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Annual Review of Nutrition, ISSN 0199-9885, 08/2012, Volume 32, Issue 1, pp. 147 - 160
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 2013, Volume 216, Issue 1, pp. 1 - 11
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2008, Volume 105, Issue 46, pp. 17712 - 17717
Refsum disease is caused by a deficiency of phytanoyl-CoA hydroxylase (PHYH), the first enzyme of the peroxisomal a-oxidation system, resulting in the... 
Cerebellum | Peroxisomes | Diet | Gait | Purkinje cells | Alleles | Central nervous system | Mice | Fatty acids | Refsum disease | Fatty acid oxidation | Metabolic disorder | PPAR-ALPHA | RAT | MULTIDISCIPLINARY SCIENCES | PRISTANIC ACID | ACTIVATED RECEPTOR-ALPHA | ASTROCYTES | GAS-CHROMATOGRAPHY | METABOLISM | CHAIN FATTY-ACIDS | metabolic disorder | peroxisomes | MICE | fatty acid oxidation | PHYTANIC ACID | Mixed Function Oxygenases - deficiency | Central Nervous System - abnormalities | Central Nervous System - pathology | Lipidoses - pathology | Male | Purkinje Cells - enzymology | Refsum Disease - enzymology | Behavior, Animal - drug effects | Peripheral Nervous System Diseases - pathology | Purkinje Cells - drug effects | Spermatogonia - pathology | Ataxia - pathology | Disease Models, Animal | Spermatogonia - drug effects | Ataxia - physiopathology | Gene Targeting | Automation | Refsum Disease - pathology | Peripheral Nervous System Diseases - enzymology | Lipidoses - enzymology | Gait - drug effects | Phenotype | Phytol - pharmacology | Animals | Ataxia - enzymology | Refsum Disease - physiopathology | Central Nervous System - drug effects | Spermatogonia - enzymology | Genetic Vectors | Mixed Function Oxygenases - genetics | Phytol - administration & dosage | Dietary Supplements | Purkinje Cells - pathology | Central Nervous System - enzymology | Phytanic Acid - blood | Ataxia | Models | Biological oxidation (Metabolism) | Research | Properties | Health aspects | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2009, Volume 106, Issue 49, pp. 20960 - 20965
During injury to the nervous system, innate immune cells mediate phagocytosis of debris, cytokine production, and axon regeneration. In the neuro-degenerative... 
Axons | Motor neurons | Spinal cord | Nerves | Central nervous system | Disease progression | Antibodies | Amyotrophic lateral sclerosis | Macrophages | Microglia | Peripheral nervous system | Innate immunity | Neuroimmunology | Macrophage | MOTOR-NEURONS | CELLS | COMPLEMENT | MULTIDISCIPLINARY SCIENCES | WALLERIAN DEGENERATION | INJURY | macrophage | peripheral nervous system | AMYOTROPHIC-LATERAL-SCLEROSIS | amyotrophic lateral sclerosis | FAMILIAL ALS | INHERITED ALS | neuroimmunology | innate immunity | MOUSE MODEL | DISEASE PROGRESSION | Superoxide Dismutase - genetics | Immunity, Humoral - immunology | Muscles - pathology | Peripheral Nervous System - immunology | Peripheral Nervous System - pathology | Aging - immunology | Flow Cytometry | Myeloid Cells - immunology | Spinal Cord - pathology | Amyotrophic Lateral Sclerosis - enzymology | Peripheral Nervous System - enzymology | Amyotrophic Lateral Sclerosis - immunology | Green Fluorescent Proteins - metabolism | Macrophage Activation - immunology | Sciatic Nerve - pathology | Mice, Transgenic | Nerve Degeneration - immunology | Complement C4 - immunology | Mutation - genetics | Nerve Degeneration - pathology | Aging - pathology | Spinal Cord - immunology | Immunity, Innate - immunology | Amyotrophic Lateral Sclerosis - pathology | Phenotype | Animals | Staining and Labeling | Muscles - innervation | Mice | Myeloid Cells - pathology | Immunity | Research | Biological Sciences
Journal Article
Neuroscience, ISSN 0306-4522, 2016, Volume 343, pp. 355 - 363
Highlights • GCDH is widely expressed in embryonic and adult rat tissues. • A high GCDH expression in embryonic rat brain suggests an important role in brain... 
