1965, 353
Book
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 12/2010, Volume 5, Issue 12, pp. 2305 - 2314
Background. and objectives: Cinacalcet is effective in reducing serum parathyroid hormone (PTH) in patients with secondary hyperparathyroidism. However, it has...
CALCIMIMETIC AGENT | HCL | CALCITRIOL-PULSE THERAPY | SENSING RECEPTOR | CALCIUM RECEPTOR | UROLOGY & NEPHROLOGY | VITAMIN-D-RECEPTOR | PROLIFERATION | HYPERPLASIA | EXPRESSION | DIALYSIS | Hyperparathyroidism, Secondary - metabolism | Naphthalenes - adverse effects | Parathyroid Hormone - secretion | Hyperparathyroidism, Secondary - drug therapy | Parathyroid Glands - drug effects | Humans | Middle Aged | Male | Parathyroid Hormone - blood | Cinacalcet Hydrochloride | Female | Hyperparathyroidism, Secondary - pathology | Aged | Parathyroid Glands - pathology | Naphthalenes - therapeutic use | Original
CALCIMIMETIC AGENT | HCL | CALCITRIOL-PULSE THERAPY | SENSING RECEPTOR | CALCIUM RECEPTOR | UROLOGY & NEPHROLOGY | VITAMIN-D-RECEPTOR | PROLIFERATION | HYPERPLASIA | EXPRESSION | DIALYSIS | Hyperparathyroidism, Secondary - metabolism | Naphthalenes - adverse effects | Parathyroid Hormone - secretion | Hyperparathyroidism, Secondary - drug therapy | Parathyroid Glands - drug effects | Humans | Middle Aged | Male | Parathyroid Hormone - blood | Cinacalcet Hydrochloride | Female | Hyperparathyroidism, Secondary - pathology | Aged | Parathyroid Glands - pathology | Naphthalenes - therapeutic use | Original
Journal Article
Endocrine-Related Cancer, ISSN 1351-0088, 01/2016, Volume 23, Issue 1, pp. R1 - R14
Five syndromes share predominantly hyperplastic glands with a primary excess of hormones: neonatal severe primary hyperparathyroidism, from homozygous mutated...
Signal transduction | Neonate | Gonad | Adrenal cortex | Cyclic AMP | GPCR | Proliferation | Parathyroid | Thyroid | ALDOSTERONE-PRODUCING ADENOMAS | proliferation | signal transduction | LEYDIG-CELL HYPERPLASIA | PROTEIN-COUPLED RECEPTORS | neonate | parathyroid | adrenal cortex | MALE SEXUAL PRECOCITY | thyroid | CALCIUM-SENSING RECEPTOR | ONCOLOGY | NON-AUTOIMMUNE HYPERTHYROIDISM | gonad | ENDOCRINOLOGY & METABOLISM | cyclic AMP | OVARIAN HYPERSTIMULATION SYNDROME | THYROTROPIN-RECEPTOR | FAMILIAL HYPOCALCIURIC HYPERCALCEMIA | SEVERE PRIMARY HYPERPARATHYROIDISM | Adenoma - genetics | Parathyroid Neoplasms - pathology | Humans | Hormones, Ectopic - secretion | Hyperthyroidism - metabolism | Male | Puberty, Precocious - genetics | Parathyroid Neoplasms - secretion | Endocrine Glands - pathology | Adult | Female | Child | Endocrine Glands - secretion | Hyperplasia - metabolism | Puberty, Precocious - metabolism | Hyperthyroidism - pathology | Puberty, Precocious - pathology | Parathyroid Neoplasms - genetics | Hyperplasia - pathology | Pregnancy | Parathyroid Glands - secretion | Adenoma - secretion | Hyperthyroidism - genetics | Adenoma - pathology | Hyperthyroidism - congenital | Parathyroid Glands - pathology
Signal transduction | Neonate | Gonad | Adrenal cortex | Cyclic AMP | GPCR | Proliferation | Parathyroid | Thyroid | ALDOSTERONE-PRODUCING ADENOMAS | proliferation | signal transduction | LEYDIG-CELL HYPERPLASIA | PROTEIN-COUPLED RECEPTORS | neonate | parathyroid | adrenal cortex | MALE SEXUAL PRECOCITY | thyroid | CALCIUM-SENSING RECEPTOR | ONCOLOGY | NON-AUTOIMMUNE HYPERTHYROIDISM | gonad | ENDOCRINOLOGY & METABOLISM | cyclic AMP | OVARIAN HYPERSTIMULATION SYNDROME | THYROTROPIN-RECEPTOR | FAMILIAL HYPOCALCIURIC HYPERCALCEMIA | SEVERE PRIMARY HYPERPARATHYROIDISM | Adenoma - genetics | Parathyroid Neoplasms - pathology | Humans | Hormones, Ectopic - secretion | Hyperthyroidism - metabolism | Male | Puberty, Precocious - genetics | Parathyroid Neoplasms - secretion | Endocrine Glands - pathology | Adult | Female | Child | Endocrine Glands - secretion | Hyperplasia - metabolism | Puberty, Precocious - metabolism | Hyperthyroidism - pathology | Puberty, Precocious - pathology | Parathyroid Neoplasms - genetics | Hyperplasia - pathology | Pregnancy | Parathyroid Glands - secretion | Adenoma - secretion | Hyperthyroidism - genetics | Adenoma - pathology | Hyperthyroidism - congenital | Parathyroid Glands - pathology
Journal Article
PLoS Genetics, ISSN 1553-7390, 2013, Volume 9, Issue 12, p. e1003975
Klotho acts as a co-receptor for and dictates tissue specificity of circulating FGF23. FGF23 inhibits PTH secretion, and reduced Klotho abundance is considered...
