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Lancet Neurology, The, ISSN 1474-4422, 2012, Volume 11, Issue 5, pp. 429 - 442
Summary Deep brain stimulation (DBS) is an established procedure for the symptomatic treatment of Parkinson's disease... 
Neurology | SUBTHALAMIC NUCLEUS STIMULATION | IMPULSE CONTROL DISORDERS | LA-TOURETTE-SYNDROME | INTERMEDIATE THALAMIC NUCLEUS | HUMAN PEDUNCULOPONTINE NUCLEUS | INTERNAL PALLIDAL STIMULATION | HIGH-FREQUENCY STIMULATION | QUALITY-OF-LIFE | RANDOMIZED CONTROLLED-TRIAL | CAUDAL ZONA INCERTA | CLINICAL NEUROLOGY | Parkinson Disease - therapy | Motor Activity - physiology | Mental Disorders - therapy | Follow-Up Studies | Speech Disorders - therapy | Humans | Deep Brain Stimulation - adverse effects | Intralaminar Thalamic Nuclei - physiopathology | Globus Pallidus - physiopathology | Disability Evaluation | Motor Skills - physiology | Pedunculopontine Tegmental Nucleus - physiopathology | Subthalamic Nucleus - physiopathology | Basal Ganglia - physiopathology | Dyskinesias - physiopathology | Parkinson Disease - physiopathology | Disease Progression | Speech Disorders - physiopathology | Efferent Pathways - physiopathology | Mental Disorders - physiopathology | Ventral Thalamic Nuclei - physiopathology | Magnetic Resonance Imaging | Dyskinesias - therapy | Neurologic Examination | Brain Mapping | Quality of Life | Care and treatment | Parkinson's disease | Drugs | Brain | tremor | Neurodegenerative diseases | Deep brain stimulation | Globus pallidus | Data processing | Pedunculopontine tegmental nucleus | Electrical stimuli | subthalamic nucleus | Thalamus | Movement disorders
Journal Article
Trends in Neurosciences, ISSN 0166-2236, 2009, Volume 32, Issue 11, pp. 591 - 601
.... Treatment with various HDAC inhibitors can correct these deficiencies and has emerged as a promising new strategy for therapeutic intervention in neurodegenerative disease... 
Neurology | SPINAL MUSCULAR-ATROPHY | SUBEROYLANILIDE HYDROXAMIC ACID | PROTECTS DOPAMINERGIC-NEURONS | INCREASES SMN EXPRESSION | VALPROIC ACID | HISTONE DEACETYLASE INHIBITORS | BDNF GENE-TRANSCRIPTION | TRANSGENIC MOUSE MODEL | HUNTINGTONS-DISEASE | NEUROSCIENCES | HEAT-SHOCK-PROTEIN | Neuroprotective Agents - therapeutic use | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Muscular Atrophy, Spinal - enzymology | Parkinson Disease - drug therapy | Neurodegenerative Diseases - drug therapy | Stroke - physiopathology | Amyotrophic Lateral Sclerosis - drug therapy | Amyotrophic Lateral Sclerosis - enzymology | Acetylation | Huntington Disease - drug therapy | Huntington Disease - enzymology | Muscular Atrophy, Spinal - physiopathology | Huntington Disease - physiopathology | Disease Models, Animal | Alzheimer Disease - physiopathology | Neurodegenerative Diseases - pathology | Alzheimer Disease - drug therapy | Rats | Histone Deacetylases - metabolism | Stroke - drug therapy | Alzheimer Disease - enzymology | Parkinson Disease - physiopathology | Stroke - enzymology | Animals | Neurodegenerative Diseases - physiopathology | Neuroprotective Agents - classification | Parkinson Disease - enzymology | Muscular Atrophy, Spinal - drug therapy | Histone Deacetylase Inhibitors - therapeutic use | Histones - metabolism | Neurodegenerative Diseases - enzymology | Histones | Nervous system diseases | Analysis
Journal Article
Lancet Neurology, The, ISSN 1474-4422, 2009, Volume 8, Issue 5, pp. 464 - 474
Journal Article
Journal of the neurological sciences, ISSN 0022-510X, 2012, Volume 323, Issue 1-2, pp. 16 - 24
Abstract It is known that the olfactory dysfunction is involved in various neurological diseases, such as Parkinson's disease, Alzheimer's disease, multiple sclerosis, Huntington's disease and motor neuron disease... 
