals | Tooth - physiopathology | Mice | Bone Development - genetics | Chorea - genetics | Metabolism, Inborn Errors - physiopathology | Index Medicus
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4. Full Text Defective kinesin binding of TUBB2A causes progressive spastic ataxia syndrome resembling sacsinopathy
by Sferra, Antonella and Fattori, Fabiana and Rizza, Teresa and Flex, Elsabetta and Bellacchio, Emanuele and Bruselles, Alessandro and Petrini, Stefania and Cecchetti, Serena and Teson, Massimo and Restaldi, Fabrizia and Ciolfi, Andrea and Santorelli, Filippo M and Zanni, Ginevra and Barresi, Sabina and Castiglioni, Claudia and Tartaglia, Marco and Bertini, Enrico
Human molecular genetics, ISSN 0964-6906, 06/2018, Volume 27, Issue 11, pp. 1892 - 1904
Biochemistry & Molecular Biology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Neurons - pathology | Humans | Paraplegia - genetics | Male | Tubulin - genetics | Cell Movement - genetics | Intellectual Disability - genetics | Paraplegia - physiopathology | Spindle Apparatus - genetics | Kinesin - genetics | Adult | Female | Neurons - metabolism | Child | Optic Atrophy - genetics | Spinocerebellar Ataxias - genetics | Cell Proliferation - genetics | Intellectual Disability - diagnostic imaging | Spinocerebellar Ataxias - diagnostic imaging | Optic Atrophy - physiopathology | Spinocerebellar Ataxias - physiopathology | Polyneuropathies - physiopathology | Spinocerebellar Degenerations - physiopathology | Microtubules - pathology | Spinocerebellar Degenerations - genetics | Sensorimotor Cortex - physiopathology | Intellectual Disability - physiopathology | Microtubules - genetics | Adolescent | Polyneuropathies - genetics | Protein Binding | Muscle Spasticity - physiopathology | Muscle Spasticity - diagnostic imaging | Sensorimotor Cortex - metabolism | Muscle Spasticity - genetics | Optic Atrophy - diagnostic imaging | Index Medicus
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5. Full Text Sternomastoid, rib cage, and expiratory muscle activity during weaning failure
by Sairam Parthasarathy and Amal Jubran and Franco Laghi and Martin J. Tobin
Journal of Applied Physiology, ISSN 8750-7587, 07/2007, Volume 103, Issue 1, pp. 140 - 147
Sternomastoid muscles | Respiratory muscles | Mechanical ventilation | Physiology | Sport Sciences | Life Sciences & Biomedicine | Science & Technology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Pneumonia - physiopathology | Pneumonia - therapy | Intercostal Muscles - innervation | Humans | Middle Aged | Heart Failure - physiopathology | Male | Pulmonary Disease, Chronic Obstructive - physiopathology | Heart Failure - blood | Paraplegia - blood | Esophagus - physiopathology | Heart Failure - therapy | Time Factors | Paraplegia - physiopathology | Treatment Failure | Pulmonary Disease, Chronic Obstructive - blood | Aged, 80 and over | Adult | Pneumonia - blood | Electromyography | Diaphragm - innervation | Diaphragm - physiopathology | Stomach - physiopathology | Neck Muscles - physiopathology | Respiratory Center - physiopathology | Neck Muscles - innervation | Sepsis - physiopathology | Carbon Dioxide - blood | Pressure | Oxygen - blood | Inhalation | Pulmonary Disease, Chronic Obstructive - therapy | Paraplegia - therapy | Sepsis - therapy | Aged | Sepsis - blood | Exhalation | Respiratory Function Tests | Ventilator Weaning | Intercostal Muscles - physiopathology | Hydrogen-Ion Concentration | Throat | Muscular system | Respiratory system | Patients | Index Medicus
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6. Full Text Neuromodulation of lumbosacral spinal networks enables independent stepping after complete paraplegia
by Gill, Megan L and Grahn, Peter J and Calvert, Jonathan S and Linde, Margaux B and Lavrov, Igor A and Strommen, Jeffrey A and Beck, Lisa A and Sayenko, Dimitry G and Van Straaten, Meegan G and Drubach, Dina I and Veith, Daniel D and Thoreson, Andrew R and Lopez, Cesar and Gerasimenko, Yury P and Edgerton, V. Reggie and Lee, Kendall H and Zhao, Kristin D
Nature medicine, ISSN 1078-8956, 11/2018, Volume 24, Issue 11, pp. 1677 - 1682
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Motor Activity - physiology | Electric Stimulation | Transcutaneous Electric Nerve Stimulation | Humans | Male | Paralysis - rehabilitation | Paraplegia - physiopathology | Muscle, Skeletal - physiopathology | Paralysis - physiopathology | Adult | Spinal Cord Injuries - rehabilitation | Electromyography | Paraplegia - rehabilitation | Spinal Cord Injuries - physiopathology | Nerve Net - physiopathology | Paraplegia | Care and treatment | Patient outcomes | Lumbosacral region | Neural stimulation | Health aspects | Methods | Spinal cord injuries | Brain | Networks | Spinal cord | Stability | Motor activity | Spinal cord injury | Neuromodulation | Hip | Weight | Electrical stimuli | Support systems | Training | Sensorimotor system | Rehabilitation | Paralysis | Extremities | Index Medicus
