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European Heart Journal, ISSN 0195-668X, 08/2014, Volume 35, Issue 29, pp. 1917 - 1924
Epidemiological research over the last 50 years has discovered a plethora of biomarkers (including molecules, traits or other diseases) that associate with... 
Coronary artery disease | Mendelian randomization | C-REACTIVE PROTEIN | FUTURE MYOCARDIAL-INFARCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | ESTER TRANSFER PROTEIN | CARDIOVASCULAR EVENTS | SUBTILISIN/KEXIN TYPE 9 | ISCHEMIC-HEART-DISEASE | PHOSPHOLIPASE A | DENSITY-LIPOPROTEIN CHOLESTEROL | LAPLACE-TIMI 57 | GENOME-WIDE ASSOCIATION | Triglycerides - physiology | Cholesterol Ester Transfer Proteins - physiology | Humans | Obesity - genetics | Cholesterol, HDL - physiology | C-Reactive Protein - metabolism | Cholesterol Ester Transfer Proteins - genetics | Mendelian Randomization Analysis | Diabetes Complications - genetics | Genetic Pleiotropy - genetics | Hypertension - genetics | Phospholipases A2 - physiology | Telomere - genetics | Fibrinogen - physiology | Obesity - complications | Receptors, Interleukin-6 - genetics | Risk Factors | Linkage Disequilibrium - genetics | Lipoprotein(a) - physiology | C-Reactive Protein - genetics | Cholesterol, LDL - physiology | Cholesterol, HDL - genetics | Randomized Controlled Trials as Topic | Cholesterol, LDL - genetics | Serum Amyloid P-Component - genetics | Genetic Markers - genetics | Coronary Artery Disease - genetics | Hypertension - complications | Polymorphism, Single Nucleotide - genetics | Triglycerides - genetics | Lipoprotein(a) - genetics | Phospholipases A2 - genetics
Journal Article
Nature Genetics, ISSN 1061-4036, 06/2009, Volume 41, Issue 6, pp. 666 - 676
Journal Article
by Duffy, David L and Zhu, Gu and Li, Xin and Sanna, Marianna and Iles, Mark M and Jacobs, Leonie C and Evans, David M and Yazar, Seyhan and Beesley, Jonathan and Law, Matthew H and Kraft, Peter and Visconti, Alessia and Taylor, John C and Lui, Fan and Wright, Margaret J and Henders, Anjali K and Bowdler, Lisa and Glass, Dan and Ikram, Arfan M and Uitterlinden, André G and Madden, Pamela A and Heath, Andrew C and Nelson, Elliot C and Green, Adele C and Chanock, Stephen and Barrett, Jennifer H and Brown, Matthew A and Hayward, Nicholas K and MacGregor, Stuart and Sturm, Richard A and Hewitt, Alex W and Lee, Jeffrey E and Brossard, Myriam and Moses, Eric K and Song, Fengju and Kumar, Rajiv and Easton, Douglas F and Pharoah, Paul D. P and Swerdlow, Anthony J and Kypreou, Katerina P and Harland, Mark and Randerson-Moor, Juliette and Akslen, Lars A and Andresen, Per A and Avril, Marie-Françoise and Azizi, Esther and Scarrà, Giovanna Bianchi and Brown, Kevin M and Dębniak, Tadeusz and Elder, David E and Fang, Shenying and Friedman, Eitan and Galan, Pilar and Ghiorzo, Paola and Gillanders, Elizabeth M and Goldstein, Alisa M and Gruis, Nelleke A and Hansson, Johan and Helsing, Per and Hočevar, Marko and Höiom, Veronica and Ingvar, Christian and Kanetsky, Peter A and Chen, Wei V and Landi, Maria Teresa and Lang, Julie and Lathrop, G. Mark and Lubiński, Jan and Mackie, Rona M and Mann, Graham J and Molven, Anders and Novaković, Srdjan and Olsson, Håkan and Puig, Susana and Puig-Butille, Joan Anton and Radford-Smith, Graham L and van der Stoep, Nienke and van Doorn, Remco and Whiteman, David C and Craig, Jamie E and Schadendorf, Dirk and Simms, Lisa A and Burdon, Kathryn P and Nyholt, Dale R and Pooley, Karen A and Orr, Nicholas and Stratigos, Alexander J and Cust, Anne E and Ward, Sarah V and Schulze, Hans-Joachim and Dunning, Alison M and Demenais, Florence and Amos, Christopher I and Kayser, Manfred and Hunter, David J and Newton Bishop, Julia A and Spector, Timothy D and Montgomery, Grant W and Mackey, David A and Smith, George Davey and ... and Melanoma GWAS Consortium and Surgery (Lund) and Oncology and Pathology, Kamprad Lab and Lund University and Lunds Melanomstudiegrupp and EpiHealth: Epidemiology for Health and Lund Melanoma Study Group and Kirurgi, Lund and BioCARE: Biomarkers in Cancer Medicine improving Health Care, Education and Innovation and Lunds universitet and Onkologi och Patologi, Kampradlab
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 4774 - 10
The total number of acquired melanocytic nevi on the skin is strongly correlated with melanoma risk. Here we report a meta-analysis of 11 nevus GWAS from... 