RECEPTOR 1 | HYPERPARATHYROIDISM | HORMONE | FIBROBLAST-GROWTH-FACTOR-23 | IN-VIVO | GENETICS & HEREDITY | GENE-EXPRESSION | RENAL-FUNCTION | FIBROBLAST GROWTH FACTOR-23 | CHRONIC KIDNEY-DISEASE | LEFT-VENTRICULAR HYPERTROPHY | Sequence Deletion | Cyclosporine - pharmacology | Membrane Proteins - genetics | Humans | Fibroblast Growth Factors - genetics | Signal Transduction - genetics | Parathyroid Hormone - blood | Parathyroid Hormone - genetics | Gene Expression Regulation - drug effects | Calcineurin Inhibitors | Calcium - blood | Fibroblast Growth Factors - metabolism | Kidney - metabolism | Animals | Parathyroid Glands - secretion | Vitamin D - metabolism | Membrane Proteins - metabolism | Mice | Calcineurin - metabolism | Parathyroid Glands - metabolism | Physiological aspects | Genetic research | Genetic aspects | Health aspects | Calcineurin | Hyperparathyroidism | Bone density | Statistical analysis | Hormones | Gene expression | Endocrine system | Rodents
RECEPTOR 1 | HYPERPARATHYROIDISM | HORMONE | FIBROBLAST-GROWTH-FACTOR-23 | IN-VIVO | GENETICS & HEREDITY | GENE-EXPRESSION | RENAL-FUNCTION | FIBROBLAST GROWTH FACTOR-23 | CHRONIC KIDNEY-DISEASE | LEFT-VENTRICULAR HYPERTROPHY | Sequence Deletion | Cyclosporine - pharmacology | Membrane Proteins - genetics | Humans | Fibroblast Growth Factors - genetics | Signal Transduction - genetics | Parathyroid Hormone - blood | Parathyroid Hormone - genetics | Gene Expression Regulation - drug effects | Calcineurin Inhibitors | Calcium - blood | Fibroblast Growth Factors - metabolism | Kidney - metabolism | Animals | Parathyroid Glands - secretion | Vitamin D - metabolism | Membrane Proteins - metabolism | Mice | Calcineurin - metabolism | Parathyroid Glands - metabolism | Physiological aspects | Genetic research | Genetic aspects | Health aspects | Calcineurin | Hyperparathyroidism | Bone density | Statistical analysis | Hormones | Gene expression | Endocrine system | Rodents
Journal Article
Thyroid, ISSN 1050-7256, 12/2010, Volume 20, Issue 12, pp. 1371 - 1375
Background: Thyroidectomy continues to pose the risk of typical complications, including postoperative hypocalcemia and permanent hypoparathyroidism. The...
