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Journal of Clinical Endocrinology and Metabolism, ISSN 0021-972X, 05/2017, Volume 102, Issue 5, pp. 1642 - 1651
Context: Low-grade inflammation is associated with obesity and the metabolic syndrome (MetS). Preclinical evidence suggests that resveratrol (RSV) has... 
SUPPLEMENTATION | OBESE MEN | ALKALINE-PHOSPHATASE | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | SECRETION | HEALTH | CORONARY-ARTERY-DISEASE | CHALLENGES | Blood Pressure | Body Composition | Stilbenes - therapeutic use | Humans | Leptin - metabolism | Middle Aged | Male | Metabolic Syndrome - metabolism | Muscle, Skeletal - metabolism | Adipose Tissue - immunology | Metabolic Syndrome - drug therapy | Fructosamine - metabolism | Adipose Tissue - metabolism | Muscle, Skeletal - immunology | Liver - diagnostic imaging | Quadriceps Muscle - immunology | Metabolic Syndrome - immunology | Real-Time Polymerase Chain Reaction | Double-Blind Method | Magnetic Resonance Spectroscopy | Cholesterol, HDL - metabolism | Liver - metabolism | Insulin Resistance | Cholesterol - metabolism | Intra-Abdominal Fat - metabolism | Absorptiometry, Photon | Blotting, Western | Triglycerides - metabolism | Receptors, Urokinase Plasminogen Activator - metabolism | Intra-Abdominal Fat - diagnostic imaging | Magnetic Resonance Imaging | Antioxidants - therapeutic use | Insulin - metabolism | Muscle, Skeletal - diagnostic imaging | Interleukin-6 - immunology | Cholesterol, LDL - metabolism | Blood Glucose - metabolism | Quadriceps Muscle - metabolism | C-Reactive Protein - immunology | C-reactive protein | Pressure effects | Liver | Interleukin | Medical services | Homeostasis | Clinical trials | Lipids | Standard error | Glucose | Metabolic syndrome | Composition effects | Blood | Body composition | Interleukin 6 | Body mass index | Urokinase | Glucose metabolism | Randomization | Body composition (biology) | Resveratrol | Blood pressure | Bioindicators | Lipid metabolism | Supplementation | Deposition | Middle age | Dietary supplements | Muscles | Inflammation | Gene expression | Metabolism | Low density lipoprotein | Cholesterol | Skeletal muscle | U-Plasminogen activator | Body mass | Biopsy | Body size | Men | Plasma levels | Metabolic disorders | Index Medicus | Abridged Index Medicus
Journal Article
Aging Cell, ISSN 1474-9718, 08/2015, Volume 14, Issue 4, pp. 644 - 658
The healthspan of mice is enhanced by killing senescent cells using a transgenic suicide gene. Achieving the same using small molecules would have a tremendous... 
