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Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 09/2018, Volume 19, Issue 9, p. 2487
Lymphatic vessels drain excess tissue fluids to maintain the interstitial environment. Lymphatic capillaries develop during the progression of tissue fibrosis... 
Vascular endothelial growth factor-C | Lymphangiogenesis | Transforming growth factor-β | Fibrosis | TISSUE-GROWTH-FACTOR | TYPE-1 DIABETIC-PATIENTS | PERITONEAL MESOTHELIAL CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | vascular endothelial growth factor-C | MONOCLONAL-ANTIBODY | FACTOR RECEPTOR-3 | CHEMISTRY, MULTIDISCIPLINARY | SOLUTE TRANSPORT | transforming growth factor-beta | LYMPHATIC ABSORPTION RATE | ULTRAFILTRATION FAILURE | DIALYSIS PATIENTS | lymphangiogenesis | fibrosis | GLYCATION END-PRODUCTS | Epithelial Cells - metabolism | Humans | Peritoneal Fibrosis - genetics | Nephrogenic Fibrosing Dermopathy - metabolism | Renal Insufficiency, Chronic - metabolism | Lymphatic Vessels - metabolism | Peritoneal Fibrosis - metabolism | Renal Insufficiency, Chronic - genetics | Vascular Endothelial Growth Factor D - genetics | Macrophages - pathology | Signal Transduction | Gene Expression Regulation | Nephrogenic Fibrosing Dermopathy - genetics | Epithelial Cells - pathology | Nephrogenic Fibrosing Dermopathy - pathology | Peritoneal Fibrosis - pathology | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Vascular Endothelial Growth Factor C - genetics | Renal Insufficiency, Chronic - pathology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Transforming Growth Factor beta - genetics | Vascular Endothelial Growth Factor C - metabolism | Lymphatic Vessels - pathology | Connective Tissue Growth Factor - genetics | Lymphangiogenesis - genetics | Transforming Growth Factor beta - metabolism | Connective Tissue Growth Factor - metabolism | Vascular Endothelial Growth Factor Receptor-3 - genetics | Lymphatic system | Epithelial cells | Transforming growth factor-a | Blood vessels | Connective tissue growth factor | Macrophages | Endothelial cells | Peritoneal dialysis | Vascular endothelial growth factor receptors | Connective tissues | Signal transduction | Signaling | Etiology | Capillaries | Vascular endothelial growth factor | Growth factors | Peritoneum | Nephrology | Homeostasis | Mitochondrial DNA | Proteins | Fibroblasts | Cytokines | Inflammation | Permeability | Diabetes | Kidney diseases | transforming growth factor-β
Journal Article
Immunity, ISSN 1074-7613, 01/2014, Volume 40, Issue 1, pp. 40 - 50
Journal Article
Kidney International, ISSN 0085-2538, 08/2013, Volume 84, Issue 2, pp. 297 - 307
Mesenchymal stem cells (MSCs) are multipotent adult stem cells that have regenerative capability and exert paracrine actions on damaged tissues. Since... 
peritoneal fibrosis | transforming growth factor-β1 | hepatocyte growth factor | mesenchymal stem cells | epithelial-to-mesenchymal transition | Peritoneal fibrosis | Transforming growth factor-β1 | Hepatocyte growth factor | Epithelial-to-mesenchymal transition | Mesenchymal stem cells | Coculture Techniques | Humans | Rats, Inbred F344 | Transforming Growth Factor beta1 - metabolism | Male | Green Fluorescent Proteins - genetics | Mesenchymal Stromal Cells - immunology | Peritoneum - pathology | Time Factors | Peritoneal Fibrosis - prevention & control | Epithelial-Mesenchymal Transition | Inflammation Mediators - metabolism | Peritoneal Fibrosis - metabolism | Peritoneum - immunology | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Disease Models, Animal | Green Fluorescent Proteins - metabolism | Paracrine Communication | Signal Transduction | Animals, Genetically Modified | Cells, Cultured | Smad2 Protein - metabolism | Mesenchymal Stromal Cells - metabolism | Rats | Peritoneal Fibrosis - pathology | Peritoneal Fibrosis - chemically induced | Chemotaxis | Rats, Sprague-Dawley | Hepatocyte Growth Factor - metabolism | Macrophages - metabolism | Animals | Chlorhexidine - analogs & derivatives | Glucose - metabolism | Peritoneum - metabolism | Mesenchymal Stem Cell Transplantation | Culture Media, Conditioned - metabolism | Basic Research
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e76808
Background: During the development and progression of endometriotic lesions, excess fibrosis may lead to scarring, chronic pain, and altered tissue function.... 
