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PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, pp. e0161803 - 161803
...), a previously described marker for melanoma-initiating cells, on cellular metabolism. Metabolic profiles for two groups of human G3361 melanoma cells were compared, i.e... 
OVEREXPRESSION | STEM-CELLS | ACTIVATION | MULTIPARAMETRIC OPTIMIZATION | MULTIDISCIPLINARY SCIENCES | NMR SPECTROSCOPIC ANALYSIS | MULTIDRUG-RESISTANCE | ENERGY-METABOLISM | HYPOXIA | CANCER | P-31 | Melanoma - metabolism | Magnetic Resonance Spectroscopy | Humans | Gene Expression Regulation, Neoplastic | Alanine - metabolism | Melanoma - pathology | Metabolomics - methods | ATP Binding Cassette Transporter, Subfamily B, Member 1 - genetics | ATP Binding Cassette Transporter, Subfamily B, Member 1 - metabolism | Amino Acids - metabolism | Pilot Projects | Lactates - metabolism | Neoplastic Stem Cells - metabolism | RNA Interference | Melanoma - genetics | Glycerylphosphorylcholine - metabolism | Biomarkers, Tumor - metabolism | Cell Line, Tumor | Glucose - metabolism | Biomarkers, Tumor - genetics | Cell Membrane - metabolism | Phosphatidylethanolamines - metabolism | Phospholipids - metabolism | Cell metabolism | Physiological aspects | Genetic aspects | Research | Binding proteins | Melanoma | Energy metabolism | Nuclear magnetic resonance--NMR | Lung cancer | Amino acids | Lipids | Phospholipids | Glucose | Drug resistance | Cell surface | Magnetic resonance spectroscopy | Metabolites | Energy | Spectrum analysis | Interleukin 1 | Inhibition | Lipid metabolism | Water chemistry | Alanine | Degradation products | Statistical analysis | Cold | Phosphorus | Breast cancer | Metabolism | Gene expression | Acids | Stem cells | Glycolysis | Monoclonal antibodies | Hypoxia | Lactic acid | NMR spectroscopy | Surface markers | Tumors | Life Sciences | Cellular Biology | Nuclear magnetic resonance | NMR
Journal Article
Biotechnology and Bioengineering, ISSN 0006-3592, 10/2003, Volume 84, Issue 2, pp. 170 - 178
Simultaneous nitrification and denitrification (SND) via the nitrite pathway and anaerobic–anoxic‐enhanced biological phosphorus removal (EBPR) are two... 
simultaneous nitrification | nitrous oxide (N2O) | nitrite pathway | denitrification and phosphorus removal (SNDPR) | titration and off‐gas analysis (TOGA) | denitrifying glycogen‐accumulating organisms (DGAOs) | Nitrite pathway | Nitrous oxide (N | Simultaneous nitrification | Titration and off-gas analysis (TOGA) | Denitrification and phosphorus removal (SNDPR) | Denitrifying glycogen-accumulating organisms (DGAOs) | STOICHIOMETRY | SND | GLYCOGEN-ACCUMULATING ORGANISMS | RELEASE | NITROGEN REMOVAL | denitrifying glycogen-accumulating organisms (DGAOs) | ACTIVATED-SLUDGE SYSTEMS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | WASTE-WATER | METABOLIC MODEL | titration and off-gas analysis (TOGA) | NITRITE | Anaerobiosis | Oxygen - pharmacology | Phosphates - chemistry | Nitrites - metabolism | Chromatography, Gas | Quaternary Ammonium Compounds - chemistry | Nitric Oxide - analysis | Oxygen - metabolism | Bacteria, Aerobic - growth & development | Nitrates - chemistry | Glycogen - metabolism | Mass Spectrometry | Nitrous Oxide - analysis | Oxygen - chemistry | Hydroxybutyrates - analysis | Bacteria, Anaerobic - metabolism | Ammonia - chemistry | Bacteria, Aerobic - metabolism | Acetic Acid - metabolism | Bacteria, Anaerobic - drug effects | Glycogen - analysis | Biodegradation, Environmental | Phosphorus - analysis | Nitrogen - metabolism | Aerobiosis | Ammonia - metabolism | Nitrogen - analysis | Valerates - metabolism | Nitrites - chemistry | Nitrous Oxide - metabolism | Valerates - analysis | Nitrates - metabolism | Phosphates - metabolism | Algorithms | Waste Disposal, Fluid - methods | Phosphorus - metabolism | Bioreactors - microbiology | Quaternary Ammonium Compounds - metabolism | Bacteria, Aerobic - drug effects | Acetic Acid - analysis | Hydroxybutyrates - metabolism | Nitric Oxide - metabolism | Bacteria, Anaerobic - growth & development
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 2, p. e0147818
Rhabdomyolysis is common in very long-chain acyl-CoA dehydrogenase deficiency (VLCADD) and other metabolic myopathies, but its pathogenic basis is poorly... 