CUTANEOUS MALIGNANT-MELANOMA | METAANALYSIS | SUN EXPOSURE | VARIANTS | MULTIDISCIPLINARY SCIENCES | GENETIC INFLUENCES | PREVALENCE | MELANOCYTIC NEVI | CANCER | TANNING RESPONSE | GENOME-WIDE ASSOCIATION | Cytochrome P-450 CYP1B1 - genetics | Humans | Stem Cell Factor - genetics | RNA - genetics | Telomere-Binding Proteins - genetics | Telomerase - genetics | Nevus, Pigmented - genetics | Group VI Phospholipases A2 - genetics | Melanoma - genetics | Genetic Pleiotropy - genetics | Nuclear Proteins - genetics | Microfilament Proteins - genetics | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Guanine Nucleotide Exchange Factors - genetics | Histone Deacetylases - genetics | Interferon Regulatory Factors - genetics | Repressor Proteins - genetics | Nerve Tissue Proteins - genetics | Carrier Proteins - genetics | Skin Neoplasms - genetics | MicroRNAs - genetics | Polymorphism, Single Nucleotide | Receptors, G-Protein-Coupled - genetics | Bivariate analysis | Pathways | Interferon regulatory factor 4 | Genes | Melanoma | Nevus | Risk | Skin | Single-nucleotide polymorphism | Loci | PLA2G6 protein, human | risk assessment | cutaneous melanoma | Interferon Regulatory Factors | RNA | Medical and Health Sciences | KITLG protein, human | single nucleotide polymorphism | Repressor Proteins | carrier protein | repressor protein | Group VI Phospholipases A2 | Stn1 protein, human | pigmented nevus | G protein coupled receptor | genetic risk | genetic predisposition | skin tumor | PPARGC1B protein, human | telomerase RNA | Carrier Proteins | Basic Medicine | peroxisome proliferator activated receptor gamma coactivator 1beta | nerve protein | biology | gene | HDAC4 protein, human | Receptors, G-Protein-Coupled | Caucasian | melanoma | European Continental Ancestry Group | Genetic Pleiotropy | Nerve Tissue Proteins | histone deacetylase 4 | Guanine Nucleotide Exchange Factors | Nuclear Proteins | Clinical Medicine | Skin Neoplasms | Cytochrome P-450 CYP1B1 | interferon regulatory factor | DOCK8 protein, human | gene expression | cytochrome P450 1B1 | United States | meta analysis | Medicin och hälsovetenskap | Article | skin | pleiotropy | Klinisk medicin | nuclear protein | Medicinsk genetik | Medical Genetics | Netherlands | telomerase | genetics | human | stem cell factor | GPRC5A protein, human | phospholipase A2 group VI | telomere binding protein | meta-analysis | United Kingdom | Histone Deacetylases | interferon regulatory factor 4 | actin binding protein | SYNE2 protein, human | histone deacetylase | Telomere-Binding Proteins | gene locus | guanine nucleotide exchange factor | Microfilament Proteins | MicroRNAs | Nevus, Pigmented | genome-wide association study | Medicinska och farmaceutiska grundvetenskaper | microRNA | meta analysis (topic) | MIRN146 microRNA, human | cancer | Australia | Cancer and Oncology | CYP1B1 protein, human | interferon regulatory factor-4 | telomere homeostasis | Cancer och onkologi
Journal Article
Toxicon, ISSN 0041-0101, 12/2013, Volume 75, pp. 63 - 89
Clostridia comprise a heterogenous group of environmental bacteria containing 15 pathogenic species, which produce the most potent toxins. The origin of toxins... 
