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Kidney International, ISSN 0085-2538, 11/2015, Volume 88, Issue 5, pp. 1030 - 1046
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2007, Volume 13, Issue 11, pp. 1349 - 1358
Data providing direct evidence for a causative link between endothelial dysfunction, microvascular disease and diabetic end-organ damage are scarce. Here we... 
MEDICINE, RESEARCH & EXPERIMENTAL | POLY(ADP-RIBOSE) POLYMERASE INHIBITORS | ANTICOAGULANT PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | ALPHA | CELL BIOLOGY | THROMBOMODULIN | INFLAMMATION | DYSFUNCTION | CELL | RECEPTOR-1 | SEVERE SEPSIS | Kidney Glomerulus - blood supply | Diabetic Nephropathies - enzymology | Protein C - physiology | Diabetes Mellitus, Experimental - enzymology | Humans | Diabetes Mellitus, Experimental - genetics | Podocytes - enzymology | Apoptosis - genetics | Endothelium, Vascular - enzymology | Thrombomodulin - physiology | Diabetes Mellitus, Experimental - prevention & control | Mice, Mutant Strains | Diabetic Nephropathies - prevention & control | Kidney Glomerulus - enzymology | Diabetic Nephropathies - pathology | Mice, Inbred C57BL | Cells, Cultured | Diabetic Nephropathies - genetics | Mice, Transgenic | Signal Transduction - genetics | Kidney Glomerulus - pathology | Podocytes - pathology | Protein C - biosynthesis | Animals | Microcirculation - pathology | Microcirculation - enzymology | Diabetes Mellitus, Experimental - pathology | Amino Acid Substitution - genetics | Cytoprotection - genetics | Endothelium, Vascular - pathology | Mice | Enzyme Activation - genetics | Cell Line, Transformed | Protein C - genetics | Proteins | Diabetes | Kidney diseases | Cellular biology | Rodents | Apoptosis
Journal Article
Kidney International, ISSN 0085-2538, 2014, Volume 85, Issue 2, pp. 457 - 470
Renal toxicity constitutes a dose-limiting side effect of anticancer therapies targeting vascular endothelial growth factor (VEGF). In order to study this... 
anti-VEGF | FSGS | NF-κB | c-mip | thrombotic microangiopathy | MCNS | ANGIOGENESIS | PHOSPHORYLATION | PROTEINURIA | PODOCYTES | CANCER | CELL CARCINOMA | DAMAGE | RENAL THROMBOTIC MICROANGIOPATHY | NF-kappa B | AUTOCRINE | UROLOGY & NEPHROLOGY | NF-KAPPA-B | Glomerulosclerosis, Focal Segmental - enzymology | Predictive Value of Tests | Humans | Middle Aged | Male | Protein Kinase Inhibitors - adverse effects | Young Adult | Proteinuria - chemically induced | Renal Insufficiency - chemically induced | Base Sequence | Kidney Diseases - chemically induced | Glomerulosclerosis, Focal Segmental - chemically induced | Transcription, Genetic | Hypertension - enzymology | Angiogenesis Inhibitors - adverse effects | Binding Sites | Biomarkers - metabolism | Niacinamide - adverse effects | Kidney Glomerulus - pathology | Thrombotic Microangiopathies - enzymology | Mice, Knockout | Glomerulosclerosis, Focal Segmental - diagnosis | Adaptor Proteins, Signal Transducing | Nephrosis, Lipoid - chemically induced | Thrombotic Microangiopathies - diagnosis | Mice | Transcription Factor RelA - deficiency | Niacinamide - analogs & derivatives | Renal Insufficiency - enzymology | Renal Insufficiency - diagnosis | Vascular Endothelial Growth Factors - antagonists & inhibitors | Kidney Diseases - diagnosis | Molecular Sequence Data | Nephrosis, Lipoid - enzymology | Kidney Diseases - enzymology | Transcription Factor RelA - genetics | Case-Control Studies | Transfection | Phenylurea Compounds - adverse effects | Hypertension - chemically induced | Adult | Female | Proteinuria - diagnosis | Hypertension - diagnosis | Kidney Glomerulus - enzymology | Cell Line | Promoter Regions, Genetic | Nephrosis, Lipoid - diagnosis | Kidney Glomerulus - drug effects | Gene Expression Regulation | Carrier Proteins - genetics | Proteinuria - enzymology | Animals | Carrier Proteins - metabolism | Transcription Factor RelA - metabolism | Vascular Endothelial Growth Factors - metabolism | Aged | Thrombotic Microangiopathies - chemically induced | Life Sciences | Biochemistry, Molecular Biology
Journal Article
Journal Article
American Journal of Pathology, ISSN 0002-9440, 05/2009, Volume 174, Issue 5, pp. 1663 - 1674
Hypoxia-inducible transcription factors (HIFs) play important roles in the response of the kidney to systemic and regional hypoxia. Degradation of HIFs is... 