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7. Full Text Spinal cord injury alters purinergic neurotransmission to mesenteric arteries in rats
by Sangsiri, Sutheera and Xu, Hui and Fernandes, Roxanne and Fink, Greg D and Lujan, Heidi L and DiCarlo, Stephen E and Galligan, James J
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 02/2020, Volume 318, Issue 2, pp. H223 - H237
Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Blood Pressure | Mesenteric Arteries - physiopathology | Bradycardia - physiopathology | Rats | Male | Sympathetic Nervous System - physiopathology | Tachycardia - etiology | Rats, Sprague-Dawley | Tachycardia - physiopathology | Receptors, Purinergic - metabolism | Synaptic Transmission | Animals | Norepinephrine - metabolism | Quadriplegia - physiopathology | Excitatory Postsynaptic Potentials | Paraplegia - physiopathology | Adenosine Triphosphate - metabolism | Bradycardia - etiology | Mesenteric Arteries - innervation | Spinal Cord Injuries - physiopathology | Frequency dependence | Spinal cord | Spinal cord injury | Arteries | Paraplegia | Rodents | Nerves | Amperometry | Frequency response | Blood pressure | Reflexes | Spleen | Cardiac ventricles | Neurotransmitter release | Kidneys | Complications | Paraplegics | Quality of life | Electrical measurement | Tachycardia | Bradycardia | Neurotransmitters | Nitric oxide | Norepinephrine | Spinal cord injuries | ATP | Neurotransmission | Index Medicus
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8. Full Text Syndrome disintegration: Exome sequencing reveals that Fitzsimmons syndrome is a co-occurrence of multiple events
by Armour, Christine M and Smith, Amanda and Hartley, Taila and Chardon, Jodi Warman and Sawyer, Sarah and Schwartzentruber, Jeremy and Hennekam, Raoul and Majewski, Jacek and Bulman, Dennis E and Suri, Mohnish and Boycott, Kym M and FORGE Canada Consortium
American journal of medical genetics. Part A, ISSN 1552-4825, 2016, Volume 170A, Issue 7, pp. 1820 - 1825
Trichorhinophalangeal syndrome type 1 | concomitant mutations | exome sequencing | ARSACS | syndrome | Fitzsimmons syndrome | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Spastic Paraplegia, Hereditary - genetics | Dysarthria - physiopathology | Hair Diseases - genetics | Nose - abnormalities | Brachydactyly - physiopathology | Dysarthria - diagnosis | Spastic Paraplegia, Hereditary - diagnosis | Humans | Male | Fingers - physiopathology | Spinocerebellar Ataxias - congenital | Heat-Shock Proteins - genetics | Spinocerebellar Ataxias - diagnosis | Hair Diseases - physiopathology | Female | Fingers - abnormalities | Nuclear Proteins - genetics | Child | Spastic Paraplegia, Hereditary - physiopathology | Langer-Giedion Syndrome - physiopathology | Spinocerebellar Ataxias - genetics | Nose - physiopathology | Langer-Giedion Syndrome - genetics | Repressor Proteins - genetics | Spinocerebellar Ataxias - physiopathology | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Exome - genetics | Brachydactyly - genetics | Brachydactyly - diagnosis | Muscle Spasticity - diagnosis | High-Throughput Nucleotide Sequencing | Muscle Spasticity - physiopathology | Muscle Spasticity - genetics | Dysarthria - genetics | Autism | Boycotts | Twins | Analysis | Genomics | Index Medicus
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9. Full Text Lipids in the Physiopathology of Hereditary Spastic Paraplegias
by Darios, Frederic and Mochel, Fanny and Stevanin, Giovanni
Frontiers in neuroscience, ISSN 1662-4548, 02/2020, Volume 14, pp. 74 - 74
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cytochrome | Membranes | Cognitive ability | Homeostasis | Cytotoxicity | Lipids | Identification | Neurotoxicity | Mitochondria | Enzymatic activity | Sterols | Neurodegeneration | Ataxia | Lipid metabolism | Motor neurons | Enzymes | Phenotypes | Neurodegenerative diseases | Neurons | Spasticity | Metabolism | Cholesterol | Hereditary spastic paraplegia | Axons | Acids | Mutation | Endoplasmic reticulum | Bile | Life Sciences | Neurons and Cognition | Neurobiology | myelin | cholesterol | metabolism | phospholipids | fatty acids | sphingolipids
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