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2012, ISBN 9780199642243, 1 v.
Parasites that manipulate the behaviour of their hosts represent striking examples of adaptation by natural selection. This text provides an up-to-date and... 
Host-parasite relationships | Biology, life sciences
Book
2009, ISBN 9789812835604, viii, 85
Book
2009, ISBN 1555814050, xv, 391
This book uniquely addresses the impact, both positive and negative, of food-borne microbes on the host and their role in shaping the ecosystems of the gut and... 
Host-Pathogen Interactions | Microbiology | Food Contamination | Gastrointestinal system | Ecosystem | Microbial ecology | Food Microbiology | Food
Book
2002, illustrated edition, Oxford series in ecology and evolution., ISBN 0195146530, 329
This volume presents ecological consequences and evolutionary mechanisms that may be associated with behavioral alterations in parasitized hosts. Alterations... 
Host-parasite relationships | Animal behavior | Biology, life sciences | Parasitism
eBook
2009, ISBN 9789812833792, xxx, 691
Book
09/2016, Drug discovery in infectious diseases, ISBN 3527337458, 237
In light of the rapidity increasing incidence rate of bacterial and fungal infections with multi-resistant pathogens, the metabolic changes associated with... 
Host-parasite relationships
eBook
Sexually Transmitted Infections, ISSN 1368-4973, 07/2019, Volume 95, Issue Suppl 1, p. A338
BackgroundSyndromic management remains the standard nongonococcal urethritis (NGU) treatment approach. Whether pathogen-specific signs/symptoms inform... 
Journal Article
Cellular Microbiology, ISSN 1462-5814, 10/2015, Volume 17, Issue 10, pp. 1494 - 1509
  Summary The multifunctional-autoprocessing repeats-in-toxin (MARTXVv) toxin that harbours a varied repertoire of effector domains is the primary virulence... 
Pathogens
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 01/2017, Volume 195
O2 sats in 80s prompted another CT scan 3 days later which showed findings of worsening diffuse ground glass opacities suggestive of ARDS. [...]it is uncertain... 
Pathogens
Journal Article
AIP Conference Proceedings, ISSN 0094-243X, 04/2019, Volume 2086, Issue 1
In this paper the triple sequence space are the generalization of the classical Maddox’s paranormed sequence space have been introduced and investigated some... 
Pathogens
Journal Article
Sexually Transmitted Infections, ISSN 1368-4973, 07/2017, Volume 93, Issue Suppl 2, p. A64
IntroductionSexually Transmitted Infections (STIs) are increasing worldwide. Innovative approaches are required to eliminate barriers to STIs testing such as... 
Pathogens
Journal Article
2013, ISBN 9780470958605, viii, 360 p., [24] p. of plates
Phloem: Molecular Cell Biology, Systemic Communication, Biotic Interactions is a timely collection of research on the cellular and molecular biology of this... 
Phloem | Plant-pathogen relationships | Sieve elements | Plant cell interaction | Development | Plants | Growth (Plants) | Plant molecular biology | Life Sciences | Science | General | Biology
Book
2009, ISBN 9781555814014, xv, 559 p., [8] p. of plates
Book
European Journal of Immunology, ISSN 0014-2980, 12/2015, Volume 45, Issue 12, p. NA
Our cover features an H&E staining of liver sections from Cryptococcus neoformans‐infected RAG‐1−/− mice after immune reconstitution with CD4+ T cells.... 
Pathogens
Journal Article
Annual Review of Physiology, ISSN 0066-4278, 03/2009, Volume 72, Issue 1, pp. 413 - 435
Most studies of innate immunity have focused on leukocytes such as neutrophils, macrophages, and natural killer cells. However, epithelial cells play key roles... 
