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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 05/2013, Volume 110, Issue 21, pp. 8708 - 8713
Stress and glucocorticoid hormones regulate hippocampal neurogenesis, but the molecular mechanisms mediating these effects are poorly understood. Here we... 
Phosphorylation | Messenger RNA | Glucocorticoids | Antidepressants | Genetic variation | Rodents | Neurogenesis | Progenitor cells | Hippocampus | Depressive disorders | Neuroplasticity | Hypothalamus-pituitary-adrenal axis | Stem cells | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Receptors, Glucocorticoid - antagonists & inhibitors | Depression - pathology | Humans | Middle Aged | Stress, Physiological | Hedgehog Proteins - metabolism | Hydrocortisone - metabolism | Male | Receptors, Glucocorticoid - metabolism | RNA, Messenger - metabolism | Immediate-Early Proteins - metabolism | Cell Nucleus - metabolism | Cell Nucleus - pathology | Glucocorticoids - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Adult | Female | Hippocampus - enzymology | Protein-Serine-Threonine Kinases - metabolism | Nerve Tissue Proteins - antagonists & inhibitors | Rats | Hippocampus - pathology | Rats, Sprague-Dawley | Depression - enzymology | Nerve Tissue Proteins - metabolism | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Animals | Signal Transduction - drug effects | Active Transport, Cell Nucleus - drug effects | Benzoates - pharmacology | Mifepristone - pharmacology | Cell Line, Transformed | Immediate-Early Proteins - antagonists & inhibitors | Physiological aspects | Corticosteroids | Hippocampus (Brain) | Phosphotransferases | Depression, Mental | Brain | Molecules | Hormones | Mental depression | Gene expression | Stress | Index Medicus | Biological Sciences | stem cells | antidepressants | hypothalamus–pituitary–adrenal axis | neuroplasticity
Journal Article
Journal of diabetes research, ISSN 2314-6745, 4/2014, Volume 2014, pp. 391476 - 10
This study aimed to explore the effect of angiotensin (1–7) (Ang (1–7)) on palmitate-induced apoptosis in islet endothelial cells and the mechanism of action.... 
Endocrinology & Metabolism | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Islets of Langerhans - drug effects | Antihypertensive Agents - pharmacology | Receptors, G-Protein-Coupled - metabolism | Antihypertensive Agents - antagonists & inhibitors | Apoptosis - drug effects | Microvessels - metabolism | Endothelium, Vascular - drug effects | Protective Agents - chemistry | Nitric Oxide Synthase Type III - chemistry | Fatty Acids, Nonesterified - metabolism | Microvessels - enzymology | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Islets of Langerhans - blood supply | Angiotensin II - pharmacology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Peptide Fragments - antagonists & inhibitors | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Receptors, G-Protein-Coupled - antagonists & inhibitors | Fatty Acids, Nonesterified - antagonists & inhibitors | MAP Kinase Kinase 4 - chemistry | Mice | p38 Mitogen-Activated Protein Kinases - chemistry | Palmitic Acid - metabolism | Peptide Fragments - pharmacology | Receptors, G-Protein-Coupled - agonists | Endothelium, Vascular - enzymology | Palmitic Acid - antagonists & inhibitors | MAP Kinase Kinase 4 - metabolism | Islets of Langerhans - metabolism | Protective Agents - pharmacology | Proto-Oncogene Proteins - agonists | MAP Kinase Kinase 4 - antagonists & inhibitors | Angiotensin I - pharmacology | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Microvessels - drug effects | Angiotensin II - analogs & derivatives | Enzyme Activation - drug effects | Animals | MAP Kinase Signaling System - drug effects | Proto-Oncogene Proteins c-akt - agonists | Protein Kinase Inhibitors - pharmacology | Cell Line, Transformed | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Angiotensin I - antagonists & inhibitors | Proteins | Flow cytometry | Enzymes | Biotechnology | Phosphorylation | Statistical analysis | Diabetes | Experiments | Deoxyribonucleic acid--DNA | Apoptosis | Index Medicus
Journal Article
Biochemical and biophysical research communications, ISSN 0006-291X, 08/2015, Volume 464, Issue 1, pp. 221 - 228
Adult T cell leukemia and lymphoma (ATL) is a highly aggressive form of hematological malignancy and is caused by chronic infection of human T cell leukemia... 