RNA INTEGRITY | ACTIVATION | NODULES | EPITHELIAL-CELLS | PERITONEAL ENDOMETRIOSIS | MULTIDISCIPLINARY SCIENCES | DISEASE | IDIOPATHIC PULMONARY-FIBROSIS | MESENCHYMAL TRANSITION | EXPRESSION | BETA | Endometriosis - genetics | Stromal Cells - pathology | Humans | Actins - metabolism | Fibrosis - metabolism | Wnt Proteins - metabolism | Actins - genetics | Young Adult | Heterografts | Wnt Proteins - genetics | RNA Interference | Adult | Female | Collagen - genetics | Wnt Signaling Pathway | Disease Models, Animal | Perylene - analogs & derivatives | Fibrosis - genetics | Stromal Cells - metabolism | Endometriosis - pathology | beta Catenin - metabolism | Perylene - pharmacology | beta Catenin - genetics | Collagen - metabolism | Animals | Endometriosis - metabolism | Connective Tissue Growth Factor - genetics | Connective Tissue Growth Factor - metabolism | Collagen (type I) | Wnt protein | Disease | Xenotransplantation | Collagens | Smooth muscle | Antagonists | Activation | Contraction | Fibronectin | Proteins | Connective tissues | β-catenin | Signal transduction | Pain | Actin | Surgery | Implants | Xenografts | Lesions | Growth factors | Endometrium | Recombinant | Menstruation | Gels | Immunodeficiency | Muscles | Endometriosis | Connective tissue growth factor | Laparoscopy | Gene expression | Patients | Molecular chains | Studies | Signaling | Scars | Molecular modelling | Pulmonary fibrosis | Stromal cells | Infertility | Fibrosis | Mice
Journal Article
Kidney International, ISSN 0085-2538, 06/2018, Volume 93, Issue 6, pp. 1384 - 1396
Ultrafiltration failure is a major complication of long-term peritoneal dialysis, resulting in dialysis failure. Peritoneal fibrosis induced by continuous... 
α-1,6 fucosyltransferase | fibrosis | peritoneal dialysis | core fucosylation | peritoneal membrane | cell signaling | CELLS | ACTIVATION | MEMBRANE | MOUSE | E-CADHERIN | MESENCHYMAL TRANSITION | DIALYSIS | BETA | PATHWAY | IMATINIB | UROLOGY & NEPHROLOGY | alpha-1,6 fucosyltransferase | RNA, Small Interfering - genetics | Dialysis Solutions | Fucose - metabolism | Transforming Growth Factor beta1 - metabolism | Peritoneum - drug effects | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Glucose | Peritoneum - pathology | RNA Interference | Peritoneal Fibrosis - prevention & control | Peritoneal Fibrosis - metabolism | Disease Models, Animal | Fucosyltransferases - genetics | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Signal Transduction | Imatinib Mesylate - pharmacology | Peritoneal Fibrosis - pathology | Peritoneal Fibrosis - chemically induced | Rats, Sprague-Dawley | Fucosyltransferases - metabolism | Platelet-Derived Growth Factor - metabolism | Animals | Receptors, Transforming Growth Factor beta - metabolism | Receptors, Platelet-Derived Growth Factor - metabolism | Peritoneal Dialysis - methods | Protein Kinase Inhibitors - pharmacology | Protein Processing, Post-Translational | Peritoneum - metabolism | RNA, Small Interfering - metabolism | Platelet-derived growth factor | Transforming growth factor | Drug development | Kinases | Cybernetics | Proteins | Signal transduction | Ultrafiltration | Rodents | Extracellular matrix | Growth factors | Antigens | Imatinib | Immunoglobulins | Statistical analysis | Fluid | Equilibrium | Polymerase chain reaction | Collagen | Adenoviruses | Fibrosis | Protein expression | Software | Peritoneum
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, p. e0183013
Background Abdominal surgery and disease cause persistent abdominal adhesions, pelvic pain, infertility and occasionally, bowel obstruction. Current treatments... 