Neurology | expression | central and peripheral nervous system | glutaryl-CoA-dehydrogenase | embryonic development | glutaric aciduria type-I | rat tissues | 3-HYDROXYGLUTARIC ACID | MITOCHONDRIA | AXONAL-TRANSPORT | ORGANIC-ACIDS | NEUROSCIENCES | DEFICIENCY | MOUSE MODEL | ACIDURIA TYPE-I | ACIDEMIA TYPE-I | PART 2 | TYPE-1 | Glutaryl-CoA Dehydrogenase - metabolism | Liver - enzymology | Kidney - enzymology | Brain - enzymology | Neurons - cytology | Peripheral Nervous System - growth & development | Brain - growth & development | Lung - cytology | Liver - growth & development | Peripheral Nervous System - cytology | Intestinal Mucosa - growth & development | Lung - enzymology | Intestinal Mucosa - cytology | Intestinal Mucosa - enzymology | Female | Neurons - metabolism | Peripheral Nervous System - enzymology | Epithelial Cells - cytology | Glutaryl-CoA Dehydrogenase - genetics | Brain - cytology | Muscle Development - physiology | Kidney - growth & development | Kidney - cytology | Rats, Sprague-Dawley | Mice, Knockout | Animals | Muscles - enzymology | Epithelial Cells - enzymology | Fluorescent Antibody Technique | Liver - cytology | Lung - growth & development | Microscopy, Fluorescence | Muscles - cytology | Brain | Enzymes | Embryonic development | Neurons | Islamic law | Tryptophan | Albumin | Skin | Movement disorders
Journal Article
Pain, ISSN 0304-3959, 06/2008, Volume 137, Issue 1, pp. 81 - 95
Neuropathic pain consequent to peripheral injury is associated with local inflammation and overexpression of nitric oxide synthases (NOS) and inflammatory... 
Nitric oxide synthase | Purinergic antagonism | IL-1β | PPADS | IL-6 | Neuropathic pain | ACTIVATION | RAT | IL-1 beta | MECHANISMS | CYTOKINE EXPRESSION | NEUROSCIENCES | SENSORY NEURONS | CLINICAL NEUROLOGY | neuropathic pain | MICROGLIA | P2X RECEPTORS | GENE-EXPRESSION | ANESTHESIOLOGY | nitric oxide synthase | SCHWANN-CELLS | purinergic antagonising | ATP | Nitric Oxide Synthase Type II - biosynthesis | Central Nervous System - metabolism | Interleukin-6 - antagonists & inhibitors | Nitric Oxide Synthase Type I - biosynthesis | Male | Peripheral Nervous System Diseases - physiopathology | Pain - metabolism | Drug Delivery Systems | Peripheral Nervous System Diseases - prevention & control | Nitric Oxide Synthase Type II - antagonists & inhibitors | Nitric Oxide Synthase Type I - antagonists & inhibitors | Behavior, Animal - drug effects | Interleukin-1beta - biosynthesis | Pyridoxal Phosphate - analogs & derivatives | Interleukin-1beta - antagonists & inhibitors | Pyridoxal Phosphate - pharmacology | Pyridoxal Phosphate - therapeutic use | Pain - prevention & control | Mice, Inbred C57BL | Peripheral Nervous System Diseases - enzymology | Purinergic P2 Receptor Antagonists | Behavior, Animal - physiology | Receptors, Purinergic P2 - physiology | Peripheral Nervous System Diseases - metabolism | Animals | Pain - physiopathology | Peripheral Nervous System - metabolism | Central Nervous System - drug effects | Interleukin-6 - biosynthesis | Mice | Central Nervous System - enzymology | Peripheral Nervous System - drug effects
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2018, Volume 314, Issue 3, pp. H580 - H592
Despite advances in antihypertensive therapeutics, at least 15-20% of hypertensive patients have resistant hypertension through mechanisms that remain poorly... 
Renin angiotensin system | Neurogenic hypertension | (pro)renin receptor | NADPH oxidase | Central nervous system | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ACTIVATED PROTEIN-KINASE | RESISTANT HYPERTENSION | neurogenic hypertension | NADPH OXIDASE ACTIVITY | renin angiotensin system | central nervous system | SIGNAL-TRANSDUCTION | NAD(P)H OXIDASE | PARAVENTRICULAR NUCLEUS | HUMAN RENIN | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | HYDROGEN-PEROXIDE | TRANSGENIC MICE | Up-Regulation | Vacuolar Proton-Translocating ATPases - genetics | Blood Pressure - genetics | Humans | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Synapsins - genetics | Vacuolar Proton-Translocating ATPases - metabolism | Hypertension - chemically induced | Renin - administration & dosage | Female | Blood Pressure - drug effects | Hypertension - enzymology | Hypertension - genetics | Angiotensin II | Neurons - drug effects | Disease Models, Animal | Promoter Regions, Genetic | NADPH Oxidase 4 - genetics | Signal Transduction | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Mice, Transgenic | Renin-Angiotensin System - genetics | Hypertension - physiopathology | NADPH Oxidase 4 - metabolism | Animals | Neurons - enzymology | Central Nervous System - drug effects | Renin-Angiotensin System - drug effects | Central Nervous System - physiopathology | Infusions, Intraventricular | Central Nervous System - enzymology | Receptors, Cell Surface - genetics | Genetic aspects | Blood pressure | Gene expression | Health aspects | Angiotensin | (pro)renin receptor, renin angiotensin system
Journal Article
Circulation Research, ISSN 0009-7330, 08/2016, Volume 119, Issue 5, pp. 607 - 620
Journal Article
Journal Article