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The Journal of Immunology, ISSN 0022-1767, 12/2008, Volume 181, Issue 12, pp. 8677 - 8687
Resolution of inflammation is essential. Although supplementation of omega-3 fatty acids is widely used, their availability at sites of inflammation is not... 
RHEUMATOID-ARTHRITIS | ANTIINFLAMMATORY PROPERTIES | MYOCARDIAL-INFARCTION | SUDDEN-DEATH | DOCOSAHEXAENOIC ACID | HUMANS | POLYUNSATURATED FATTY-ACIDS | HUMAN BLOOD | LIPID MEDIATORS | IMMUNOLOGY | ALPHA-LINOLENIC ACID | Inflammation Mediators - administration & dosage | Eicosapentaenoic Acid - analogs & derivatives | Humans | Ascitic Fluid - pathology | Male | Eicosapentaenoic Acid - administration & dosage | Anti-Inflammatory Agents, Non-Steroidal - blood | Cell Migration Inhibition | Exudates and Transudates - metabolism | Peritonitis - pathology | Ascitic Fluid - metabolism | Time Factors | Inflammation Mediators - metabolism | Peritonitis - blood | Anti-Inflammatory Agents, Non-Steroidal - metabolism | Neutrophils - metabolism | Fatty Acids, Omega-3 - administration & dosage | Neutrophils - pathology | Ascitic Fluid - immunology | Fatty Acids, Omega-3 - metabolism | Eicosapentaenoic Acid - metabolism | Inflammation Mediators - blood | Neutrophils - immunology | Exudates and Transudates - chemistry | Peritonitis - immunology | Docosahexaenoic Acids - administration & dosage | Mice, Inbred Strains | Exudates and Transudates - immunology | Eicosapentaenoic Acid - blood | Animals | Diffusion Chambers, Culture | Docosahexaenoic Acids - blood | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | Fatty Acids, Omega-3 - blood | Mice | Docosahexaenoic Acids - metabolism | lipid mediators | inflammation | neutrophils
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 07/2017, Volume 102, Issue 1, pp. 7 - 17
Multiple cathepsins promote particle‐induced cell death independently of inflammasomes, suggests that cathepsin inhibitors may therapeutically target multiple... 
pyroptosis | macrophage | inflammation | peritonitis | caspase‐1 | Inflammation | Pyroptosis | Peritonitis | Caspase-1 | Macrophage | HEPATOCYTE APOPTOSIS | IMMUNE-RESPONSE | NLRP3 INFLAMMASOME | DEFICIENT MICE | IMMUNOLOGY | NALP3 INFLAMMASOME | CELL BIOLOGY | CHOLESTEROL CRYSTALS | DANGER SIGNAL | LYSOSOMAL MEMBRANE PERMEABILIZATION | DYING CELLS | HEMATOLOGY | caspase-1 | CONVERTING-ENZYME | Inflammasomes - metabolism | Macrophages - pathology | Apoptosis - drug effects | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Cathepsin B - genetics | Particulate Matter - adverse effects | Peritonitis - metabolism | Caspase 1 - metabolism | Cathepsin B - metabolism | Peritonitis - chemically induced | Interleukin-1beta - genetics | Inflammasomes - genetics | Mice, Knockout | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Peritonitis - pathology | Macrophages - metabolism | Animals | Cathepsins - genetics | Caspase 1 - genetics | Cathepsins - metabolism | Interleukin-1beta - metabolism | Particulate Matter - pharmacology | Peritonitis - genetics | Mice | Secretion | Mortality | Lysosomes | Particulates | Caspase | Cathepsins | Macrophages | Cytosol | Inflammatory diseases | Nigericin | Inhibitors | Cell death | Interleukin 1 | Cathepsin B | Phagocytosis | Apoptosis | Spotlight on Leading Edge Research
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 9, pp. 5823 - 5829
Macrophages play a crucial role in the innate immune response against the human pathogen Streptococcus pyogenes, yet the innate immune response against the... 
