X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (4789) 4789
Book Review (417) 417
Publication (405) 405
Newsletter (30) 30
Book Chapter (26) 26
Conference Proceeding (6) 6
Book / eBook (5) 5
Data Set (1) 1
Dissertation (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (4454) 4454
animals (2734) 2734
humans (2458) 2458
male (2228) 2228
female (1623) 1623
mice (1422) 1422
peritonitis (1353) 1353
peritonitis - metabolism (1224) 1224
middle aged (952) 952
rats (867) 867
adult (761) 761
inflammation (750) 750
abridged index medicus (684) 684
disease models, animal (672) 672
aged (664) 664
peritonitis - immunology (649) 649
peritonitis - etiology (638) 638
peritonitis - drug therapy (620) 620
immunology (617) 617
peritonitis - pathology (573) 573
sepsis (567) 567
mice, inbred c57bl (561) 561
peritonitis - microbiology (530) 530
surgery (494) 494
peritonitis - chemically induced (466) 466
urology & nephrology (417) 417
time factors (365) 365
peritonitis - physiopathology (351) 351
macrophages (330) 330
mice, knockout (329) 329
expression (326) 326
peritoneal dialysis (326) 326
spontaneous bacterial peritonitis (318) 318
cytokines (313) 313
peritonitis - complications (309) 309
cells, cultured (307) 307
rats, wistar (304) 304
gastroenterology & hepatology (291) 291
pharmacology & pharmacy (281) 281
peritoneum - metabolism (277) 277
septic shock (266) 266
analysis (264) 264
cell biology (260) 260
peritonitis - blood (257) 257
mortality (256) 256
hematology (249) 249
acute disease (248) 248
infection (245) 245
adolescent (237) 237
health aspects (234) 234
research (234) 234
cytokines - metabolism (232) 232
peritoneal dialysis, continuous ambulatory - adverse effects (227) 227
neutrophils (223) 223
tumor-necrosis-factor (221) 221
peritonitis - therapy (217) 217
rats, sprague-dawley (217) 217
peritoneal dialysis - adverse effects (216) 216
microbiology (211) 211
neutrophils - metabolism (211) 211
tumor necrosis factor-alpha - metabolism (210) 210
activation (208) 208
cells (206) 206
biochemistry & molecular biology (203) 203
ascitic fluid - metabolism (202) 202
rodents (199) 199
critical care medicine (198) 198
infections (198) 198
macrophages - metabolism (196) 196
prospective studies (196) 196
kidney failure, chronic - therapy (195) 195
mice, inbred balb c (193) 193
neutrophils - immunology (193) 193
infectious diseases (190) 190
cirrhosis (186) 186
in-vivo (182) 182
medicine, research & experimental (182) 182
peritonitis - prevention & control (182) 182
risk factors (180) 180
peritonitis - diagnosis (179) 179
research article (174) 174
liver - metabolism (171) 171
multidisciplinary sciences (169) 169
proteins (169) 169
peritonitis - mortality (165) 165
prognosis (165) 165
ascites (164) 164
medicine & public health (163) 163
apoptosis (161) 161
sepsis - metabolism (157) 157
disease (155) 155
liver cirrhosis - complications (155) 155
sepsis - immunology (155) 155
bacteria (153) 153
liver cirrhosis (153) 153
article (151) 151
care and treatment (151) 151
peritoneum - pathology (151) 151
physiological aspects (151) 151
nitric-oxide (150) 150
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (4306) 4306
Russian (279) 279
Japanese (71) 71
German (53) 53
French (24) 24
Chinese (22) 22
Polish (18) 18
Spanish (17) 17
Ukrainian (12) 12
Italian (10) 10
Bulgarian (5) 5
Czech (3) 3
Hungarian (3) 3
Portuguese (3) 3
Croatian (2) 2
Korean (2) 2
Serbian (2) 2
Turkish (2) 2
Dutch (1) 1
Romanian (1) 1
Slovak (1) 1
Swedish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Nature, ISSN 0028-0836, 08/2012, Volume 488, Issue 7413, pp. 670 - 674
The inflammasome regulates the release of caspase activation-dependent cytokines, including interleukin (IL)-1 beta, IL-18 and high-mobility group box 1... 
