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Nature (London), ISSN 1476-4687, 12/2015, Volume 529, Issue 7584, pp. 110 - 114
...)-binding sites, compromising binding of this methylation-sensitive insulator protein. Reduced CTCF binding is associated with loss of insulation between topological domains and aberrant gene activation... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Chromatin - metabolism | Up-Regulation | Humans | Glioma - genetics | Base Sequence | Glioma - pathology | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Binding Sites | Chromatin - drug effects | Repressor Proteins - metabolism | Oncogenes - genetics | Insulator Elements - genetics | Glioma - enzymology | Chromosomal Proteins, Non-Histone - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Isocitrate Dehydrogenase - genetics | DNA Methylation - genetics | Down-Regulation - drug effects | Mutation - genetics | CRISPR-Cas Systems - genetics | Insulator Elements - drug effects | Phenotype | Isocitrate Dehydrogenase - chemistry | Receptor, Platelet-Derived Growth Factor alpha - genetics | CCCTC-Binding Factor | CpG Islands - genetics | Protein Binding | Isocitrate Dehydrogenase - metabolism | Cell Proliferation - drug effects | Enhancer Elements, Genetic - genetics | Glutarates - metabolism | Cell Transformation, Neoplastic - drug effects | Chromatin - genetics | DNA Methylation - drug effects | Glioma - drug therapy | Complications and side effects | Care and treatment | Platelet-derived growth factor | Gliomas | Analysis | Influence | Genetic aspects | Research | Methylation | Oncogenes | DNA methylation | Epigenetics | Genomes | Mutation | Gene expression | Binding sites | Deoxyribonucleic acid--DNA | Tumors | Index Medicus
Journal Article
Journal of acquired immune deficiency syndromes (1999), ISSN 1525-4135, 07/2013, Volume 63, Issue 3, pp. 280 - 288
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 09/2016, Volume 311, Issue 3, pp. H725 - H734
Journal Article
PLoS medicine, ISSN 1549-1676, 07/2006, Volume 3, Issue 7, pp. e270 - 1151
...), essential thrombocytosis (ET), and myelofibrosis with myeloid metaplasia (MF). Subsequent analysis has shown that constitutive activation of the JAK-STAT signal... 
Recombinant Fusion Proteins - adverse effects | Humans | Myeloproliferative Disorders - pathology | Mutation, Missense | Primary Myelofibrosis - pathology | Receptors, Cytokine - physiology | Janus Kinases - physiology | Bone Marrow Transplantation | Hematopoiesis - physiology | Myeloid Cells - drug effects | Transcription, Genetic | Colony-Forming Units Assay | Phosphorylation - drug effects | mRNA Cleavage and Polyadenylation Factors - genetics | Hematologic Neoplasms - physiopathology | Disease Models, Animal | Cells, Cultured - drug effects | Bone Marrow Cells - pathology | Hematopoiesis - genetics | Signal Transduction - genetics | Thrombocytosis - etiology | Sequence Analysis, DNA | Cell Division - drug effects | mRNA Cleavage and Polyadenylation Factors - physiology | Receptor, Platelet-Derived Growth Factor alpha - physiology | Hematologic Neoplasms - drug therapy | Mice | Mice, Inbred BALB C | Myeloid Cells - pathology | Protein Processing, Post-Translational - genetics | Myeloproliferative Disorders - genetics | Oncogene Proteins, Fusion - physiology | STAT Transcription Factors - physiology | Protein Processing, Post-Translational - drug effects | Cells, Cultured - pathology | Bone Marrow Cells - drug effects | Spleen - pathology | Thrombocytosis - pathology | Gene Expression Regulation | Janus Kinases - antagonists & inhibitors | Point Mutation | Megakaryocytes - pathology | Primary Myelofibrosis - genetics | Animals | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Myeloproliferative Disorders - etiology | Signal Transduction - physiology | Protein Kinase Inhibitors - pharmacology | Genetic Vectors | Megakaryocytes - drug effects | Cytokines - pharmacology | Amino Acid Substitution | Thrombocytosis - genetics | Genetic aspects | Myelofibrosis | Research | Index Medicus | Hematology | Gene Therapy | Genomics | Genetics | Oncology | Cancer Biology | Hematology (including Blood Transfusion)
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 08/2016, Volume 36, Issue 8, pp. 1534 - 1548
OBJECTIVE—Drug-eluting coronary stents reduce restenosis rate and late lumen loss compared with bare-metal stents... 