GROUP-A-STREPTOCOCCUS | PATTERN-RECOGNITION | INTERLEUKIN-1-BETA | INFECTION | SECRETION | IMMUNOLOGY | CASPASE-1 ACTIVATION | ATP | IPAF | NALP3 INFLAMMASOME | INNATE IMMUNITY | NLR Family, Pyrin Domain-Containing 3 Protein | Streptococcal Infections - metabolism | Protein Precursors - secretion | Caspase 1 - metabolism | Multiprotein Complexes - genetics | NF-kappa B - metabolism | Streptococcal Infections - mortality | Streptococcus pyogenes - genetics | Multiprotein Complexes - metabolism | Signal Transduction - immunology | Streptolysins - physiology | Macrophages - secretion | Peritonitis - microbiology | Streptolysins - deficiency | Receptors, Purinergic P2 - genetics | Inflammation Mediators - metabolism | Toll-Like Receptors - physiology | Interleukin-1beta - biosynthesis | Inflammation Mediators - physiology | Macrophages - immunology | Disease Models, Animal | Carrier Proteins - physiology | Interleukin-1beta - secretion | Streptolysins - metabolism | Macrophages - pathology | Streptococcus pyogenes - immunology | Mice, Inbred C57BL | Cells, Cultured | Receptors, Purinergic P2 - physiology | Signal Transduction - genetics | Peritonitis - immunology | Bacterial Proteins - physiology | Peritonitis - mortality | Mice, Knockout | Multiprotein Complexes - physiology | Receptors, Purinergic P2X7 | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Protein Precursors - biosynthesis | NF-kappa B - physiology | Survival Analysis | Bacterial Proteins - metabolism | Mice | Streptococcal Infections - immunology | Receptors, Purinergic P2 - deficiency
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e62792 - e62792
Background: Patients admitted to the intensive care unit with alcohol use disorders have increased morbidity and mortality. The purpose of this study was to... 
BARRIER DYSFUNCTION | INCREASED INTESTINAL PERMEABILITY | CHRONIC ETHANOL INGESTION | CECAL LIGATION | PSEUDOMONAS-AERUGINOSA PNEUMONIA | MULTIDISCIPLINARY SCIENCES | ACUTE LUNG INJURY | CRITICALLY-ILL | EPITHELIAL APOPTOSIS | INTENSIVE-CARE | RESPIRATORY-DISTRESS-SYNDROME | Intestines - drug effects | Metabolomics | Intestinal Mucosa - metabolism | Apoptosis - drug effects | Male | Intestines - metabolism | Intestinal Mucosa - drug effects | Intestines - immunology | Occludin - metabolism | Immunity - drug effects | Liver - immunology | Liver - drug effects | Intestinal Mucosa - immunology | Sepsis - metabolism | Ethanol - adverse effects | Peritonitis - blood | Zonula Occludens-1 Protein - metabolism | Cytokines - blood | Tight Junctions - drug effects | Tight Junctions - metabolism | Permeability - drug effects | Sepsis - immunology | Liver - metabolism | Peritonitis - metabolism | Immunity - immunology | Pancreas - drug effects | Pancreas - metabolism | Peritonitis - immunology | Pancreas - immunology | Alcohol Drinking - adverse effects | Peritonitis - mortality | Sepsis - mortality | Animals | Tight Junctions - immunology | Mice | Sepsis - blood | Peroxidase - metabolism | Infection | Phosphates | Killer cells | Drinking of alcoholic beverages | Permeability | T cells | Creatine | Health aspects | Apoptosis | CD8 antigen | Liver | Alcohol | Betaine | Surgery | Bone marrow | Peritonitis | Spleen | Cytokines | Ethanol | Mortality | Cecum | Cell division | Respiratory distress syndrome | γ-Interferon | Sepsis | Acetic acid | Animal models | Intensive care | Critical care | Lymphocytes T | Biology | Ingestion | Alcohol use | Granulocyte colony-stimulating factor | Lymphocytes | Intestine | Rodents | Tumor necrosis factor-TNF | Pancreas | Immune system | Adenosine | Immune response | Histology | Bacteremia | Morbidity | CD4 antigen | Medicine | Lungs | Choline | Interferon | Bone | Laboratory animals | Uracil | Peritoneum | Integrity | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2014, Volume 289, Issue 1, pp. 41 - 52
Journal Article
Immunology, ISSN 0019-2805, 01/2018, Volume 153, Issue 1, pp. 71 - 83
The Nod‐like receptor protein 3 ( NLRP 3) inflammasome plays roles in host defence against invading pathogens and in the development of autoimmune damage.... 