SURGERY | PRESERVATION | RECOVERY | IMPACT | MULTICENTER | MORBIDITY | HYPOPARATHYROIDISM | ENDOCRINOLOGY & METABOLISM | GRAVES-DISEASE | TERM-FOLLOW-UP | COMPLICATIONS | Intraoperative Period | Parathyroid Hormone - secretion | Prospective Studies | Hypocalcemia - etiology | Humans | Middle Aged | Color | Postoperative Period | Male | Transplantation, Autologous | Parathyroid Hormone - blood | Thyroidectomy - adverse effects | Parathyroid Glands - transplantation | Parathyroid Glands - secretion | Adult | Female | Aged | Retrospective Studies | Parathyroid hormone | Complications | Surgery | Physiological aspects | Transplantation of organs, tissues, etc | Research | Health aspects | Thyroidectomy
SURGERY | PRESERVATION | RECOVERY | IMPACT | MULTICENTER | MORBIDITY | HYPOPARATHYROIDISM | ENDOCRINOLOGY & METABOLISM | GRAVES-DISEASE | TERM-FOLLOW-UP | COMPLICATIONS | Intraoperative Period | Parathyroid Hormone - secretion | Prospective Studies | Hypocalcemia - etiology | Humans | Middle Aged | Color | Postoperative Period | Male | Transplantation, Autologous | Parathyroid Hormone - blood | Thyroidectomy - adverse effects | Parathyroid Glands - transplantation | Parathyroid Glands - secretion | Adult | Female | Aged | Retrospective Studies | Parathyroid hormone | Complications | Surgery | Physiological aspects | Transplantation of organs, tissues, etc | Research | Health aspects | Thyroidectomy
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 09/2015, Volume 26, Issue 9, pp. 2163 - 2171
The calcium-sensing receptor (CaR) modulates renal calciumreabsorption and parathyroid hormone (PTH) secretion and is involved in the etiology of secondary...
HORMONE SECRETION | DOMAIN | PROTONATION SITE | CA2+-BINDING SITES | RESPIRATORY-ACIDOSIS | INCREASES | UROLOGY & NEPHROLOGY | METABOTROPIC GLUTAMATE RECEPTORS | DOG | CA2+-SENSING RECEPTOR | DEPENDENCE | Parathyroid Hormone - secretion | Phosphorylation | Humans | Histidine - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Histidine - metabolism | Acidosis - metabolism | Receptors, Calcium-Sensing - metabolism | Alkalosis - metabolism | Cysteine - genetics | Animals | Cattle | Parathyroid Glands - secretion | HEK293 Cells | Cysteine - metabolism | Hydrogen-Ion Concentration | Parathyroid Glands - metabolism | parathyroid hormone | calcium-sensing receptor | mineral metabolism | hyperparathyroidism | acidosis | Basic Research
HORMONE SECRETION | DOMAIN | PROTONATION SITE | CA2+-BINDING SITES | RESPIRATORY-ACIDOSIS | INCREASES | UROLOGY & NEPHROLOGY | METABOTROPIC GLUTAMATE RECEPTORS | DOG | CA2+-SENSING RECEPTOR | DEPENDENCE | Parathyroid Hormone - secretion | Phosphorylation | Humans | Histidine - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Histidine - metabolism | Acidosis - metabolism | Receptors, Calcium-Sensing - metabolism | Alkalosis - metabolism | Cysteine - genetics | Animals | Cattle | Parathyroid Glands - secretion | HEK293 Cells | Cysteine - metabolism | Hydrogen-Ion Concentration | Parathyroid Glands - metabolism | parathyroid hormone | calcium-sensing receptor | mineral metabolism | hyperparathyroidism | acidosis | Basic Research
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 11/2013, Volume 98, Issue 11, pp. 4285 - 4290
Context: Transient and permanent postoperative hypoparathyroidism are recognized complications of neck surgery. Postoperative hypoparathyroidism is usually...
CELLS | PTH(1-84) | THERAPY | THYROIDECTOMY | ENDOCRINOLOGY & METABOLISM | Parathyroid Hormone - secretion | Graves Disease - surgery | Parathyroidectomy - adverse effects | Humans | Middle Aged | Postoperative Complications - metabolism | Transplantation, Autologous | Thyroidectomy - adverse effects | Hypoparathyroidism - metabolism | Parathyroid Glands - transplantation | Parathyroid Glands - secretion | Recovery of Function - physiology | Adult | Female | Aged | Hyperparathyroidism - surgery
CELLS | PTH(1-84) | THERAPY | THYROIDECTOMY | ENDOCRINOLOGY & METABOLISM | Parathyroid Hormone - secretion | Graves Disease - surgery | Parathyroidectomy - adverse effects | Humans | Middle Aged | Postoperative Complications - metabolism | Transplantation, Autologous | Thyroidectomy - adverse effects | Hypoparathyroidism - metabolism | Parathyroid Glands - transplantation | Parathyroid Glands - secretion | Recovery of Function - physiology | Adult | Female | Aged | Hyperparathyroidism - surgery
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 08/2013, Volume 98, Issue 8, pp. E1345 - E1351
Context and Objective: Caffeine is a highly consumed psychoactive substance present in our daily drinks. Independent studies have reported associations between...
