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Journal Article
Lancet Oncology, The, ISSN 1470-2045, 2013, Volume 14, Issue 7, pp. 590 - 598
Summary Background Currently, crizotinib is the only drug that has been approved for treatment of ALK -rearranged non-small-cell lung cancer (NSCLC). We aimed... 
Hematology, Oncology and Palliative Medicine | FUSION | GENE | ONCOLOGY | C-MET | KINASE | CRIZOTINIB | ANAPLASTIC LYMPHOMA | INHIBITOR | ROS1 | Carcinoma, Large Cell - drug therapy | Lung Neoplasms - drug therapy | Prognosis | Follow-Up Studies | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Humans | Lung Neoplasms - metabolism | Middle Aged | Male | Immunoenzyme Techniques | Adenocarcinoma - metabolism | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Adult | Female | Piperidines - pharmacokinetics | Adenocarcinoma - genetics | Carcinoma, Large Cell - genetics | Lung Neoplasms - genetics | Protein Kinase Inhibitors - pharmacokinetics | Carcinoma, Large Cell - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Adenocarcinoma - drug therapy | Maximum Tolerated Dose | Carcinoma, Squamous Cell - drug therapy | Receptor Protein-Tyrosine Kinases - genetics | Piperidines - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Gene Rearrangement | Carbazoles - pharmacokinetics | Aged | Carbazoles - therapeutic use | Carcinoma, Non-Small-Cell Lung - drug therapy | Neoplasm Staging | Care and treatment | Oncology, Experimental | Research | College teachers | Lung cancer, Non-small cell | Drug approval | Cancer
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 02/2015, Volume 91, Issue 2, pp. 351 - 359
Journal Article
Circulation, ISSN 0009-7322, 07/2014, Volume 130, Issue 4, pp. 298 - 307
BACKGROUND—Fibroblast growth factor-23 (FGF-23) is a hormone that promotes urinary phosphate excretion and regulates vitamin D metabolism. Circulating FGF-23... 
fibroblast growth factors | minerals | atrial fibrillation | chronic | renal insufficiency | UNITED-STATES | CARDIAC & CARDIOVASCULAR SYSTEMS | renal insufficiency, chronic | EVENTS | VASCULAR DYSFUNCTION | RENAL-FUNCTION | RISK | LEFT-VENTRICULAR HYPERTROPHY | COMMUNITY | CYSTATIN-C | PARATHYROID-HORMONE | PERIPHERAL VASCULAR DISEASE | CHRONIC KIDNEY-DISEASE | United States - epidemiology | Follow-Up Studies | Humans | Middle Aged | Fibroblast Growth Factor 3 - physiology | Male | Renal Insufficiency, Chronic - complications | Vitamin D - biosynthesis | Renal Insufficiency, Chronic - epidemiology | Ventricular Remodeling | Ventricular Dysfunction, Left - blood | Aged, 80 and over | Female | Heart Failure - epidemiology | Ethnic Groups - statistics & numerical data | Glomerular Filtration Rate | Hypertrophy, Left Ventricular - blood | Comorbidity | Phosphates - pharmacokinetics | Risk Factors | Proportional Hazards Models | Ventricular Dysfunction, Left - etiology | Fibroblast Growth Factor 3 - blood | Ventricular Dysfunction, Left - physiopathology | Atrial Fibrillation - blood | Renal Insufficiency, Chronic - blood | Phosphates - metabolism | Atrial Fibrillation - epidemiology | Atrial Fibrillation - etiology | Vitamin D - analogs & derivatives | Aged | Demographic aspects | Atrial fibrillation | Atherosclerosis | Physiological aspects | Fibroblast growth factors | Research | Cardiovascular diseases | chronic kidney disease | fibroblast growth factor | mineral
Journal Article