SKELETAL-MUSCLE | MAGNETIC-RESONANCE-SPECTROSCOPY | ACID BETA-OXIDATION | MAXIMAL FAT OXIDATION | FATIGUE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | KINASE | RESISTANCE | DUCHENNE MUSCULAR-DYSTROPHY | MITOCHONDRIAL MYOPATHIES | Mitochondrial Diseases - pathology | Humans | Muscle Fibers, Fast-Twitch - metabolism | Mitochondrial Diseases - metabolism | Rhabdomyolysis - complications | Male | Muscle Fibers, Slow-Twitch - metabolism | Adenosine Triphosphate - biosynthesis | Case-Control Studies | Mitochondrial Diseases - complications | Exercise | Lipid Metabolism, Inborn Errors - pathology | Muscular Diseases - physiopathology | Rhabdomyolysis - pathology | Adult | Female | Lipid Metabolism, Inborn Errors - metabolism | Acyl-CoA Dehydrogenase, Long-Chain - metabolism | Muscular Diseases - metabolism | Oxidative Phosphorylation | Rhabdomyolysis - physiopathology | Mitochondria - metabolism | Muscular Diseases - pathology | Models, Statistical | Acetylcarnitine - blood | Rhabdomyolysis - metabolism | Lipid Metabolism, Inborn Errors - physiopathology | Phosphates - metabolism | Muscle Fibers, Slow-Twitch - pathology | Adolescent | Muscle Fibers, Fast-Twitch - pathology | Muscular Diseases - complications | Phosphocreatine - metabolism | Lipid Metabolism, Inborn Errors - complications | Mitochondrial Diseases - physiopathology | Acyl-CoA Dehydrogenase, Long-Chain - deficiency | Physiological aspects | Cycling | Research | Rhabdomyolysis | Risk factors | Phosphates | Pediatrics | Neurosciences | Nuclear magnetic resonance--NMR | Laboratories | Oxidative metabolism | Homeostasis | Chains | Dehydrogenase | Muscular dystrophy | Acyl-CoA dehydrogenase | Defects | Mitochondria | Child development | Metabolites | Rodents | Workloads | Mathematical models | Bioinformatics | Enzymes | Departments | Phosphorus | Phosphocreatine | Earth science | Metabolism | Medical screening | Patients | Fatty acids | Carnitine | Children & youth | Energy balance | Musculoskeletal system | Chemistry | Hypotheses | Hospitals | Bicycles | Quadriceps muscle | Respiration | ATP | Metabolic disorders | Nuclear magnetic resonance | NMR
Journal Article
Brain, ISSN 0006-8950, 12/2009, Volume 132, Issue 12, pp. 3285 - 3297
Mitochondrial dysfunction hypothetically contributes to neuronal degeneration in patients with Parkinson's disease. While several in vitro data exist, the... 
P MRS | Energy metabolism | Mitochondrial dysfunction | Parkinson's disease | MR spectroscopy | OXIDATIVE-PHOSPHORYLATION | HUMAN BRAIN | mitochondrial dysfunction | NEUROSCIENCES | P-31 MR SPECTROSCOPY | CLINICAL NEUROLOGY | MULTIPLE SYSTEM ATROPHY | COENZYME Q | IN-VIVO ASSESSMENT | COMPLEX-I | P-31 MRS | PLACEBO-CONTROLLED TRIAL | ENERGY-METABOLISM | energy metabolism | PROGRESSIVE SUPRANUCLEAR PALSY | Protons | Substantia Nigra - physiopathology | Predictive Value of Tests | Humans | Middle Aged | Mitochondrial Diseases - metabolism | Male | Substantia Nigra - metabolism | Brain - metabolism | Brain Diseases, Metabolic - diagnosis | Putamen - physiopathology | Adenosine Triphosphate - metabolism | Female | Parkinson Disease - metabolism | Energy Metabolism - physiology | Biomarkers - metabolism | Magnetic Resonance Spectroscopy - methods | Adenosine Triphosphate - analysis | Brain - physiopathology | Biomarkers - analysis | Oxidative Phosphorylation | Mitochondria - metabolism | Phosphocreatine - analysis | Brain Diseases, Metabolic - metabolism | Parkinson Disease - physiopathology | Disease Progression | Brain Chemistry - physiology | Brain Diseases, Metabolic - physiopathology | Phosphorus - metabolism | Parkinson Disease - diagnosis | Aged | Phosphocreatine - metabolism | Mitochondrial Diseases - physiopathology | Mitochondrial Diseases - diagnosis | Putamen - metabolism
Journal Article