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Autophagy, ISSN 1554-8627, 09/2012, Volume 8, Issue 9, pp. 1333 - 1341
Photodynamic therapy (PDT) involves photosensitizing agents that, in the presence of oxygen and light, initiate formation of cytotoxic reactive oxygen species... 
apoptosis | lysosomes | autophagy | subcellular localization | photodynamic therapy | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Lysosomes | Photodynamic therapy | Autophagy | Subcellular localization | Apoptosis | CANCER-CELLS | ACTIVATION | AUTOPHAGIC RESPONSES | WST11 | CELL BIOLOGY | CYTOCHROME-C | BCL-2 | PHOTOSENSITIZER NPE6 | REACTIVE OXYGEN | MEMBRANE PERMEABILIZATION | Apoptosis - drug effects | Apoptosis - radiation effects | Microtubule-Associated Proteins - metabolism | Fluorescence | Phagosomes - radiation effects | Protein Transport - drug effects | Recombinant Fusion Proteins - metabolism | Vacuoles - drug effects | Cell Shape - radiation effects | Lysosomes - radiation effects | Photosensitizing Agents - pharmacology | Cell Nucleus - metabolism | Lysosomes - metabolism | Cell Nucleus - radiation effects | Light | Phagosomes - ultrastructure | Microtubule-Associated Proteins - deficiency | Phagosomes - drug effects | Lysosomes - drug effects | Cell Survival - drug effects | Green Fluorescent Proteins - metabolism | Permeability - drug effects | Subcellular Fractions - drug effects | Bacteriochlorophylls - pharmacology | Porphyrins - pharmacology | Cell Nucleus - ultrastructure | Amines - metabolism | Cell Survival - radiation effects | Subcellular Fractions - metabolism | Cell Shape - drug effects | Vacuoles - radiation effects | Autophagy-Related Protein 7 | Animals | Permeability - radiation effects | Protein Transport - radiation effects | Subcellular Fractions - radiation effects | Bacteriochlorophylls - chemistry | Cell Line, Tumor | Vacuoles - metabolism | Mice | Cell Nucleus - drug effects | Basic Research Paper
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 08/2013, Volume 288, Issue 33, pp. 24247 - 24263
Journal Article
Clinical and experimental immunology, ISSN 1365-2249, 07/2010, Volume 161, Issue 1, pp. 1 - 9
.... The use of some of these drugs has been linked to reactivation TB. In addition to blocking TNF-mediated immune responses, some anti-TNF drugs have been found... 
certolizumab | mycobacteria | etanercept | infliximab | adalimumab | golimumab | Golimumab | Etanercept | Infliximab | Adalimumab | Certolizumab | Mycobacteria | BACILLUS-CALMETTE-GUERIN | INTRACELLULAR MYCOBACTERIUM-TUBERCULOSIS | ACTIVE RHEUMATOID-ARTHRITIS | IMMUNOLOGY | PHAGOSOME MATURATION ARREST | HOST-DEFENSE MECHANISM | REGULATORY T-CELLS | INFLIXIMAB INDUCES APOPTOSIS | FACTOR ANTAGONISTS | ANTI-TNF-ALPHA | CASPASE-DEPENDENT PATHWAY | Apoptosis - drug effects | Humans | Antibodies, Monoclonal - adverse effects | Mycobacterium tuberculosis - immunology | Autophagy - drug effects | Inflammation - complications | Immunity, Cellular - drug effects | Inflammation - drug therapy | T-Lymphocyte Subsets - drug effects | Immunoglobulin G - pharmacology | Dendritic Cells - drug effects | Immunosuppressive Agents - pharmacology | Autoimmune Diseases - drug therapy | Phagosomes - drug effects | Disease Susceptibility | Antibodies, Monoclonal - pharmacology | Autoimmune Diseases - complications | Immunoglobulin G - adverse effects | Tuberculosis - immunology | Animals | Receptors, Tumor Necrosis Factor | Lymphocyte Activation - drug effects | Tumor Necrosis Factor-alpha - physiology | Immunosuppressive Agents - adverse effects | Mice | Macrophage Activation - drug effects | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Mycobacterium tuberculosis - growth & development | Rheumatoid factor | Tuberculosis | Bacterial infections | Rheumatoid arthritis | Biological response modifiers | T cells | Necrosis | Tumors | Interferon gamma | TNF inhibitors | Immune system | Review
Journal Article
The Journal of cell biology, ISSN 1540-8140, 2010, Volume 188, Issue 4, pp. 537 - 546
Journal Article
Autophagy, ISSN 1554-8635, 2014, Volume 7, Issue 11, pp. 1359 - 1370