WOMEN | RISK-FACTORS | CA2 | CALCIUM | ENDOCRINOLOGY & METABOLISM | RELEASE | BONE | EXPRESSION | BRAIN | OSTEOPOROSIS | CONSUMPTION | Cyclic AMP - analysis | Cyclic AMP-Dependent Protein Kinases - metabolism | Parathyroid Hormone - secretion | Receptors, Purinergic P1 - genetics | Calcium - metabolism | Parathyroid Glands - drug effects | Humans | Middle Aged | Male | Parathyroid Hormone - genetics | Parathyroid Glands - cytology | Parathyroid Glands - secretion | Aged, 80 and over | Caffeine - pharmacology | Adult | Female | Aged | Receptors, Purinergic P1 - analysis | Hot Topics in Translational Endocrinology
WOMEN | RISK-FACTORS | CA2 | CALCIUM | ENDOCRINOLOGY & METABOLISM | RELEASE | BONE | EXPRESSION | BRAIN | OSTEOPOROSIS | CONSUMPTION | Cyclic AMP - analysis | Cyclic AMP-Dependent Protein Kinases - metabolism | Parathyroid Hormone - secretion | Receptors, Purinergic P1 - genetics | Calcium - metabolism | Parathyroid Glands - drug effects | Humans | Middle Aged | Male | Parathyroid Hormone - genetics | Parathyroid Glands - cytology | Parathyroid Glands - secretion | Aged, 80 and over | Caffeine - pharmacology | Adult | Female | Aged | Receptors, Purinergic P1 - analysis | Hot Topics in Translational Endocrinology
Journal Article
Kidney International, ISSN 0085-2538, 09/2005, Volume 68, Issue 3, pp. 998 - 1007
Structure of non-(1-84) PTH fragments secreted by parathyroid glands in primary and secondary hyperparathyroidism. Non-(1-84) parathyroid hormone (PTH)...
parathyroid hormone | secondary hyperparathyroidism | calcium | primary hyperparathyroidism | non-(1-84) parathyroid hormone | Primary hyperparathyroidism | Non-(1-84) parathyroid hormone | Calcium | Parathyroid hormone | Secondary hyperparathyroidism | CELLS | CARBOXYL-TERMINAL FRAGMENTS | RATS | RECEPTOR | IDENTIFICATION | PTH-(1-84) | HORMONE | RENAL-FAILURE | UROLOGY & NEPHROLOGY | ASSAYS | BONE | Amino Acid Sequence | Hyperparathyroidism, Secondary - metabolism | Parathyroid Hormone - secretion | Peptide Fragments - metabolism | Humans | Peptide Fragments - secretion | Molecular Sequence Data | Male | Chromatography, High Pressure Liquid | Parathyroid Hormone - chemistry | Hyperparathyroidism, Primary - metabolism | Peptide Fragments - chemistry | Parathyroid Glands - secretion | Parathyroid Hormone - metabolism | Female | Parathyroid Glands - metabolism
parathyroid hormone | secondary hyperparathyroidism | calcium | primary hyperparathyroidism | non-(1-84) parathyroid hormone | Primary hyperparathyroidism | Non-(1-84) parathyroid hormone | Calcium | Parathyroid hormone | Secondary hyperparathyroidism | CELLS | CARBOXYL-TERMINAL FRAGMENTS | RATS | RECEPTOR | IDENTIFICATION | PTH-(1-84) | HORMONE | RENAL-FAILURE | UROLOGY & NEPHROLOGY | ASSAYS | BONE | Amino Acid Sequence | Hyperparathyroidism, Secondary - metabolism | Parathyroid Hormone - secretion | Peptide Fragments - metabolism | Humans | Peptide Fragments - secretion | Molecular Sequence Data | Male | Chromatography, High Pressure Liquid | Parathyroid Hormone - chemistry | Hyperparathyroidism, Primary - metabolism | Peptide Fragments - chemistry | Parathyroid Glands - secretion | Parathyroid Hormone - metabolism | Female | Parathyroid Glands - metabolism
Journal Article
10.