PATHWAYS | APOPTOSIS | ACID | PROTEIN-KINASE PKR | MULTIDISCIPLINARY SCIENCES | INFECTION | NLRP3 INFLAMMASOME | ELF2-ALPHA | STRESS | BINDING | SUBSTRATE RECOGNITION | Crystallins - metabolism | Interleukin-6 - analysis | Phosphorylation | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | eIF-2 Kinase - metabolism | Humans | Salmonella Infections - immunology | Male | Salmonella Infections - metabolism | HMGB1 Protein - secretion | Interleukin-1beta - blood | eIF-2 Kinase - deficiency | Adenosine Triphosphate - pharmacology | CARD Signaling Adaptor Proteins - metabolism | Transfection | Interleukin-6 - blood | Rotenone - pharmacology | Female | Membrane Proteins - metabolism | Escherichia coli - physiology | Macrophages, Peritoneal - drug effects | eIF-2 Kinase - genetics | Calcium-Binding Proteins - metabolism | Cell Line | Salmonella typhimurium - physiology | Interleukin-18 - blood | Salmonella typhimurium - immunology | Inflammasomes - agonists | Mice, Inbred C57BL | Peritonitis - metabolism | Cells, Cultured | Escherichia coli - immunology | Antigens, Bacterial - pharmacology | Escherichia coli Infections - metabolism | eIF-2 Kinase - antagonists & inhibitors | Apoptosis Regulatory Proteins - metabolism | Animals | Carrier Proteins - metabolism | Bacterial Toxins - pharmacology | Uric Acid - pharmacology | RNA, Double-Stranded - immunology | RNA, Double-Stranded - pharmacology | Escherichia coli Infections - immunology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Macrophages, Peritoneal - metabolism | HMGB1 Protein - blood | Physiological aspects | Chromosomal proteins | Research | Cytokines | Aluminum | Health aspects | Proteins | E coli | Pathogenesis | Plasmids | Bone marrow | Bacteria | Kinases | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 03/2006, Volume 440, Issue 7081, pp. 237 - 241
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2015, Volume 5, Issue 1, pp. 17935 - 17935
Inflammasomes are cytoplasmic, multiprotein complexes that trigger caspase-1 activation and IL-1 beta maturation in response to diverse stimuli. Although... 
ASC | OXYGEN SPECIES PRODUCTION | STIMULATION | CASPASE-1 | MULTIDISCIPLINARY SCIENCES | SALMONELLA | IL-1-BETA | UNCOUPLING PROTEIN-2 | NALP3 INFLAMMASOME | CELL-DEATH | BETA | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Ion Channels - genetics | Caspase 1 - metabolism | Mitochondrial Proteins - genetics | Lipopolysaccharides - immunology | Autophagy - drug effects | Mitochondrial Proteins - metabolism | Flagellin - metabolism | Macrophages - immunology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Peritonitis - etiology | Flagellin - immunology | Pneumonia - etiology | Cytokines - metabolism | Macrophage Activation - immunology | Apoptosis Regulatory Proteins - chemistry | Peritonitis - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Ion Channels - metabolism | Iridoids - pharmacology | CARD Signaling Adaptor Proteins | Chemokines - metabolism | Mice | Uncoupling Protein 2 | Pneumonia - metabolism | Protein Multimerization - drug effects | Mitochondrial uncoupling protein 2 | Reactive oxygen species | Oligomerization | Lung | Neutrophils | Aluminum sulfate | Caspase | Flagellin | IL-1β | Autophagy | Inflammatory diseases | Caspase-1 | Lipopolysaccharides | Genipin | Peritonitis | Phagocytosis | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 01/2009, Volume 206, Issue 1, pp. 15 - 23
The endogenous cellular and molecular mechanisms that control acute inflammation and its resolution are of wide interest. Using self-resolving inflammatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | RESOLVINS | DOCOSATRIENES | DOCOSAHEXAENOIC ACID | INFLAMMATION | OMEGA-3-FATTY-ACIDS | RESOLUTION | ATHEROSCLEROSIS | LIPID MEDIATORS | IMMUNOLOGY | NEUROPROTECTIN-D1 | Leukocytes, Mononuclear - metabolism | Neutrophils - cytology | Stereoisomerism | Eicosapentaenoic Acid - analogs & derivatives | Humans | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Macrophages, Peritoneal - cytology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Lipoxygenase - metabolism | Autacoids - metabolism | Eicosapentaenoic Acid - pharmacology | Inflammation Mediators - pharmacology | Inflammation Mediators - metabolism | Autacoids - pharmacology | Molecular Structure | Anti-Inflammatory Agents, Non-Steroidal - metabolism | Macrophages, Peritoneal - drug effects | Neutrophils - metabolism | Dinoprostone - pharmacology | Phagocytosis - drug effects | Leukocytes, Mononuclear - drug effects | Docosahexaenoic Acids - chemistry | Neutrophils - drug effects | Peritonitis - metabolism | Peritonitis - chemically induced | Macrophages - cytology | Autacoids - chemistry | Mice, Inbred Strains | Cell Movement - drug effects | Docosahexaenoic Acids - pharmacology | Macrophages - metabolism | Animals | Lipid Metabolism - drug effects | Lipoxygenase - genetics | Inflammation Mediators - chemistry | Leukocytes, Mononuclear - cytology | Macrophages - drug effects | Zymosan - toxicity | Mice | Macrophages, Peritoneal - metabolism | Docosahexaenoic Acids - metabolism | Index Medicus | Brief Definitive Reports
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 6, pp. 1037 - 1048
Resolution of inflammation is now recognized as a biosynthetically active process involving pro-resolving mediators. Here, we show in zymosan-initiated... 
