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Kidney international, ISSN 0085-2538, 06/2013, Volume 83, Issue 6, pp. 1065 - 1075
Collapsing focal segmental glomerulosclerosis (cFSGS) is a progressive kidney disease characterized by glomerular collapse with epithelial hyperplasia. Here we... 
parietal epithelial cell | focal segmental glomerulosclerosis | podocyte loss | Notch signaling | Focal segmental glomerulosclerosis | Parietal epithelial cell | Podocyte loss | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Cell Proliferation | RNA, Untranslated - metabolism | Homeodomain Proteins - metabolism | Humans | Hyperplasia | Transforming Growth Factor beta1 - metabolism | Proteinuria - pathology | RNA, Untranslated - genetics | Dibenzazepines - pharmacology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Time Factors | Serrate-Jagged Proteins | Disease Models, Animal | Jagged-1 Protein | Biomarkers - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Membrane Proteins - genetics | Enzyme Inhibitors - pharmacology | Epithelial Cells - pathology | Mice, Transgenic | Kidney Glomerulus - pathology | Podocytes - pathology | Proteinuria - genetics | Amyloid Precursor Protein Secretases - metabolism | Interleukin-2 Receptor alpha Subunit - metabolism | Claudin-1 - genetics | Mice | Receptor, Notch1 - antagonists & inhibitors | Calcium-Binding Proteins - genetics | Cell Movement | Transcription Factor HES-1 | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Glomerulosclerosis, Focal Segmental - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Interleukin-2 Receptor alpha Subunit - genetics | Claudin-1 - metabolism | Kidney Glomerulus - metabolism | Integrases - metabolism | Membrane Proteins - metabolism | Calcium-Binding Proteins - metabolism | Cell Line | Podocytes - metabolism | Kidney Glomerulus - drug effects | Intercellular Signaling Peptides and Proteins - genetics | Proteinuria - metabolism | Antibodies, Monoclonal | Receptor, Notch1 - metabolism | Homeodomain Proteins - genetics | Glomerulosclerosis, Focal Segmental - metabolism | Animals | Exotoxins | Podocytes - drug effects | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Integrases - genetics | Glomerulosclerosis, Focal Segmental - pathology | Apoptosis | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e62068 - e62068
The NF-kappa B pathway plays an important role in chronic inflammatory and autoimmune diseases. Recently, NF-kappa B has also been suggested as an important... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Albuminuria - complications | Diabetes Mellitus, Experimental - drug therapy | Liver - pathology | Triterpenes - pharmacology | Kidney - pathology | Glycated Hemoglobin A - metabolism | Triterpenes - therapeutic use | Kidney Glomerulus - physiopathology | Male | NF-kappa B - metabolism | Kidney Function Tests | Diabetes Mellitus, Experimental - blood | Adipose Tissue - metabolism | Albuminuria - physiopathology | Kidney - metabolism | Liver - drug effects | Lipids - blood | Inflammation Mediators - metabolism | Lipid Peroxidation - drug effects | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Diabetes Mellitus, Experimental - metabolism | Fatty Acids - metabolism | Kidney - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Albuminuria - pathology | NF-kappa B - antagonists & inhibitors | Kidney - drug effects | Podocytes - metabolism | Cytokines - metabolism | Kidney Glomerulus - drug effects | Liver - metabolism | Adipose Tissue - pathology | Cells, Cultured | Insulin Resistance | Kidney Glomerulus - pathology | Podocytes - pathology | Animals | Podocytes - drug effects | Mice | Oxidative Stress - drug effects | Adipose Tissue - drug effects | Transcription factors | Nephrology | Adipose tissue | Disease | Syngeneic grafts | Oxidative metabolism | Liver | Body weight | Neuropeptides | Adipocytes | Glucose | Animal tissues | Lipid metabolism | Obesity | Medical research | NF-κB protein | Cytokines | Internal medicine | Mortality | Abnormalities | Polyamide-imides | Metabolism | Insulin | Fatty acids | Inhibitors | Nephropathy | Weight reduction | Metabolic disorders | Structure-function relationships | Oxidative stress | Animal models | Homeostasis | Adiponectin | Oxidation resistance | Rodents | Collagen (type IV) | Creatinine | Urine | Hypertension | Excretion | Fasting | Kidneys | Diabetes mellitus | Organs | Inflammation | Medicine | Insulin resistance | Diabetes | Autoimmune diseases | Index Medicus
Journal Article
Journal of cell science, ISSN 0021-9533, 01/2014, Volume 127, Issue 1, pp. 158 - 171
Here, we show that epithelial-mesenchymal status influences how cells deposit extracellular matrix. Retinal pigmented epithelial (RPE) cells that expressed... 
