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Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 7602 - 8
Verteporfin (VP), a light-activated drug used in photodynamic therapy for the treatment of choroidal neovascular membranes, has also been shown to be an... 
TRANSCRIPTION FACTORS | BREAST-CANCER | PHOTODYNAMIC THERAPY | CANCER CELLS | MULTIDISCIPLINARY SCIENCES | HIPPO PATHWAY | OVARIAN-CANCER | YAP-TEAD | EXPRESSION | OPTIC PATHWAY GLIOMA | NERVE GLIOMA | Neuroglia - pathology | Humans | Verteporfin - pharmacology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Octamer Transcription Factor-3 - genetics | Photosensitizing Agents - pharmacology | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Survivin - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Microtubule-Associated Proteins - agonists | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Receptor Protein-Tyrosine Kinases - metabolism | Cysteine-Rich Protein 61 - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Cell Line, Tumor | Connective Tissue Growth Factor - genetics | Proto-Oncogene Proteins c-myc - genetics | Connective Tissue Growth Factor - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Gene Expression Regulation, Neoplastic | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | Cysteine-Rich Protein 61 - metabolism | Neuroglia - drug effects | DNA-Binding Proteins - metabolism | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Nuclear Proteins - genetics | Proto-Oncogene Proteins - metabolism | Survivin - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Connective Tissue Growth Factor - antagonists & inhibitors | Cysteine-Rich Protein 61 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Transcription Factors - antagonists & inhibitors | Phosphoproteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Octamer Transcription Factor-3 - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - metabolism | Transcription Factors - metabolism | Receptor Protein-Tyrosine Kinases - genetics | Nuclear Proteins - antagonists & inhibitors | Octamer Transcription Factor-3 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Neuroglia - metabolism | Cell Proliferation - drug effects | Adaptor Proteins, Signal Transducing - metabolism | TOR protein | Photodynamic therapy | Brain tumors | Oct-4 protein | Glioblastoma | c-Myc protein | MAP kinase | Connective tissue growth factor | AKT protein | Survivin | Myc protein | Tumor cell lines | Ovarian cancer | Axl protein | Yes-associated protein | Cell activation | Glioma cells | CYR61 protein | Retinoblastoma | Pluripotency | Index Medicus
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 04/2011, Volume 22, Issue 8, pp. 1191 - 1206
Mitosis requires precise coordination of multiple global reorganizations of the nucleus and cytoplasm. Cyclin-dependent kinase 1 (Cdk1) is the primary upstream... 
ANAPHASE-PROMOTING COMPLEX | XENOPUS EGG EXTRACTS | PROTEIN PHOSPHATASES | CYCLE-DEPENDENT PHOSPHORYLATION | UBIQUITIN LIGASE | M-PHASE | CHROMOSOME ALIGNMENT | CELL-CYCLE | TUMOR-CELLS | SPINDLE-ASSEMBLY CHECKPOINT | CELL BIOLOGY | Cyclin-Dependent Kinases - metabolism | Gene Expression - drug effects | Xenopus Proteins - genetics | Mitosis | Protein-Tyrosine Kinases - metabolism | Humans | Phosphoprotein Phosphatases - metabolism | G2 Phase - drug effects | CDC2 Protein Kinase - metabolism | Cell Cycle Proteins - antagonists & inhibitors | Protein-Tyrosine Kinases - genetics | Cyclin B - genetics | cdc25 Phosphatases - antagonists & inhibitors | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Prometaphase - drug effects | Female | Cyclin-Dependent Kinases - antagonists & inhibitors | Phosphorylation - drug effects | cdc25 Phosphatases - metabolism | Cyclin-Dependent Kinases - genetics | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | CDC2 Protein Kinase - genetics | Protein Phosphatase 2 - antagonists & inhibitors | CDC2 Protein Kinase - antagonists & inhibitors | Xenopus laevis | Cell Cycle Proteins - metabolism | Protein Phosphatase 2 - genetics | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Phosphoprotein Phosphatases - antagonists & inhibitors | cdc25 Phosphatases - genetics | Xenopus Proteins - antagonists & inhibitors | Membrane Proteins | Animals | Phosphoprotein Phosphatases - genetics | Nuclear Proteins - antagonists & inhibitors | Protein Phosphatase 2 - metabolism | Cyclin B - metabolism | Feedback, Physiological - drug effects | Xenopus Proteins - metabolism | Protein Kinase Inhibitors - pharmacology | S Phase - drug effects | HeLa Cells | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 04/2012, Volume 130, Issue 7, pp. 1695 - 1705
Lung cancer is one of the most commonly occurring malignancies. It has been reported that mammalian target of rapamycin (mTOR) is phosphorylated in lung cancer... 
