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Neuron, ISSN 0896-6273, 01/2015, Volume 85, Issue 2, pp. 296 - 302
Journal Article
Genes and Development, ISSN 0890-9369, 08/2003, Volume 17, Issue 15, pp. 1835 - 1840
Several platelet-derived growth factor (PDGF) and vascular endothelial growth factor (VEGF) family members display C-terminal protein motifs that confer... 
Pericyte | Cell retention | Retina | Mesangial cell | PDGF | Heparan sulphate proteoglycan | DIABETIC-RETINOPATHY | VEGF ISOFORMS | pericyte | ANGIOGENESIS | A-CHAIN | DEVELOPMENTAL BIOLOGY | mesangial cell | CELL BIOLOGY | cell retention | retina | IN-VIVO | GENETICS & HEREDITY | MICE | heparan sulphate proteoglycan | HEPARAN-SULFATE PROTEOGLYCANS | GROWTH-FACTOR | BINDING | SMOOTH-MUSCLE CELLS | Kidney - physiology | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Microcirculation - metabolism | Retina - metabolism | Vascular Endothelial Growth Factors | Glomerulosclerosis, Focal Segmental - genetics | Molecular Sequence Data | Intercellular Signaling Peptides and Proteins - metabolism | Lymphokines - metabolism | Protein Structure, Tertiary | Amino Acid Sequence | Proto-Oncogene Proteins c-sis - genetics | Proto-Oncogene Proteins c-sis - metabolism | Retinal Degeneration - genetics | Pericytes - metabolism | Proteinuria - genetics | Amino Acid Motifs | Retina - physiology | Phenotype | Platelet-Derived Growth Factor - metabolism | Animals | Endothelium, Vascular - metabolism | Alleles | Mice | Models, Genetic | Mutation | Microscopy, Fluorescence | Cell Movement | Proteins | Blood platelets | Secretion | Analysis | Genetic research | Genetic aspects | Growth factors | Endothelium | Index Medicus | Research Communications | Glomerulosclerosis; Focal Segmental/genetics | Intercellular Signaling Peptides and Proteins/metabolism | Platelet-Derived Growth Factor/metabolism | Proto-Oncogene Proteins c-sis/genetics/metabolism | Proteinuria/genetics | Models; Genetic | Microcirculation/metabolism | Lymphokines/metabolism | Microscopy; Fluorescence | Pericytes/metabolism | Kidney/physiology | Protein Structure; Tertiary | Retinal Degeneration/genetics | Endothelial Growth Factors/metabolism | Endothelium; Vascular/metabolism | Retina/metabolism/physiology
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2011, Volume 286, Issue 20, pp. 17536 - 17542
Apolipoprotein E (apoE) is a major apolipoprotein in the brain. The epsilon 4 allele of apoE is a major risk factor for Alzheimer disease, and apoE deficiency... 
CHOLESTEROL-METABOLISM | PROTEIN-KINASE-C | RECEPTOR-RELATED PROTEIN | LIPOPROTEIN RECEPTORS | LDL-RECEPTOR | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE MODEL | ENDOTHELIAL-CELLS | A-BETA | PLAQUE-FORMATION | Protein Kinase C - genetics | Apolipoprotein E4 - genetics | Humans | Astrocytes - pathology | Low Density Lipoprotein Receptor-Related Protein-1 - genetics | Antigens, CD - genetics | Alzheimer Disease - pathology | Antigens, CD - metabolism | Occludin | Phosphorylation - genetics | Protein Isoforms - metabolism | Pericytes - pathology | Protein Kinase C - metabolism | Tumor Suppressor Proteins - genetics | Membrane Proteins - metabolism | Tight Junctions - metabolism | Receptors, LDL - genetics | Tumor Suppressor Proteins - metabolism | Apolipoprotein E4 - metabolism | Endothelial Cells - metabolism | Membrane Proteins - genetics | Pericytes - metabolism | Cells, Cultured | Low Density Lipoprotein Receptor-Related Protein-1 - metabolism | Receptors, LDL - metabolism | Permeability | Apolipoprotein E3 - metabolism | Blood-Brain Barrier - metabolism | Gene Knock-In Techniques | Mice, Knockout | Blood-Brain Barrier - pathology | Apolipoprotein E3 - genetics | Animals | Models, Biological | Alzheimer Disease - metabolism | Mice | Alzheimer Disease - genetics | Endothelial Cells - pathology | Enzyme Activation - genetics | Tight Junctions - pathology | Astrocytes - metabolism | Protein Isoforms - genetics | Index Medicus | HDL | Neurobiology | PKC | Alzheimer Disease | Apolipoproteins | Endothelium | Tight Junction | Blood-Brain Barrier
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2013, Volume 19, Issue 8, pp. 1047 - 1053
Journal Article
Nature Medicine, ISSN 1078-8956, 06/2017, Volume 23, Issue 6, pp. 733 - 741
Blood vessels in the central nervous system (CNS) are controlled by neuronal activity. For example, widespread vessel constriction (vessel tone) is induced by... 