CIRCUITS | MEDICINE, RESEARCH & EXPERIMENTAL | PROTECTINS | CHEMOTAXIS | INHIBITS LEUKOCYTE MIGRATION | NETRIN-1 | ATHEROSCLEROSIS | MECHANISMS | IMMUNOLOGY | RECEPTORS | HUMAN PHAGOCYTES | PRORESOLVING LIPID MEDIATORS | Netrin-1 | Humans | Lipids - immunology | Monocytes - metabolism | Monocytes - immunology | Nerve Growth Factors - metabolism | Docosahexaenoic Acids - immunology | Inflammation - metabolism | Lipids - chemistry | Tumor Suppressor Proteins - genetics | Inflammation Mediators - metabolism | Chemotaxis - immunology | Neutrophils - metabolism | Macrophages - immunology | Inflammation Mediators - immunology | Recombinant Proteins - metabolism | Zymosan | Tumor Suppressor Proteins - metabolism | Vagus Nerve - metabolism | Mice, Inbred C57BL | Neutrophils - drug effects | Peritonitis - metabolism | Cells, Cultured | Lipoxins - immunology | Neutrophils - immunology | Vagus Nerve - surgery | Inflammation - immunology | Nerve Growth Factors - immunology | Peritonitis - chemically induced | Chemotaxis - drug effects | Peritonitis - immunology | Recombinant Proteins - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Tumor Suppressor Proteins - immunology | Vagotomy | Blotting, Western | Mice, Knockout | Macrophages - metabolism | Animals | Recombinant Proteins - immunology | Nerve Growth Factors - genetics | Vagus Nerve - immunology | Inflammation - genetics | Mice | Lipoxins - metabolism | Microscopy, Fluorescence | Docosahexaenoic Acids - metabolism | Index Medicus | 303
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 01/2017, Volume 26, Issue 1, pp. 28 - 43
Journal Article
Scientific Reports, ISSN 2045-2322, 03/2016, Volume 6, Issue 1, pp. 23240 - 23240
Sepsis is the principal cause of fatality in the intensive care units worldwide. It involves uncontrolled inflammatory response resulting in multi-organ... 
CELLS | SHOCK | ACTIVATION | RECOGNITION | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | IL-10 PRODUCTION | PROINFLAMMATORY CYTOKINES | INTERVENTION | SESQUITERPENE LACTONES | SEVERE SEPSIS | Tumor Necrosis Factor-alpha - metabolism | Sepsis - etiology | Sepsis - prevention & control | Apoptosis - drug effects | Monocytes - cytology | Humans | Monocytes - metabolism | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Interleukin-1beta - metabolism | Interleukin-10 - metabolism | Female | Chemokine CCL2 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Cell Line | Sesquiterpenes, Guaiane - pharmacology | Lung - pathology | Lipopolysaccharides - toxicity | Anti-Inflammatory Agents - pharmacology | Liver - metabolism | Mice, Inbred C57BL | Macrophages - cytology | Toll-Like Receptor 4 - metabolism | Monocytes - drug effects | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Macrophages - drug effects | RAW 264.7 Cells | Cell Proliferation - drug effects | Mice | Mitogen-Activated Protein Kinases - metabolism | Liver | AKT protein | Arthritis | Kinases | Macrophages | Immunosuppressive agents | Lipopolysaccharides | Interleukin 6 | Intensive care units | Cell activation | Intestine | Rodents | Gram-negative bacteria | Peritonitis | Dendritic cells | Secretion | Cell walls | MAP kinase | 1-Phosphatidylinositol 3-kinase | Dextran | Monocytes | Protein kinase | Interleukin 10 | Sepsis | Interferon | Colitis | Monocyte chemoattractant protein 1 | Cancer | Index Medicus
Journal Article
SHOCK, ISSN 1073-2322, 04/2017, Volume 47, Issue 4, pp. 463 - 473
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 04/2018, Volume 217, Issue 4, pp. 1503 - 1519
In ischemic vascular diseases, leukocyte recruitment and polarization are crucial for revascularization and tissue repair. We investigated the role of... 