Clostridium | Toxin | Evolution | Genetics | PERFRINGENS TYPE-A | NEUROTOXIN TYPE-A | FRAGMENT LENGTH POLYMORPHISM | BINARY TOXIN | CHOLESTEROL-DEPENDENT CYTOLYSINS | INFANT BOTULISM | PORE-FORMING TOXIN | TANDEM-REPEAT ANALYSIS | BOTULINUM TYPE-A | PHARMACOLOGY & PHARMACY | TOXICOLOGY | ENCODING BETA2 TOXIN | Botulinum Toxins - genetics | Clostridium botulinum - classification | Clostridium difficile - classification | Pore Forming Cytotoxic Proteins - genetics | Clostridium perfringens - classification | Clostridium difficile - genetics | Genetic Loci | Clostridium - isolation & purification | Clostridium - classification | Genetic Variation | Plasmids - genetics | Bacterial Toxins - genetics | Clostridium botulinum - genetics | Genes, Bacterial | Gene Transfer, Horizontal | Clostridium - genetics | Chromosomes, Bacterial | Sequence Analysis, DNA | DNA Transposable Elements | Enterotoxins - genetics | DNA, Bacterial - genetics | Clostridium perfringens - genetics | RNA, Ribosomal, 16S - genetics | Type C Phospholipases - genetics | Protein Conformation | Calcium-Binding Proteins - genetics | Evolution, Molecular | Enzymes | Bacteria | Genes | Index Medicus | Spreads | Toxins | Chromosomes | Strain | Clostridium botulinum | Calcium-Binding Proteins | Enterotoxins | Clostridium difficile | Clostridium perfringens | DNA, Bacterial | Botulinum Toxins | Life Sciences | Toxicology | Bacterial Toxins | Plasmids | Type C Phospholipases | Pore Forming Cytotoxic Proteins | RNA, Ribosomal, 16S
Journal Article
Journal Article
Blood, ISSN 0006-4971, 02/2017, Volume 129, Issue 6, pp. 759 - 770
Kinases downstream of B-cell antigen receptor (BCR) represent attractive targets for therapy in non-Hodgkin lymphoma (NHL). As clinical responses vary,... 
SURFACE IGM | BCR | CHRONIC-LYMPHOCYTIC-LEUKEMIA | BRUTONS TYROSINE KINASE | HUMAN FOLLICULAR LYMPHOMA | GENE-EXPRESSION | KINASE INHIBITOR IBRUTINIB | MUTATIONS | TARGETING BTK | HEMATOLOGY | NF-KAPPA-B | Lymphoma, Follicular - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Lymphoma, Mantle-Cell - pathology | Leukemia, Lymphocytic, Chronic, B-Cell - genetics | Receptors, Antigen, B-Cell - metabolism | STAT1 Transcription Factor - metabolism | Lymphoma, Follicular - genetics | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Single-Cell Analysis | Immunoglobulin M - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | STAT1 Transcription Factor - genetics | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Lymphoma, Large B-Cell, Diffuse - diagnosis | Mitogen-Activated Protein Kinase 3 - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - metabolism | Receptors, Antigen, B-Cell - genetics | CD79 Antigens - genetics | src-Family Kinases - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Phosphorylation | Gene Expression Regulation, Neoplastic | Lymphoma, Follicular - diagnosis | Lymphoma, Large B-Cell, Diffuse - metabolism | Phosphoproteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Lymphoma, Mantle-Cell - diagnosis | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Protein-Tyrosine Kinases - genetics | Flow Cytometry | Syk Kinase - genetics | Phospholipase C gamma - genetics | src-Family Kinases - metabolism | Lymphoma, Mantle-Cell - genetics | Lymphoma, Mantle-Cell - metabolism | Diagnosis, Differential | Phospholipase C gamma - metabolism | Lymphoma, Large B-Cell, Diffuse - pathology | Lymphoma, Follicular - pathology | p38 Mitogen-Activated Protein Kinases - genetics | Phosphoproteins - genetics | CD79 Antigens - metabolism | Syk Kinase - metabolism | Lymphoma, Large B-Cell, Diffuse - genetics | Leukemia, Lymphocytic, Chronic, B-Cell - diagnosis | Life Sciences | Human health and pathology | Hematology | Cancer | Lymphoid Neoplasia
Journal Article
Nature Genetics, ISSN 1061-4036, 09/2017, Volume 49, Issue 9, pp. 1373 - 1384
Journal Article