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Nature cell biology, ISSN 1476-4679, 02/2018, Volume 20, Issue 3, pp. 307 - 319
Reactive oxygen species (ROS) contribute to tissue damage and remodelling mediated by the inflammatory response after injury... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Ganglia, Spinal - injuries | Sciatic Nerve - injuries | Axons - enzymology | Reactive Oxygen Species - metabolism | Exosomes - pathology | Sciatic Nerve - physiopathology | Spinal Cord Injuries - genetics | Spinal Cord Injuries - pathology | Endocytosis | CX3C Chemokine Receptor 1 - metabolism | Ganglia, Spinal - enzymology | Peripheral Nerve Injuries - physiopathology | Spinal Cord Injuries - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Dyneins - metabolism | Cell Line | NADPH Oxidase 2 - deficiency | Macrophages - pathology | Signal Transduction | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Endosomes - pathology | Nerve Regeneration | Macrophages - enzymology | Mice, Knockout | Animals | Peripheral Nerve Injuries - enzymology | NADPH Oxidase 2 - metabolism | Axons - pathology | Ganglia, Spinal - pathology | Exosomes - enzymology | Endosomes - enzymology | Spinal Cord Injuries - physiopathology | NADPH Oxidase 2 - genetics | Nerve Degeneration | Peripheral Nerve Injuries - genetics | Sciatic Nerve - enzymology | Axons | Regeneration | Reactive oxygen species | NADP (Coenzyme) | Nervous system | Cellular signal transduction | Research | Gene expression | Oxidase | Recovery of function | Spinal injuries | CX3CR1 protein | Inflammatory response | AKT protein | Exosomes | Macrophages | Inactivation | NAD(P)H oxidase | Signal transduction | Dorsal root ganglia | Neurodegeneration | Dieback | CYBB protein | Degeneration | Cell body | Deactivation | Inflammation | Ganglia | 1-Phosphatidylinositol 3-kinase | Signaling | Dynein | PTEN protein | Sciatic nerve | Endosomes | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0123122 - e0123122
Neuropathic pain is common in peripheral nerve injury and often fails to respond to ordinary medication... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fatty Acid Synthase, Type I - biosynthesis | Spinal Cord Injuries - drug therapy | Neuralgia - metabolism | Catechin - administration & dosage | Male | Pain - metabolism | Pain - drug therapy | Cell Membrane - metabolism | Neuralgia - physiopathology | Fatty Acids - metabolism | Cell Membrane - drug effects | Cytokines - metabolism | Hyperalgesia - metabolism | Spinal Cord Injuries - metabolism | Fatty Acid Synthase, Type I - metabolism | Neuralgia - drug therapy | Hyperalgesia - physiopathology | Gene Expression Regulation - drug effects | Animals | Hyperalgesia - drug therapy | Pain - physiopathology | Pain Threshold | Catechin - analogs & derivatives | Mice | Spinal Cord Injuries - physiopathology | Cytokines - biosynthesis | Peripheral Nerve Injuries | Nerves, Peripheral | Catechin | Care and treatment | Pain | Physiological aspects | Research | Injuries | Methods | Spinal cord | Laboratories | Nervous system | Glutamic acid receptors | Neuropathy | Kinases | Proteins | Rodents | Polyphenols | Localization | Dorsal horn | NF-κB protein | Pain perception | Cytokines | Epigallocatechin-3-gallate | N-Methyl-D-aspartic acid receptors | Inflammation | Derivatives | Fatty acids | Fatty-acid synthase | Latency | Injury prevention | Molecular modelling | Nitric oxide | Index Medicus | Dolor neuropàtic | Tractament | Treatment | Neuràlgia | Dolor | Neuralgia
Journal Article
Cellular and molecular neurobiology, ISSN 0272-4340, 1/2016, Volume 36, Issue 1, pp. 37 - 46
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 01/2017, Volume 37, Issue 4, pp. 871 - 881
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 03/2015, Volume 290, Issue 11, pp. 6937 - 6950
.... After peripheral nerve injury, Schwann cells transition from axon myelination to a demyelinated state that supports neuronal survival and ultimately remyelination of axons... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Sciatic Nerve - injuries | Demyelinating Diseases - genetics | Early Growth Response Protein 2 - genetics | Epigenesis, Genetic | Demyelinating Diseases - complications | Male | Demyelinating Diseases - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Myelin Sheath - metabolism | Sciatic Nerve - metabolism | Peripheral Nerve Injuries - metabolism | Demyelinating Diseases - pathology | Peripheral Nerve Injuries - complications | Myelin Sheath - pathology | Signal Transduction | SOXE Transcription Factors - metabolism | Sciatic Nerve - pathology | Cells, Cultured | Gene Expression Regulation | Peripheral Nerve Injuries - pathology | Rats | Schwann Cells - pathology | Schwann Cells - metabolism | Transcription Factors - genetics | Rats, Sprague-Dawley | Transcription Factors - metabolism | Animals | Histones - genetics | Enhancer Elements, Genetic | Proto-Oncogene Proteins c-jun - metabolism | Myelin Sheath - genetics | Early Growth Response Protein 2 - metabolism | Mice | Histones - metabolism | Peripheral Nerve Injuries - genetics | Core Binding Factor Alpha 1 Subunit - genetics | SOXE Transcription Factors - genetics | Index Medicus | Gene Regulation | Glial Cell | ChIP Sequencing (ChIP-seq) | Schwann Cells | Histone Modification | Neurobiology | Chromatin Immunoprecipitation (ChiP)
Journal Article
Molecular therapy, ISSN 1525-0016, 03/2015, Volume 23, Issue 3, pp. 423 - 433
Journal Article