dasatinib | plasminogen‐activated inhibitor | dependence receptors | quercetin | ephrins | 3K delta | p21 | Dasatinib | Dependence receptors | Ephrins | Plasminogen-activated inhibitor | Quercetin | PI3K delta | P21 | ENDOTHELIAL DYSFUNCTION | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | EXPRESSION PROFILES | plasminogen-activated inhibitor | CELLULAR SENESCENCE | CANCER-THERAPY | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | LUNG-CANCER | SET ENRICHMENT ANALYSIS | GENE-EXPRESSION | IONIZING-RADIATION | TUMOR-GROWTH | Carotid Arteries - drug effects | Endonucleases - genetics | Plasminogen Activator Inhibitor 2 - genetics | Transcriptome | Gene Expression Profiling | Adipocytes - drug effects | Aging - genetics | Osteoporosis - metabolism | Ephrins - metabolism | Plasminogen Activator Inhibitor 2 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Osteoporosis - genetics | Intervertebral Disc - pathology | Fibroblasts - metabolism | Endothelial Cells - metabolism | Intervertebral Disc - chemistry | Fibroblasts - pathology | Mesenchymal Stem Cells - pathology | Mice, Knockout | Dasatinib - pharmacology | Osteoporosis - pathology | Ephrins - genetics | Fibroblasts - drug effects | Mice | Endothelial Cells - pathology | bcl-X Protein - metabolism | Aging - metabolism | Aging - drug effects | bcl-X Protein - genetics | Cellular Senescence - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Endonucleases - metabolism | DNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Mesenchymal Stem Cells - drug effects | Mesenchymal Stem Cells - metabolism | Heart - physiopathology | Cellular Senescence - genetics | Osteoporosis - prevention & control | DNA-Binding Proteins - genetics | Adipocytes - pathology | Aging - pathology | Phosphatidylinositol 3-Kinases - genetics | Animals | Quercetin - pharmacology | Adipocytes - metabolism | Heart - drug effects | Carotid Arteries - pathology | Intervertebral Disc - drug effects | Drug Combinations | Endothelial Cells - drug effects | Index Medicus | Original
Journal Article
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 04/2010, Volume 103, Issue 4, pp. 788 - 796
CD40L figures prominently in atherogenesis. Recent data demonstrate elevated levels of sCD40L in the serum of patients with the metabolic syndrome (MS). This... 
metabolic syndrome | CD40L | preadipocytes | inflammation | adipocytes | Cardiovascular Biology and Cell Signalling | Preadipocytes | Inflammation | Adipocytes | Metabolic syndrome | ACTIVATOR INHIBITOR TYPE-1 | ENHANCED SOLUBLE CD40L | ACUTE CORONARY SYNDROMES | KAPPA-B | HUMAN ADIPOCYTES | DIABETES-MELLITUS | OBESITY | INSULIN-RESISTANCE | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ARTERY-DISEASE | Obesity - immunology | CD40 Ligand - blood | Humans | Metabolic Syndrome - metabolism | NF-kappa B - metabolism | PPAR gamma - metabolism | RNA, Messenger - metabolism | Cardiovascular Diseases - genetics | Obesity - genetics | Case-Control Studies | Inflammation - metabolism | CD40 Antigens - metabolism | CD40 Ligand - metabolism | Plasminogen Activator Inhibitor 1 - metabolism | Time Factors | Interleukin-6 - blood | Inflammation Mediators - metabolism | Chemokine CCL2 - metabolism | Metabolic Syndrome - immunology | Interleukin-8 - metabolism | Adipocytes - immunology | Adipogenesis | CCAAT-Enhancer-Binding Proteins - metabolism | Recombinant Proteins - metabolism | Cardiovascular Diseases - metabolism | Signal Transduction | Endothelial Cells - metabolism | Inflammation - immunology | 3T3-L1 Cells | Obesity - metabolism | Endothelial Cells - immunology | Cardiovascular Diseases - immunology | Animals | CD40 Ligand - genetics | Metabolic Syndrome - genetics | Adipocytes - metabolism | Inflammation - genetics | Mice | Culture Media, Conditioned - metabolism | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Journal Article
Journal of Surgical Research, ISSN 0022-4804, 2014, Volume 192, Issue 2, pp. 268 - 275
Journal Article
Cell Metabolism, ISSN 1550-4131, 07/2011, Volume 14, Issue 1, pp. 67 - 79
Imbalances in glucose and energy homeostasis are at the core of the worldwide epidemic of obesity and diabetes. Here, we illustrate an important role of the... 