MEDICINE, RESEARCH & EXPERIMENTAL | PIAL VESSELS | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRACE AMINES | SYNAPTIC-TRANSMISSION | HYPOXIA | CELL BIOLOGY | CENTRAL-NERVOUS-SYSTEM | LOCOMOTOR FUNCTION | CEREBRAL-ARTERIES | RECEPTORS | BRAIN | Capillaries - pathology | Serotonin 5-HT1 Receptor Antagonists - pharmacology | Receptors, Adrenergic, alpha-2 - metabolism | Transcriptome | Capillaries - drug effects | RNA, Messenger - metabolism | Oxygen - metabolism | Hypoxia - metabolism | Receptors, Serotonin, 5-HT1 - metabolism | Spinal Cord Injuries - pathology | Receptor, Serotonin, 5-HT1B - metabolism | Aromatic-L-Amino-Acid Decarboxylases - metabolism | Tyramine - metabolism | Capillaries - metabolism | Locomotion - drug effects | Injections, Spinal | Capillaries - physiopathology | Spinal Cord Injuries - metabolism | Pericytes - metabolism | Vasoconstriction | Rats | Microscopy, Interference | Oxygen Inhalation Therapy | Microscopy, Confocal | Animals | Norepinephrine - metabolism | Tryptamines - metabolism | Serotonin - metabolism | Spinal Cord Injuries - physiopathology | Biogenic Monoamines - metabolism | Locomotion - physiology | Enzymes | Care and treatment | Analysis | Tryptophan | Spinal cord injuries | Dosage and administration | Drug therapy | Health aspects | Noradrenaline | Blood flow | Index Medicus | spinal cord injury | AADC | hypoxia | pericyte | capillary | locomotion | Trace amines | neurovascular coupling | 5-HT1B receptor | motoneurons | ischemia
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2013, Volume 15, Issue 7, pp. 807 - 817
In a significant fraction of breast cancer patients, distant metastases emerge after years or even decades of latency. How disseminated tumour cells (DTCs) are... 
CANCER-CELLS | METASTATIC NICHE | EPITHELIAL-CELLS | HEMATOPOIETIC STEM-CELLS | ENDOTHELIAL-CELLS | SELF-RENEWAL | 3-DIMENSIONAL CULTURE | EXTRACELLULAR-MATRIX | ANGIOCRINE FACTORS | BASEMENT-MEMBRANE | CELL BIOLOGY | Neoplasm, Residual - pathology | Neoplasm, Residual - metabolism | Humans | Lung Neoplasms - metabolism | Neovascularization, Pathologic | Tumor Microenvironment | Neoplasm, Residual - blood supply | Brain Neoplasms - blood supply | Brain Neoplasms - metabolism | Breast Neoplasms - metabolism | Brain Neoplasms - secondary | Lung Neoplasms - secondary | Pericytes - pathology | Female | Tumor Cells, Cultured | Lung Neoplasms - blood supply | Pericytes - metabolism | Breast Neoplasms - blood supply | Cell Adhesion Molecules - metabolism | Zebrafish - growth & development | Bone Marrow Neoplasms - secondary | Animals | Bone Marrow Neoplasms - metabolism | Breast Neoplasms - pathology | Endothelium, Vascular - metabolism | Zebrafish - metabolism | Fluorescent Antibody Technique | Endothelium, Vascular - pathology | Stem Cell Niche - physiology | Mice | Thrombospondin 1 - metabolism | Bone Marrow Neoplasms - blood supply | Transforming Growth Factor beta - metabolism | Physiological aspects | Bone morphogenetic proteins | Genetic aspects | Breast cancer | Research | Transforming growth factors | Index Medicus
Journal Article
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 3/2013, Volume 33, Issue 3, pp. 428 - 439
Despite its limited regenerative capacity, the central nervous system (CNS) shares more repair mechanisms with peripheral tissues than previously recognized.... 
