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1. Full Text Upregulation of TGF-β, FOXP3, and CD4 +CD25 + Regulatory T Cells Correlates with More Rapid Parasite Growth in Human Malaria Infection
by Walther, Michael and Tongren, Jon Eric and Andrews, Laura and Korbel, Daniel and King, Elizabeth and Fletcher, Helen and Andersen, Rikke F and Bejon, Philip and Thompson, Fiona and Dunachie, Susanna J and Edele, Fanny and de Souza, J. Brian and Sinden, Robert E and Gilbert, Sarah C and Riley, Eleanor M and Hill, Adrian V.S
Immunity, ISSN 1074-7613, 2005, Volume 23, Issue 3, pp. 287 - 296
Understanding the regulation of immune responses is central for control of autoimmune and infectious disease. In murine models of autoimmunity and chronic... 
IN-VITRO | SUPPRESSIVE ACTIVITY | POLYMERASE-CHAIN-REACTION | CYTOKINE PRODUCTION | TRANSCRIPTION FACTOR FOXP3 | CD4(+) | PERIPHERAL-BLOOD | IMMUNOLOGY | EX-VIVO | PLASMODIUM-FALCIPARUM MALARIA | CUTTING EDGE | Transforming Growth Factor beta - immunology | CD4 Antigens - immunology | Up-Regulation | DNA-Binding Proteins - immunology | Humans | T-Lymphocytes - parasitology | Clinical Trials as Topic | Plasmodium falciparum - immunology | Receptors, Interleukin-2 - immunology | Transforming Growth Factor beta - blood | DNA-Binding Proteins - metabolism | Malaria, Falciparum - parasitology | Animals | Malaria, Falciparum - immunology | T-Lymphocytes - immunology | Forkhead Transcription Factors | Index Medicus
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2. Full Text The Plasmodium translocon of exported proteins (PTEX) component thioredoxin‐2 is important for maintaining normal blood‐stage growth
by Matthews, Kathryn and Kalanon, Ming and Chisholm, Scott A and Sturm, Angelika and Goodman, Christopher D and Dixon, Matthew W. A and Sanders, Paul R and Nebl, Thomas and Fraser, Fiona and Haase, Silvia and McFadden, Geoffrey I and Gilson, Paul R and Crabb, Brendan S and Koning‐Ward, Tania F
Molecular Microbiology, ISSN 0950-382X, 09/2013, Volume 89, Issue 6, pp. 1167 - 1186
P lasmodium parasites remodel their vertebrate host cells by translocating hundreds of proteins across an encasing membrane into the host cell cytosol via a... 
LIFE-CYCLE | CELLS | FALCIPARUM-INFECTED ERYTHROCYTES | LIVER STAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYTOADHERENCE | MALARIA PARASITE | MICROBIOLOGY | HOST ERYTHROCYTE | VACUOLAR MEMBRANE | BERGHEI | VIRULENCE | Virulence Factors - genetics | Malaria, Cerebral - parasitology | Mice, Inbred C57BL | Plasmodium berghei - genetics | Virulence | Animals | Thioredoxins - genetics | Gene Deletion | Plasmodium berghei - enzymology | Survival Analysis | Parasitemia - parasitology | Plasmodium berghei - growth & development | Thioredoxins - metabolism | Virulence Factors - metabolism | Mice | Mice, Inbred BALB C | Plasmodium berghei - pathogenicity | Disease Models, Animal | Malaria | Thioredoxin | Growth | Analysis | Proteins | Survival analysis | Parasitology | Parasites | Molecular biology | Bacterial proteins | Index Medicus
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3. Full Text Plasmodium-specific antibodies block in vivo parasite growth without clearing infected red blood cells
by Akter, Jasmin and Khoury, David S and Aogo, Rosemary and Lansink, Lianne I. M and SheelaNair, Arya and Thomas, Bryce S and Laohamonthonkul, Pawat and Pernold, Clara P. S and Dixon, Matthew W. A and Soon, Megan S. F and Fogg, Lily G and Engel, Jessica A and Elliott, Trish and Sebina, Ismail and James, Kylie R and Cromer, Deborah and Davenport, Miles P and Haque, Ashraful
PLoS Pathogens, ISSN 1553-7366, 02/2019, Volume 15, Issue 2, pp. e1007599 - e1007599
Plasmodium parasites invade and multiply inside red blood cells (RBC). Through a cycle of maturation, asexual replication, rupture and release of multiple... 