lung cancer | signaling | PI3K | fisetin | mTOR | Akt | flavonoid | dual inhibition | ACTIVATED PROTEIN-KINASE | CYCLE ARREST | KAPPA-B | TUBEROUS-SCLEROSIS | ONCOLOGY | PROSTATE-CANCER | GROWTH | TUMOR-SUPPRESSOR | MTOR INHIBITORS | PI3K/AKT/MTOR PATHWAY | EXPRESSION | Protein Kinases - metabolism | Lung Neoplasms - drug therapy | TOR Serine-Threonine Kinases - metabolism | Tumor Suppressor Proteins - antagonists & inhibitors | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Lung Neoplasms - metabolism | Eukaryotic Initiation Factor-4E - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | TOR Serine-Threonine Kinases - antagonists & inhibitors | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Regulatory-Associated Protein of mTOR | Flavonoids - pharmacology | Phosphorylation - drug effects | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Rapamycin-Insensitive Companion of mTOR Protein | Tumor Suppressor Proteins - metabolism | Lung Neoplasms - enzymology | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Ribosomal Protein S6 Kinases, 70-kDa - antagonists & inhibitors | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | MTOR Associated Protein, LST8 Homolog | Carcinoma, Non-Small-Cell Lung - metabolism | Carrier Proteins - antagonists & inhibitors | PTEN Phosphohydrolase - metabolism | Eukaryotic Initiation Factor-4E - antagonists & inhibitors | Carrier Proteins - metabolism | Cell Line, Tumor | Stem Cells - drug effects | Cell Proliferation - drug effects | Carcinoma, Non-Small-Cell Lung - drug therapy | Adaptor Proteins, Signal Transducing - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Kinases | Phosphorylation | Lung cancer | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 09/2016, Volume 538, Issue 7623, pp. 114 - 117
The common participation of oncogenic KRAS proteins in many of the most lethal human cancers, together with the ease of detecting somatic KRAS mutant alleles... 
TARGET | CRM1/XPO1 | ACTIVATION | MODELS | MULTIDISCIPLINARY SCIENCES | KINASE | YAP | MUTATIONS | INHIBITORS | IDENTIFICATION | Lung Neoplasms - drug therapy | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Cell Survival - genetics | Lung Neoplasms - pathology | NF-kappa B - metabolism | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Cell Nucleus - metabolism | RNA Interference | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Female | Verteporfin | Protein-Serine-Threonine Kinases - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Follistatin-Related Proteins - genetics | Lung Neoplasms - genetics | Cell Survival - drug effects | NF-kappa B - antagonists & inhibitors | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Porphyrins - pharmacology | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Animals | Karyopherins - metabolism | Active Transport, Cell Nucleus - drug effects | Genes, Lethal - genetics | NF-KappaB Inhibitor alpha - metabolism | Nuclear Proteins - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | RNA, Small Interfering | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Cell Nucleus - drug effects | Karyopherins - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - metabolism | Care and treatment | Genetic aspects | Gene mutations | Health aspects | Guanosine triphosphatase | Lung cancer | Genomics | Genomes | Kinases | Drug resistance | Gene expression | Drug dosages | Index Medicus
Journal Article
Cell Reports, ISSN 2211-1247, 01/2016, Volume 14, Issue 3, pp. 598 - 610
Ewing sarcoma cells depend on the EWS-FLI1 fusion transcription factor for cell survival. Using an assay of EWS-FLI1 activity and genome-wide RNAi screening,... 
