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Annual review of immunology, ISSN 1545-3278, 2009, Volume 27, Issue 1, pp. 621 - 668
.... This nosology has taken root because of the dramatic advances in our knowledge of the genetic basis of both mendelian and complex autoinflammatory diseases, and with the... 
Pulmonary fibrosis | Atherosclerosis | Innate immunity | Crohn's disease | Inflammasome | Type 2 diabetes mellitus | IL-1β | Ankylosing spondylitis | atherosclerosis | HEMOLYTIC-UREMIC SYNDROME | INTERLEUKIN-1 RECEPTOR ANTAGONIST | ankylosing spondylitis | JUVENILE IDIOPATHIC ARTHRITIS | IL-1 beta | INNATE IMMUNE-SYSTEM | ENDOPLASMIC-RETICULUM STRESS | IMMUNOLOGY | ENCODING MEVALONATE KINASE | UNFOLDED PROTEIN RESPONSE | innate immunity | FAMILIAL MEDITERRANEAN FEVER | inflammasome | MACROPHAGE ACTIVATION SYNDROME | pulmonary fibrosis | type 2 diabetes mellitus | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Phosphotransferases (Alcohol Group Acceptor) - immunology | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Tumor Necrosis Factor-alpha - genetics | NF-kappa B - immunology | NF-kappa B - metabolism | Interleukin-1beta - genetics | Autoimmune Diseases - genetics | Interleukin-1beta - metabolism | Tumor Necrosis Factor-alpha - immunology | Cytokines - genetics | Carrier Proteins - immunology | Cytokines - immunology | Autoimmune Diseases - classification | Cytokines - metabolism | Autoimmune Diseases - immunology | Interleukin-1beta - immunology | Phosphotransferases (Alcohol Group Acceptor) - genetics | Inflammation - immunology | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | NF-kappa B - genetics | Inflammation - classification | Inflammation - genetics | Crohn’s disease
Journal Article
Cell metabolism, ISSN 1550-4131, 2014, Volume 19, Issue 3, pp. 431 - 444
Journal Article
Cell metabolism, ISSN 1550-4131, 2012, Volume 15, Issue 6, pp. 813 - 826
Immune cells are somewhat unique in that activation responses can alter quantitative phenotypes upwards of 100,000-fold. To date little is known about the... 
PROTEIN | GENE | CELL ACTIVATION | GLUTATHIONE | MURINE MACROPHAGES | TRANSCRIPTION | ENDOCRINOLOGY & METABOLISM | RECEPTOR | BINDING-ACTIVITY | DIFFERENTIATION | INFLAMMATORY RESPONSE | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Cell Polarity | Transcription Factors - chemistry | Endotoxemia - enzymology | Phosphotransferases (Alcohol Group Acceptor) - chemistry | Humans | Tumor Necrosis Factor-alpha - genetics | Male | Endotoxemia - immunology | Interleukin-6 - metabolism | Macrophages - immunology | Macrophages - physiology | Protein Structure, Tertiary | Amino Acid Sequence | Cell Line | Double-Blind Method | Interleukin-6 - genetics | Down-Regulation | Mice, Inbred C57BL | Carbohydrate Metabolism | Models, Molecular | Phosphotransferases (Alcohol Group Acceptor) - genetics | Transcription Factors - genetics | Macrophages - enzymology | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Gene Expression Regulation, Enzymologic | Transcription Factors - metabolism | Macrophages - metabolism | Phenotype | Animals | Energy Metabolism | Lipopolysaccharides - pharmacology | Glucose - metabolism | Mice | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Phosphates | Glucose metabolism | Medical colleges | Orphans | Physiological aspects | Glucose | Macrophages | Phosphotransferases | Dextrose | Sugars | Monosaccharides | Adaptations | Carbohydrates | Polarization | Cell activation | Immunomodulation | Pentose phosphate pathway | Lipopolysaccharides
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2009, Volume 30, Issue 6, pp. 832 - 844
Effector T cell differentiation requires the simultaneous integration of multiple, and sometimes opposing, cytokine signals. We demonstrated mTOR's role in... 