COMPLEMENT | PHAGOCYTOSIS | FALCIPARUM-MALARIA | IMMUNOGLOBULIN | HOST | MICROBIOLOGY | MECHANISMS | IMMUNIZATION | PROTECTIVE IMMUNITY | VIROLOGY | VITRO GROWTH | PARASITOLOGY | OPSONIC ACTIVITY | Plasmodium - pathogenicity | Humans | Malaria - immunology | Plasmodium - metabolism | Parasites | Plasmodium yoelii - immunology | Phagocytes | Plasmodium chabaudi - immunology | Erythrocytes - microbiology | Plasmodium yoelii - pathogenicity | Animals | Plasmodium - growth & development | Erythrocytes - physiology | Mice | Malaria - metabolism | Antibodies, Protozoan - metabolism | Disease Models, Animal | Plasmodium chabaudi - pathogenicity | Medical research | Malaria | Dendritic cells | Analysis | Immunoglobulin G | Medicine, Experimental | Research | Health aspects | Animal models | Funding | Erythrocytes | Antibodies | Homology | Infections | Macrophages | Immunity | Bisphosphonates | Blood | Proteins | Cobra venom factor | Red blood cells | Immunity (humoral) | Parasitemia | Research centers | Vector-borne diseases | Spleen | Immunoglobulins | Schizonts | Blood cells | Rupture | Disease control | Merozoites | Exports | Clodronic acid | Depletion | Complement component C5 | Complement component C3 | Venom | In vivo methods and tests | Liposomes | Index Medicus
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4. Full Text Placental Malaria-Associated Inflammation Disturbs the Insulin-like Growth Factor Axis of Fetal Growth Regulation
by Alexandra J. Umbers and Philippe Boeuf and Caroline Clapham and Danielle I. Stanisic and Francesca Baiwog and Ivo Mueller and Peter Siba and Christopher L. King and James G. Beeson and Jocelyn Glazier and Stephen J. Rogerson
The Journal of Infectious Diseases, ISSN 0022-1899, 2/2011, Volume 203, Issue 4, pp. 561 - 569
Background. The pathogenetic mechanisms of fetal growth restriction associated with placental malaria are largely unknown. We sought to determine whether... 
Women | Pregnancy | PARASITES | Somatomedins | Monocytes | Placenta | Malaria | Birth weight | Infants | Inflammation | Infections | INFECTIOUS DISEASES | LOW-BIRTH-WEIGHT | I IGF-I | AMINO-ACID-TRANSPORT | CORD-BLOOD | IFN-GAMMA | FACTOR RECEPTORS | MICROBIOLOGY | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | PLASMODIUM-FALCIPARUM MALARIA | BINDING PROTEIN-1 IGFBP-1 | PREGNANCY OUTCOMES | Papua New Guinea | RNA, Messenger - isolation & purification | Humans | Malaria, Falciparum - pathology | RNA, Messenger - genetics | Insulin-Like Growth Factor Binding Proteins - blood | Gene Expression Profiling | Insulin-Like Growth Factor I - analysis | Pregnancy Complications, Infectious - pathology | Fetal Growth Retardation - pathology | Insulin-Like Growth Factor II - analysis | Plasma - chemistry | Placenta - pathology | Insulin-Like Growth Factor Binding Protein 3 | Plasmodium falciparum - pathogenicity | Adult | Female | Insulin-Like Growth Factor Binding Protein 1 - blood | Complications and side effects | Growth | Physiological aspects | Fetus | Research | Insulin-like growth factor 1 | Risk factors | Index Medicus | Abridged Index Medicus | Major and Brief Reports
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5. Full Text Mapping child growth failure in Africa between 2000 and 2015
by Osgood-Zimmerman, Aaron and Millear, Anoushka I and Stubbs, Rebecca W and Shields, Chloe and Pickering, Brandon V and Earl, Lucas and Graetz, Nicholas and Kinyoki, Damaris K and Ray, Sarah E and Bhatt, Samir and Browne, Annie J and Burstein, Roy and Cameron, Ewan and Casey, Daniel C and Deshpande, Aniruddha and Fullman, Nancy and Gething, Peter W and Gibson, Harry S and Henry, Nathaniel J and Herrero, Mario and Krause, L. Kendall and Letourneau, Ian D and Levine, Aubrey J and Liu, Patrick Y and Longbottom, Joshua and Mayala, Benjamin K and Mosser, Jonathan F and Noor, Abdisalan M and Pigott, David M and Piwoz, Ellen G and Rao, Puja and Rawat, Rahul and Reiner, Robert C and Smith, David L and Weiss, Daniel J and Wiens, Kirsten E and Mokdad, Ali H and Lim, Stephen S and Murray, Christopher J.L and Kassebaum, Nicholas J and Hay, Simon I
Nature, ISSN 0028-0836, 02/2018, Volume 555, Issue 7694, pp. 41 - 47
Insufficient growth during childhood is associated with poor health outcomes and an increased risk of death. Between 2000 and 2015, nearly all African... 