OXYGEN | CELLS | DOMAIN PROTEINS | ERYTHROPOIETIN | MESSENGER-RNA | HYPOXIA-INDUCIBLE-FACTOR | ACUTE-RENAL-FAILURE | INDUCTION | PATHOLOGY | UP-REGULATION | FACTOR-ALPHA | Fibroblasts - enzymology | Prognosis | Homeodomain Proteins - metabolism | Kidney - enzymology | Podocytes - enzymology | Male | Gene Expression Regulation, Enzymologic - physiology | RNA, Messenger - metabolism | Immunoenzyme Techniques | Oxygen - metabolism | Antineoplastic Agents - toxicity | Procollagen-Proline Dioxygenase - metabolism | DNA-Binding Proteins - metabolism | Immediate-Early Proteins - metabolism | Contrast Media - pharmacology | Cisplatin - toxicity | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Kidney Tubules, Collecting - enzymology | Ischemia - pathology | Kidney Tubules, Collecting - drug effects | Kidney - drug effects | Kidney Medulla - enzymology | Kidney Tubules - drug effects | Kidney Medulla - drug effects | Mice, Inbred C57BL | RNA, Messenger - genetics | Ischemia - metabolism | Rats | Hypoxia-Inducible Factor-Proline Dioxygenases | Acute Kidney Injury - enzymology | DNA-Binding Proteins - genetics | Kidney - injuries | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Blotting, Western | Homeodomain Proteins - genetics | Animals | Immediate-Early Proteins - genetics | Podocytes - drug effects | Procollagen-Proline Dioxygenase - genetics | Kidney Tubules - enzymology | Fibroblasts - drug effects | Mice | Index Medicus | Abridged Index Medicus | Regular
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 06/2014, Volume 25, Issue 6, pp. 1237 - 1254
Journal Article
Kidney International, ISSN 0085-2538, 04/2017, Volume 91, Issue 4, pp. 896 - 913
The glycoprotein CD44 is barely detected in normal mouse and human glomeruli, but is increased in glomerular parietal epithelial cells following podocyte... 
collagen | FSGS | glomerulosclerosis | mitogen-activated protein kinases | glomerulus | podocyte | RENIN LINEAGE | INDUCED APOPTOSIS | RECEPTOR | NEPHROPATHY | PODOCYTE PROTEINS | DISEASE | UROLOGY & NEPHROLOGY | EXTRACELLULAR-MATRIX | CANCER PROGRESSION | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | EXPRESSION | Glomerulosclerosis, Focal Segmental - enzymology | Phosphorylation | Glomerulosclerosis, Focal Segmental - genetics | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Podocytes - enzymology | Male | Transfection | Time Factors | Hyaluronan Receptors - metabolism | Disease Models, Animal | Collagen Type IV - metabolism | Kidney Glomerulus - enzymology | Genetic Predisposition to Disease | Signal Transduction | Extracellular Matrix - drug effects | Kidney Glomerulus - drug effects | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Cells, Cultured | Kidney Glomerulus - pathology | Podocytes - pathology | Albuminuria - prevention & control | Hyaluronan Receptors - genetics | Albuminuria - enzymology | Mice, Knockout | Albuminuria - genetics | Extracellular Matrix - enzymology | Phenotype | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Podocytes - drug effects | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Extracellular Matrix - pathology | Glomerulosclerosis, Focal Segmental - pathology | Cell Movement | Mitogen-Activated Protein Kinase 1 - metabolism | Mitogen-activated protein kinases
Journal Article
Kidney International, ISSN 0085-2538, 01/2018, Volume 93, Issue 1, pp. 110 - 127
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 11/2013, Volume 84, Issue 5, pp. 920 - 930
Podocytes are highly specialized epithelial cells with complex actin cytoskeletal architecture crucial for maintenance of the glomerular filtration barrier.... 
glomerular filtration barrier | glomerular disease | cytoskeleton | transgenic mouse | podocyte | NEPHRIN | PROTEINURIA | DIRECT TARGET | CELL BIOLOGY | NEURONAL DEVELOPMENT | ACTIN REORGANIZATION | DISEASE | UROLOGY & NEPHROLOGY | KIDNEY PODOCYTES | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | Renal Insufficiency - enzymology | Phosphorylation | Podocytes - enzymology | rac1 GTP-Binding Protein - deficiency | Desoxycorticosterone Acetate | Male | cdc42 GTP-Binding Protein - metabolism | Acute Kidney Injury - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Mice, 129 Strain | Renal Insufficiency - etiology | Actin Depolymerizing Factors - metabolism | Nephrectomy | Time Factors | Cell Shape | Hypertension - chemically induced | Acute Kidney Injury - chemically induced | Albuminuria - metabolism | Membrane Proteins - metabolism | Hypertension - enzymology | Hypertension - genetics | Neuropeptides - genetics | Disease Models, Animal | Protamines | Neuropeptides - deficiency | Renal Insufficiency - genetics | Signal Transduction | Acute Kidney Injury - pathology | Mice, Inbred C57BL | Genotype | Neuropeptides - metabolism | Podocytes - pathology | Acute Kidney Injury - enzymology | Mice, Knockout | Phenotype | Animals | Renal Insufficiency - pathology | cdc42 GTP-Binding Protein - genetics | Mice | rac1 GTP-Binding Protein - metabolism | cdc42 GTP-Binding Protein - deficiency | rac1 GTP-Binding Protein - genetics
Journal Article
BBA - General Subjects, ISSN 0304-4165, 11/2017, Volume 1861, Issue 11, pp. 2758 - 2765
Journal Article