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1. Full Text NLRP3 Inflammasome Inhibition by MCC950 Reduces Atherosclerotic Lesion Development in Apolipoprotein E-Deficient Mice - Brief Report
by Van Der Heijden, Thomas and Kritikou, Eva and Venema, Wouter and Van Duijn, Janine and Van Santbrink, Peter J and Slütter, Bram and Foks, Amanda C and Bot, Ilze and Kuiper, Johan
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 08/2017, Volume 37, Issue 8, pp. 1457 - 1461
atherosclerosis | macrophages | inflammasomes | interleukin-1beta | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Carotid Artery Diseases - prevention & control | Inflammasomes - metabolism | Apolipoproteins E - deficiency | Atherosclerosis - genetics | RNA, Messenger - metabolism | Sulfones - pharmacology | Heterocyclic Compounds, 4 or More Rings - pharmacology | Necrosis | Inflammasomes - antagonists & inhibitors | Time Factors | Carotid Artery Diseases - pathology | Interleukin-1beta - metabolism | Dendritic Cells - drug effects | Female | Vascular Cell Adhesion Molecule-1 - genetics | Carotid Artery Diseases - metabolism | Diet, Western | Dendritic Cells - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Genetic Predisposition to Disease | Anti-Inflammatory Agents - pharmacology | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Crystallization | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Intercellular Adhesion Molecule-1 - metabolism | Macrophages - metabolism | Phenotype | Animals | Signal Transduction - drug effects | Apolipoproteins E - genetics | Intercellular Adhesion Molecule-1 - genetics | Carotid Artery Diseases - genetics | Lipopolysaccharides - pharmacology | Cholesterol - pharmacology | Macrophages - drug effects | Carotid Arteries - pathology | Vascular Cell Adhesion Molecule-1 - metabolism | Atherosclerosis - prevention & control | Index Medicus
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2. Full Text Single-cell immune landscape of human atherosclerotic plaques
by Fernandez, Dawn M and Rahman, Adeeb H and Fernandez, Nicolas F and Chudnovskiy, Aleksey and Amir, El-ad David and Amadori, Letizia and Khan, Nayaab S and Wong, Christine K and Shamailova, Roza and Hill, Christopher A and Wang, Zichen and Remark, Romain and Li, Jennifer R and Pina, Christian and Faries, Christopher and Awad, Ahmed J and Moss, Noah and Bjorkegren, Johan L. M and Kim-Schulze, Seunghee and Gnjatic, Sacha and Ma'ayan, Avi and Mocco, J and Faries, Peter and Merad, Miriam and Giannarelli, Chiara
Nature medicine, ISSN 1078-8956, 10/2019, Volume 25, Issue 10, pp. 1576 - 1588
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Proteome - genetics | Atherosclerosis - genetics | Humans | Immunity, Innate - genetics | Male | Transcriptome - immunology | Interleukin-1beta - genetics | Leukocytes, Mononuclear | Adaptive Immunity - genetics | Cell Differentiation - genetics | Endarterectomy, Carotid | T-Lymphocytes - metabolism | Proteome - immunology | Female | Plaque, Atherosclerotic - pathology | Single-Cell Analysis | Macrophages - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Plaque, Atherosclerotic - metabolism | Interleukin-1beta - immunology | Signal Transduction - genetics | Transcriptome - genetics | Plaque, Atherosclerotic - immunology | Macrophages - metabolism | T-Lymphocytes - immunology | Aged | Usage | Care and treatment | Immunotherapy | Atherosclerosis | Genetic aspects | Research | Genetic transcription | T cells | Risk factors | Myocardial infarction | Cerebral infarction | Phenotypes | Stroke | Transient ischemic attack | Interleukin | Lymphocytes T | Cell differentiation | Macrophages | IL-1β | CD4 antigen | Ischemia | Lymphocytes | Exhaustion | Arteriosclerosis | Carotid artery | Plaques | Immune system | Index Medicus