MALNUTRITION | NIGERIA | INTERVENTIONS | PLASMODIUM-FALCIPARUM | MULTIDISCIPLINARY SCIENCES | INLA | TRENDS | HEALTH SYSTEM PERFORMANCE | Wasting Syndrome - prevention & control | Africa - epidemiology | Prevalence | World Health Organization | Growth | Humans | Growth Disorders - prevention & control | Wasting Syndrome - epidemiology | Child, Preschool | Infant | Male | Thinness - epidemiology | Malnutrition - prevention & control | Malnutrition - epidemiology | Growth Disorders - epidemiology | Public Health - statistics & numerical data | Thinness - prevention & control | Female | Child Development | Goals | Infant, Newborn | Children | Health aspects | Analysis | Public health | Nutrition | Malaria | Partial differential equations | Geostatistics | Sustainable development | Data bases | Wetlands | Children & youth | Datasets | Underweight | Child mortality | Trends | Malnutrition | Failure | Age | Index Medicus
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6. Full Text Plasmodium falciparum Bloom homologue, a nucleocytoplasmic protein, translocates in 3′ to 5′ direction and is essential for parasite growth
by Rahman, Farhana and Tarique, Mohammed and Tuteja, Renu
BBA - Proteins and Proteomics, ISSN 1570-9639, 05/2016, Volume 1864, Issue 5, pp. 594 - 608
Malaria caused by , particularly , is the most serious and widespread parasitic disease of humans. RecQ helicase family members are essential in homologous... 
DNA helicase | Plasmodium falciparum | Malaria | Repair | Gene disruption | ATPase | RECOMBINATION | SYNDROME GENE-PRODUCT | BIOCHEMISTRY & MOLECULAR BIOLOGY | SYNDROME HELICASE | DNA-INTERACTING COMPOUNDS | BLM HELICASE | GENOME | BIOPHYSICS | ESCHERICHIA-COLI RECQ | REPLICATION FORK | Malaria, Falciparum - enzymology | Plasmodium falciparum - enzymology | Malaria, Falciparum - genetics | RecQ Helicases - metabolism | Humans | Adenosine Triphosphatases - metabolism | RecQ Helicases - genetics | Malaria, Falciparum - parasitology | Animals | Plasmodium falciparum - genetics | RNA, Double-Stranded - genetics | DNA, Protozoan - genetics | Plasmodium falciparum - metabolism | Adenosine Triphosphatases - genetics | RecQ Helicases - chemistry | Plasmodium falciparum - growth & development | Proteins | Parasitic diseases | Growth | Physiological aspects | Adenosine triphosphatase
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7. Full Text New Assays to Characterise Growth-Related Phenotypes of Plasmodium falciparum Reveal Variation in Density-Dependent Growth Inhibition between Parasite Lines
by Rovira-Graells, N and Aguilera-Simon, S and Tinto-Font, E and Cortes, A
PLOS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, pp. e0165358 - e0165358
The growth phenotype of asexual blood stage malaria parasites can influence their virulence and also their ability to survive and achieve transmission to the... 
