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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2016, Volume 311, Issue 4, pp. H871 - H880
We previously reported that endoplasmic reticulum (ER) stress is induced in the subfornical organ (SFO) and the hypothalamic paraventricular nucleus (PVN) of... 
Heart failure | Brain | Sympathetic activity | Hypothalamic paraventricular nucleus | Mitogen-activated protein kinase | Subfornical organ | Endoplasmic reticulum stress | heart failure | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | INDUCED PHOSPHORYLATION | MYOCARDIAL-INFARCTION | ER STRESS | subfornical organ | KINASE | RATS | sympathetic activity | KAPPA-B | brain | endoplasmic reticulum stress | hypothalamic paraventricular nucleus | mitogen-activated protein kinase | UNFOLDED PROTEIN RESPONSE | PERIPHERAL VASCULAR DISEASE | UP-REGULATION | Cholagogues and Choleretics - pharmacology | Tumor Necrosis Factor-alpha - genetics | Heart Failure - physiopathology | Male | NF-KappaB Inhibitor alpha - genetics | Peptidyl-Dipeptidase A - drug effects | Interleukin-1beta - genetics | Sympathetic Nervous System - physiopathology | RNA, Messenger - metabolism | Activating Transcription Factor 6 - genetics | Subfornical Organ - drug effects | Brain - metabolism | Heat-Shock Proteins - genetics | Inflammation - metabolism | Receptor, Angiotensin, Type 1 - genetics | Cyclooxygenase 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Real-Time Polymerase Chain Reaction | Echocardiography | Signal Transduction | Rats | Cyclooxygenase 2 - drug effects | Heart Failure - metabolism | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | Tumor Necrosis Factor-alpha - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Endoplasmic Reticulum Stress | Paraventricular Hypothalamic Nucleus - metabolism | Infusions, Intraventricular | Mitogen-Activated Protein Kinases - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Interleukin-1beta - drug effects | Sympathetic Nervous System - drug effects | Mitogen-Activated Protein Kinase 1 - drug effects | X-Box Binding Protein 1 - drug effects | Sympathetic Nervous System - metabolism | Transcription Factor RelA - genetics | Activating Transcription Factor 6 - drug effects | Mitogen-Activated Protein Kinase 3 - drug effects | Taurochenodeoxycholic Acid - pharmacology | Peptidyl-Dipeptidase A - genetics | Renin-Angiotensin System | Receptor, Angiotensin, Type 1 - drug effects | RNA, Messenger - drug effects | Subfornical Organ - metabolism | Heat-Shock Proteins - drug effects | Activating Transcription Factor 4 - genetics | Activating Transcription Factor 4 - drug effects | NF-KappaB Inhibitor alpha - drug effects | Paraventricular Hypothalamic Nucleus - drug effects | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Transcription Factor RelA - drug effects | X-Box Binding Protein 1 - genetics | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Cellular signal transduction | Endoplasmic reticulum | Health aspects | Mitogen-activated protein kinases | Cardiovascular Neurohormonal Regulation
Journal Article
Free Radical Research, ISSN 1071-5762, 05/2011, Volume 45, Issue 5, pp. 575 - 584
Abstract The beneficial effects of telmisartan on Angiotensin (Ang)-II mediated oxidative stress and renal fibrosis in streptozotocin (STZ)-induced diabetic... 
Renin-angiotensin-aldosterone system | fibrosis | telmisartan | diabetic nephropathy | oxidative stress | Kidney - pathology | Receptors, G-Protein-Coupled - metabolism | Intracellular Signaling Peptides and Proteins - drug effects | Transforming Growth Factor beta1 - metabolism | Collagen Type III - metabolism | NADPH Oxidases - metabolism | Diabetic Nephropathies - drug therapy | Receptor, Angiotensin, Type 2 - drug effects | Intracellular Signaling Peptides and Proteins - metabolism | Peptidyl-Dipeptidase A - drug effects | Angiotensin II Type 1 Receptor Blockers - pharmacology | Kidney - metabolism | NADPH Oxidases - drug effects | Diabetes Mellitus, Experimental | Peptidyl-Dipeptidase A - metabolism | Superoxides - metabolism | Transforming Growth Factor beta1 - drug effects | Receptor, Angiotensin, Type 1 - drug effects | Receptors, G-Protein-Coupled - drug effects | Protein-Serine-Threonine Kinases - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - drug effects | Kidney - drug effects | Angiotensin II - metabolism | Antioxidants - pharmacology | Collagen Type III - drug effects | Diabetic Nephropathies - complications | Up-Regulation - drug effects | Hypertrophy - complications | Animals | Receptor, Angiotensin, Type 2 - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Fibrosis | PPAR gamma - agonists | Protein-Serine-Threonine Kinases - drug effects | Benzoates - pharmacology | Benzimidazoles - pharmacology | Mice | Oxidative Stress - drug effects | Hypertrophy - drug therapy
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 309, Issue 10, pp. H1697 - H1707
CD34(+) stem/progenitor cells have been identified as a promising cell population for the autologous cell-based therapies in patients with cardiovascular... 
