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Journal Article
Nature Medicine, ISSN 1078-8956, 07/2011, Volume 17, Issue 7, pp. 822 - 829
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2013, Volume 110, Issue 10, pp. 3743 - 3748
Despite the significance of Alzheimer's disease, the link between metal-associated amyloid-β (metal-Aβ) and disease etiology remains unclear. To elucidate this... 
Aggregation | Metal aggregates | Reactivity | Cell aggregates | Dimers | Amyloids | Alzheimers disease | Solar fibrils | Monomers | Metal ions | Amyloidogenesis | Amyloid-β peptide | Natural products | FIBRIL FORMATION | ALZHEIMERS-DISEASE | EGCG | MULTIDISCIPLINARY SCIENCES | amyloid-beta peptide | metal ions | MASS-SPECTROMETRY | PEPTIDE | natural products | MEROZOITE SURFACE PROTEIN-2 | amyloidogenesis | EPIGALLOCATECHIN GALLATE | ZINC-BINDING | AGGREGATION | POLYPEPTIDE | Alzheimer Disease - etiology | Camellia sinensis - chemistry | Plant Extracts - chemistry | Peptide Fragments - toxicity | Plant Extracts - pharmacology | Humans | Protein Conformation - drug effects | Amyloid beta-Peptides - drug effects | Metals - toxicity | Zinc - chemistry | Tandem Mass Spectrometry | Copper - chemistry | Neuroprotective Agents - pharmacology | Metals - chemistry | Alzheimer Disease - prevention & control | Nuclear Magnetic Resonance, Biomolecular | Catechin - pharmacology | Copper - toxicity | Neuroprotective Agents - chemistry | Amyloid beta-Peptides - toxicity | Copper - pharmacology | Models, Molecular | Peptide Fragments - chemistry | Alzheimer Disease - metabolism | Peptide Fragments - drug effects | Protein Binding | Zinc - toxicity | Catechin - analogs & derivatives | Amyloid beta-Peptides - chemistry | Zinc - pharmacology | Catechin - chemistry | Protein Multimerization - drug effects | Metals - pharmacology | Biological Sciences | Physical Sciences | amyloid-β peptide
Journal Article
Neuroscience, ISSN 0306-4522, 11/2006, Volume 142, Issue 4, pp. 941 - 952
A recent epidemiological study suggested that higher caffeine intake over decades reduces the risk of Alzheimer's disease (AD). The present study sought to... 
adenosine | learning | memory | transgenic mice | BACE | PS1 | COFFEE | PERFORMANCE | RISK | NEUROSCIENCES | OLIGOMERS | DISEASE | A-BETA | CONSUMPTION | TG2576 MOUSE MODEL | Memory Disorders - physiopathology | Purinergic P1 Receptor Antagonists | Presenilin-1 - drug effects | Amyloid beta-Peptides - drug effects | Memory, Short-Term - physiology | Brain - metabolism | Dose-Response Relationship, Drug | Cognition Disorders - prevention & control | Neuroprotective Agents - pharmacology | Alzheimer Disease - prevention & control | Amyloid beta-Peptides - metabolism | Caffeine - pharmacology | Disease Models, Animal | Alzheimer Disease - physiopathology | Cognition Disorders - physiopathology | Peptide Fragments - metabolism | Amyloid beta-Peptides - biosynthesis | Brain - physiopathology | Mice, Inbred C57BL | Alzheimer Disease - drug therapy | Memory, Short-Term - drug effects | Mice, Transgenic | Treatment Outcome | Cognition Disorders - drug therapy | Brain - drug effects | Neuropsychological Tests | Amyloid Precursor Protein Secretases - metabolism | Memory Disorders - prevention & control | Animals | Memory Disorders - drug therapy | Amyloid Precursor Protein Secretases - drug effects | Presenilin-1 - metabolism | Peptide Fragments - drug effects | Adenosine - metabolism | Cell Line, Tumor | Mice | Receptors, Purinergic P1 - metabolism
Journal Article
JAMA, ISSN 0098-7484, 01/2009, Volume 301, Issue 4, pp. 383 - 392
Journal Article
Journal of Medicinal Chemistry, ISSN 0022-2623, 01/2005, Volume 48, Issue 1, pp. 141 - 151
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2009, Volume 110, Issue 6, pp. 1784 - 1795
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2017, Volume 313, Issue 4, pp. H690 - H699
Journal Article
Life Sciences, ISSN 0024-3205, 11/2018, Volume 213, pp. 74 - 81
Increasing evidence displays that deposition of aggregated β-amyloid (Aβ) leads to neuronal cell apoptosis, thus aggravates the pathological progression of... 
ERK1/2 | D3T | PC12 cells | Apoptosis | MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | ALZHEIMERS-DISEASE | IMPAIRMENT | STRATEGIES | PATHOGENESIS | A beta | HONOKIOL | HYPOTHESIS | PHARMACOLOGY & PHARMACY | MICE | INDUCED NEUROTOXICITY | CYTOTOXICITY | Cell Survival - drug effects | Peptide Fragments - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Calcium - metabolism | Amyloid beta-Peptides - drug effects | Rats | Thiophenes - pharmacology | Thiones - metabolism | Thiophenes - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Antioxidants - pharmacology | L-Lactate Dehydrogenase - drug effects | PC12 Cells - drug effects | Animals | MAP Kinase Signaling System - drug effects | Neuroprotective Agents - pharmacology | Signal Transduction - drug effects | Peptide Fragments - drug effects | Oxidative Stress - drug effects | Thiones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Proteins | Development and progression | Alzheimer's disease | Protein kinases | Neuroprotection | Flow cytometry | Bax protein | Calcium | AKT protein | Activation | Kinases | Assaying | Caspase-3 | Pretreatment | Calcium (intracellular) | Neurodegenerative diseases | Neurons | Extracellular signal-regulated kinase | Caspase | Lactate dehydrogenase | Cells | L-Lactate dehydrogenase | 1-Phosphatidylinositol 3-kinase | Pathology | Inhibitors | Protein kinase | Pheochromocytoma cells | β-Amyloid | Lactic acid | Intracellular | Alzheimers disease | Fluo-3 | Viability | Calcium ions
Journal Article