Niclosamide | HTLV-1 | Tax | Down-regulation | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Gene Expression Regulation, Viral - drug effects | Gene Products, tax - antagonists & inhibitors | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Myeloid Cell Leukemia Sequence 1 Protein - metabolism | Gene Products, tax - genetics | Host-Pathogen Interactions - drug effects | Proteolysis - drug effects | Proto-Oncogene Proteins c-bcl-2 - metabolism | Proteasome Endopeptidase Complex - drug effects | Gene Products, tax - metabolism | I-kappa B Kinase - metabolism | I-kappa B Kinase - antagonists & inhibitors | Niclosamide - pharmacology | Mitogen-Activated Protein Kinase 1 - genetics | Human T-lymphotropic virus 1 - drug effects | Human T-lymphotropic virus 1 - genetics | Gene Expression Regulation, Neoplastic - drug effects | Antinematodal Agents - pharmacology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Myeloid Cell Leukemia Sequence 1 Protein - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | T-Lymphocytes | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | I-kappa B Kinase - genetics | Myeloid Cell Leukemia Sequence 1 Protein - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Human T-lymphotropic virus 1 - metabolism | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Proteasome Endopeptidase Complex - metabolism | STAT3 Transcription Factor - antagonists & inhibitors | Proto-Oncogene Proteins c-bcl-2 - genetics | Cell Line, Transformed | Apoptosis | Mitogen-Activated Protein Kinase 1 - metabolism | Pharmacy | Leukemia | Genomics | Drugstores | Antiparasitic agents | Lymphomas | Taxation | T cells | Health aspects | Phosphotransferases | Index Medicus | HUMAN POPULATIONS | APOPTOSIS | BORON CHLORIDES | TAXES | TRANSCRIPTION | SIGNALS | 60 APPLIED LIFE SCIENCES | MOLECULES | LYMPHOMAS | LEUKEMIA VIRUSES | GENES | PHOSPHOTRANSFERASES | LEUKEMIA | TRANSFORMATIONS | LYMPHOCYTES | PLANT GROWTH
Journal Article
PLoS pathogens, ISSN 1553-7374, 07/2013, Volume 9, Issue 7, pp. e1003499 - e1003499
Defining the mechanisms of Mycobacterium tuberculosis (Mtb) persistence in the host macrophage and identifying mycobacterial factors responsible for it are... 
Parasitology | Life Sciences & Biomedicine | Microbiology | Science & Technology | Virology | Tuberculosis, Pulmonary - microbiology | Phagosomes - enzymology | Bone Marrow Cells - enzymology | Humans | Virulence | Mycobacterium tuberculosis - immunology | NADPH Oxidases - metabolism | Nucleoside-Diphosphate Kinase - metabolism | Mycobacterium tuberculosis - pathogenicity | Bone Marrow Cells - immunology | Mycobacterium tuberculosis - physiology | Female | Phagosomes - ultrastructure | Tuberculosis, Pulmonary - enzymology | Neuropeptides - genetics | Mutant Proteins - antagonists & inhibitors | Macrophages - immunology | Macrophages - microbiology | Nucleoside-Diphosphate Kinase - genetics | Recombinant Proteins - metabolism | Bacterial Proteins - antagonists & inhibitors | Macrophages - ultrastructure | Oligoribonucleotides, Antisense | Bone Marrow Cells - cytology | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Bacterial Proteins - genetics | Cells, Cultured | Mutant Proteins - genetics | Recombinant Proteins - chemistry | Mutant Proteins - metabolism | Neuropeptides - metabolism | Tuberculosis, Pulmonary - pathology | Immunity, Innate | Mice, SCID | Nucleoside-Diphosphate Kinase - antagonists & inhibitors | Macrophages - enzymology | Tuberculosis, Pulmonary - immunology | Animals | Mycobacterium tuberculosis - enzymology | rac1 GTP-Binding Protein - antagonists & inhibitors | Bone Marrow Cells - microbiology | Bacterial Proteins - metabolism | Mice | Cell Line, Transformed | rac1 GTP-Binding Protein - metabolism | Neuropeptides - antagonists & inhibitors | rac1 GTP-Binding Protein - genetics | Mycobacterium tuberculosis | Physiological aspects | Host-parasite relationships | Research | Health aspects | Phosphotransferases | Guanosine triphosphatase | Index Medicus | Medical research | Tuberculosis | Biosynthesis | Kinases | Charitable foundations | Experiments | Apoptosis
Journal Article