cells | Migration | ACE-2 | Ang-(1-7) | Proliferation | Mas receptor | CD34 | STEM-CELLS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | CD34(+) cells | proliferation | PULMONARY-HYPERTENSION | BONE-MARROW | PERIPHERAL-BLOOD | ANGIOTENSIN-CONVERTING ENZYME | ADHESION | IN-VITRO | HEMATOPOIETIC PROGENITOR CELLS | migration | PERIPHERAL VASCULAR DISEASE | EXPRESSION | Antigens, CD34 | Leukocytes, Mononuclear - metabolism | Reactive Oxygen Species - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Receptor, Angiotensin, Type 2 - drug effects | Peptide Fragments - pharmacology | Peptidyl-Dipeptidase A - drug effects | Xanthones - pharmacology | Peptidyl-Dipeptidase A - metabolism | Angiotensin I - pharmacology | Receptor, Angiotensin, Type 1 - drug effects | Receptors, G-Protein-Coupled - drug effects | Wound Healing - drug effects | Proto-Oncogene Proteins - metabolism | Vasoconstrictor Agents - pharmacology | Diminazene - pharmacology | Angiotensin II - pharmacology | Proto-Oncogene Proteins - drug effects | Leukocytes, Mononuclear - drug effects | Vasodilator Agents - pharmacology | Diminazene - analogs & derivatives | Cell Adhesion - drug effects | Cell Movement - drug effects | Receptor, Angiotensin, Type 2 - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Cell Proliferation - drug effects | Vascular Biology and Microcirculation | Ang-(1–7) | CD34+ cells
Journal Article
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 05/2008, Volume 51, Issue 5, pp. 1312 - 1317
Journal Article
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 06/2013, Volume 61, Issue 6, pp. 1233 - 1238
Journal Article
European Journal of Pain, ISSN 1090-3801, 04/2019, Volume 23, Issue 4, pp. 739 - 749
Journal Article
American Journal of Clinical Nutrition, ISSN 0002-9165, 02/2003, Volume 77, Issue 2, pp. 326 - 330
Background: Angiotensin-converting enzyme (ACE; EC 3.4.15.1) plays a dual role in the regulation of hypertension: it catalyzes the production of the... 
Angiotensin-converting enzyme inhibitors | Hypertension | Valine-proline-proline | Lactobacillus helveticus | Isoleusine-proline- proline | Fermented milk | Blood pressure | Bioactive peptides | isoleusine-proline-proline | blood pressure | angiotensin-converting enzyme inhibitors | I-CONVERTING ENZYME | RATS | fermented milk | SOUR MILK | bioactive peptides | NUTRITION & DIETETICS | Lactobacillus helvetacus | REDUCTION | ANTIHYPERTENSIVE PEPTIDES | PURIFICATION | valine-proline-proline | INHIBITORS | hypertension | Milk - chemistry | Lactobacillus - growth & development | Milk - microbiology | Humans | Middle Aged | Antihypertensive Agents - administration & dosage | Hypertension - drug therapy | Male | Peptidyl-Dipeptidase A - drug effects | Peptides - administration & dosage | Milk Proteins - administration & dosage | Milk Proteins - chemistry | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Peptidyl-Dipeptidase A - metabolism | Adult | Female | Milk Proteins - therapeutic use | Blood Pressure - drug effects | Antihypertensive Agents - therapeutic use | Fermentation | Lactobacillus - metabolism | Angiotensin-Converting Enzyme Inhibitors - administration & dosage | Animals | Milk - metabolism | Renin-Angiotensin System - drug effects | Peptides - therapeutic use | Acidophilus milk | Care and treatment | Cookery for hypertensives | Health aspects | ACE inhibitors
Journal Article
Brain Research, ISSN 0006-8993, 2010, Volume 1352, pp. 176 - 186
Journal Article