blood platelets | endothelial cells | peroxisome proliferator-activated receptors | vascular smooth muscle cells | coronary in-stent restenosis | Recurrence | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Glucose Transporter Type 1 - metabolism | Aorta - metabolism | Angioplasty, Balloon - adverse effects | Aortic Diseases - metabolism | Neointima | Time Factors | Aortic Diseases - prevention & control | Myocytes, Smooth Muscle - drug effects | PPAR delta - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Atherosclerosis - pathology | Drug-Eluting Stents | Human Umbilical Vein Endothelial Cells - drug effects | Coronary Artery Disease - metabolism | Rats | Rats, Sprague-Dawley | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Cell Movement - drug effects | Thrombosis - pathology | Thrombosis - metabolism | Coronary Artery Disease - therapy | Signal Transduction - drug effects | Blood Platelets - metabolism | PPAR delta - agonists | PPAR delta - metabolism | Energy Metabolism - drug effects | Steroids - administration & dosage | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Dose-Response Relationship, Drug | Transfection | Coronary Artery Disease - pathology | Thrombosis - prevention & control | Blood Platelets - drug effects | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Aortic Diseases - pathology | Carotid Artery Injuries - pathology | Cardiovascular Agents - administration & dosage | PPAR delta - deficiency | Aorta - drug effects | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Re-Epithelialization - drug effects | Angioplasty, Balloon - instrumentation | Muscle, Smooth, Vascular - pathology | Animals | Glucose Transporter Type 1 - genetics | Thrombosis - etiology | Cell Proliferation - drug effects | Platelet Activation - drug effects | Atherosclerosis - prevention & control | Index Medicus
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 10/2016, Volume 65, Issue 10, pp. 3111 - 3128
Decreased heat shock protein (Hsp) expression in type 1 and type 2 diabetes has been implicated as a primary factor contributing to diabetes-induced organ... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Exosomes - drug effects | Exosomes - metabolism | Neovascularization, Physiologic - drug effects | Reactive Oxygen Species - metabolism | Collagen Type III - metabolism | HSP20 Heat-Shock Proteins - genetics | Male | Cell Movement - physiology | HSP20 Heat-Shock Proteins - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Myocardium - metabolism | Superoxide Dismutase-1 - metabolism | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Aniline Compounds - pharmacology | Collagen Type I - metabolism | Endothelial Cells - metabolism | Neovascularization, Physiologic - genetics | Cell Proliferation - physiology | Cells, Cultured | Mice, Transgenic | Cell Movement - drug effects | Neovascularization, Physiologic - physiology | Animals | Benzylidene Compounds - pharmacology | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - physiology | Heart - drug effects | Myocytes, Cardiac - metabolism | Protein Binding | Cell Proliferation - drug effects | Mice | Endothelial Cells - drug effects | Genetically modified mice | Complications and side effects | Care and treatment | Cardiomyopathy | Heat shock proteins | Genetic aspects | Diabetes | Research | Heart diseases | Risk factors | Angiogenesis | Cardiomyocytes | Protein expression | Biosynthesis | Index Medicus | Abridged Index Medicus | Pathophysiology
Journal Article
PloS one, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e90699 - e90699
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 02/2014, Volume 81, Issue 3, pp. 588 - 602
.... We also discovered that ET-1 acts mechanistically by promoting Notch activation in OPCs during remyelination through induction of Jagged1 expression in reactive... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Endothelin | Neurophysiology | Medical research | Experiments | Multiple sclerosis | Index Medicus
Journal Article