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Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 09/2007, Volume 34, Issue 9, pp. 938 - 945
SUMMARY • In the present review, we addressed studies in humans and rats to determine the role that oxidative stress may play in mediating cardiovascular... 
kidney | aopcynin | F2-isoprostanes | superoxide | tempol | molsidomine | F2‐isoprostanes | Tempol | Aopcynin | Superoxide | Molsidomine | Kidney | PHYSIOLOGY | DIMORPHISM | SPONTANEOUSLY HYPERTENSIVE-RATS | QUANTIFICATION | PROSTAGLANDIN-F2-ALPHA METABOLITE | F-2-ISOPROSTANES | IN-VIVO | PHARMACOLOGY & PHARMACY | VASODILATOR | POSTMENOPAUSAL WOMEN | PROGRESSION | Cardiovascular Diseases - prevention & control | Humans | Kidney - enzymology | NADPH Oxidases - metabolism | Nitric Oxide Synthase - antagonists & inhibitors | Hypertension - drug therapy | Male | Spin Labels | Kidney - metabolism | Cyclic N-Oxides - pharmacology | Acetophenones - pharmacology | Female | Blood Pressure - drug effects | Nitric Oxide Donors - pharmacology | Superoxide Dismutase - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Rats, Inbred SHR | Cardiovascular Diseases - etiology | Glutathione Peroxidase - metabolism | Cardiovascular Diseases - physiopathology | Kidney - drug effects | Cardiovascular Diseases - metabolism | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Antioxidants - pharmacology | Hypertension - physiopathology | Hypertension - metabolism | Catalase - metabolism | Antioxidants - therapeutic use | Animals | Ascorbic Acid - pharmacology | Molsidomine - pharmacology | Sex Factors | Hypertension - complications | Nitric Oxide Synthase - metabolism | Oxidative Stress - drug effects | Nitric Oxide - metabolism | Vitamin E - pharmacology | Kidney, drug effects | NG-Nitroarginine Methyl Ester, pharmacology | Nitric Oxide Synthase, antagonists and inhibitors | Oxidative Stress, drug effects | Ascorbic Acid, pharmacology | Hypertension, drug therapy | Cardiovascular Diseases, etiology | Cardiovascular Diseases, prevention and control | Cyclic N-Oxides, pharmacology | Enzyme Inhibitors, pharmacology | Superoxide Dismutase, metabolism | Nitric Oxide Synthase, metabolism | Hypertension, physiopathology | Antioxidants, pharmacology | Kidney, metabolism | NADPH Oxidase, antagonists and inhibitors | Blood Pressure, drug effects | Catalase, metabolism | Cardiovascular Diseases, metabolism | Hypertension, metabolism | Acetophenones, pharmacology | Vitamin E, pharmacology | Antioxidants, therapeutic use | Kidney, enzymology | Nitric Oxide, metabolism | Molsidomine, pharmacology | NADPH Oxidase, metabolism | Hypertension, complications | Nitric Oxide Donors, pharmacology | Glutathione Peroxidase, metabolism | Cardiovascular Diseases, physiopathology | Hypertension | Oxidative stress | Blood pressure
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2004, Volume 36, Issue 8, pp. 994 - 1010
As rat spermatozoa undergo epididymal maturation, they acquire the ability to exhibit a spontaneous burst of luminol-peroxidase-dependent chemiluminescence... 
Epididymis | Peroxynitrite | Free radicals | Sperm | Nitric oxide | Luminol | Peroxidase | NADPH OXIDASE | nitric oxide | free radicals | LUMINOL CHEMILUMINESCENCE | peroxynitrite | LIPID-PEROXIDATION | peroxidase | NITRIC-OXIDE SYNTHASE | MOUSE SPERMATOZOA | BIOCHEMISTRY & MOLECULAR BIOLOGY | HUMAN SPERM FUNCTION | PROTEIN-TYROSINE PHOSPHORYLATION | epididytnis | sperm | SUPEROXIDE ANION | ENDOCRINOLOGY & METABOLISM | HYDROGEN-PEROXIDE | CELLULAR MECHANISMS | luminol | Catalase - pharmacology | Epididymis - metabolism | Temperature | Reactive Oxygen Species | Uncoupling Agents - pharmacology | Rats, Wistar | Calcium - metabolism | Capsaicin - pharmacology | Nitric Oxide Synthase - antagonists & inhibitors | Male | Spermatozoa - metabolism | Culture Media - metabolism | Acrosome Reaction | Time Factors | Indicators and Reagents - pharmacology | Rotenone - pharmacology | Bicarbonates - chemistry | NADH, NADPH Oxidoreductases - metabolism | Diterpenes - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Deferoxamine - pharmacology | Superoxide Dismutase - pharmacology | Oxidation-Reduction | Enzyme Inhibitors - pharmacology | Rats | Onium Compounds - pharmacology | Arginine - chemistry | Luminol - pharmacology | Microscopy, Confocal | Animals | Iron Chelating Agents - pharmacology | Models, Biological | Acridines - pharmacology | Glucose - chemistry | 4-Chloromercuribenzenesulfonate - pharmacology | Fructose - chemistry | Microscopy, Fluorescence | Peroxidase - metabolism
Journal Article
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 09/2017, Volume 140, pp. 41 - 52
Ferroptosis has recently been identified as a mode of programmed cell death. However, little is yet known about the signaling mechanism. Here, we report that... 
