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American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2015, Volume 308, Issue 4, pp. L344 - L357
Pathological fibrosis is driven by a feedback loop in which the fibrotic extracellular matrix is both a cause and consequence of fibroblast activation.... 
Plasminogen activator inhibitor 1 | Extracellular matrix | Mechanotransduction | Hippo | Idiopathic pulmonary fibrosis | LUNG | DOMAIN | PHYSIOLOGY | mechanotransduction | HIPPO PATHWAY | idiopathic pulmonary fibrosis | TRANSCRIPTIONAL COACTIVATOR | plasminogen activator inhibitor 1 | GROWTH-FACTOR-BETA | MECHANICAL MEMORY | MATRIX STIFFNESS | INHIBITION | RESPIRATORY SYSTEM | DIFFERENTIATION | IDIOPATHIC PULMONARY-FIBROSIS | extracellular matrix | NIH 3T3 Cells | Humans | Male | Intracellular Signaling Peptides and Proteins - metabolism | Pulmonary Fibrosis - genetics | Phosphoproteins - metabolism | Serpin E2 - biosynthesis | Gene Knockdown Techniques | Female | Lung - metabolism | Pulmonary Fibrosis - metabolism | Plasminogen Activator Inhibitor 1 - genetics | Intracellular Signaling Peptides and Proteins - genetics | Mechanotransduction, Cellular - genetics | Fibroblasts - metabolism | Lung - pathology | Pulmonary Fibrosis - pathology | Phosphoproteins - genetics | Fibroblasts - pathology | Plasminogen Activator Inhibitor 1 - biosynthesis | Serpin E2 - genetics | Animals | Transforming Growth Factor beta - genetics | Adaptor Proteins, Signal Transducing - genetics | Mice, Inbred NOD | Mice | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Transforming Growth Factor beta - metabolism | Transcriptional coactivators | Fibroblasts | Genetic aspects | Pulmonary fibrosis | Health aspects | Oncogenes | Call for Papers
Journal Article
Journal Article
ONCOGENE, ISSN 0950-9232, 09/2012, Volume 31, Issue 37, pp. 4150 - 4163
MicroRNAs (miRNAs) as modulators of gene expression have been described to display both tumor-promoting and tumor-suppressive functions. Although their role... 
ACTIVATION | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | miR-520c | breast cancer | NF-kappaB | PROLIFERATION | MICRORNAS | ER-negative | CELL BIOLOGY | TGF-beta | INVASION | miR-373 | ONCOLOGY | EMBRYONIC STEM-CELLS | GENETICS & HEREDITY | BONE METASTASIS | GROWTH-FACTOR | UP-REGULATION | EXPRESSION | Receptors, Estrogen - metabolism | Receptors, Transforming Growth Factor beta - genetics | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | NF-kappa B - metabolism | Gene Expression Profiling | Receptors, Estrogen - deficiency | Transcription Factor RelA - genetics | Breast Neoplasms - metabolism | Parathyroid Hormone-Related Protein - biosynthesis | Angiopoietin-like 4 Protein | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Female | Protein-Serine-Threonine Kinases - metabolism | Angiopoietins - biosynthesis | Signal Transduction | Interleukin-8 - biosynthesis | Protein-Serine-Threonine Kinases - genetics | Parathyroid Hormone-Related Protein - metabolism | Neoplasm Metastasis - genetics | Plasminogen Activator Inhibitor 1 - biosynthesis | Breast Neoplasms - genetics | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Transcription Factor RelA - metabolism | Cell Line, Tumor | Interleukin-6 - biosynthesis | Transforming Growth Factor beta - metabolism | Tumor cells | Estrogen receptors | Breast cancer | Genomes | Inflammation | Transforming growth factor- beta | Gene expression | Lymph nodes | Metastases | Parathyroid hormone-related protein | plasminogen | Signal transduction | NF- Kappa B protein | Tumor suppressor genes | miRNA | RelA protein | Interleukin 8 | Tumors
Journal Article
Circulation Research, ISSN 0009-7330, 03/2005, Volume 96, Issue 5, pp. e48 - e57
The vascular endothelium maintains blood fluidity by inhibiting blood coagulation, inhibiting platelet aggregation, and promoting fibrinolysis. Endothelial... 