INFECTIONS | MULTIDISCIPLINARY SCIENCES | ABILITY | RED-BLOOD-CELLS | MALARIA PARASITES | EXPRESSION | COMMUNICATION | CLAG3 GENES | CYCLE | Parasitemia - diagnosis | Merozoites - physiology | Genomics | Humans | Life Cycle Stages | Malaria, Falciparum - parasitology | Phenotype | Flow Cytometry | Microarray Analysis | Malaria, Falciparum - diagnosis | Proteomics | Parasitemia - parasitology | Schizonts - growth & development | Plasmodium falciparum - growth & development | Plasmodium falciparum - physiology | Erythrocytes - parasitology | Plasmodium falciparum | Genetic aspects | Virulence (Microbiology) | Malaria | Growth | Vector-borne diseases | Flow cytometry | Phenotypes | Growth rate | Virulence | Erythrocytes | Parasites | Gene deletion | Parameter identification | Assaying | Density | Blood | Cytometry | Disease transmission | Clonal deletion | Cell cycle | Deletion | Inhibition | Variation | Index Medicus | Parasitic diseases | Malalties parasitàries
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8. Full Text Transcriptional profiling of growth perturbations of the human malaria parasite Plasmodium falciparum
by Engelberg, Klemens and Spielmann, Tobias and Preiser, Peter R and Hu, Guangan and Cheemadan, Sabna and Cabrera, Ana and Mok, Sachel and Chaal, Balbir K and Bozdech, Zbynek and Haase, Silvia and Gilberger, Tim-W and Kono, Maya
Nature Biotechnology, ISSN 1087-0156, 01/2010, Volume 28, Issue 1, pp. 91 - 98
Functions have yet to be defined for the majority of genes of Plasmodium falciparum, the agent responsible for the most serious form of human malaria. Here we... 
LIFE-CYCLE | INVASION | NETWORK | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | RECONSTRUCTION | IDENTIFICATION | PROTEIN FUNCTION | GENOME | DISCOVERY | GENE-FUNCTION | PREDICTION | Transcription, Genetic - drug effects | Markov Chains | Humans | Gene Expression Profiling | Plasmodium falciparum - drug effects | Gene Regulatory Networks | Antimalarials - pharmacology | Gene Expression Regulation - drug effects | Malaria, Falciparum - parasitology | Algorithms | Animals | Protozoan Proteins - metabolism | Plasmodium falciparum - genetics | Merozoites - drug effects | Plasmodium falciparum - pathogenicity | Plasmodium falciparum - growth & development | Merozoites - metabolism | Plasmodium falciparum | Malaria | Growth | Causes of | Genetic aspects | Research | Gene expression | Health aspects | Biotechnology | Parasites | Developmental biology | Index Medicus
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9. Full Text Imaging of Plasmodium Liver Stages to Drive Next-Generation Antimalarial Drug Discovery
by Stephan Meister and David M. Plouffe and Kelli L. Kuhen and Ghislain M. C. Bonamy and Tao Wu and S. Whitney Barnes and Selina E. Bopp and Rachel Borboa and A. Taylor Bright and Jianwei Che and Steve Cohen and Neekesh V. Dharia and Kerstin Gagaring and Montip Gettayacamin and Perry Gordon and Todd Groessl and Nobutaka Kato and Marcus C. S. Lee and Case W. McNamara and David A. Fidock and Advait Nagle and Tae-gyu Nam and Wendy Richmond and Jason Roland and Matthias Rottmann and Bin Zhou and Patrick Froissard and Richard J. Glynne and Dominique Mazier and Jetsumon Sattabongkot and Peter G. Schultz and Tove Tuntland and John R. Walker and Yingyao Zhou and Arnab Chatterjee and Thierry T. Diagana and Elizabeth A. Winzeler
Science, ISSN 0036-8075, 12/2011, Volume 334, Issue 6061, pp. 1372 - 1377
Most malaria drug development focuses on parasite stages detected in red blood cells, even though, to achieve eradication, next-generation drugs active against... 