fibrosis | pericyte | cerebral ischemia | extracellular matrix | platelet-derived growth factor receptor beta | neurovascular unit | MOUSE-BRAIN | APOPTOSIS | ACTIVATION | MICROVASCULATURE | ANGIOGENESIS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | MESENCHYMAL STEM-CELLS | ENGRAFTMENT | ENDOCRINOLOGY & METABOLISM | HEMATOLOGY | RETINAL PERICYTES | MOLECULAR-MECHANISMS | Capillaries - pathology | Stromal Cells - pathology | Humans | Astrocytes - pathology | Brain Ischemia - metabolism | Male | Intracellular Signaling Peptides and Proteins - metabolism | Antigens, CD - metabolism | Brain - metabolism | Stroke - pathology | Brain - blood supply | Cicatrix - metabolism | Endoglin | Mice, Mutant Strains | Pericytes - pathology | Female | Capillaries - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Pericytes - metabolism | Stromal Cells - metabolism | Receptors, Cell Surface - metabolism | Cerebrovascular Circulation | Stroke - metabolism | Animals | Brain Ischemia - pathology | Brain - pathology | Cicatrix - pathology | Mice | Astrocytes - metabolism | Index Medicus | Cell proliferation | Platelet-derived growth factor receptors | Brain | Stroke | Deposits | Astrocytes | pericytes | Central nervous system | Recovery of function | Patching | Spinal cord injury | Ischemia | Cerebral blood flow | stromal cells | Extracellular matrix | Original
Journal Article
Cell Stem Cell, ISSN 1934-5909, 01/2015, Volume 16, Issue 1, pp. 51 - 66
Mesenchymal stem cells (MSCs) reside in the perivascular niche of many organs, including kidney, lung, liver, and heart, although their roles in these tissues... 
REGULATOR | ORIGIN | SONIC HEDGEHOG | PATHWAY | BONE-MARROW NICHE | MYOFIBROBLASTS | NG2 PROTEOGLYCAN | EXPRESSION | MESENCHYMAL STEM-CELLS | GROWTH FACTOR-AA | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Organ Specificity - drug effects | Diphtheria Toxin - pharmacology | Neovascularization, Physiologic - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Fibrosis - metabolism | Cell Lineage - drug effects | Myofibroblasts - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Kruppel-Like Transcription Factors - metabolism | Pericytes - pathology | Bone Marrow Cells - drug effects | Colony-Forming Units Assay | Aorta - physiopathology | Homeostasis - drug effects | Heart Ventricles - pathology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Endothelial Cells - metabolism | Aorta - drug effects | Pericytes - metabolism | Cells, Cultured | Proteoglycans - metabolism | Antigens - metabolism | Aorta - pathology | Myofibroblasts - cytology | Animals | Heart Ventricles - physiopathology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Blood Vessels - drug effects | Mice | Stem Cell Niche - drug effects | Zinc Finger Protein GLI1 | Fibrosis - pathology | Bone Marrow Cells - metabolism | Endothelial Cells - drug effects | Heart Ventricles - drug effects | Index Medicus
Journal Article