by Meade, T and Sleight, P and Collins, R and Armitage, J and Parish, S and Barton, J and Bray, C and Wincott, E and Bowman, L and Clarke, R and Graham, I and Simpson, D and Warlow, C and Wilcken, D and Tobert, J and Musliner, T and Wilhelmsen, L and Doll, R and Fox, K.M and Hill, C and Sandercock, P and Peto, R and Webster, J and Jamieson, J and Nixon, A and Lackie, S and Thompson, J and Brown, M and Blackwood, S and Morgan, M and Rhoden, W and Saeed, B and Houghton, M and Nicholson, A and Simpson, C and Hoburn, B and Cooper, I and Gallivan, A and Pickerell, E and Hancock, J and Watkinson, J and Ryder, B and Jones, S and Burbridge, W and Kitchen, M and O'Leary, H and Verow, C and Meynell, L and Rollinson, L and Bain, S and Jones, A and Jewkes, C and Russon, C and Bateson, M and Gill, P and Nicol, J and Stansbie, D and Bayly, G and Andrews, G and Halestrap, M and Meredith, J and Best, R and Appleyard, D and Briggs, R and Wareing, H and Holmes, K and Holt, J and Kenyon, M and White, C and Khalifa, M and Newton, D and Wass, A and Watkinson, R and Creamer, J and Anderson, S and Bethell, A and Butler, C and Washington, M and Weston, E and Machin, J and Cleaver, K and Wray, R and Sinclair, J and Van Aalst, A and Been, M and Mattu, R and Bates, D and Burke, A and Gill, L and Walton, E and Cowley, M and Robson, H and Graham, A and Rose, G and Kerr, M and Mallinson, J and Peascod, B and Kalk, J and Scott, A and Donnelly, R and ... and The SEARCH Collaborative Group and SEARCH Collaborative Grp and SEARCH Collaborative Group and Sahlgrenska akademin and Institutionen för medicin, avdelningen för akut och kardiovaskulär medicin and Institute of Medicine, Department of Emergeny and Cardiovascular Medicine and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
The New England Journal of Medicine, ISSN 0028-4793, 08/2008, Volume 359, Issue 8, pp. 789 - 799
A genomewide screen of patients with myopathy who were taking high-dose simvastatin (80 mg per day) showed a strong association between myopathy and variants... 
POPULATION | TRIALS | MEDICINE, GENERAL & INTERNAL | METAANALYSIS | EFFICACY | SAFETY | WIDE ASSOCIATION | TRANSPORTING POLYPEPTIDE 1B1 | SIMVASTATIN | SINGLE NUCLEOTIDE POLYMORPHISMS | ATORVASTATIN | Simvastatin - therapeutic use | Simvastatin - adverse effects | Humans | Middle Aged | Diabetes Mellitus - drug therapy | Genotype | Male | Risk | Genetic Markers | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacokinetics | Organic Anion Transporters - genetics | Hydroxymethylglutaryl-CoA Reductase Inhibitors - adverse effects | Myocardial Infarction - drug therapy | Arterial Occlusive Diseases - drug therapy | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Female | Aged | Myocardial Infarction - prevention & control | Polymorphism, Single Nucleotide | Chromosomes, Human, Pair 12 | Muscular Diseases - chemically induced | Muscular Diseases - genetics | Solute Carrier Organic Anion Transporter Family Member 1b1 | Complications and side effects | Analysis | Muscle diseases | Genetic aspects | Dosage and administration | Single nucleotide polymorphisms | Risk factors | Statins | Drug therapy | Kinases | Drug dosages | Cholesterol | Index Medicus | Abridged Index Medicus | MEDICIN OCH HÄLSOVETENSKAP | Single Nucleotide | genetics | Arterial Occlusive Diseases | pharmacokinetics | Muscular Diseases | Hydroxymethylglutaryl-CoA Reductase Inhibitors | Chromosomes | MEDICAL AND HEALTH SCIENCES | Human | drug therapy | chemically induced | Simvastatin | prevention & control | Diabetes Mellitus | Myocardial Infarction | Pair 12 | Organic Anion Transporters | adverse effects | therapeutic use | Polymorphism
Journal Article
Journal Article
Clinical Pharmacokinetics, ISSN 0312-5963, 01/2014, Volume 53, Issue 11, pp. 1045 - 1051
Journal Article
Journal Article