MIGRATION | CELLS | ACTIVATION | ARTERY GROWTH ARTERIOGENESIS | PHOSPHORYLATION | DYNAMICS | ACTIN CYTOSKELETON | ENA/VASP PROTEINS | MACROPHAGE POLARIZATION | CCR2 | CELL BIOLOGY | Chemotaxis, Leukocyte | Cell Adhesion Molecules - genetics | Hindlimb | STAT Transcription Factors - metabolism | beta-Arrestin 2 - metabolism | Ischemia - genetics | Phosphoproteins - metabolism | Peritonitis - pathology | Time Factors | Peritonitis - genetics | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Ischemia - pathology | Disease Models, Animal | Muscle, Skeletal - blood supply | Actin Cytoskeleton - metabolism | Microfilament Proteins - deficiency | Signal Transduction | Peritonitis - metabolism | Cell Adhesion Molecules - deficiency | Ischemia - metabolism | Neuropeptides - metabolism | Ischemia - physiopathology | Phosphoproteins - genetics | Cell Adhesion Molecules - metabolism | Mice, Knockout | Protein Transport | Macrophages - metabolism | Phenotype | Receptors, CCR2 - metabolism | Animals | Phosphoproteins - deficiency | Peritonitis - physiopathology | Chemokines - metabolism | Leukocytes - metabolism | Neovascularization, Physiologic | rac1 GTP-Binding Protein - metabolism | CC chemokine receptors | Polarization | Transcription | Arrestin | Leukocytes | Macrophages | Recovery | Blood | Recruitment | Leucocytes | Angiogenesis | Immunology | Vascular diseases | Ischemia | CCR2 protein | Clonal deletion | Deletion | Bone marrow | Repair | Protein transport | Growth factors | Cytokines | Muscles | Leukocytes (neutrophilic) | Chemotaxis | Vasodilator-stimulated phosphoprotein | Blood flow | Bone blood flow | White blood cells | Monocytes | Internalization | Infiltration | Mice | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Journal Article
Cellular Immunology, ISSN 0008-8749, 2012, Volume 272, Issue 2, pp. 251 - 258
► TREM-1 is expressed on neutrophils and macrophages but not on lymphocytes. ► Blockade of TREM-1 signaling significantly prolongs survival of mice with... 
TREM-1 | Pseudomonas aeruginosa | Macrophages | Bacterial sepsis | RECOGNITION | INFLAMMATION | HOST-DEFENSE | INNATE | Pseudomonos aeruginosa | IMMUNOLOGY | NF-KAPPA-B | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | B7-2 Antigen - genetics | Pseudomonas aeruginosa - isolation & purification | Membrane Glycoproteins - metabolism | Recombinant Fusion Proteins - pharmacology | Male | Interferon-gamma - metabolism | Recombinant Fusion Proteins - metabolism | Receptors, Immunologic - antagonists & inhibitors | Bacteremia - microbiology | Pseudomonas Infections - metabolism | Membrane Glycoproteins - antagonists & inhibitors | Interleukin-1beta - metabolism | Peritonitis - genetics | Inflammation Mediators - metabolism | CD40 Antigens - genetics | Chemokine CCL2 - metabolism | Neutrophils - metabolism | Lymphocytes - metabolism | Signal Transduction | Peritonitis - metabolism | Pseudomonas Infections - therapy | Bacteremia - genetics | Membrane Glycoproteins - genetics | Pseudomonas Infections - genetics | Bacteremia - metabolism | Animals | Triggering Receptor Expressed on Myeloid Cells-1 | Blood Platelets - metabolism | Recombinant Fusion Proteins - genetics | Mice | Mice, Inbred BALB C | Macrophages, Peritoneal - metabolism | Receptors, Immunologic - genetics | Leukocytes - metabolism | Phagocytosis | Receptors, Immunologic - metabolism | Index Medicus
Journal Article