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International journal of cancer, ISSN 0020-7136, 08/2008, Volume 123, Issue 3, pp. 552 - 560
Genistein is a phytoestrogen that has been reported to suppress the AKT signaling pathway in several malignancies. However, the molecular mechanism of... 
Prostate cancer | Genistein | Tumor suppressor gene | Life Sciences & Biomedicine | Oncology | Science & Technology | PTEN Phosphohydrolase - drug effects | Prostatic Neoplasms - metabolism | Deubiquitinating Enzyme CYLD | CpG Islands - drug effects | Humans | Male | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Genes, Tumor Suppressor - drug effects | Genes, p53 - drug effects | Prostatic Neoplasms - genetics | Chromatin Immunoprecipitation | Forkhead Transcription Factors - metabolism | Sirtuin 1 | Antimetabolites, Antineoplastic - pharmacology | Electrophoretic Mobility Shift Assay | Gene Expression Regulation, Neoplastic - drug effects | Chromones - pharmacology | Anticarcinogenic Agents - pharmacology | Hydroxamic Acids - pharmacology | Phytoestrogens - pharmacology | Prostatic Neoplasms - drug therapy | PTEN Phosphohydrolase - genetics | Tumor Suppressor Proteins - metabolism | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Azacitidine - analogs & derivatives | Down-Regulation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Histones - drug effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Azacitidine - pharmacology | Acetylation - drug effects | Methylation - drug effects | Genistein - pharmacology | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Histones - metabolism | Forkhead Box Protein O3 | Sirtuins - metabolism | NF-kappa B - drug effects | Index Medicus
Journal Article
Toxicology and applied pharmacology, ISSN 0041-008X, 06/2017, Volume 325, pp. 61 - 70
Estrogen receptors (ERs) α and β are distributed in most tissues of women and men. ERs are bound by estradiol (E2), a natural hormone, and mediate the... 
PC12 | Phytoestrogens | SERM | Proliferation | Differentiation | Breast cancer cell lines | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Adrenal Gland Neoplasms - drug therapy | Transcription, Genetic - drug effects | Humans | Isoflavones - pharmacology | Stilbenes - pharmacology | PC12 Cells | Breast Neoplasms - metabolism | Dose-Response Relationship, Drug | Chemokine CXCL12 - genetics | Transfection | MCF-7 Cells | Pheochromocytoma - pathology | Estrogen Receptor alpha - metabolism | Female | Neurogenesis - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Adrenal Gland Neoplasms - pathology | Neurites - drug effects | Phytoestrogens - pharmacology | Pheochromocytoma - metabolism | Response Elements | Rats | Neurites - metabolism | Breast Neoplasms - drug therapy | Estrogen Receptor alpha - drug effects | Nerve Tissue Proteins - metabolism | Neurites - pathology | Pheochromocytoma - drug therapy | Animals | Breast Neoplasms - genetics | Chemokine CXCL12 - metabolism | Estrogen Receptor alpha - genetics | Diet | Breast Neoplasms - pathology | Adrenal Gland Neoplasms - metabolism | Pheochromocytoma - genetics | Cell Proliferation - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Adrenal Gland Neoplasms - genetics | Selective Estrogen Receptor Modulators - pharmacology | Apigenin - pharmacology | Zearalenone - pharmacology | Analysis | Resveratrol | Phenols | Nerve growth factor | Hormones | Cell differentiation | Estradiol | Isoflavones | Therapeutics | Homeopathy | Materia medica and therapeutics | Index Medicus | Life Sciences | Pharmacology | Pharmaceutical sciences | PUBLIC HEALTH | GROWTH FACTORS | MAMMARY GLANDS | NEOPLASMS | NERVES | ESTRADIOL | PHENOTYPE | CELL PROLIFERATION | ANIMAL TISSUES | RECEPTORS | 60 APPLIED LIFE SCIENCES | BRAIN
Journal Article
Toxicology and applied pharmacology, ISSN 0041-008X, 2009, Volume 236, Issue 1, pp. 85 - 96
Several anthropogenous and naturally occurring substances, referred to as estrogen active compounds (EACs), are able to interfere with hormone and in... 
Endocrine active compound | Estrogen receptor | Gene expression | Ishikawa cells | Illumina | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Estradiol - analogs & derivatives | Receptors, Estrogen - metabolism | Estrogens - pharmacology | Oligonucleotide Array Sequence Analysis | Humans | Endometrial Neoplasms - metabolism | Estrogen Antagonists - pharmacology | Stilbenes - pharmacology | RNA, Messenger - metabolism | Diethylstilbestrol - pharmacology | Endocrine Disruptors - pharmacology | Endometrial Neoplasms - genetics | Time Factors | Polymerase Chain Reaction | Female | Gene Expression Regulation, Neoplastic - drug effects | Estradiol - pharmacology | Phytoestrogens - pharmacology | Phenols - pharmacology | Reproducibility of Results | Receptors, Estrogen - genetics | Benzhydryl Compounds | Risk Assessment | Gene Expression Profiling - methods | Estrogens - toxicity | Endocrine Disruptors - toxicity | DDT - pharmacology | Receptors, Estrogen - drug effects | Genistein - pharmacology | Cell Line, Tumor | Cluster Analysis | Zearalenone - pharmacology | Endometrial cancer | Bisphenol-A | Resveratrol | Genetic research | Phenols | Diethylstilbestrol | Angiogenesis inhibitors | Hormones | Persistent organic pollutants | Isoflavones | Index Medicus | ESTROGENS | NEOPLASMS | DDT | DOSES | GENES | SCREENING | GENE REGULATION | RECEPTORS | 60 APPLIED LIFE SCIENCES
Journal Article
Neuroscience, ISSN 0306-4522, 05/2013, Volume 238, pp. 345 - 360
Highlights ► This study has demonstrated a key role of ERβ and GPR30 in the neuroprotective action of daidzein. ► Daidzein inhibited glutamate-induced... 
Neurology | phytoestrogen | apoptosis | membrane estrogen receptor | primary neuronal cell cultures | neurotoxicity | Phytoestrogen | Neurotoxicity | Membrane estrogen receptor | Apoptosis | Primary neuronal cell cultures | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Estradiol - analogs & derivatives | Receptors, G-Protein-Coupled - metabolism | Apoptosis - drug effects | Isoflavones - pharmacology | Caspase 3 - metabolism | Cerebellum - drug effects | Neurons - cytology | Hippocampus - drug effects | Membrane Potential, Mitochondrial - drug effects | Quinolines - pharmacology | Estrogen Receptor beta - metabolism | Neocortex - metabolism | Neocortex - drug effects | Neuroprotective Agents - pharmacology | Piperidines - pharmacology | Membrane Potential, Mitochondrial - physiology | Neurons - metabolism | Phosphorylation - drug effects | Neurons - drug effects | Estrogen Receptor beta - antagonists & inhibitors | Estradiol - pharmacology | Pyrazoles - pharmacology | Glutamic Acid - pharmacology | Receptors, Estrogen | Cerebellum - metabolism | Cells, Cultured | Hippocampus - cytology | Hippocampus - metabolism | Animals | Signal Transduction - drug effects | Cerebellum - cytology | Receptors, G-Protein-Coupled - antagonists & inhibitors | Neocortex - cytology | Signal Transduction - physiology | Mice | Apoptosis - physiology | Benzodioxoles - pharmacology | Index Medicus
Journal Article
Advanced drug delivery reviews, ISSN 0169-409X, 01/2009, Volume 61, Issue 1, pp. 14 - 25
Journal Article