TARGET | CHROMOSOME-TRANSLOCATION | DNA-BINDING | SOLID TUMORS | GENE | EWS/FLI-1 FUSION | PLADIENOLIDE | PHASE-I | CANCER | FAMILY | CELL BIOLOGY | RNA-Binding Proteins - genetics | Oncogene Proteins, Fusion - metabolism | Exons | Humans | Gene Expression Regulation, Neoplastic | Sarcoma, Ewing - pathology | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | RNA Splicing | Ribonucleoprotein, U2 Small Nuclear - metabolism | RNA-Binding Protein EWS - antagonists & inhibitors | RNA Interference | Proto-Oncogene Protein c-fli-1 - metabolism | Base Sequence | Calmodulin-Binding Proteins - antagonists & inhibitors | Ribonucleoprotein, U2 Small Nuclear - genetics | RNA Precursors - metabolism | Microfilament Proteins - metabolism | Heterogeneous-Nuclear Ribonucleoprotein Group F-H - antagonists & inhibitors | Binding Sites | Microfilament Proteins - genetics | RNA-Binding Proteins - antagonists & inhibitors | Calmodulin-Binding Proteins - genetics | Proto-Oncogene Protein c-fli-1 - genetics | Cell Survival | RNA-Binding Protein EWS - genetics | RNA Splicing Factors | Transcription Factors - antagonists & inhibitors | Calmodulin-Binding Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Phosphoproteins - genetics | Transcription Factors - genetics | RNA-Binding Protein EWS - metabolism | Heterogeneous-Nuclear Ribonucleoprotein Group F-H - genetics | Heterogeneous-Nuclear Ribonucleoprotein Group F-H - metabolism | Transcription Factors - metabolism | Proto-Oncogene Protein c-fli-1 - antagonists & inhibitors | Microfilament Proteins - antagonists & inhibitors | Oncogene Proteins, Fusion - genetics | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Cell Line, Tumor | Ribonucleoprotein, U2 Small Nuclear - antagonists & inhibitors | Oncogene Proteins, Fusion - antagonists & inhibitors | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2018, Volume 200, Issue 7, pp. 2291 - 2303
Binge/moderate alcohol suppresses TLR4-MyD88 proinflammatory cytokines; however, alcohol's effects on TLR-TRIF signaling, especially after in vivo exposure in... 
TOLL-LIKE RECEPTOR | IN-VIVO | FACTOR-KAPPA-B | ENDOPLASMIC-RETICULUM STRESS | PROINFLAMMATORY CYTOKINES | IMMUNOLOGY | PATTERN-RECOGNITION RECEPTORS | HUMAN MONOCYTES | RESPIRATORY SYNCYTIAL VIRUS | IFN-BETA INDUCTION | INNATE IMMUNITY | Inflammation - pathology | Interleukin-6 - antagonists & inhibitors | Binge Drinking - pathology | Ethanol - toxicity | Humans | Middle Aged | Receptors, Neuropeptide Y - metabolism | Male | Monocytes - immunology | X-Box Binding Protein 1 - drug effects | Toll-Like Receptor 3 - antagonists & inhibitors | Lipopolysaccharides - immunology | Chemokine CCL5 - antagonists & inhibitors | Young Adult | Myeloid Differentiation Factor 88 - antagonists & inhibitors | Adult | Female | Poly I-C - immunology | Toll-Like Receptor 4 - antagonists & inhibitors | Macrophages - immunology | Cell Line | Adaptor Proteins, Vesicular Transport - antagonists & inhibitors | Chemokine CXCL10 - antagonists & inhibitors | Interferon-beta - antagonists & inhibitors | HSP70 Heat-Shock Proteins - metabolism | Monocytes - drug effects | Animals | Signal Transduction - drug effects | Adolescent | Macrophages - drug effects | RAW 264.7 Cells | Mice | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Phosphoprotein phosphatase | β-Interferon | RANTES | Alcohol | Macrophages | Lipopolysaccharides | Interleukin 6 | Proteins | Signal transduction | Drug tolerance | Rodents | Toll-like receptors | IP-10 protein | Inhibition | Enzyme-linked immunosorbent assay | Ethanol | Cytokines | Immunoregulation | Exposure | Inflammation | Poly (I:C) | TLR4 protein | TLR3 protein | Signaling | Monocytes | Interferon regulatory factor 3 | Tumor necrosis factor | Alcoholic beverages | MyD88 protein | Protein phosphatase | Stress proteins | Chemokines | Index Medicus | Abridged Index Medicus
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 2014, Volume 10, Issue 6, pp. 477 - 482
Journal Article
Oncogene, ISSN 0950-9232, 04/2013, Volume 32, Issue 14, pp. 1821 - 1830
The Salvador/Warts/Hippo (Hippo) signaling pathway defines a novel signaling cascade regulating cell contact inhibition, organ size control, cell growth,... 
