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Brain Research, ISSN 0006-8993, 2008, Volume 1227, pp. 128 - 141
Abstract Rats can anticipate a daily mealtime when they are maintained on restricted feeding schedules (RFS). Neural substrates of the food-entrainable... 
Neurology | FEO, food-entrainable oscillator | AHA, anterior hypothalamic area | LEO, light-entrainable oscillator | PVTp, paraventricular thalamic nucleus, posterior part | Restricted feeding schedule | PVHp, paraventricular hypothalamic nucleus, parvocellular part | SuM, supramammillary nucleus | PVH, paraventricular hypothalamic nucleus | HPA, hypothalamic–pituitary adrenal axis | CeA, central nucleus of amygdala | DLG, dorsal lateral geniculate nucleus | PVT, paraventricular thalamic nucleus | Food-entrainable oscillator | AD, anterodorsal thalamic nucleus | SG, suprageniculate thalamic nucleus | BLA, basolateral amygdala | LHA, lateral hypothalamic area | Food anticipation | DMHv, dorsomedial hypothalamic nucleus, ventral part | DTg, dorsal tegmental nucleus | SON, supraoptic nucleus | AH/SHi, anterior hippocampal continuation and septohippocampal nucleus | PVTa, paraventricular thalamic nucleus, anterior part | DMH, dorsomedial hypothalamic nucleus | PVHam, paraventricular hypothalamic nucleus, anterior magnocellular part | c- fos | CRF, corticotropin releasing factor | FAA, food-anticipatory activity | Neuronal activation | ACh, acetylcholine | VMH, ventromedial hypothalamic nucleus | LM, lateral mammillary nucleus | ACTH, adrenocorticotropic hormone | NTS, nucleus of the solitary tract | PB, parabrachial nuclei | CRF | SCN, suprachiasmatic nucleus | RFS, restricted feeding schedules | PVHmm, paraventricular hypothalamic nucleus, medial magnocellular part | SuG, superficial gray layer of the superior colliculus | DMHc, dorsomedial hypothalamic nucleus, compact part | HPA, hypothalamic-pituitary adrenal axis | c-fos | ENHANCED ACETYLCHOLINE-RELEASE | restricted feeding schedule | DORSOMEDIAL HYPOTHALAMIC NUCLEUS | CIRCADIAN-RHYTHMS | food-entrainable oscillator | HORMONE MESSENGER-RNA | NEUROSCIENCES | NEUROSECRETORY NEURONS | PLASMA-CORTICOSTERONE | ZUCKER RATS | PARAVENTRICULAR THALAMIC NUCLEUS | neuronal activation | AGED RATS | food anticipation | C-FOS EXPRESSION | Paraventricular Hypothalamic Nucleus - physiology | Insulin - physiology | Rats, Wistar | Dorsomedial Hypothalamic Nucleus - physiology | Male | Insulin - blood | RNA, Messenger - metabolism | Brain - physiology | Eating - physiology | Brain - metabolism | Paraventricular Hypothalamic Nucleus - cytology | In Situ Hybridization | Neurons - physiology | Corticotropin-Releasing Hormone - genetics | Neurons - metabolism | Corticosterone - blood | Gene Expression | Enzyme-Linked Immunosorbent Assay | Dorsomedial Hypothalamic Nucleus - cytology | RNA, Messenger - genetics | Amygdala - metabolism | Proto-Oncogene Proteins c-fos - metabolism | Rats | Amygdala - cytology | Dorsomedial Hypothalamic Nucleus - metabolism | Hippocampus - cytology | Corticotropin-Releasing Hormone - metabolism | Feeding Behavior - physiology | Food Deprivation - physiology | Hippocampus - metabolism | Animals | Glucose - physiology | Paraventricular Hypothalamic Nucleus - metabolism | Proto-Oncogene Proteins c-fos - genetics | Corticosterone - metabolism | Body Weight - physiology | Glucose - analysis | Hippocampus - physiology | Amygdala - physiology | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2010, Volume 16, Issue 9, pp. 1001 - 1008
Thyroid hormones have widespread cellular effects; however it is unclear whether their effects on the central nervous system (CNS) contribute to global energy... 
MEDICINE, RESEARCH & EXPERIMENTAL | NERVOUS-SYSTEM | ACTIVATED PROTEIN-KINASE | FOOD-INTAKE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADULT HUMANS | BROWN ADIPOSE-TISSUE | ACETYL-COA CARBOXYLASE | INSULIN SENSITIVITY | VENTROMEDIAL HYPOTHALAMUS | CELL BIOLOGY | SKELETAL-MUSCLE | MALONYL-COA | AMP-Activated Protein Kinases - metabolism | Thyroxine - blood | Hyperthyroidism - metabolism | Thermogenesis - physiology | Thyrotropin-Releasing Hormone - genetics | Cerulenin - pharmacology | Energy Metabolism - physiology | Fatty Acids - metabolism | Hyperphagia - etiology | Arcuate Nucleus of Hypothalamus - metabolism | RNA, Messenger - genetics | Adipose Tissue, Brown - physiology | Rats | Pro-Opiomelanocortin - metabolism | Triiodothyronine - blood | Animals | Agouti-Related Protein - genetics | Hypothalamus - enzymology | Paraventricular Hypothalamic Nucleus - metabolism | Hyperthyroidism - complications | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Fatty Acid Synthesis Inhibitors - pharmacology | Thyroid Gland - metabolism | Thyroxine - pharmacology | Physiological aspects | Thyroid hormones | Adenylic acid | Health aspects | Protein kinases | Thyroid diseases | Lipids | Thyroid gland | Hormones | Fatty acids | Rodents | Index Medicus | Medical Biotechnology | Medical and Health Sciences | Medicin och hälsovetenskap | Medical Biotechnology (with a focus on Cell Biology (including Stem Cell Biology), Molecular Biology, Microbiology, Biochemistry or Biopharmacy) | Medicinsk bioteknologi (med inriktning mot cellbiologi (inklusive stamcellsbiologi), molekylärbiologi, mikrobiologi, biokemi eller biofarmaci) | Medicinsk bioteknologi
Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 06/2017, Volume 595, Issue 11, pp. 3425 - 3447
Relaxin‐3 is a stress‐responsive neuropeptide that acts at its cognate receptor, RXFP3, to alter behaviours including feeding. In this study, we have... 
