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Journal of Bone and Mineral Research, ISSN 0884-0431, 11/2013, Volume 28, Issue 11, pp. 2266 - 2276
Journal Article
Cell Metabolism, ISSN 1550-4131, 02/2016, Volume 23, Issue 2, pp. 315 - 323
Cachexia is a wasting syndrome associated with elevated basal energy expenditure and loss of adipose and muscle tissues. It accompanies many chronic diseases... 
chronic kidney disease | skeletal muscle atrophy | cachexia | adipose tissue browning | parathyroid hormone (PTH) | cancer | PTHrP | THERMOGENESIS | BROWN-ADIPOSE-TISSUE | RESTING ENERGY-EXPENDITURE | VITAMIN-D | PROTEIN | MUSCLE STRENGTH | PTH | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | PARATHYROID-HORMONE | HEMODIALYSIS | CELL BIOLOGY | Cachexia - complications | Thermogenesis - genetics | Receptor, Parathyroid Hormone, Type 1 - metabolism | Ion Channels - genetics | Hyperparathyroidism - complications | Male | Parathyroid Hormone - pharmacology | Mitochondrial Proteins - genetics | Atrophy | Adipose Tissue - metabolism | Carcinoma, Lewis Lung - complications | Hyperparathyroidism - genetics | Nephrectomy | Mitochondrial Proteins - metabolism | Parathyroid Hormone - metabolism | Renal Insufficiency - complications | Disease Models, Animal | Mice, Inbred C57BL | Rats | Mice, Knockout | Parathyroid Hormone-Related Protein - pharmacology | Gene Expression Regulation - drug effects | Animals | Ion Channels - metabolism | Signal Transduction - drug effects | Renal Insufficiency - pathology | Cachexia - pathology | Carcinoma, Lewis Lung - pathology | Muscle, Skeletal - pathology | Thermogenesis - drug effects | Uncoupling Protein 1 | Adipose Tissue - drug effects | Kidney failure | Parathyroid hormone | Mortality | Chronic diseases | Muscles | Models | Cachexia | Health aspects | Cancer | Index Medicus | Parathyroid hormone (PTH)
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2010, Volume 285, Issue 36, pp. 28164 - 28173
PTH stimulates osteoblastic cells to form new bone and to produce osteoblast-osteoclast coupling factors such as RANKL. Whether osteoclasts or their activity... 
DENSITY | CORTICAL POROSITY | RESORPTION | OOPHORECTOMIZED MICE | BIOCHEMISTRY & MOLECULAR BIOLOGY | OSTEOPROTEGERIN LIGAND | RECEPTOR | TERIPARATIDE | STRENGTH | POSTMENOPAUSAL WOMEN | OSTEOPOROSIS | Humans | Male | Parathyroid Hormone - pharmacology | Bone and Bones - drug effects | Dose-Response Relationship, Drug | Antibodies, Monoclonal, Humanized | RANK Ligand - pharmacology | Receptor Activator of Nuclear Factor-kappa B - deficiency | Bone and Bones - metabolism | Female | Receptor Activator of Nuclear Factor-kappa B - metabolism | Bone and Bones - cytology | Biomarkers - metabolism | Gene Expression | Receptor Activator of Nuclear Factor-kappa B - antagonists & inhibitors | Bone and Bones - physiology | Ovariectomy | Antibodies, Monoclonal - pharmacology | Osteogenesis - drug effects | Bone Resorption - drug therapy | RANK Ligand - administration & dosage | Denosumab | Osteoclasts - metabolism | Gene Knock-In Techniques | Bone Density - drug effects | Animals | Antibodies, Monoclonal - administration & dosage | Alendronate - pharmacology | Receptor Activator of Nuclear Factor-kappa B - genetics | Parathyroid Hormone - administration & dosage | Mice | Osteoclasts - drug effects | Alendronate - administration & dosage | Index Medicus | Rankl | Mouse | Mouse Genetics | Osteoclast | Bone | Hormones | Metabolism | Parathyroid Hormone
Journal Article
Bone, ISSN 8756-3282, 2005, Volume 37, Issue 2, pp. 148 - 158
Intermittent parathyroid hormone (PTH) application is an established pharmacological principle to stimulate bone formation. Yet, the molecular mechanisms... 