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3. Full Text Suppressed monocyte recruitment drives macrophage removal from atherosclerotic plaques of Apoe(-/-) mice during disease regression
by Potteaux, S and Gautier, E.L and Hutchison, S.B and van Rooijen, N and Rader, D.J and Thomas, M.J and Sorci-Thomas, M.G and Randolph, GJ
The Journal of clinical investigation, ISSN 0021-9738, 2011, Volume 121, Issue 5, pp. 2025 - 2036
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Atherosclerosis - pathology | Flow Cytometry - methods | Plaque, Atherosclerotic - metabolism | Monocytes - cytology | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Male | Mice, Transgenic | Macrophages - cytology | Cholesterol - metabolism | Genetic Complementation Test | Atherosclerosis - metabolism | Macrophages - metabolism | Animals | Receptors, CCR7 - metabolism | Apolipoproteins E - genetics | Adenoviridae - genetics | Female | Mice | Physiological aspects | Development and progression | Monocytes | Research | Macrophages | Atherosclerotic plaque | Index Medicus | Abridged Index Medicus
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4. Full Text KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis
by Shankman, Laura S and Gomez, Delphine and Cherepanova, Olga A and Salmon, Morgan and Alencar, Gabriel F and Haskins, Ryan M and Swiatlowska, Pamela and Newman, Alexandra A C and Greene, Elizabeth S and Straub, Adam C and Isakson, Brant and Randolph, Gwendalyn J and Owens, Gary K
Nature medicine, ISSN 1078-8956, 06/2015, Volume 21, Issue 6, pp. 628 - 637
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Promoter Regions, Genetic | Atherosclerosis - pathology | Plaque, Atherosclerotic - genetics | Macrophages - pathology | Atherosclerosis - genetics | Humans | Myocytes, Smooth Muscle - pathology | Mesenchymal Stromal Cells - metabolism | Cell Tracking | Cell Differentiation - genetics | Cell Lineage | Animals | Apolipoproteins E - antagonists & inhibitors | Mice | Mesenchymal Stromal Cells - pathology | Plaque, Atherosclerotic - pathology | Kruppel-Like Transcription Factors - genetics | Atherosclerosis | Physiological aspects | Development and progression | Smooth muscle | Genetic aspects | Research | Macrophages | Studies | Cellular biology | Genotype & phenotype | Index Medicus
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5. Full Text Lack of myeloid Fatp1 increases atherosclerotic lesion size in Ldlr−/− mice
by Zhao, Liyang and Cozzo, Alyssa J and Johnson, Amy R and Christensen, Taylor and Freemerman, Alex J and Bear, James E and Rotty, Jeremy D and Bennett, Brian J and Makowski, Liza
Atherosclerosis, ISSN 0021-9150, 11/2017, Volume 266, pp. 182 - 189
FATP1 | Metabolic reprogram | Fatty acid transport protein | Macrophage | LDL receptor | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Inflammation - pathology | Oxidative Stress | Fatty Acid Transport Proteins - deficiency | Atherosclerosis - genetics | Male | Aorta - metabolism | Aortic Diseases - metabolism | Inflammation - metabolism | Lipids - blood | Bone Marrow Transplantation | Receptors, LDL - deficiency | Aortic Diseases - pathology | Disease Models, Animal | Receptors, LDL - genetics | Atherosclerosis - pathology | Genetic Predisposition to Disease | Cells, Cultured | Cellular Microenvironment | Plaque, Atherosclerotic | Transplantation Chimera | Atherosclerosis - metabolism | Macrophage Activation | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Phenotype | Animals | Inflammation - genetics | Blood Glucose - metabolism | Macrophages, Peritoneal - metabolism | Fatty Acid Transport Proteins - genetics | Index Medicus | macrophage | fatty acid transport protein | metabolic reprogram
