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Nature, ISSN 0028-0836, 2014, Volume 508, Issue 1, pp. 55 - 60
Journal Article
Circulation, ISSN 0009-7322, 04/2014, Volume 129, Issue 15, pp. 1586 - 1597
BACKGROUND—Pericytes and their crosstalk with endothelial cells are critical for the development of a functional microvasculature and vascular remodeling. It... 
cell communication | transforming growth factor beta | fibroblast growth factor 2 | pericytes | endothelilal cells | pulmonary arterial hypertension | interleukin-6 | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | BONE MORPHOGENETIC PROTEIN | MICROVASCULAR PERICYTES | INHIBITION | ARTERIAL-HYPERTENSION | ADAMS-OLIVER SYNDROME | PERIPHERAL VASCULAR DISEASE | MICE | DIFFERENTIATION | CONTRIBUTES | MOLECULAR-MECHANISMS | Pericytes - drug effects | Rats, Wistar | Humans | Middle Aged | Pericytes - cytology | Fibroblast Growth Factor 2 - pharmacology | Hypertension, Pulmonary - physiopathology | Cell Communication - physiology | Male | Pericytes - physiology | Retinal Vessels - physiology | Microcirculation - physiology | Pulmonary Circulation - physiology | Interleukin-6 - physiology | Retinal Vessels - cytology | Adult | Female | Fibroblast Growth Factor 2 - physiology | Cell Differentiation - physiology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Cells, Cultured | Rats | Mice, Transgenic | Muscle, Smooth, Vascular - cytology | Neovascularization, Physiologic - physiology | Animals | Interleukin-6 - pharmacology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Cell Communication - drug effects | Mice | Hypertension, Pulmonary - pathology | Endothelial Cells - drug effects | Physiological aspects | Smooth muscle | Fibroblast growth factors | Research | Pulmonary hypertension | Interleukin-6 | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 3/2013, Volume 33, Issue 3, pp. 428 - 439
Despite its limited regenerative capacity, the central nervous system (CNS) shares more repair mechanisms with peripheral tissues than previously recognized.... 
fibrosis | pericyte | cerebral ischemia | extracellular matrix | platelet-derived growth factor receptor beta | neurovascular unit | MOUSE-BRAIN | APOPTOSIS | ACTIVATION | MICROVASCULATURE | ANGIOGENESIS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | MESENCHYMAL STEM-CELLS | ENGRAFTMENT | ENDOCRINOLOGY & METABOLISM | HEMATOLOGY | RETINAL PERICYTES | MOLECULAR-MECHANISMS | Capillaries - pathology | Stromal Cells - pathology | Humans | Astrocytes - pathology | Brain Ischemia - metabolism | Male | Intracellular Signaling Peptides and Proteins - metabolism | Antigens, CD - metabolism | Brain - metabolism | Stroke - pathology | Brain - blood supply | Cicatrix - metabolism | Endoglin | Mice, Mutant Strains | Pericytes - pathology | Female | Capillaries - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Pericytes - metabolism | Stromal Cells - metabolism | Receptors, Cell Surface - metabolism | Cerebrovascular Circulation | Stroke - metabolism | Animals | Brain Ischemia - pathology | Brain - pathology | Cicatrix - pathology | Mice | Astrocytes - metabolism | Index Medicus | Cell proliferation | Platelet-derived growth factor receptors | Brain | Stroke | Deposits | Astrocytes | pericytes | Central nervous system | Recovery of function | Patching | Spinal cord injury | Ischemia | Cerebral blood flow | stromal cells | Extracellular matrix | Original
Journal Article
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 05/2015, Volume 116, Issue 10, pp. e81 - e94
RATIONALE:Optimization of cell therapy for cardiac repair may require the association of different cell populations with complementary activities.... 
Cardiac remodeling, ventricular | Pericytes | Acute inferior myocardial infarction | Cell transplantation | Cell- and tissue-based therapy | Stem cells | cell- and tissue-based therapy | PROGENITOR CELLS | stem cells | TISSUE KALLIKREIN | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | pericytes | REGENERATION | cardiac remodeling, ventricular | MODEL | CARDIOMYOCYTES | TRANSPLANTATION | acute inferior myocardial infarction | cell transplantation | IN-VITRO | ISCHEMIC CARDIOMYOPATHY | INTRACORONARY | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Cell Proliferation | Coculture Techniques | Humans | Recovery of Function | Stem Cell Transplantation | Ventricular Remodeling | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Pericytes - transplantation | Myocardial Infarction - physiopathology | Cell Differentiation | Saphenous Vein - cytology | Disease Models, Animal | Myocardial Contraction | Myocardial Infarction - surgery | Paracrine Communication | Cell Survival | Pericytes - metabolism | Cells, Cultured | Myocardium - pathology | Myocardial Infarction - metabolism | Mice, SCID | Myocytes, Cardiac - pathology | Myocytes, Cardiac - transplantation | Regeneration | Phenotype | Animals | Fibrosis | Myocytes, Cardiac - metabolism | Hemodynamics | Angiogenic Proteins - metabolism | Neovascularization, Physiologic | Index Medicus
Journal Article
Journal Article