Schizonts | Malaria | RESEARCH ARTICLES | Liver | Antimalarials | Infections | Parasites | Inhibitory concentration 50 | Scaffolds | Blood | Chemicals | INFECTIONS | FALCIPARUM | MULTIDISCIPLINARY SCIENCES | METHEMOGLOBINEMIA | RESISTANCE | MALARIA | TARGETS | MUTATIONS | Plasmodium berghei - physiology | Humans | Imidazoles - chemistry | Plasmodium - physiology | Piperazines - chemistry | Plasmodium falciparum - drug effects | Plasmodium - growth & development | Imidazoles - therapeutic use | Plasmodium falciparum - physiology | Antimalarials - pharmacokinetics | Antimalarials - therapeutic use | Imidazoles - pharmacology | Piperazines - therapeutic use | Random Allocation | Piperazines - pharmacology | Plasmodium berghei - cytology | Small Molecule Libraries | Malaria - parasitology | Cell Line, Tumor | Plasmodium berghei - growth & development | Mice | Mice, Inbred BALB C | Liver - parasitology | Erythrocytes - parasitology | Plasmodium berghei - drug effects | Plasmodium - cytology | Plasmodium falciparum - cytology | Plasmodium - drug effects | Imidazoles - pharmacokinetics | Plasmodium yoelii - growth & development | Protozoan Proteins - genetics | Sporozoites - growth & development | Protozoan Proteins - metabolism | Plasmodium yoelii - drug effects | Protozoan Proteins - chemistry | Molecular Structure | Piperazines - pharmacokinetics | Drug Evaluation, Preclinical | Sporozoites - drug effects | Drug Resistance | Plasmodium yoelii - physiology | Plasmodium yoelii - cytology | Drug Discovery | Antimalarials - pharmacology | Malaria - prevention & control | Animals | Antimalarials - chemistry | Polymorphism, Single Nucleotide | Malaria - drug therapy | Plasmodium falciparum - growth & development | Pharmaceutical research | Plasmodium | Care and treatment | Analysis | Research | Drug therapy | Index Medicus
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10. Full Text Graphene oxide inhibits malaria parasite invasion and delays parasitic growth: In vitro
by Kenry, Kenry and Lim, Ying Bena and Nai, Mui Hoon and Cao, Jianshu and Loh, Kian Ping and Lim, Chwee Teck
Nanoscale, ISSN 2040-3364, 10/2017, Volume 9, Issue 37, pp. 14065 - 14073
The interactions between graphene oxide (GO) and various biological entities have been actively investigated in recent years, resulting in numerous potential... 
CELLS | PHYSICS, APPLIED | HUMAN SERUM-ALBUMIN | MATERIALS SCIENCE, MULTIDISCIPLINARY | SIZE | CHEMISTRY | NANOSCIENCE & NANOTECHNOLOGY | LATERAL DIMENSION | PLASMODIUM-FALCIPARUM MALARIA | CHEMISTRY, MULTIDISCIPLINARY | CHALLENGES | DELIVERY | Malaria | Humans | Oxides | Nanostructures | Plasmodium falciparum - drug effects | Erythrocytes - parasitology | Graphite - pharmacology | Index Medicus
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11. Full Text ALBA4 modulates its stage‐specific interactions and specific mRNA fates during Plasmodium yoelii growth and transmission
by Muñoz, Elyse E and Hart, Kevin J and Walker, Michael P and Kennedy, Mark F and Shipley, Mackenzie M and Lindner, Scott E
Molecular Microbiology, ISSN 0950-382X, 10/2017, Volume 106, Issue 2, pp. 266 - 284
Transmission of the malaria parasite occurs in an unpredictable moment, when a mosquito takes a blood meal. Plasmodium has therefore evolved strategies to... 
PROTEOMIC ANALYSES | P-BODIES | DNA-BINDING | TRANSLATIONAL REPRESSION | SURFACE PROTEIN | FALCIPARUM | BIOCHEMISTRY & MOLECULAR BIOLOGY | BINDING PROTEINS | MICROBIOLOGY | MALARIA PARASITES | STRESS GRANULES | SEXUAL DEVELOPMENT | Plasmodium yoelii - physiology | Anopheles - parasitology | Transcriptome | Parasites | Plasmodium yoelii - growth & development | RNA, Messenger - metabolism | Plasmodium yoelii - genetics | RNA, Messenger - biosynthesis | Animals | Protozoan Proteins - metabolism | Sporozoites - metabolism | Malaria - parasitology | Proteomics | Parasitic Diseases - genetics | Enzyme Repression | RNA-Binding Proteins - metabolism | Plasmodium falciparum |