NLRP | macrophage | 3 inflammasome | Tim‐3 | NLRP3 inflammasome | Tim-3 | HOMEOSTASIS | INHIBITION | IMMUNE-RESPONSES | MACROPHAGES | DISEASE | SECRETION | IMMUNOLOGY | SUPPRESSION | EXPRESSION | Potassium - metabolism | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Hepatitis A Virus Cellular Receptor 2 - metabolism | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Adenosine Triphosphate - biosynthesis | Case-Control Studies | Young Adult | Peritonitis - pathology | Interleukin-1beta - metabolism | Adult | Female | Caspase 1 | Protein Interaction Domains and Motifs | Macrophages - immunology | Disease Models, Animal | Hepatitis A Virus Cellular Receptor 2 - chemistry | Cell Line | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Peritonitis - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Peritonitis - immunology | Macrophages - metabolism | Animals | Inflammasomes - immunology | Protein Binding | Mice | Interleukins | Genetic engineering | T cells | Macrophages | Mitogens | Peritonitis | Efflux | Immunoglobulins | Reactive oxygen species | Interleukin | Aluminum sulfate | Transgenic | Priming | Lymphocytes T | Activation | Cell activation | Mucin | Immune checkpoint | Rodents | Interleukin 1 | Alum | ATP | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2014, Volume 9, Issue 11, p. e113480
Objective: Inflammation and its resolution are central to vascular injury and repair. Maresins comprise a new family of bioactive lipid mediators synthesized... 
ADHESION MOLECULE-1 | OXIDATIVE STRESS | MULTIDISCIPLINARY SCIENCES | E-SELECTIN | TRANSCRIPTION | NECROSIS-FACTOR-ALPHA | NF-KAPPA-B | HUMAN PHAGOCYTES | EXPRESSION | MICROARRAY ANALYSIS | CAMP | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Endothelial Cells - metabolism | Humans | Cells, Cultured | NADPH Oxidases - metabolism | E-Selectin - metabolism | NF-kappa B - metabolism | Cell Adhesion - drug effects | Down-Regulation - drug effects | Muscle, Smooth, Vascular - cytology | Up-Regulation - drug effects | Docosahexaenoic Acids - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Endothelial Cells - cytology | U937 Cells | Oxidative Stress - drug effects | Cyclic AMP - metabolism | Endothelial Cells - drug effects | Muscle, Smooth, Vascular - drug effects | Oxidative stress | Animal models | Angioplasty | Epithelial cells | Gene regulation | Homeostasis | Intracellular signalling | Smooth muscle | Lipids | Kinases | Macrophages | Docosahexaenoic acid | Cell surface | NAD(P)H oxidase | Cell adhesion & migration | Signal transduction | Cell activation | NOX4 protein | Pathways | Surgery | Penicillin | CYBB protein | Attenuation | Peritonitis | Translocation | Edema | NF-κB protein | AMP | Neutrophils | Muscles | Cyclic AMP | Cultures | Leukocytes (neutrophilic) | Inflammation | Fatty acids | Adhesion | Biological activity | Nuclear transport | Endothelial cells | Studies | E-selectin | Monocytes | Intracellular | Colitis | Alzheimers disease | Human behavior
Journal Article