Fibrillin-1 | Cell-cell junction | Integrin | Perlecan | Epithelial cell | Mesenchymal cell | Fibronectin | Syndecan | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Retinal Pigment Epithelium - metabolism | Epithelial Cells - metabolism | Cadherins - metabolism | Humans | Syndecan-4 - genetics | Actins - metabolism | Integrins - genetics | Epithelial-Mesenchymal Transition - genetics | Mammary Glands, Human - metabolism | Fibroblasts - ultrastructure | Heparitin Sulfate - metabolism | Integrins - metabolism | Actins - genetics | Actomyosin - genetics | Retinal Pigment Epithelium - ultrastructure | Female | Microfilament Proteins - metabolism | Zonula Occludens-1 Protein - metabolism | Cadherins - genetics | Microfilament Proteins - genetics | Fibroblasts - metabolism | Microfibrils - ultrastructure | Zonula Occludens-1 Protein - genetics | Cell Line | Podocytes - metabolism | Syndecan-4 - metabolism | Gene Expression Regulation | Intercellular Junctions - metabolism | Epithelial Cells - ultrastructure | Fibrillins | Organ Specificity | Actomyosin - metabolism | beta Catenin - metabolism | Fibronectins - metabolism | beta Catenin - genetics | Transforming Growth Factor beta - pharmacology | Podocytes - ultrastructure | Mammary Glands, Human - ultrastructure | Fibronectins - genetics | Intercellular Junctions - ultrastructure | Microfibrils - metabolism | Index Medicus | Cell–cell junction
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 12, pp. 4375 - 4387
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 2009, Volume 20, Issue 12, pp. 2593 - 2603
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 03/2016, Volume 65, Issue 3, pp. 755 - 767
Overcoming refractory massive proteinuria remains a clinical and research issue in diabetic nephropathy. This study was designed to investigate the... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Kidney - pathology | Microtubule-Associated Proteins - genetics | Diabetic Nephropathies - etiology | Humans | Middle Aged | Rats, Long-Evans | Male | Intracellular Signaling Peptides and Proteins - metabolism | Diabetes Mellitus, Type 2 - metabolism | Proteinuria - pathology | Autophagy | Young Adult | Kidney - metabolism | Lysosomes - metabolism | Diet, High-Fat | Adult | Female | Lysosomes - pathology | Diabetes Mellitus, Experimental - complications | Membrane Proteins - metabolism | Microfilament Proteins - metabolism | RNA-Binding Proteins - blood | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Type 2 - complications | Intravital Microscopy | Proteinuria - etiology | Severity of Illness Index | Cell Line | Microscopy, Electron, Transmission | Diabetic Nephropathies - pathology | Microscopy, Electron, Scanning | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Proteinuria - metabolism | Rats | Mice, Transgenic | Blotting, Western | Mice, Knockout | Autophagy-Related Protein 7 | Animals | Autophagy-Related Protein 5 | Diabetes Mellitus, Experimental - pathology | Aged | Mice | Diabetes Mellitus, Type 2 - pathology | Apoptosis | Care and treatment | Diabetic nephropathies | Homeostasis | Influence | Dosage and administration | Research | Insulin | Proteinuria | Index Medicus | Abridged Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 12/2011, Volume 6, Issue 12, pp. e28710 - e28710
Actin dynamics has emerged at the forefront of podocyte biology. Slit diaphragm junctional adhesion protein Nephrin is necessary for development of the... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Phosphorylation | Phosphatidylinositol Phosphates - metabolism | Humans | Actins - metabolism | Inositol Polyphosphate 5-Phosphatases | Gene Knockdown Techniques | Phosphotyrosine - metabolism | Multiprotein Complexes - metabolism | Cell Membrane - metabolism | Membrane Proteins - metabolism | Microfilament Proteins - metabolism | Contractile Proteins - metabolism | Podocytes - metabolism | Signal Transduction | Oncogene Proteins - metabolism | Protamines - metabolism | Podocytes - pathology | p21-Activated Kinases - metabolism | Filamins | src Homology Domains | Animals | Carrier Proteins - metabolism | Pseudopodia - metabolism | Models, Biological | Protein Binding | Mice | Adaptor Proteins, Signal Transducing - metabolism | Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases | Phosphoric Monoester Hydrolases - metabolism | RNA, Small Interfering - metabolism | Polymerization | Fc receptors | Inositol | Phosphatases | Muscle proteins | Actin | Cell culture | GTP-binding protein | Nephrology | Leukocyte migration | Kinases | Phosphatase | Medical schools | Cell adhesion & migration | Proteins | Cell activation | Cell growth | Epidermal growth factor | Dynamics | Fibroblasts | Diaphragm (anatomy) | Inositol polyphosphate 5-phosphatase | Trends | myo-Inositol-1 (or 4)-monophosphatase | p21-activated kinase | Cell division | Crosslinking | Pseudopodia | Breast cancer | Clustering | Adhesion | Dynamic tests | CD16 antigen | Morphology | Cytoskeleton | Ligands | Mutation | Diaphragm | Cell migration | Index Medicus | Activation | Myo-Inositol-1 (or 4)-monophosphatase
Journal Article
PloS one, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, pp. e25065 - e25065
Injury to the glomerular podocyte is a key mechanism in human glomerular disease and podocyte repair is an important therapeutic target. In Fabry disease,... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Surface Plasmon Resonance | alpha-Galactosidase - genetics | Cell Membrane | Iodine Radioisotopes | Fabry Disease - genetics | Humans | Adaptor Proteins, Vesicular Transport - genetics | Male | alpha-Galactosidase - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Immunoenzyme Techniques | Receptor, IGF Type 2 - metabolism | Receptor, IGF Type 2 - genetics | Kidney - metabolism | Insulin-Like Growth Factor II - genetics | Adult | Low Density Lipoprotein Receptor-Related Protein-2 - genetics | Recombinant Proteins - metabolism | Trihexosylceramides - metabolism | Podocytes - metabolism | RNA, Messenger - genetics | Cells, Cultured | Low Density Lipoprotein Receptor-Related Protein-2 - metabolism | Kidney - cytology | Recombinant Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Insulin-Like Growth Factor II - metabolism | Endocytosis - physiology | Podocytes - cytology | Fabry Disease - metabolism | Phosphates | Globotriaosylceramide | Drug delivery systems | Disease | Galactosidase | Biochemistry | Mannose | Biology | Drug delivery | Insulin-like growth factors | Proteins | Receptors | Endocytosis | Labeling | Localization | Carbohydrates | Antigens | Enzymes | Kidneys | Organs | Insulin | Patients | Fabry's disease | Biopsy | Intracellular | Kidney transplantation | Index Medicus
Journal Article