Cystic fibrosis (CF) is the most common inherited lethal disease of Caucasians which results in multi organ dysfunction. However, 85% of the deaths are due to... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Burkholderia cenocepacia | Cystic fibrosis | Inflammation | Rapamycin | Macrophages | Autophagy | Host-pathogen interaction | host-pathogen interaction | MAMMALIAN TARGET | LISTERIA-MONOCYTOGENES | FRANCISELLA | autophagy | PROTEIN | MATURATION | CONJUGATION SYSTEM | INTRACELLULAR SURVIVAL | CELL BIOLOGY | macrophages | inflammation | cystic fibrosis | INFANTS | CEPACIA COMPLEX | rapamycin | LOWER AIRWAY INFLAMMATION | Phagosomes - microbiology | Burkholderia Infections - complications | Microtubule-Associated Proteins - metabolism | Cystic Fibrosis - pathology | Vacuoles - drug effects | Autophagy - drug effects | Burkholderia Infections - pathology | Burkholderia cenocepacia - ultrastructure | Phagosomes - ultrastructure | Interleukin-1beta - biosynthesis | Autophagy - genetics | Burkholderia Infections - microbiology | Macrophages - microbiology | Disease Models, Animal | Phagosomes - drug effects | Burkholderia cenocepacia - growth & development | Lysosomes - drug effects | Macrophages - ultrastructure | Sirolimus - therapeutic use | Vacuoles - microbiology | Intracellular Space - drug effects | Microbial Viability - drug effects | Cystic Fibrosis - microbiology | Mice, Inbred C57BL | Burkholderia cenocepacia - physiology | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Pneumonia - microbiology | Cystic Fibrosis - complications | Mutation - genetics | Sirolimus - pharmacology | Down-Regulation - genetics | Macrophages - metabolism | Animals | Lysosomes - microbiology | Pneumonia - drug therapy | Intracellular Space - microbiology | Burkholderia cenocepacia - drug effects | Burkholderia Infections - drug therapy | Pneumonia - complications | Macrophages - drug effects | Mice | Cystic Fibrosis - drug therapy | RNA, Small Interfering - metabolism | Translational Research Paper
Journal Article
Autophagy, ISSN 1554-8635, 2014, Volume 8, Issue 9, pp. 1357 - 1370
Emerging evidence points to an important role of autophagy in the immune response mediated by dendritic cells (DC) against Mycobacterium tuberculosis (Mtb).... 
vaccine | autophagy | dendritic cells | Mycobacterium tuberculosis | BCG | RD1 region | ESX1/type VII secretion system | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Dendritic cells | Vaccine | Autophagy | Rd1 region | SYSTEM | MACROPHAGES | VIRULENCE | ESAT-6 SECRETION | CELL BIOLOGY | ADAPTIVE IMMUNITY | PHAGOSOME | IMMUNOGENICITY | GENE-EXPRESSION | T-CELLS | Biomarkers - metabolism | Th1 Cells - drug effects | Humans | Phagosomes - metabolism | Cells, Cultured | Mycobacterium tuberculosis - ultrastructure | Dendritic Cells - pathology | Tuberculosis - microbiology | Th1 Cells - immunology | Mycobacterium tuberculosis - drug effects | Sirolimus - pharmacology | Autophagy - drug effects | Tuberculosis - immunology | Mycobacterium bovis - drug effects | Bacterial Secretion Systems - drug effects | Cell Differentiation - drug effects | Dendritic Cells - ultrastructure | Mycobacterium tuberculosis - physiology | Dendritic Cells - microbiology | Dendritic Cells - drug effects | Phagosomes - ultrastructure | Mycobacterium bovis - physiology | Phagosomes - drug effects | Tuberculosis - pathology | Dendritic Cells | Bacterial Secretion Systems | Sirolimus | Mycobacterium bovis | Phagosomes | Life Sciences | Tuberculosis | Th1 Cells | Biomarkers | Cell Differentiation | ESX1 | Basic Research Paper | type VII secretion system
Journal Article
Cell death & disease, ISSN 2041-4889, 04/2010, Volume 1, Issue 4, pp. e32 - e32
We previously identified a novel estrogen-induced gene, EIG121, as being differentially regulated in endometrioid and nonendometrioid endometrial carcinoma.... 
EIG121 | Starvation | Cell survival | Autophagy | Lysosome | Apoptosis | CARCINOMA-CELLS | autophagy | cell survival | CANCER CELLS | apoptosis | DEATH | MAMMALIAN-CELLS | CELL BIOLOGY | starvation | THERAPY | INHIBITION | PATHWAY | MONITORING AUTOPHAGY | GROWTH | lysosome | TUMORIGENESIS | Vacuoles - ultrastructure | Estrogens - pharmacology | Apoptosis - drug effects | Humans | Transcriptional Activation - drug effects | Protein Transport - drug effects | Neoplasm Proteins - metabolism | Vacuoles - drug effects | Autophagy - drug effects | Endosomes - metabolism | Gene Knockdown Techniques | trans-Golgi Network - metabolism | Lysosomes - metabolism | Protein Processing, Post-Translational - drug effects | Endosomes - drug effects | Endosomes - ultrastructure | Phagosomes - ultrastructure | Cell Membrane - metabolism | Stress, Physiological - drug effects | Transport Vesicles - drug effects | Neoplasm Proteins - genetics | Cell Membrane - drug effects | Phagosomes - drug effects | Lysosomes - drug effects | Cell Survival - drug effects | Phagosomes - metabolism | Cell Membrane - ultrastructure | trans-Golgi Network - drug effects | Lysosomes - ultrastructure | trans-Golgi Network - ultrastructure | Transport Vesicles - metabolism | Cell Compartmentation - drug effects | Cell Line, Tumor | Vacuoles - metabolism | Cell Proliferation - drug effects | Original
Journal Article
Autophagy, ISSN 1554-8635, 2014, Volume 8, Issue 3, pp. 297 - 309
Journal Article
Annals of neurology, ISSN 1531-8249, 2009, Volume 66, Issue 3, pp. 378 - 389
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 527, Issue 7578, pp. 323 - 328
Journal Article