Full Text
Direct effect of phosphorus on PTH secretion from whole rat parathyroid glands in vitro
Journal of Bone and Mineral Research, ISSN 0884-0431, 07/1996, Volume 11, Issue 7, pp. 970 - 976
Phosphorus retention is an important factor in the development of hyperparathyroidism secondary to renal failure. In vivo manipulation of phosphorus is...
SERUM CONCENTRATION | UREMIA | HORMONE | CALCIUM | ENDOCRINOLOGY & METABOLISM | RENAL-FAILURE | SECONDARY HYPERPARATHYROIDISM | RECEPTOR | RESTRICTION | 1,25-DIHYDROXYVITAMIN-D | CALCITRIOL | Phosphorus - pharmacology | Parathyroid Hormone - secretion | Animals | Calcium - pharmacology | Analysis of Variance | Rats, Wistar | Culture Techniques | Parathyroid Glands - drug effects | Parathyroid Glands - secretion | Secretory Rate - drug effects | Arachidonic Acid - pharmacology | Rats
SERUM CONCENTRATION | UREMIA | HORMONE | CALCIUM | ENDOCRINOLOGY & METABOLISM | RENAL-FAILURE | SECONDARY HYPERPARATHYROIDISM | RECEPTOR | RESTRICTION | 1,25-DIHYDROXYVITAMIN-D | CALCITRIOL | Phosphorus - pharmacology | Parathyroid Hormone - secretion | Animals | Calcium - pharmacology | Analysis of Variance | Rats, Wistar | Culture Techniques | Parathyroid Glands - drug effects | Parathyroid Glands - secretion | Secretory Rate - drug effects | Arachidonic Acid - pharmacology | Rats
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 2005, Volume 16, Issue 7, pp. 2190 - 2197
In uremic patients, severe parathyroid hyperplasia is associated with reduced parathyroid calcium-sensing receptor (CaR) expression. Thus, in these patients, a...
CELLS | RESPONSIVENESS | MESSENGER-RIBONUCLEIC-ACID | PROTEIN | PTH | SET-POINT | TISSUE | SECONDARY HYPERPARATHYROIDISM | UROLOGY & NEPHROLOGY | RELEASE | CHRONIC-RENAL-FAILURE | Humans | Hyperplasia | Hyperparathyroidism, Secondary - physiopathology | Male | Uremia - therapy | Kidney Failure, Chronic - therapy | Parathyroid Glands - secretion | Parathyroid Hormone - biosynthesis | Kidney Failure, Chronic - complications | Female | Uremia - etiology | Hyperparathyroidism, Secondary - etiology | In Vitro Techniques | Parathyroid Glands - pathology | Receptors, Calcium-Sensing - biosynthesis | Renal Dialysis
CELLS | RESPONSIVENESS | MESSENGER-RIBONUCLEIC-ACID | PROTEIN | PTH | SET-POINT | TISSUE | SECONDARY HYPERPARATHYROIDISM | UROLOGY & NEPHROLOGY | RELEASE | CHRONIC-RENAL-FAILURE | Humans | Hyperplasia | Hyperparathyroidism, Secondary - physiopathology | Male | Uremia - therapy | Kidney Failure, Chronic - therapy | Parathyroid Glands - secretion | Parathyroid Hormone - biosynthesis | Kidney Failure, Chronic - complications | Female | Uremia - etiology | Hyperparathyroidism, Secondary - etiology | In Vitro Techniques | Parathyroid Glands - pathology | Receptors, Calcium-Sensing - biosynthesis | Renal Dialysis
Journal Article
Saudi journal of kidney diseases and transplantation : an official publication of the Saudi Center for Organ Transplantation, Saudi Arabia, ISSN 1319-2442, 03/2016, Volume 27, Issue 2, pp. 263 - 269
Characteristics of parathyroid glands usually determined by ultrasonography such as its total weight or volume might be a good indicator for the induction or...