TO-MESENCHYMAL TRANSITION | PROGESTERONE | MULTIDISCIPLINARY SCIENCES | INJURED PERITONEUM | SURFACE EPITHELIAL-CELLS | FOLLICULAR-FLUID | MICE | CARBON NANOTUBES | ABDOMINAL-CAVITY | ADHESION FORMATION | MESOTHELIAL CELLS | Humans | Peritoneal Fibrosis - genetics | MicroRNAs - metabolism | Molecular Targeted Therapy | RNA, Messenger - metabolism | Tissue Adhesions - chemically induced | Tissue Adhesions - pathology | Dexamethasone - pharmacology | Nanotubes, Carbon - toxicity | Peritoneal Fibrosis - prevention & control | Tissue Adhesions - genetics | Female | Collagen - genetics | Progesterone - pharmacology | Extracellular Matrix Proteins - metabolism | Epithelium - drug effects | Epithelium - pathology | Gene Expression | Epithelium - metabolism | Tissue Adhesions - prevention & control | Extracellular Matrix Proteins - antagonists & inhibitors | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Collagen - antagonists & inhibitors | Peritoneal Fibrosis - pathology | Peritoneal Fibrosis - chemically induced | Collagen - metabolism | Protein-Lysine 6-Oxidase - antagonists & inhibitors | Animals | Protein-Lysine 6-Oxidase - metabolism | Aminopropionitrile - pharmacology | Mice | MicroRNAs - genetics | Protein-Lysine 6-Oxidase - genetics | Abdominal Cavity - surgery | Primary Cell Culture | Interleukin-1alpha - pharmacology | Lysine | Fibrosis | Research | Reproductive health | Collagen (type I) | Interleukin | Oxidase | Nanotubes | mRNA | Medical schools | Prevention | Pain | Intestine | Surgery | Interleukin 1 | Diaphragm (anatomy) | Fibre | miRNA | Life sciences | Inhibition | Lesions | Multi wall carbon nanotubes | Deposition | Medical research | Dexamethasone | Bone morphogenetic protein 1 | Crosslinking | Laparoscopy | Inflammation | Gene expression | Lysyl oxidase | Abdomen | Collagen | Infertility | Liquid oxygen | Abdominal wall | Diaphragm | Progesterone | In vitro methods and tests | Nanotechnology | Steroid hormones | Steroids | Peritoneum
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2017, Volume 12, Issue 9, p. e0184302
Background Progressive fibrous thickening of the peritoneal membrane is a complication of long-term peritoneal dialysis (PD). TGF-beta/Smad pathway activation,... 
TO-MESENCHYMAL TRANSITION | HISTONE DEACETYLASE INHIBITION | ACETYLATION | MULTIDISCIPLINARY SCIENCES | HEMODIALYSIS-PATIENTS | SUPPRESSES | MODEL | TGF-BETA-1 | Inflammation - pathology | Rats, Wistar | Bone Morphogenetic Protein 7 - genetics | Cell Count | Peritoneal Fibrosis - genetics | Peritoneum - drug effects | Body Weight - drug effects | Male | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Valproic Acid - pharmacology | Myofibroblasts - metabolism | Peritoneal Fibrosis - drug therapy | Peritoneum - pathology | Valproic Acid - therapeutic use | Biological Transport - drug effects | Capillaries - metabolism | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | Myofibroblasts - pathology | Inflammation Mediators | Cytokines - metabolism | RNA, Messenger - genetics | Peritoneal Fibrosis - pathology | Treatment Outcome | Peritoneal Fibrosis - chemically induced | Myofibroblasts - drug effects | Fibronectins - metabolism | Gene Expression Regulation - drug effects | Animals | Transforming Growth Factor beta - genetics | Signal Transduction - drug effects | Bone Morphogenetic Protein 7 - metabolism | Fibronectins - genetics | Peritoneum - metabolism | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | Neovascularization, Physiologic | Calcium-Binding Proteins - genetics | Divalproex | Treatment outcome | Analysis | Fibrosis | Development and progression | Dosage and administration | Research | Valproic acid | Drug therapy | Immunohistochemistry | Multiplexing | Drugs | Chromatin | Nephrology | Smad protein | Laboratories | Smad3 protein | Medical schools | Peritoneal dialysis | Fibronectin | Proteins | Signal transduction | Ultrafiltration | Rodents | Interleukin 1 | Histones | Chlorhexidine | Acetylation | Thickening | Cytokines | Complications | Mortality | Rats | Inflammation | Gene expression | Studies | Acids | Tumor necrosis factor | Deacetylation | Smad7 protein | Dialysis | Kidney diseases | Immunofluorescence | Monocyte chemoattractant protein 1 | Peritoneum
Journal Article