PTH and PTH/RP | Gene expression | Osteoblasts | Histomorphometry-animal | INTERMITTENT TREATMENT | ANABOLIC ACTION | PARATHYROID-HORMONE 1-34 | OSTEOPROGENITOR CELLS | OSTEOCLAST DIFFERENTIATION FACTOR | histomorphometry-animal | OVARIECTOMIZED RATS | TRABECULAR BONE | CANCELLOUS BONE | BMP ANTAGONIST | ENDOCRINOLOGY & METABOLISM | osteoblasts | gene expression | GROWTH-FACTOR-I | Bone Morphogenetic Proteins - antagonists & inhibitors | Tetradecanoylphorbol Acetate - pharmacology | Rats, Wistar | Parathyroid Hormone - pharmacology | Peptide Fragments - pharmacology | RNA, Messenger - metabolism | Skull - physiology | Peptide Fragments - physiology | Bone Morphogenetic Proteins - metabolism | Female | Protein Synthesis Inhibitors - pharmacology | Bone Morphogenetic Proteins - genetics | Skull - drug effects | Parathyroid Hormone - physiology | Cell Line | Bone Density | Ovariectomy | Colforsin - pharmacology | Tissue Culture Techniques | Rats | Femur - drug effects | Parathyroid Hormone - antagonists & inhibitors | Glycoproteins | RNA Stability | Cycloheximide - pharmacology | Femur - physiology | Cyclic AMP - antagonists & inhibitors | Animals | Peptide Fragments - antagonists & inhibitors | Genetic Markers - genetics | Ionomycin - pharmacology | Mice | Osteogenesis | Messenger RNA | Parathyroid hormone | Analysis | Genetic research | Protein biosynthesis | Bones | Peptide hormones | Density | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 03/2017, Volume 25, Issue 3, pp. 661 - 672
Journal Article
Kidney International, ISSN 0085-2538, 12/2014, Volume 86, Issue 6, pp. 1106 - 1115
Journal Article
Journal Article
Cellular Signalling, ISSN 0898-6568, 03/2016, Volume 28, Issue 3, pp. 204 - 213
Parathyroid hormone (PTH) is secreted from the parathyroid glands in response to low plasma calcium levels. Besides its classical actions on bone and kidney,... 
PDE | Adipocyte | cAMP | PTH | Lipolysis | PKA | CAMP | PRIMARY HYPERPARATHYROIDISM | ACTIVATION | PROTEIN-KINASE | GLUCOSE-INTOLERANCE | IDENTIFICATION | INSULIN SENSITIVITY | CELL BIOLOGY | HUMAN ADIPOSE-TISSUE | DISEASE | FAT | RESISTANCE | Lipolysis - drug effects | Receptor, Parathyroid Hormone, Type 1 - metabolism | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Sterol Esterase - metabolism | Adipocytes - cytology | Male | Parathyroid Hormone - pharmacology | Phosphoproteins - metabolism | Receptor, Parathyroid Hormone, Type 2 - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Cyclic Nucleotide Phosphodiesterases, Type 4 - metabolism | Cyclic Nucleotide Phosphodiesterases, Type 4 - chemistry | Cyclic AMP-Dependent Protein Kinases - metabolism | Adenylyl Cyclase Inhibitors - pharmacology | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Cells, Cultured | Receptor, Parathyroid Hormone, Type 2 - genetics | Imines - pharmacology | Phosphodiesterase 4 Inhibitors - pharmacology | Animals | Carrier Proteins - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Adipocytes - metabolism | Receptor, Parathyroid Hormone, Type 1 - genetics | Mice | Protein Kinase Inhibitors - pharmacology | Mitogen-Activated Protein Kinase 1 - metabolism | Perilipin-1 | Vitamin D | Parathyroid hormone | Insulin resistance | Development and progression | Calcifediol | Alfacalcidol | Calcium-binding proteins | Lipase | Type 2 diabetes | Protein kinases | Index Medicus | Proteins | Phosphorylation | Inhibitors | Pathways | Hormones | Kinases | Vitamins | Cell and Molecular Biology | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Cell- och molekylärbiologi | Medicinska och farmaceutiska grundvetenskaper
Journal Article