Antimicrobial | Innate immunity | Pneumonia | Microbial killing | antimicrobial | PHYSIOLOGY | HUMAN AIRWAY EPITHELIA | TOLL-LIKE-RECEPTOR | CATHELICIDIN ANTIMICROBIAL PEPTIDE | ADAPTIVE IMMUNITY | microbial killing | GROWTH-FACTOR-BETA | innate immunity | pneumonia | PSEUDOMONAS-AERUGINOSA | IMMUNE-RESPONSES | HOST-DEFENSE | COMPLEMENT ANAPHYLATOXIN C3A | PATTERN-RECOGNITION RECEPTORS | Respiratory Mucosa - anatomy & histology | Pneumonia - therapy | Infection - therapy | Lung Diseases - diagnosis | Infection - immunology | Lung Diseases - microbiology | Humans | Infection - microbiology | Lung Diseases - therapy | Pneumonia - microbiology | Blood Bactericidal Activity | Lung Diseases - immunology | Respiratory Mucosa - microbiology | Animals | Immunity, Innate - physiology | Infection - diagnosis | Pneumonia - diagnosis | Respiratory Mucosa - immunology | Pneumonia - immunology | Cytokines | Lung diseases | Physiological aspects | Causes of | Anti-infective agents | Research | Leukocytes | Health aspects | Non-typeable Haemophilus influenzae (NTHi): an unencapsulated (hence non-typeable) strain of a Gram-negative bacterial pathogen that is frequently cultured from the lungs of patients with chronic respiratory disease | Interferon (IFN): a subset of cytokines that inhibit viral replication within host cells and activate leukocytes. There are three classes – Type I (α, β, ω, ε, and κ), Type II (γ), and Type III (λ1-3, also known as IL-28A | Interleukin (IL): a widely expressed and highly variable group of cytokine signaling molecules involved in both innate and adaptive immune responses | Innate immunity: host antimicrobial defenses involving detection of conserved molecular motifs by germline-encoded pattern recognition receptors, and characterized by rapid but transient responses of both leukocytes and parenchymal cells | Leucine rich repeats (LRR): common molecular sequences of many pattern recognition receptors that generally occur within pathogen recognition domains | B and IL-29), each with distinct receptors | Transcytosis: mechanism of transport across polarized epithelial cells involving endocytois of extracellular macromolecules or particles on one surface, transcellular vesicle trafficking, and exocytosis on the other surface | Pattern recognition receptors (PRR): membrane-associated, cytosolic or secreted host products that recognize conserved molecular patterns on pathogens, initiating innate immune responses. These include Toll-like receptors (TLR), NOD-like receptors (NLR), RIG-I-like receptors (RLR), class A scavenger receptors (SR-A), and macrophage receptor with collagenous structure (MARCO) | Receptor for advanced glycation end-products (RAGE): a multifunctional member of the immunoglobulin superfamily that recognizes several host immunomodulatory proteins, including HMGB1 and S100, as well as host proteins without immunomodulatory activity that become glycated during aging or inflammation | Resistance: the strategy of host survival of infection that is associated with a reduction in pathogen burden; this is contrasted to tolerance, the strategy of generating a host phenotype indifferent to the pathogen burden | Lipopolysaccharide (LPS): cell wall component of Gram-negative bacteria, the lipid A portion of which is recognized by TLR4 in association with MD2 and CD14 | Complement: a system of more than thirty proteins activated by three pathways that permeabilize pathogens with the membrane attack complex, opsonize microbes and release fragments with signaling properties such as the anaphylotoxins C3a and C5a | Hemolymph: fluid in the body cavity of insects, homologous to vertebrate blood, with most proteins produced by the fat body, homologous to the vertebrate liver, with functions in both metabolism and immunity | Secretory leukocyte proteinase inhibitor (SLPI): epithelium-derived protease inhibitor with intrinsic antibacterial activity | Transmigration: paracellular migration of leukocytes or pathogens through epithelial or endothelial barriers and associated basement membranes | Anoikis: apoptosis of epithelial cells induced by detachment from the extracellular matrix | Toll | Pathogen-associated molecular pattern (PAMP): stereotypic molecular motifs conserved across microbial species that are recognized by pattern recognition receptors, triggering innate immune responses | Damage-associated molecular pattern (DAMP): molecular motifs expressed on or released by infected or injured host cells, also known as danger signals or alarmins | Adaptive immunity: host antimicrobial defenses involving pathogen detection of specific antigens by somatically recombined receptors, and characterized by clonal expansion of pathogen-specific lymphocytes and immunologic memory | Interleukin-1 receptor adaptors (TIR adaptors): host peptides that are selectively recruited to Toll-like receptors and IL-1 receptor upon ligand binding and are required for signal propagation
Journal Article
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