Neurology | Multiple sclerosis | Parkinson's disease | Olfactory dysfunction | Motor neuron disease | Functional magnetic resonance imaging | Huntington's disease | Olfactory event-related potentials | Alzheimer's disease | ALZHEIMERS-DISEASE | ENSHEATHING CELLS | PIRIFORM CORTEX | MOTOR-NEURON DISEASE | TIME-COURSE | REGIONAL BRAIN RESPONSES | NEUROSCIENCES | CLINICAL NEUROLOGY | MULTIPLE-SCLEROSIS | ODOR IDENTIFICATION | OF-PENNSYLVANIA SMELL | PARKINSONS-DISEASE | Parkinson Disease - complications | Alzheimer Disease - physiopathology | Alzheimer Disease - complications | Motor Neuron Disease - complications | Sensory Thresholds | Humans | Evoked Potentials | Olfaction Disorders - diagnosis | Parkinson Disease - physiopathology | Neurodegenerative Diseases - complications | Odorants | Huntington Disease - complications | Multiple Sclerosis - complications | Neuropsychological Tests | Olfactory Pathways - physiopathology | Magnetic Resonance Imaging | Multiple Sclerosis - physiopathology | Neurodegenerative Diseases - physiopathology | Aging - physiology | Discrimination (Psychology) | Olfaction Disorders - physiopathology | Motor Neuron Disease - physiopathology | Olfaction Disorders - etiology | Huntington Disease - physiopathology | Nervous system diseases | Diagnostic imaging | Huntington's chorea
Journal Article
Trends in neurosciences (Regular ed.), ISSN 0166-2236, 2010, Volume 33, Issue 10, pp. 474 - 484
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2007, Volume 30, Issue 5, pp. 759 - 768
Abstract In Alzheimer's disease (AD), Parkinson's disease (PD), dementia with Lewy bodies (DLB... 
Neurology | Internal Medicine | Lewy bodies dementia | Brain aging | Parkinson's disease | Neurodegenerative diseases | Toll-like receptors | Innate immunity | Amyotrophic lateral sclerosis | CD14 | Alzheimer's disease | MICROGLIAL ACTIVATION | HSP70 | LPS RECEPTOR | ALPHA-SYNUCLEIN | TRANSGENIC MODEL | PATHOLOGY | NEUROSCIENCES | GERIATRICS & GERONTOLOGY | CELL UPTAKE | MOUSE MODEL | PARKINSONS-DISEASE | Amyotrophic Lateral Sclerosis - physiopathology | Lewy Body Disease - immunology | Species Specificity | Extracellular Fluid - immunology | Humans | Male | Neurodegenerative Diseases - immunology | Lipopolysaccharide Receptors - analysis | Lipopolysaccharide Receptors - metabolism | Brain - metabolism | Lewy Body Disease - genetics | Encephalitis - physiopathology | Immunity, Innate - physiology | Encephalitis - metabolism | Female | Toll-Like Receptors - metabolism | Alzheimer Disease - immunology | Disease Models, Animal | Biomarkers - metabolism | Amyotrophic Lateral Sclerosis - immunology | Alzheimer Disease - physiopathology | Parkinson Disease - immunology | Amyotrophic Lateral Sclerosis - genetics | Brain - physiopathology | Mice, Inbred C57BL | Biomarkers - analysis | Encephalitis - immunology | Mice, Transgenic | Neurodegenerative Diseases - genetics | Up-Regulation - genetics | Parkinson Disease - genetics | Parkinson Disease - physiopathology | Toll-Like Receptors - analysis | Animals | Neurodegenerative Diseases - physiopathology | Up-Regulation - immunology | Mice | Alzheimer Disease - genetics | Brain - immunology | Lewy Body Disease - physiopathology | Proteins | Neurons | Analysis | Nervous system | Degeneration | Epidemiology | Brain diseases
Journal Article
Neurobiology of disease, ISSN 0969-9961, 2012, Volume 46, Issue 3, pp. 559 - 564
Journal Article
Molecular neurobiology, ISSN 1559-1182, 2008, Volume 38, Issue 1, pp. 78 - 100
.... However, alterations in calcium homeostasis lead to persistent, pathologic activation of calpain in a number of neurodegenerative diseases... 
Multiple sclerosis | Calcium | Amylotrophic lateral sclerosis | Neurobiology | Calpain | Prion-related encephalopathy | Huntington’s | Alzheimer’s | Cell Biology | Parkinson’s | Life Sciences | Neurology | Cerebral ischemia | Excitotoxicity | Neurodegenerative disease | Parkinson's | Alzheimer's | Huntington's | APOPTOSIS-INDUCING FACTOR | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | CONSTITUTIVELY ACTIVE CALCINEURIN | neurodegenerative disease | calcium | SELECTIVE STRIATAL DEGENERATION | multiple sclerosis | FOCAL CEREBRAL-ISCHEMIA | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | prion-related encephalopathy | AMYOTROPHIC-LATERAL-SCLEROSIS | AMYLOID PRECURSOR PROTEIN | D-ASPARTATE RECEPTOR | NEUROSCIENCES | calpain | cerebral ischemia | amylotrophic lateral sclerosis | excitotoxicity | CYCLIN-DEPENDENT KINASE-5 | Prion Diseases - physiopathology | Alzheimer Disease - physiopathology | Parkinson Disease - pathology | Trauma, Nervous System - pathology | Calpain - metabolism | Neurodegenerative Diseases - pathology | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Huntington Disease - pathology | Brain Ischemia - physiopathology | Prion Diseases - pathology | Calpain - genetics | Parkinson Disease - physiopathology | Calpain - chemistry | Alzheimer Disease - pathology | Multiple Sclerosis - physiopathology | Amyotrophic Lateral Sclerosis - pathology | Trauma, Nervous System - physiopathology | Animals | Neurodegenerative Diseases - physiopathology | Brain Ischemia - pathology | Multiple Sclerosis - pathology | Signal Transduction - physiology | Huntington Disease - physiopathology | Brain damage | Proteases | Neurodegeneration |