MOLIMMUNO | CELLIMMUNO | MAMMALIAN TARGET | RAPAMYCIN | FOXP3 EXPRESSION | MAINTENANCE | ACTIVATION | PATHWAY | AKT | INDUCTION | IMMUNOLOGY | TH17 | INSIGHTS | Phosphotransferases (Alcohol Group Acceptor) - immunology | STAT Transcription Factors - metabolism | STAT Transcription Factors - immunology | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Interleukin-2 - immunology | T-Lymphocytes, Helper-Inducer - immunology | T-Lymphocytes, Helper-Inducer - enzymology | Receptors, Antigen, T-Cell - immunology | Transcription Factors - immunology | Carrier Proteins - immunology | Trans-Activators - immunology | Transforming Growth Factor beta - immunology | Receptors, Antigen, T-Cell - metabolism | Phosphotransferases (Alcohol Group Acceptor) - genetics | Transcription Factors - genetics | Mice, Knockout | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Transcription Factors - metabolism | Carrier Proteins - genetics | Cell Differentiation - immunology | Animals | Carrier Proteins - metabolism | Trans-Activators - metabolism | Mice | TOR Serine-Threonine Kinases | Interleukin-2 - biosynthesis | Transforming Growth Factor beta - metabolism | T-Lymphocytes, Regulatory - enzymology | Genetic research | DNA binding proteins | Transforming growth factors | T cells | Proteins | Cell growth | Phosphorylation | T cell receptors | Kinases | Rodents
Journal Article
The Journal of cell biology, ISSN 1540-8140, 2009, Volume 187, Issue 6, pp. 859 - 874
Mammalian target of rapamycin (mTOR) is a key regulator of cell growth that associates with raptor and rictor to form the mTOR complex 1 (mTORC1) and mTORC2,... 
Birds of prey | Messenger RNA | Glycogen | Stem cells | Muscles | Mice | Gene expression regulation | Muscle fibers | Insulin | Skeletal muscle | MAMMALIAN TARGET | RAPAMYCIN | GLYCOGEN-SYNTHASE | CELLS | SIGNALING PATHWAYS | GLYCOPROTEIN COMPLEX | MUSCULAR-DYSTROPHY | ANIMAL-MODELS | DEFICIENT SKELETAL-MUSCLE | MDX MICE | CELL BIOLOGY | Muscular Dystrophy, Animal - genetics | Utrophin - metabolism | Age Factors | Muscular Dystrophy, Animal - physiopathology | Glycogen - metabolism | Muscle, Skeletal - drug effects | Female | Regulatory-Associated Protein of mTOR | Proto-Oncogene Proteins c-akt - metabolism | Dystrophin - metabolism | Rapamycin-Insensitive Companion of mTOR Protein | Severity of Illness Index | Muscle, Skeletal - enzymology | Mitochondria, Muscle - enzymology | Transduction, Genetic | Oxidation-Reduction | Mice, Inbred C57BL | Phosphotransferases (Alcohol Group Acceptor) - deficiency | Carrier Proteins - antagonists & inhibitors | Cells, Cultured | Rats | Electroporation | Phosphotransferases (Alcohol Group Acceptor) - genetics | Sirolimus - pharmacology | Mice, Knockout | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Carrier Proteins - genetics | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Adaptor Proteins, Signal Transducing | Animals | Carrier Proteins - metabolism | Muscle Contraction | Muscular Dystrophy, Animal - enzymology | Dystrophin - genetics | Energy Metabolism | Muscle, Skeletal - physiopathology | Glucose - metabolism | TOR Serine-Threonine Kinases | Enzyme Activation | Mutation | Glucose metabolism | Utrophin | Physiological aspects | Glucose | Dystrophin | Dextrose | Life Sciences
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2010, Volume 207, Issue 1, pp. 17 - 27
Lymphocyte egress from lymph nodes (LNs) is dependent on sphingosine-1-phosphate (S1P), but the cellular source of this S1P is not defined. We generated mice... 
MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | ZEBRAFISH | VASCULATURE | SPHINGOSINE-1-PHOSPHATE | LYMPHANGIOGENESIS | IMMUNOLOGY | RECEPTOR-1 | EXPRESSION | T-CELLS | 1-PHOSPHATE | BLOOD | GTP-Binding Protein alpha Subunits, Gi-Go - genetics | Lysophospholipids - metabolism | Intercellular Junctions - enzymology | Phosphotransferases (Alcohol Group Acceptor) - immunology | Peyer's Patches - metabolism | Glycoproteins - metabolism | Intercellular Junctions - immunology | Cell Movement - genetics | Lysophospholipids - immunology | Cell Movement - immunology | GTP-Binding Protein alpha Subunits, Gi-Go - immunology | Lymph Nodes - enzymology | Signal Transduction - immunology | Lymphocytes - immunology | Sphingosine - metabolism | Pertussis Toxin - pharmacology | Glycoproteins - genetics | Phosphotransferases (Alcohol Group Acceptor) - genetics | Sphingosine - immunology | Signal Transduction - genetics | Lymph Nodes - immunology | Intercellular Junctions - genetics | Lymph Nodes - cytology | Mice, Knockout | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Endothelial Cells - immunology | Cell Movement - drug effects | Glycoproteins - immunology | Lysophospholipids - genetics | Peyer's Patches - cytology | Sphingosine - analogs & derivatives | Animals | Signal Transduction - drug effects | Lymphocytes - enzymology | Sphingosine - genetics | Mice | Peyer's Patches - immunology | Endothelial Cells - enzymology | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Brief Definitive Report
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2018, Volume 137, Issue 21, pp. 2256 - 2273
BACKGROUND:Myocardial metabolic impairment is a major feature in chronic heart failure. As the major coenzyme in fuel oxidation and oxidative phosphorylation... 
heart failure | LIFE-SPAN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ADULT HEART | PROTEIN | ACETYLATION | acetyl coenzyme A | glycolysis | HEART-FAILURE | CELL-DEATH | NAD | NAD(+) METABOLISM | cardiomyopathy, dilated | FAILING HEART | PERIPHERAL VASCULAR DISEASE | MICE | serum response factor | energy metabolism | nicotinamide-beta-riboside | Niacinamide - analogs & derivatives | Serum Response Factor - genetics | AMP-Activated Protein Kinases - metabolism | Gene Expression Profiling | Heart Failure - prevention & control | Cardiomyopathy, Dilated - drug therapy | Nicotinamide Phosphoribosyltransferase - metabolism | Cytokines - genetics | NAD - metabolism | Disease Models, Animal | Cytokines - metabolism | Myocytes, Cardiac - cytology | Niacinamide - therapeutic use | Rats | Mice, Transgenic | Phosphotransferases (Alcohol Group Acceptor) - genetics | Serum Response Factor - deficiency | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Nicotinamide Phosphoribosyltransferase - genetics | Animals | Myocytes, Cardiac - drug effects | Metabolome - drug effects | Piperidines - therapeutic use | Myocytes, Cardiac - metabolism | Acrylamides - therapeutic use | Citric Acid - metabolism | Mice | PPAR alpha - metabolism | Dietary Supplements | Niacinamide | Care and treatment | Research | Cardiomyopathy | Heart diseases | Risk factors | Life Sciences | Molecular biology | Biochemistry, Molecular Biology | Serum Response Factor | acetyl-coA | nicotinamide riboside | dilated cardiomyopathy
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2014, Volume 289, Issue 9, pp. 6120 - 6132
Journal Article
Biochemical journal, ISSN 0264-6021, 2012, Volume 447, Issue 1, pp. 149 - 157
Journal Article