endothelium | CATALYZED ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | transcription | HUMAN VASCULAR ENDOTHELIUM | PROTEIN-C | coagulation | TUMOR-NECROSIS-FACTOR | THROMBOMODULIN GENE | PLASMINOGEN-ACTIVATOR | thrombomodulin | SHEAR-STRESS | CELL COFACTOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | TRANSCRIPTION FACTOR | TISSUE FACTOR | Humans | DNA, Complementary - genetics | Recombinant Fusion Proteins - physiology | von Willebrand Factor - biosynthesis | von Willebrand Factor - genetics | Hemorheology | Nitric Oxide Synthase - genetics | Thrombomodulin - biosynthesis | Trans-Activators - physiology | RNA, Messenger - biosynthesis | Thrombin - pharmacology | Transfection | Cattle | Trans-Activators - genetics | Aorta | Nitric Oxide Synthase - biosynthesis | Trans-Activators - biosynthesis | Plasminogen Activator Inhibitor 1 - genetics | Thrombomodulin - genetics | Binding Sites | Promoter Regions, Genetic | Endothelial Cells - metabolism | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Nitric Oxide Synthase Type III | Gene Expression Regulation - physiology | Umbilical Veins | Interleukin-1 - pharmacology | Gene Expression Regulation - drug effects | Kruppel-Like Transcription Factors | Plasminogen Activator Inhibitor 1 - biosynthesis | Tumor Necrosis Factor-alpha - pharmacology | Thrombosis - metabolism | Transcription, Genetic - physiology | Animals | Blood Coagulation - genetics | Cells, Cultured - metabolism | Blood Coagulation - physiology | Thromboplastin - metabolism
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 09/2012, Volume 108, Issue 3, pp. 485 - 492
Plasminogen activator inhibitor type-1 (PAI-1) is one of the most important inhibitors of endogenous fibrinolysis. Adipose tissue is a suggested source of the... 
adipose tissue | PAI-1 | CABG | inflammation | Blood Coagulation, Fibrinolysis and Cellular Haemostasis | Inflammation | Adipose tissue | VISCERAL FAT | MYOCARDIAL-INFARCTION | NECROSIS-FACTOR-ALPHA | CARDIOPULMONARY BYPASS | INTERLEUKIN-6 RELEASE | FIBRINOLYTIC SYSTEM | HUMAN ADIPOCYTES | GROWTH-FACTOR-BETA | CONVERTING ENZYME-INHIBITION | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Heart Valve Prosthesis Implantation | Humans | Middle Aged | DNA, Complementary - genetics | Postoperative Complications - metabolism | Male | Tissue Plasminogen Activator - genetics | Angiotensin II Type 1 Receptor Blockers - pharmacology | Immunoenzyme Techniques | Adrenergic beta-Antagonists - pharmacology | RNA, Messenger - biosynthesis | Inflammation - metabolism | Subcutaneous Fat - metabolism | Postoperative Complications - genetics | Aged, 80 and over | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Plasminogen Activator Inhibitor 1 - genetics | Acute Disease | Interleukin-6 - genetics | RNA, Messenger - genetics | Omentum - metabolism | Gene Expression Regulation - drug effects | Plasminogen Activator Inhibitor 1 - biosynthesis | Abdominal Fat - metabolism | Tissue Plasminogen Activator - biosynthesis | Coronary Artery Bypass | Inflammation - genetics | Interleukin-6 - biosynthesis | Aged | Fibrinolysis - physiology | Biological Sciences | Immunology | Naturvetenskap | Immunologi | Biologiska vetenskaper | Natural Sciences
Journal Article
Journal Article
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 04/2005, Volume 288, Issue 4, pp. 731 - 740
The expression profile of a series of adipokine genes linked to inflammation has been examined by quantitative PCR during the differentiation of human... 