Multivariate Analysis | Predictive Value of Tests | Ultrasonography, Doppler, Color | Humans | Middle Aged | Male | Parathyroid Hormone - blood | Parathyroidectomy | Renal Insufficiency, Chronic - complications | Parathyroid Glands - blood supply | Renal Insufficiency, Chronic - therapy | Iran | Adult | Female | Renal Insufficiency, Chronic - diagnosis | Alkaline Phosphatase - blood | Parathyroid Hormone - secretion | Cross-Sectional Studies | Enzyme-Linked Immunosorbent Assay | Hospitals, University | Organ Size | Linear Models | Biomarkers - blood | Regional Blood Flow | Renal Insufficiency, Chronic - blood | Hyperparathyroidism, Secondary - blood | Hyperparathyroidism, Secondary - diagnosis | Parathyroid Glands - diagnostic imaging | Parathyroid Glands - secretion | Parathyroid Glands - surgery | Blood Flow Velocity | Renal Dialysis - adverse effects | Aged | Hyperparathyroidism, Secondary - etiology | Care and treatment | Parathyroid hormone | Ultrasound imaging | Hemodialysis | Analysis | Physiological aspects | Research | Health aspects | Hyperparathyroidism
Multivariate Analysis | Predictive Value of Tests | Ultrasonography, Doppler, Color | Humans | Middle Aged | Male | Parathyroid Hormone - blood | Parathyroidectomy | Renal Insufficiency, Chronic - complications | Parathyroid Glands - blood supply | Renal Insufficiency, Chronic - therapy | Iran | Adult | Female | Renal Insufficiency, Chronic - diagnosis | Alkaline Phosphatase - blood | Parathyroid Hormone - secretion | Cross-Sectional Studies | Enzyme-Linked Immunosorbent Assay | Hospitals, University | Organ Size | Linear Models | Biomarkers - blood | Regional Blood Flow | Renal Insufficiency, Chronic - blood | Hyperparathyroidism, Secondary - blood | Hyperparathyroidism, Secondary - diagnosis | Parathyroid Glands - diagnostic imaging | Parathyroid Glands - secretion | Parathyroid Glands - surgery | Blood Flow Velocity | Renal Dialysis - adverse effects | Aged | Hyperparathyroidism, Secondary - etiology | Care and treatment | Parathyroid hormone | Ultrasound imaging | Hemodialysis | Analysis | Physiological aspects | Research | Health aspects | Hyperparathyroidism
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 07/2012, Volume 97, Issue 7, pp. 2341 - 2346
Context: Hypocalcemia, transient or permanent, represents a common complication after total thyroidectomy, but data on the secretory capacity of the...
SURGERY | RISK-FACTORS | AUTOTRANSPLANTATION | HYPOPARATHYROIDISM | ENDOCRINOLOGY & METABOLISM | MULTINODULAR GOITER | BICARBONATE INFUSION TEST | HYPOCALCEMIA | COMPLICATIONS | ALENDRONATE | Parathyroid Glands - physiology | Thyroid Nodule - blood | Hypocalcemia - metabolism | Humans | Middle Aged | Hypocalcemia - blood | Male | Reference Values | Sodium Bicarbonate - administration & dosage | Calcium - analysis | Parathyroid Hormone - blood | Calcium - blood | Adult | Female | Diagnostic Techniques, Endocrine - standards | Parathyroid Hormone - secretion | Hypocalcemia - etiology | Postoperative Period | Thyroidectomy - methods | Thyroid Nodule - metabolism | Algorithms | Parathyroid Glands - secretion | Infusions, Intravenous | Thyroid Nodule - surgery | Thyroidectomy - rehabilitation
SURGERY | RISK-FACTORS | AUTOTRANSPLANTATION | HYPOPARATHYROIDISM | ENDOCRINOLOGY & METABOLISM | MULTINODULAR GOITER | BICARBONATE INFUSION TEST | HYPOCALCEMIA | COMPLICATIONS | ALENDRONATE | Parathyroid Glands - physiology | Thyroid Nodule - blood | Hypocalcemia - metabolism | Humans | Middle Aged | Hypocalcemia - blood | Male | Reference Values | Sodium Bicarbonate - administration & dosage | Calcium - analysis | Parathyroid Hormone - blood | Calcium - blood | Adult | Female | Diagnostic Techniques, Endocrine - standards | Parathyroid Hormone - secretion | Hypocalcemia - etiology | Postoperative Period | Thyroidectomy - methods | Thyroid Nodule - metabolism | Algorithms | Parathyroid Glands - secretion | Infusions, Intravenous | Thyroid Nodule - surgery | Thyroidectomy - rehabilitation
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e31376
Apart from its function as co-receptor for fibroblast growth factor-23 (FGF23), Klotho is thought to regulate insulin signaling, intracellular oxidative...