GROWTH-FACTOR-BETA | TARGETED DISRUPTION | WHITE FAT | TGF-BETA | GENETIC-CONTROL | TRANSFORMING GROWTH-FACTOR-BETA-1 | ENDOCRINOLOGY & METABOLISM | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | ADIPOCYTE DIFFERENTIATION | BROWN ADIPOSE-TISSUE | DIET-INDUCED OBESITY | CELL BIOLOGY | Adipose Tissue, White - metabolism | Smad3 Protein - metabolism | DNA-Binding Proteins - metabolism | Smad3 Protein - deficiency | Smad3 Protein - genetics | Mitochondria - genetics | Adipose Tissue, White - physiology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Antibodies - immunology | Transforming Growth Factor beta - antagonists & inhibitors | Transforming Growth Factor beta - immunology | Glucose Tolerance Test | Signal Transduction | Adipose Tissue, Brown - physiology | Diabetes Mellitus - metabolism | Mitochondria - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Diabetes Mellitus - prevention & control | Animals | Energy Metabolism | Obesity - prevention & control | Mice, Obese | Trans-Activators - metabolism | Adipose Tissue, Brown - metabolism | Mice | Mitochondria - physiology | Transforming Growth Factor beta - metabolism | Blood glucose | Obesity | Football (American) | Muscles | Strategy | Diabetes | Fats | Respiration | Animal subjects | Index Medicus
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 11/2013, Volume 49, Issue 5, pp. 751 - 758
Plasminogen has a role in airway inflammation. Airway smooth muscle (ASM) cells cleave plasminogen into plasmin, a protease with proinflammatory activity. In... 
Interleukin-8 | Plasmin | Asthma | Interleukin-6 | S100A10 | SEVERE ASTHMA | ACTIVATION | CANCER CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | asthma | INDUCTION | interleukin-8 | interleukin-6 | CELL BIOLOGY | MURINE MODEL | ACUTE EXACERBATIONS | PATHWAY | RESPIRATORY SYSTEM | plasmin | GROWTH | EXPRESSION | Humans | Annexin A2 - deficiency | Bronchoalveolar Lavage Fluid - immunology | Plasminogen - metabolism | Muscle, Smooth - immunology | RNA, Messenger - metabolism | Time Factors | Inflammation Mediators - metabolism | Fibroblast Growth Factor 2 - metabolism | Pneumonia - immunology | Interleukin-8 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Cytokines - genetics | Fibrinolysin - metabolism | Interleukin-6 - metabolism | Myocytes, Smooth Muscle - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Respiratory System - immunology | alpha-2-Antiplasmin - metabolism | Disease Models, Animal | Cytokines - metabolism | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Muscle, Smooth - metabolism | Mice, Knockout | Animals | Annexin A2 - genetics | Urokinase-Type Plasminogen Activator - metabolism | Respiratory System - metabolism | Myocytes, Smooth Muscle - immunology | Mice | Pneumonia - metabolism | Annexin A2 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Aging Cell, ISSN 1474-9718, 08/2017, Volume 16, Issue 4, pp. 837 - 846
Kallistatin, an endogenous protein, protects against vascular injury by inhibiting oxidative stress and inflammation in hypertensive rats and enhancing the... 
34a | sirtuin 1 | RNA | micro | kallistatin | vascular senescence | aging | oxidative stress | microRNA-34a | NECROSIS-FACTOR-ALPHA | TISSUE KALLIKREIN INHIBITOR | BINDING PROTEIN | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | ORGAN INJURY | CAENORHABDITIS-ELEGANS | PROTECTIVE ROLE | PROGENITOR-CELL SENESCENCE | ENDOTHELIAL-CELLS | RENAL INJURY | Sirtuin 1 - metabolism | Diabetes Mellitus, Experimental - drug therapy | Endothelial Progenitor Cells - cytology | MicroRNAs - antagonists & inhibitors | Streptozocin | Humans | Diabetes Mellitus, Experimental - genetics | NADPH Oxidases - metabolism | Caenorhabditis elegans Proteins - metabolism | Cellular Senescence - drug effects | Male | MicroRNAs - metabolism | Sirtuin 1 - genetics | Telomerase - genetics | Plasminogen Activator Inhibitor 1 - metabolism | Superoxides - metabolism | beta-Galactosidase - metabolism | NADPH Oxidases - genetics | Telomerase - metabolism | Diabetes Mellitus, Experimental - chemically induced | Superoxides - antagonists & inhibitors | Nitric Oxide Synthase Type III - metabolism | Plasminogen Activator Inhibitor 1 - genetics | Diabetes Mellitus, Experimental - metabolism | Sirtuins - genetics | Endothelial Progenitor Cells - metabolism | Cellular Senescence - genetics | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - genetics | Catalase - genetics | Endothelial Progenitor Cells - drug effects | Cells, Cultured | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Nitric Oxide Synthase Type III - genetics | Serpins - pharmacology | Catalase - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Animals | Caenorhabditis elegans - drug effects | Genistein - pharmacology | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Mice | MicroRNAs - genetics | beta-Galactosidase - genetics | Caenorhabditis elegans Proteins - genetics | Sirtuins - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Gene expression | MicroRNA | Oxidative stress | Senescence | Genistein | INK4a protein | Kinases | NAD(P)H oxidase | Catalase | Rodents | Aging | Aorta | Telomerase | Protein-tyrosine kinase | Tyrosine | Enzymes | Diabetes mellitus | p16 Protein | MiRNA | Superoxide | SIRT1 protein | Life span | Tumor necrosis factor | MicroRNAs | Stem cells | Nematodes | Diabetes | Index Medicus | microRNA‐34a | Original
Journal Article
BMC Nephrology, ISSN 1471-2369, 01/2017, Volume 18, Issue 1, pp. 35 - 35
Background: During chronic kidney disease progression, kidney-specific risk factors for cardiovascular disease come into play. The present study investigated... 
Prothrombotic state | Hemostatic disorder | Indoxyl sulfate | von Willebrand factor | Tissue factor | Tryptophan derivatives | Uremic toxin | Cardiovascular disease | Chronic kidney disease | Monocytes activation | MORTALITY | KAPPA-B ACTIVATION | OXIDATIVE STRESS | RISK-FACTORS | KYNURENINE PATHWAY | ARYL-HYDROCARBON RECEPTOR | INFLAMMATION | ENDOTHELIAL-CELLS | UROLOGY & NEPHROLOGY | CARDIORENAL SYNDROME | EXPRESSION | Prevalence | C-Reactive Protein | Oxidative Stress | Tissue Plasminogen Activator - metabolism | Humans | Middle Aged | Male | Case-Control Studies | Renal Insufficiency, Chronic - epidemiology | Renal Insufficiency, Chronic - metabolism | Plasminogen Activator Inhibitor 1 - metabolism | Hemostasis | Lipoproteins - metabolism | Cardiovascular Diseases - epidemiology | Adult | Female | Prothrombin - metabolism | von Willebrand Factor - metabolism | Peptide Fragments - metabolism | Cardiovascular Diseases - metabolism | Inflammation | Hydrogen Peroxide - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Urokinase-Type Plasminogen Activator - metabolism | Indican - metabolism | Aged | Thrombin - metabolism | Thromboplastin - metabolism | Neopterin - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Physiological aspects | Toxins | Chronic kidney failure | Cardiovascular diseases | Health aspects | Index Medicus
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 02/2016, Volume 120, Issue 4, pp. 377 - 390
Sulforaphane (SFN), an activator of NF-E2-related factor 2 (Nrf2), has been found to have an antifibrotic effect on liver and lung. However, its effects on... 
TGF-β/Smad | Nrf2 | Duchenne muscular dystrophy | Sulforaphane | Fibrosis | DUCHENNE-MUSCULAR-DYSTROPHY | SPORT SCIENCES | sulforaphane | PHYSIOLOGY | MESSENGER-RNA EXPRESSION | MOUSE | CELL THERAPY | HALOFUGINONE | SKELETAL-MUSCLE | GROWTH-FACTOR-BETA | PATHWAY | TGF-beta/Smad | TRANSFORMING GROWTH-FACTOR-BETA-1 | DISEASE | fibrosis | Tumor Necrosis Factor-alpha - metabolism