Acute leukemia | Redox | Ferroptosis | Cell death | ROS | APOPTOSIS | OXIDATIVE STRESS | ACTIVATION | NECROPTOSIS | 5-LIPOXYGENASE INHIBITOR | ARACHIDONIC-ACID | PEROXIDATION | ZILEUTON | ANTIOXIDANT | RESISTANCE | PHARMACOLOGY & PHARMACY | Masoprocol - pharmacology | Reactive Oxygen Species - metabolism | Arachidonate 12-Lipoxygenase - metabolism | Glutathione Peroxidase - antagonists & inhibitors | Humans | Arachidonate 12-Lipoxygenase - chemistry | Arachidonate 15-Lipoxygenase - chemistry | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Cyclohexylamines - pharmacology | Lipid Peroxidation - drug effects | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | alpha-Tocopherol - pharmacology | Arachidonate 12-Lipoxygenase - genetics | Fas-Associated Death Domain Protein - genetics | Glutathione Peroxidase - metabolism | Flavanones - pharmacology | Fas-Associated Death Domain Protein - metabolism | Carbolines - antagonists & inhibitors | Antioxidants - pharmacology | Antineoplastic Agents - chemistry | Arachidonate 15-Lipoxygenase - metabolism | Glutathione Peroxidase - genetics | Reactive Oxygen Species - antagonists & inhibitors | Arachidonate 15-Lipoxygenase - genetics | Cell Line, Tumor | Precursor Cell Lymphoblastic Leukemia-Lymphoma - enzymology | Phenylenediamines - pharmacology | Kinetics | Oxidative Stress - drug effects | Lipoxygenase Inhibitors - pharmacology | Carbolines - pharmacology | Biochemistry | Peroxidase | Acute lymphocytic leukemia | Index Medicus
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 02/2014, Volume 67, pp. 366 - 376
Chronic lead exposure induces hypertension affecting endothelial function. We investigated whether low-concentration lead exposure alters blood pressure and... 
Hypertension | Rat aorta | Lead acetate | Free radicals | Vasoconstrictor prostanoids | ROS | Local renin–angiotensin system | Local renin-angiotensin system | SYSTEM | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTATHIONE-PEROXIDASE | BLOOD-PRESSURE | GUANYLATE-CYCLASE | INDUCED HYPERTENSION | SMOOTH-MUSCLE | NITRIC-OXIDE METABOLISM | ENDOTHELIUM | CYCLOOXYGENASE PATHWAYS | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR-DISEASE | Rats, Wistar | Prostaglandins - pharmacology | Male | Aorta - metabolism | Lead Poisoning - metabolism | Phenylephrine - pharmacology | Acetophenones - pharmacology | Vascular Resistance - drug effects | Blood Pressure - drug effects | Aorta - physiopathology | Lead Poisoning - physiopathology | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Superoxide Dismutase - pharmacology | Vasodilator Agents - pharmacology | Cyclooxygenase 2 Inhibitors - pharmacology | Aorta - drug effects | Losartan - pharmacology | Rats | Antioxidants - pharmacology | Vasoconstriction - drug effects | Indomethacin - pharmacology | Animals | Renin-Angiotensin System - drug effects | Vasodilation - drug effects | Nitric Oxide - metabolism | Chronic Disease | COX-2 inhibitors | Prostaglandins | International agencies | Angiotensin | Dosage and administration | Superoxide | Blood pressure | Indomethacin
Journal Article
Progress in Neuropsychopharmacology & Biological Psychiatry, ISSN 0278-5846, 06/2013, Volume 43, pp. 79 - 88
While it is now well established that the independent brain renin–angiotensin system (RAS) has some important central functions besides the vascular ones, the... 
Angiotensin II | Oxidative stress | Memory | RECEPTOR ANTAGONISTS | AT RECEPTOR | PSYCHIATRY | ALZHEIMERS-DISEASE | MOTOR-ACTIVITY | CONVERTING ENZYME-INHIBITORS | DEFICIENT MICE | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | CLINICAL NEUROLOGY | VASCULAR DEMENTIA | PHARMACOLOGY & PHARMACY | CEREBRAL-BLOOD-FLOW | ANTIHYPERTENSIVE TREATMENTS | Memory - drug effects | Captopril - pharmacology | Rats, Wistar | Angiotensin Receptor Antagonists - pharmacology | Male | Hippocampus - drug effects | Angiotensin II Type 1 Receptor Blockers - pharmacology | Angiotensin II Type 2 Receptor Blockers - pharmacology | Lipid Peroxidation - drug effects | Behavior, Animal - drug effects | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Glutathione Peroxidase - metabolism | Angiotensin II - pharmacology | Losartan - pharmacology | Rats | Memory, Short-Term - drug effects | Imidazoles - pharmacology | Psychomotor Performance - drug effects | Maze Learning - drug effects | Hippocampus - metabolism | Animals | Avoidance Learning - drug effects | Pyridines - pharmacology | Oxidative Stress - drug effects | Oxidases | Phosphates | Niacinamide | Brain | Captopril | Amyloid beta-protein | Proline | Superoxide | Antioxidants | Purines | Angiotensin | Alzheimer's disease
Journal Article