TRPV5 CHANNEL | GENE | HORMONE KLOTHO | MOUSE | BIOLOGY | FUNCTIONAL VARIANT | D-RECEPTOR | DEFICIENT MICE | INSULIN SENSITIVITY | EXPRESSION | MINERAL ION HOMEOSTASIS | Bone Resorption | Glucuronidase - physiology | Parathyroid Hormone - secretion | Animals | Vitamin D | Parathyroid Glands - secretion | Receptors, Calcitriol | Glucose - metabolism | Homeostasis | Mice | Fibroblast Growth Factors | Mice, Knockout | Phosphates | Calcifediol | Glucose | Gene expression | Density | Dextrose | Glucose metabolism | Lactose | Parathyroid hormone | Physiological aspects | Bones | Fibroblast growth factors | Alfacalcidol | Body weight | Calcium metabolism | Cardiovascular disease | Veterinary medicine | Proteins | Bone histomorphometry | Aging | Biocompatibility | Physiology | Blood pressure | Spleen | Bone turnover | Metabolism | Insulin | Ablation | Steady state | Beta cells | Diet | Ligands | Mutation | Hyperparathyroidism | Fibroblast growth factor | Oxidative stress | Calcium | Fibroblast growth factor 23 | Glands | Arthritis | Insulin-like growth factors | Dwarfism | Atrophy | Biomedical materials | Calcium (blood) | Rodents | Mathematical models | Pancreas | Parathyroid | Calcium (urinary) | Excretion | Secretion | Phosphorus | Histology | Biophysics | Hypoglycemia | Mutants | Calcium phosphates | Hypercalcemia | Glucose tolerance | Coronary vessels | Insulin resistance | Bone mineral density | In vivo methods and tests | Bone | Cell size | Emphysema
TRPV5 CHANNEL | GENE | HORMONE KLOTHO | MOUSE | BIOLOGY | FUNCTIONAL VARIANT | D-RECEPTOR | DEFICIENT MICE | INSULIN SENSITIVITY | EXPRESSION | MINERAL ION HOMEOSTASIS | Bone Resorption | Glucuronidase - physiology | Parathyroid Hormone - secretion | Animals | Vitamin D | Parathyroid Glands - secretion | Receptors, Calcitriol | Glucose - metabolism | Homeostasis | Mice | Fibroblast Growth Factors | Mice, Knockout | Phosphates | Calcifediol | Glucose | Gene expression | Density | Dextrose | Glucose metabolism | Lactose | Parathyroid hormone | Physiological aspects | Bones | Fibroblast growth factors | Alfacalcidol | Body weight | Calcium metabolism | Cardiovascular disease | Veterinary medicine | Proteins | Bone histomorphometry | Aging | Biocompatibility | Physiology | Blood pressure | Spleen | Bone turnover | Metabolism | Insulin | Ablation | Steady state | Beta cells | Diet | Ligands | Mutation | Hyperparathyroidism | Fibroblast growth factor | Oxidative stress | Calcium | Fibroblast growth factor 23 | Glands | Arthritis | Insulin-like growth factors | Dwarfism | Atrophy | Biomedical materials | Calcium (blood) | Rodents | Mathematical models | Pancreas | Parathyroid | Calcium (urinary) | Excretion | Secretion | Phosphorus | Histology | Biophysics | Hypoglycemia | Mutants | Calcium phosphates | Hypercalcemia | Glucose tolerance | Coronary vessels | Insulin resistance | Bone mineral density | In vivo methods and tests | Bone | Cell size | Emphysema
Journal Article
Journal of Molecular Endocrinology, ISSN 0952-5041, 04/2011, Volume 46, Issue 2, pp. 63 - 66
Vitamin D compounds regulate PTH at the transcriptional level, presumably via binding to the vitamin D receptor (VDR), but the exact mechanism is presently...