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6. Full Text Atherosclerotic Plaque Destabilization Mechanisms, Models, and Therapeutic Strategies
by Silvestre-Roig, Carlos and de Winther, Menno P and Weber, Christian and Daemen, Mat J and Lutgens, Esther and Soehnlein, Oliver
Circulation research, ISSN 0009-7330, 2014, Volume 114, Issue 1, pp. 214 - 226
atherosclerosis | macrophages | neutrophils | necrotic core | efferocytosis | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Macrophages - physiology | Plaque, Atherosclerotic - metabolism | Anti-Inflammatory Agents - pharmacology | Humans | Neutrophils - drug effects | Neutrophils - physiology | Apolipoproteins E - metabolism | Macrophages - metabolism | Animals | Apolipoproteins E - genetics | Cell Death | Anti-Inflammatory Agents - therapeutic use | Macrophages - drug effects | Plaque, Atherosclerotic - drug therapy | Neutrophils - metabolism | Disease Models, Animal | Index Medicus
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7. Full Text Downregulated microRNA‐224 aggravates vulnerable atherosclerotic plaques and vascular remodeling in acute coronary syndrome through activation of the TGF‐β/Smad pathway
by Xu, Hai‐Ming and Sui, Feng‐Hua and Sun, Mei‐Hua and Guo, Gong‐Liang
Journal of cellular physiology, ISSN 0021-9541, 03/2019, Volume 234, Issue 3, pp. 2537 - 2551
microRNA‐224 | acute coronary syndrome | vascular remodeling | vulnerable atherosclerotic plaque | TGF‐β/Smad pathway | microRNA-224 | TGF-β/Smad pathway | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Myocardial Infarction - genetics | Plaque, Atherosclerotic - genetics | Gene Expression Regulation - genetics | Humans | Rats | Smad Proteins - genetics | Signal Transduction - genetics | Matrix Metalloproteinase 2 - genetics | Animals | Transforming Growth Factor beta - genetics | Vascular Remodeling - genetics | Acute Coronary Syndrome - pathology | Myocardial Infarction - pathology | Vascular Cell Adhesion Molecule-1 - genetics | MicroRNAs - genetics | Acute Coronary Syndrome - genetics | Plaque, Atherosclerotic - pathology | Disease Models, Animal | Myocardial infarction | Heart | Smad protein | Endothelin | Body weight | Lipids | Activation | Smad3 protein | Remodeling | Smad4 protein | Blood | Proteins | Smad2 protein | Atherosclerosis | miRNA | Plaques | Therapeutic applications | Inflammation | Ribonucleic acid--RNA | Gene expression | Cholesterol | Nitric oxide | Arteriosclerosis | Collagen | Protein expression | Smad7 protein | Infarction | Acute coronary syndromes | Index Medicus
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8. Full Text Atherosclerotic dyslipidemia revealed by plasma lipidomics on ApoE −/− mice fed a high-fat diet
by Chen, Yanyan and Wen, Shiyuan and Jiang, Miaomiao and Zhu, Yan and Ding, Liqin and Shi, Hong and Dong, Pengzhi and Yang, Jing and Yang, Yue
Atherosclerosis, ISSN 0021-9150, 2017, Volume 262, pp. 78 - 86
Cardiovascular | Sphingolipid | ApoE | Glycerophospholipid | Atherosclerosis | Dyslipidemia | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Dyslipidemias - genetics | Atherosclerosis - genetics | Male | Chromatography, High Pressure Liquid | Dyslipidemias - blood | Diet, High-Fat | Lipids - blood | Mass Spectrometry | Disease Models, Animal | Atherosclerosis - pathology | Genetic Predisposition to Disease | Phosphatidylcholines - blood | Mice, Inbred C57BL | Biomarkers - blood | Mice, Knockout, ApoE | Plaque, Atherosclerotic | Metabolomics - methods | Disease Progression | Discriminant Analysis | Atherosclerosis - blood | Phenotype | Sphingosine - analogs & derivatives | Animals | Sphingomyelins - blood | Sphingosine - blood | Pattern Recognition, Automated | Index Medicus
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