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Nature (London), ISSN 1476-4687, 03/2014, Volume 508, Issue 7494, pp. 118 - 122
.... In contrast, colon cancers that harbour the same BRAF(V600E) mutation are intrinsically resistant to BRAF inhibitors, due to feedback activation of the epidermal growth factor receptor (EGFR... 
Antineoplastic Agents/administration & dosage | Transforming Growth Factor beta/metabolism | Humans | Mitogen-Activated Protein Kinase Kinases/antagonists & inhibitors | Indoles/administration & dosage | Flow Cytometry | Receptor, Platelet-Derived Growth Factor beta/biosynthesis | Proto-Oncogene Proteins B-raf/antagonists & inhibitors | ErbB Receptors/biosynthesis | Melanoma/drug therapy | Female | Cell Proliferation/drug effects | Sulfonamides/administration & dosage | Gene Library | Cellular Senescence/drug effects | Receptor Protein-Tyrosine Kinases/biosynthesis | Drug Resistance, Neoplasm/drug effects | Gene Expression Regulation, Neoplastic/drug effects | SOXE Transcription Factors/deficiency | Signal Transduction/drug effects | Protein Kinase Inhibitors/administration & dosage | Vemurafenib | Animals | Mice | RNA, Small Interfering | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Receptor, Epidermal Growth Factor - genetics | Receptor Protein-Tyrosine Kinases - biosynthesis | Cellular Senescence - drug effects | Melanoma - enzymology | Antineoplastic Agents - administration & dosage | Receptor, Platelet-Derived Growth Factor beta - genetics | Indoles - administration & dosage | Mitogen-Activated Protein Kinase Kinases - metabolism | Receptor, Epidermal Growth Factor - metabolism | Melanoma - genetics | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | SOXE Transcription Factors - deficiency | Proto-Oncogene Proteins B-raf - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Receptor, Epidermal Growth Factor - biosynthesis | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Melanoma - pathology | Receptor Protein-Tyrosine Kinases - metabolism | Sulfonamides - pharmacology | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Protein Kinase Inhibitors - administration & dosage | Transforming Growth Factor beta - pharmacology | Drug Resistance, Neoplasm - genetics | Receptor Protein-Tyrosine Kinases - genetics | Signal Transduction - drug effects | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Transforming Growth Factor beta - metabolism | Receptor, Platelet-Derived Growth Factor beta - biosynthesis | Sulfonamides - administration & dosage | Drug Resistance, Neoplasm - drug effects | SOXE Transcription Factors - genetics | Proteins | Biopsy | Rodents | Genes | Melanoma | Mutation | Kinases | Drug resistance | Patients | Tumors | Index Medicus
Journal Article
Drugs & Aging, ISSN 1170-229X, 02/2010, Volume 27, Issue 2, pp. 95 - 115
...) sustained the previously held view that interleukin (IL)-1 or tumour necrosis factor-α (TNFα) disrupt the metabolism of synovial joint tissues... 
Osteoarthritis, pathogenesis | Cytokine-inhibitors, therapeutic use | Elderly | Osteoarthritis, treatment | Pharmacotherapy | Internal Medicine | Medicine & Public Health | Pharmacology/Toxicology | Geriatrics/Gerontology | Tumor Necrosis Factor-alpha - metabolism | Interleukin-18 - immunology | Interleukin-6 - antagonists & inhibitors | Cartilage - pathology | Humans | Interleukin-18 - antagonists & inhibitors | Antirheumatic Agents - immunology | Interleukin-17 - immunology | Synovial Fluid - metabolism | Osteoarthritis - drug therapy | Synovial Fluid - drug effects | Insulin-Like Growth Factor I - immunology | Signal Transduction - immunology | Interleukin-1 - immunology | Tumor Necrosis Factor-alpha - immunology | Fibroblast Growth Factor 2 - physiology | Interleukin-1 - antagonists & inhibitors | Joints - metabolism | Antirheumatic Agents - therapeutic use | Cytokines - immunology | Transforming Growth Factor beta - immunology | Cartilage - immunology | Interleukin-1beta - analysis | Osteoarthritis - immunology | Transforming Growth Factor beta - physiology | Interleukin-1beta - immunology | Joints - pathology | Arthritis, Experimental | Cartilage, Articular - pathology | Interleukin-7 - immunology | Interleukin-17 - antagonists & inhibitors | Joints - immunology | Interleukin-6 - immunology | Interleukin 1 Receptor Antagonist Protein | Platelet-Derived Growth Factor | Aged | Cytokines - antagonists & inhibitors | Interleukin-7 - antagonists & inhibitors | Cartilage, Articular - immunology | Insulin-Like Growth Factor I - metabolism | Interleukins | Tumor necrosis factor | Physiological aspects | Research | Drug therapy | Health aspects | Osteoarthritis | Index Medicus
Journal Article
Blood, ISSN 1528-0020, 02/2017, Volume 129, Issue 6, pp. 704 - 714
Molecular diagnostics has generated substantial dividends in dissecting the genetic basis of myeloid neoplasms with eosinophilia. The family of diseases... 