Nerve growth factor | White adipose tissue | Tumor necrosis factor-α | 3T3-L1 ADIPOCYTES | HAPTOGLOBIN GENE | PHYSIOLOGY | white adipose tissue | HUMAN PREADIPOCYTES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | METALLOTHIONEIN GENE-EXPRESSION | tumor necrosis factor-alpha | WHITE ADIPOSE-TISSUE | TUMOR-NECROSIS-FACTOR | GROWTH-HORMONE | nerve growth factor | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | Adipocytes - secretion | Haptoglobins - biosynthesis | Chemokine CCL2 - secretion | Humans | Metallothionein - biosynthesis | Middle Aged | Intercellular Signaling Peptides and Proteins - biosynthesis | Adipocytes - drug effects | Adipocytes - physiology | Adiponectin | Leptin - secretion | RNA, Messenger - biosynthesis | Angiotensinogen - biosynthesis | Adult | Female | Leptin - biosynthesis | Intercellular Signaling Peptides and Proteins - secretion | Adipocytes - immunology | Gene Expression | RNA, Messenger - genetics | Cytokines - secretion | Plasminogen Activator Inhibitor 1 - secretion | Nerve Growth Factor - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Interleukin-6 - secretion | Cell Differentiation - immunology | Plasminogen Activator Inhibitor 1 - biosynthesis | Tumor Necrosis Factor-alpha - pharmacology | Metallothionein - secretion | Angiotensinogen - secretion | Chemokine CCL2 - biosynthesis | Interleukin-6 - biosynthesis | Cytokines - biosynthesis | Haptoglobins - secretion
Journal Article
Oncogene, ISSN 0950-9232, 09/2012, Volume 31, Issue 37, pp. 4139 - 4149
The epidermal growth factor receptor (EGFR), a member of the ErbB family of receptor tyrosine kinases, is expressed in up to 70% of epithelial ovarian cancers... 
PAI-1 | epithelial ovarian cancer | NFkB | EGFR | IL-6 | CELLS | GROWTH-FACTOR-RECEPTOR | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR | KAPPA-B | CELL BIOLOGY | BREAST-CANCER | OVEREXPRESSION | PLASMINOGEN-ACTIVATOR | ONCOLOGY | GENETICS & HEREDITY | EXPRESSION | PROGRESSION | Cell Proliferation | Humans | ErbB Receptors - genetics | Neoplasms, Glandular and Epithelial - metabolism | Tumor Microenvironment | NF-kappa B - metabolism | Ovarian Neoplasms - mortality | Neoplasms, Glandular and Epithelial - mortality | Plasminogen Activator Inhibitor 1 - metabolism | RNA Interference | Female | Benzamides - pharmacology | Ovarian Neoplasms - metabolism | Interleukin-6 - metabolism | NF-kappa B - antagonists & inhibitors | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Signal Transduction | Tyrphostins - pharmacology | Cyclohexanones - pharmacology | Disease-Free Survival | Plasminogen Activator Inhibitor 1 - biosynthesis | Carcinoma, Ovarian Epithelial | Cell Line, Tumor | Interleukin-6 - biosynthesis | Biomarkers, Tumor - genetics | RNA, Small Interfering | Quinazolines - pharmacology | Physiological aspects | Development and progression | Research | Ovarian cancer | Interleukin-6 | Ligands | Oncology | Gene expression | Kinases | Immunohistochemistry | Prognosis | Epidermal growth factor receptors | Transcription | ErbB protein | Data processing | Inflammation | Survival | Interleukin 6 | Signal transduction | NF- Kappa B protein | Surgery | Plasminogen activator inhibitors | Protein-tyrosine kinase receptors | Microenvironments | Ascites | Protein-tyrosine kinase | Tumors
Journal Article
Journal Article