KIBRA | Willin | breast cancer | Hippo pathway | Merlin | claudin-low | APOPTOSIS | PROTEIN | ORGAN SIZE CONTROL | BIOCHEMISTRY & MOLECULAR BIOLOGY | YAP | PROLIFERATION | DROSOPHILA | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER CELLS | ONCOGENE | ONCOLOGY | CYCLE EXIT | GENETICS & HEREDITY | RNA, Small Interfering - genetics | Neurofibromin 2 - genetics | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Cytoskeletal Proteins - genetics | Humans | Gene Expression Profiling | Intracellular Signaling Peptides and Proteins - metabolism | Immunoenzyme Techniques | Hepatocyte Growth Factor - antagonists & inhibitors | Biomarkers, Tumor - metabolism | Hepatocyte Growth Factor - genetics | Carcinoma, Ductal, Breast - pathology | Carcinoma, Ductal, Breast - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Real-Time Polymerase Chain Reaction | Protein-Serine-Threonine Kinases - metabolism | Signal Transduction | Breast - cytology | Membrane Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Blotting, Western | Hepatocyte Growth Factor - metabolism | Breast Neoplasms - genetics | Claudin-1 - genetics | Biomarkers, Tumor - genetics | Cell Movement | Carcinoma, Ductal, Breast - genetics | Phosphorylation | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | Breast - metabolism | Breast Neoplasms - metabolism | Claudin-1 - metabolism | Epithelial-Mesenchymal Transition | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cytoskeletal Proteins - metabolism | Female | Membrane Proteins - metabolism | Nuclear Proteins - genetics | Neurofibromin 2 - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Transcription Factors - antagonists & inhibitors | Phosphoproteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Breast Neoplasms - pathology | Nuclear Proteins - antagonists & inhibitors | Apoptosis | Cellular proteins | Prognosis | Physiological aspects | Breast cancer | Genetic aspects | Cellular signal transduction | Research | Health aspects | Mammals | Gene expression | Index Medicus
Journal Article
Cancer Science, ISSN 1347-9032, 03/2017, Volume 108, Issue 3, pp. 478 - 487
Pancreatic ductal adenocarcinoma ( PDAC ) is one of the most aggressive human malignancies. The Yes‐associated protein‐1 ( YAP ) plays a critical role in cell... 
Angiogenesis | apoptosis | pancreatic cancer | vasculogenic mimicry | verteporfin | HUMAN CHOLANGIOCARCINOMA | HIPPO PATHWAY | DOWN-REGULATION | ANGIOPOIETIN-2 | CANCER | MOLECULAR PATHWAYS | HEPATOCELLULAR-CARCINOMA | IN-VITRO | ONCOLOGY | POOR-PROGNOSIS | CHEMORESISTANCE | Human Umbilical Vein Endothelial Cells | Cyclin D1 - metabolism | Apoptosis - drug effects | Humans | Male | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Proto-Oncogene Proteins c-bcl-2 - metabolism | Vesicular Transport Proteins - antagonists & inhibitors | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Cell Survival - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Matrix Metalloproteinase 2 - metabolism | Pancreatic Neoplasms - pathology | Oncogene Proteins - metabolism | Nuclear Proteins - metabolism | Poly (ADP-Ribose) Polymerase-1 - metabolism | Transcription Factors - antagonists & inhibitors | Porphyrins - pharmacology | Carcinoma, Pancreatic Ductal - pathology | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Cadherins - antagonists & inhibitors | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Cyclin E - metabolism | Adaptor Proteins, Signal Transducing - metabolism | G1 Phase Cell Cycle Checkpoints - drug effects | Antigens, CD | Adenocarcinoma | Proteins | Photochemotherapy | Analysis | Pancreatic cancer | Cancer | Cell proliferation | Metastasis | Cancer therapies | Mimicry | Cell adhesion & migration | Macular degeneration | Cell cycle | Xenografts | Conflicts of interest | G1 phase | Cell survival | Photodynamic therapy | Poly(ADP-ribose) polymerase | Gene expression | Cadherin | Gelatinase A | Yes-associated protein | Microscopy | Medical prognosis | Stem cells | Hypoxia | Apoptosis | Tumors | Index Medicus | Original
Journal Article