Life Sciences & Biomedicine | Hematology | Science & Technology | Eosinophilia - therapy | Oncogene Proteins, Fusion - metabolism | Leukemia - pathology | Proto-Oncogene Proteins c-abl - antagonists & inhibitors | Humans | Gene Expression Regulation, Neoplastic | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | fms-Like Tyrosine Kinase 3 | Antineoplastic Agents - therapeutic use | Myeloproliferative Disorders - pathology | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Myeloproliferative Disorders - genetics | Transplantation, Homologous | Eosinophilia - pathology | Hypereosinophilic Syndrome - diagnosis | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Janus Kinase 2 - metabolism | Eosinophilia - genetics | Hypereosinophilic Syndrome - pathology | Hypereosinophilic Syndrome - therapy | Eosinophilia - diagnosis | Janus Kinase 2 - antagonists & inhibitors | Leukemia - genetics | Receptor, Platelet-Derived Growth Factor beta - metabolism | Myeloproliferative Disorders - diagnosis | Proto-Oncogene Proteins c-abl - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Myeloproliferative Disorders - therapy | Janus Kinase 2 - genetics | Hematopoietic Stem Cell Transplantation | Leukemia - therapy | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Leukemia - diagnosis | Proto-Oncogene Proteins c-abl - metabolism | Hypereosinophilic Syndrome - genetics | Oncogene Proteins, Fusion - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Radiation Research, ISSN 0033-7587, 2002, Volume 157, Issue 1, pp. 45 - 51
...). Angiogenesis is critical for tumor development, growth and metastasis. The vascular endothelial growth factor (VEGF... 
Tumor burden | Angiogenesis | Receptors | Cell growth | Squamous cell carcinoma | Irradiation | Angiogenesis inhibitors | Radiotherapy | Tumors | Cancer | Biology | Life Sciences & Biomedicine - Other Topics | Biophysics | Life Sciences & Biomedicine | Radiology, Nuclear Medicine & Medical Imaging | Science & Technology | Vascular Endothelial Growth Factors | Receptors, Growth Factor - antagonists & inhibitors | Neoplasm Proteins - antagonists & inhibitors | Carcinoma, Squamous Cell - radiotherapy | Indoles - administration & dosage | Receptors, Growth Factor - genetics | RNA, Messenger - biosynthesis | Platelet-Derived Growth Factor - biosynthesis | Pyrroles - administration & dosage | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Chemotherapy, Adjuvant | Lymphokines - biosynthesis | Specific Pathogen-Free Organisms | Receptors, Fibroblast Growth Factor - biosynthesis | Angiogenesis Inhibitors - pharmacology | Receptors, Platelet-Derived Growth Factor - genetics | Endothelial Growth Factors - biosynthesis | Carcinoma, Squamous Cell - drug therapy | RNA, Neoplasm - biosynthesis | Fibroblast Growth Factor 2 - genetics | Carcinoma, Squamous Cell - blood supply | Indoles - therapeutic use | Mice | Receptors, Vascular Endothelial Growth Factor | Vascular Endothelial Growth Factor A | Carcinoma, Squamous Cell - metabolism | Receptor Protein-Tyrosine Kinases - biosynthesis | Neoplasm Proteins - metabolism | Radioisotope Teletherapy | Platelet-Derived Growth Factor - genetics | Radiation-Sensitizing Agents - therapeutic use | Angiogenesis Inhibitors - administration & dosage | Lymphokines - genetics | Radiation-Sensitizing Agents - administration & dosage | Receptors, Fibroblast Growth Factor - genetics | Angiogenesis Inhibitors - therapeutic use | Dose Fractionation | Indoles - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Pyrroles - therapeutic use | Radiation-Sensitizing Agents - toxicity | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Radiation-Sensitizing Agents - pharmacology | Fibroblast Growth Factor 2 - biosynthesis | Receptors, Fibroblast Growth Factor - antagonists & inhibitors | Indoles - toxicity | Mice, Inbred C3H | Pyrroles - pharmacology | Animals | Angiogenesis Inhibitors - toxicity | Receptor Protein-Tyrosine Kinases - genetics | Pyrroles - toxicity | Endothelial Growth Factors - genetics | Receptors, Growth Factor - biosynthesis | Receptors, Platelet-Derived Growth Factor - biosynthesis | Drug Screening Assays, Antitumor | Index Medicus | Space life sciences
Journal Article
Current medicinal chemistry, ISSN 0929-8673, 11/2016, Volume 23, Issue 37, pp. 4176 - 4220
Molecularly targeted anticancer therapy involves the use of drugs or other substances affecting specific molecular targets that play a part in the development,... 
Side effects | Monoclonal antybodies | mTOR patway | Chemical structures | Mechanism of action | Pharmacokinetic parameters | Tyrosine kinase inhibitors | Pharmacology & Pharmacy | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Chemistry, Medicinal | Science & Technology | ErbB Receptors - metabolism | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Receptors, Platelet-Derived Growth Factor - immunology | TOR Serine-Threonine Kinases - metabolism | Humans | Antibodies, Monoclonal - therapeutic use | Vascular Endothelial Growth Factor A - immunology | Antineoplastic Agents - therapeutic use | Vascular Endothelial Growth Factor A - metabolism | Antineoplastic Agents - chemistry | Neoplasms - drug therapy | Antineoplastic Agents - metabolism | Protein Kinase Inhibitors - chemistry | Receptors, Vascular Endothelial Growth Factor - metabolism | Protein Kinase Inhibitors - therapeutic use | Receptors, Platelet-Derived Growth Factor - metabolism | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - metabolism | Drugs | Therapy | Transformation | Platelet-derived growth factor | Target recognition | Molecular structure | Transcription | Clinical trials | Antibodies | mRNA | Kinases | Blockage | Proteins | Signal transduction | Synthesis | Antitumor agents | Cell cycle | Vascular endothelial growth factor | Growth factors | Protein-tyrosine kinase | Fibroblast growth factor 2 | Tyrosine | Medical research | Epidermal growth factor receptors | Structural proteins | Antisense therapy | Metabolism | Vascular endothelial growth factor receptors | Transduction | Cancer | Structure-function relationships
Journal Article