RESPONSE ELEMENT | BOVINE PARATHYROID CELLS | TRANSCRIPTION | ENDOCRINOLOGY & METABOLISM | HORMONE GENE | SECRETION | D-3 | BINDING PROTEIN | Organ Culture Techniques - methods | Gene Expression | Receptors, Calcitriol - deficiency | Parathyroid Glands - drug effects | Gene Expression Regulation | Mice, Transgenic | Parathyroid Hormone - antagonists & inhibitors | Ergocalciferols - metabolism | Receptors, Calcitriol - genetics | Calcifediol - pharmacology | Parathyroid Hormone - genetics | Parathyroid Glands - cytology | Animals | Parathyroid Glands - secretion | Parathyroid Hormone - metabolism | Calcitriol - pharmacology | Ergocalciferols - pharmacology | Calcifediol - metabolism | Transcription, Genetic | Calcitriol - metabolism | Mice | Parathyroid Glands - metabolism
RESPONSE ELEMENT | BOVINE PARATHYROID CELLS | TRANSCRIPTION | ENDOCRINOLOGY & METABOLISM | HORMONE GENE | SECRETION | D-3 | BINDING PROTEIN | Organ Culture Techniques - methods | Gene Expression | Receptors, Calcitriol - deficiency | Parathyroid Glands - drug effects | Gene Expression Regulation | Mice, Transgenic | Parathyroid Hormone - antagonists & inhibitors | Ergocalciferols - metabolism | Receptors, Calcitriol - genetics | Calcifediol - pharmacology | Parathyroid Hormone - genetics | Parathyroid Glands - cytology | Animals | Parathyroid Glands - secretion | Parathyroid Hormone - metabolism | Calcitriol - pharmacology | Ergocalciferols - pharmacology | Calcifediol - metabolism | Transcription, Genetic | Calcitriol - metabolism | Mice | Parathyroid Glands - metabolism
Journal Article
1924, 2nd ed.
Book
REVUE DE MEDECINE INTERNE, ISSN 0248-8663, 01/2017, Volume 38, Issue 1, pp. 61 - 64
Introduction. - Parathyromatosis is a rare cause of recurrent hyperparathyroidism. It results from hyper functioning parathyroid tissue scattered throughout...
MEDICINE, GENERAL & INTERNAL | Parathyroid glands | Parathyroidectomy | Surgery | Hyperparathyroidism | Recurrence | Parathyroid Hormone - secretion | Parathyroid Glands - secretion | Humans | Middle Aged | Hyperparathyroidism, Secondary - pathology | Male | Hyperparathyroidism, Secondary - etiology | Parathyroid Glands - pathology | Hyperplasia - complications
MEDICINE, GENERAL & INTERNAL | Parathyroid glands | Parathyroidectomy | Surgery | Hyperparathyroidism | Recurrence | Parathyroid Hormone - secretion | Parathyroid Glands - secretion | Humans | Middle Aged | Hyperparathyroidism, Secondary - pathology | Male | Hyperparathyroidism, Secondary - etiology | Parathyroid Glands - pathology | Hyperplasia - complications
Journal Article
Annals of Surgery, ISSN 0003-4932, 09/2016, Volume 266, Issue 6, pp. 1075 - 1083
OBJECTIVE:We asked if leptin and its cognate receptor were present in normal and diseased parathyroid glands, and if so, whether they had any functional...
parathyroid hormone | hyperparathyroidism | parathyroid | leptin | leptin receptor | SURGERY | CELLS | PRIMARY HYPERPARATHYROIDISM | ADIPOCYTES | CYCLIN D1 | MAMMALIAN PHYSIOLOGY | RECEPTOR | SERUM LEPTIN | BREAST-CANCER | BONE-FORMATION | LONG FORM | Immunohistochemistry | Receptors, Leptin - antagonists & inhibitors | Parathyroid Hormone - secretion | Prospective Studies | Humans | Leptin - metabolism | Cells, Cultured | RNA, Messenger - metabolism | Parathyroid Neoplasms - metabolism | Microscopy, Immunoelectron | Adenoma - metabolism | Mice, Knockout | Hyperparathyroidism - metabolism | Microscopy, Confocal | Animals | Receptors, Leptin - metabolism | Parathyroid Glands - pathology | Hyperplasia - metabolism | Microscopy, Fluorescence | Parathyroid Glands - metabolism
parathyroid hormone | hyperparathyroidism | parathyroid | leptin | leptin receptor | SURGERY | CELLS | PRIMARY HYPERPARATHYROIDISM | ADIPOCYTES | CYCLIN D1 | MAMMALIAN PHYSIOLOGY | RECEPTOR | SERUM LEPTIN | BREAST-CANCER | BONE-FORMATION | LONG FORM | Immunohistochemistry | Receptors, Leptin - antagonists & inhibitors | Parathyroid Hormone - secretion | Prospective Studies | Humans | Leptin - metabolism | Cells, Cultured | RNA, Messenger - metabolism | Parathyroid Neoplasms - metabolism | Microscopy, Immunoelectron | Adenoma - metabolism | Mice, Knockout | Hyperparathyroidism - metabolism | Microscopy, Confocal | Animals | Receptors, Leptin - metabolism | Parathyroid Glands - pathology | Hyperplasia - metabolism | Microscopy, Fluorescence | Parathyroid Glands - metabolism
Journal Article