oxytocin | RXFP3 receptors | vasopressin | EFFERENT PROJECTIONS | PROTEIN-KINASE-C | PHYSIOLOGY | CORTICOTROPIN-RELEASING-FACTOR | MAGNOCELLULAR NEUROSECRETORY-CELLS | FOOD-INTAKE | RECEPTOR RXFP3 | RELAXIN FAMILY PEPTIDES | ARTERIAL SMOOTH-MUSCLE | INDUCED OBESE RATS | NEUROSCIENCES | BRAIN EXPRESSION | Potassium - metabolism | Paraventricular Hypothalamic Nucleus - physiology | Rats, Wistar | Receptors, G-Protein-Coupled - metabolism | Signal Transduction | Rats | Male | Vasopressins - metabolism | Oxytocin - metabolism | Receptors, Peptide - genetics | GABA Antagonists - pharmacology | Relaxin - pharmacology | Action Potentials | Paraventricular Hypothalamic Nucleus - drug effects | Animals | Neurons - physiology | Paraventricular Hypothalamic Nucleus - metabolism | Neurons - metabolism | Receptors, G-Protein-Coupled - genetics | Tetrodotoxin - pharmacology | Neurons - drug effects | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Receptors, Peptide - metabolism | Brain | Neuropeptides | Marine toxins | RNA | Neurons | Tetrodotoxin | Glutamic acid | G protein-coupled receptors | Brain slice preparation | Activation | mRNA | Hypothalamus | Paraventricular nucleus | Fibers | Proteins | γ-Aminobutyric acid | Rodents | Terminals | Inhibition | Cadmium | Proximity | Oxytocin | Energy expenditure | Abundance | Gene expression | Relaxin | Vasopressin | Feeding | Axons | Signaling | Molecular modelling | Food intake | Nuclei (cytology) | Immunoreactivity | Potassium | In vitro methods and tests | Histochemistry | Index Medicus | Research Paper | Neuroscience ‐ Cellular | Molecular
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2014, Volume 124, Issue 10, pp. 4473 - 4488
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 05/2015, Volume 284, Issue 3, pp. 315 - 322
We hypothesized that chronic inhibition of NF-κB activity in the hypothalamic paraventricular nucleus (PVN) delays the progression of hypertension and... 
Hypertension | Cardiac hypertrophy | NF-κB | Neurohormonal excitation | Hypothalamic paraventricular nucleus | HEART-FAILURE | RATS | RECEPTOR | NECROSIS-FACTOR-ALPHA | PROINFLAMMATORY CYTOKINES | BLOOD-PRESSURE | II-INDUCED HYPERTENSION | NAD(P)H OXIDASE | NF-kappa B | RENIN-ANGIOTENSIN SYSTEM | PHARMACOLOGY & PHARMACY | TOXICOLOGY | TNF-ALPHA | Transcription Factor RelA - antagonists & inhibitors | Sympathetic Nervous System - drug effects | Cardiomegaly - etiology | Rats, Inbred WKY | Hypertension - drug therapy | Sympathetic Nervous System - physiopathology | Sympathetic Nervous System - metabolism | Pyrrolidines - pharmacology | Thiocarbamates - pharmacology | Time Factors | Arterial Pressure - drug effects | Inflammation Mediators - metabolism | Paraventricular Hypothalamic Nucleus - immunology | Disease Models, Animal | Biomarkers - metabolism | Rats, Inbred SHR | Cardiomegaly - immunology | Cytokines - metabolism | Cardiomegaly - physiopathology | Hypertension - immunology | Hypertension - physiopathology | Hypertension - metabolism | Paraventricular Hypothalamic Nucleus - drug effects | Animals | Cardiomegaly - prevention & control | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Paraventricular Hypothalamic Nucleus - metabolism | Hypertension - complications | Oxidative Stress - drug effects | Cardiomegaly - metabolism | Index Medicus | OXIDATION | EXCITATION | TIBIA | MYOSIN | MONOCYTES | INFUSION | 60 APPLIED LIFE SCIENCES | STRESSES | HEART | NAD | HYPERTROPHY | INHIBITION | PEPTIDES | LYMPHOKINES | MESSENGER-RNA | OXIDASES | INFLAMMATION | HYPERTENSION | WEIGHT
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