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Journal of Emergency Medicine, ISSN 0736-4679, 2016, Volume 50, Issue 5, pp. e231 - e236
Journal Article
PLoS Medicine, ISSN 1549-1277, 11/2012, Volume 9, Issue 11, p. e1001338
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 07/2012, Volume 303, Issue 1, pp. L20 - L32
Fisher BJ, Kraskauskas D, Martin EJ, Farkas D, Wegelin JA, Brophy D, Ward KR, Voelkel NF, Fowler AA 3rd, Natarajan R. Mechanisms of attenuation of abdominal... 
Epithelial ion channels | Coagulopathy | Barrier integrity | VITAMIN-C | OXIDATIVE STRESS | PHYSIOLOGY | NITRIC-OXIDE SYNTHASE | epithelial ion channels | coagulopathy | MULTIPLE ORGAN FAILURE | PULMONARY-EDEMA | TIGHT JUNCTIONS | RESPIRATORY SYSTEM | DISSEMINATED INTRAVASCULAR COAGULATION | ENDOTHELIAL-CELLS | SEPTIC SHOCK | barrier integrity | FACTOR PATHWAY INHIBITOR | Respiratory Mucosa - drug effects | Humans | Male | Bronchoalveolar Lavage - methods | Inflammation - blood | Sepsis - drug therapy | Inflammation - metabolism | Peritonitis - microbiology | Sepsis - metabolism | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - drug effects | Cytoskeletal Proteins - metabolism | Ion Transport - drug effects | Respiratory Mucosa - physiopathology | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Lung - metabolism | Pulmonary Alveoli - physiopathology | Neutrophils - metabolism | Cell Line | Acute Lung Injury - physiopathology | Permeability - drug effects | Abdomen - microbiology | Mice, Inbred C57BL | Neutrophils - drug effects | Peritonitis - metabolism | Neutrophils - physiology | Lung - physiopathology | Biomarkers - blood | Sepsis - physiopathology | Sodium-Potassium-Exchanging ATPase - metabolism | Acute Lung Injury - microbiology | Animals | Blood Coagulation - drug effects | Ion Channels - metabolism | Lung - drug effects | Abdomen - pathology | Ascorbic Acid - pharmacology | Peritonitis - physiopathology | Mice | Sepsis - blood | Peritonitis - drug therapy | Respiratory Mucosa - metabolism | Inflammation - physiopathology | Complications and side effects | Bacterial infections | Lung diseases | Vitamin C | Research | Health aspects | Risk factors
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 7, pp. 1680 - 1690.e1
Background & Aims Nonselective β blockers (NSBBs) reduce portal pressure and the risk for variceal hemorrhage in patients with cirrhosis. However, development... 
Gastroenterology and Hepatology | Liver Fibrosis | β Blocker | Bacterial Infection | Mortality | Adrenergic Receptor | β Blocker | SURVIVAL | ACUTE KIDNEY INJURY | beta Blocker | PROPRANOLOL | INFECTIONS | REFRACTORY ASCITES | LIVER-DISEASE | THERAPY | DYSFUNCTION | GASTROENTEROLOGY & HEPATOLOGY | PORTAL-HYPERTENSION | Liver Transplantation | Hypertension, Portal - physiopathology | Humans | Middle Aged | Liver Cirrhosis - mortality | Peritonitis - complications | Male | Hepatorenal Syndrome - diagnosis | Peritonitis - diagnosis | Hepatorenal Syndrome - physiopathology | Hypertension, Portal - diagnosis | Hypertension, Portal - mortality | Austria | Peritonitis - microbiology | Hypertension, Portal - etiology | Time Factors | Adrenergic beta-Antagonists - adverse effects | Female | Retrospective Studies | Acute Kidney Injury - etiology | Length of Stay | Liver Cirrhosis - drug therapy | Liver Cirrhosis - diagnosis | Liver Cirrhosis - complications | Hepatorenal Syndrome - etiology | Risk Factors | Kaplan-Meier Estimate | Proportional Hazards Models | Treatment Outcome | Chi-Square Distribution | Hepatorenal Syndrome - mortality | Peritonitis - mortality | Hypertension, Portal - drug therapy | Disease-Free Survival | Acute Kidney Injury - mortality | Peritonitis - physiopathology | Aged | Hemodynamics - drug effects | Liver Cirrhosis - physiopathology | Paracentesis
Journal Article
世界胃肠病学杂志:英文版, ISSN 1007-9327, 2012, Volume 18, Issue 24, pp. 3035 - 3049
Journal Article
Journal Article
Journal Article
The Journal of Pathology, ISSN 0022-3417, 10/2017, Volume 243, Issue 2, pp. 137 - 147
The peritoneum defines a confined microenvironment, which is stable under normal conditions, but is exposed to the damaging effect of infections, surgical... 
peritoneal adhesions | sterile inflammation | macrophages | angiogenesis | autoimmune serositis | endometriosis | fibrosis | peritoneal carcinomatosis | OXIDATIVE STRESS | BONE-MARROW | POSTOPERATIVE ADHESIONS | PATHOLOGY | IRON OVERLOAD | HUMAN MESOTHELIAL CELLS | ONCOLOGY | MOUSE MODEL | NITRIC-OXIDE | TUMOR-ASSOCIATED MACROPHAGES | ENDOTHELIAL GROWTH-FACTOR | Peritoneal Diseases - etiology | Peritonitis - etiology | Peritoneal Neoplasms - etiology | Humans | Peritoneum - physiology | Peritoneal Fibrosis - physiopathology | Autoimmune Diseases - etiology | Endometriosis - immunology | Peritoneum - anatomy & histology | Endometriosis - etiology | Peritonitis - pathology | Peritoneal Diseases - physiopathology | Peritoneal Fibrosis - etiology | Serositis - etiology | Immunity, Cellular - physiology | Peritoneum - immunology | Peritonitis - physiopathology | Female | Peritoneal Diseases - immunology | Wound Healing - physiology | Endometriosis - physiopathology | Peritoneal Fibrosis - immunology | Peritoneal Neoplasms - immunology | Peritoneal Neoplasms - physiopathology | Cell proliferation | Intervention | Serositis | Pathogenesis | Activation | Abatement | Macrophages | Immunity | Recruitment | Angiogenesis | Cell activation | Surgery | Damage | Injuries | Endometrium | Immune system | Antigens | Immune response | Autoantigens | Endometriosis | Inflammation | Scars | Stromal cells | Fibrosis | Cancer | Peritoneum
Journal Article