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Journal Article
American Journal of Pathology, ISSN 0002-9440, 11/2013, Volume 183, Issue 5, pp. 1364 - 1374
Journal Article
Current Opinion in Nephrology and Hypertension, ISSN 1062-4821, 11/2017, Volume 26, Issue 6, pp. 450 - 459
Journal Article
American Journal of Pathology, ISSN 0002-9440, 03/2013, Volume 182, Issue 3, pp. 727 - 741
Dyslipidemia is a frequent component of the metabolic disorder of diabetic patients contributing to organ damage. Herein, in low-density lipoprotein... 
INHIBITS DEVELOPMENT | XANTHINE OXIDOREDUCTASE | FOAM CELL-FORMATION | NUCLEAR RECEPTORS | MACROPHAGES | ATHEROSCLEROSIS | DIABETIC-NEPHROPATHY | MODIFIED LIPOPROTEINS | DEFICIENT MICE | PATHOLOGY | LXR-ALPHA | Inflammation - pathology | Hyperlipidemias - genetics | Kidney - pathology | Humans | Hyperglycemia - complications | Orphan Nuclear Receptors - metabolism | Hyperglycemia - genetics | Kidney Function Tests | Mesangial Cells - drug effects | Endothelial Cells - ultrastructure | Diabetic Nephropathies - physiopathology | Liver X Receptors | Hyperglycemia - pathology | Mesangial Cells - ultrastructure | Lipid Metabolism - genetics | Diabetes Mellitus, Experimental - complications | Hyperglycemia - physiopathology | Hyperlipidemias - physiopathology | Hyperlipidemias - complications | Kidney - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Diabetic Nephropathies - pathology | Kidney - drug effects | Podocytes - metabolism | Cytokines - metabolism | Macrophages - pathology | Mice, Inbred C57BL | Oxidative Stress - genetics | Podocytes - pathology | Kidney - ultrastructure | Diabetic Nephropathies - complications | Macrophages - metabolism | Podocytes - ultrastructure | Animals | Podocytes - drug effects | Fibrosis | Lipid Metabolism - drug effects | Mesangial Cells - pathology | Benzoates - pharmacology | Diabetes Mellitus, Experimental - pathology | Hyperlipidemias - pathology | Macrophages - drug effects | Mice | Oxidative Stress - drug effects | Endothelial Cells - pathology | Benzylamines - pharmacology | Endothelial Cells - drug effects
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 06/2015, Volume 308, Issue 11, pp. F1276 - F1287
Journal Article
Kidney International, ISSN 0085-2538, 06/2006, Volume 69, Issue 12, pp. 2131 - 2147
The terminally differentiated podocyte, also called glomerular visceral epithelial cell, are highly specialized cells. They function as a critical size and... 
glomerular epithelial cell | apoptosis | nephrin | proliferation | cell cycle | podocyte | Podocyte | Nephrin | Glomerular epithelial cell | Proliferation | Cell cycle | Apoptosis | FOOT PROCESS EFFACEMENT | GLOMERULAR EPITHELIAL-CELL | PASSIVE HEYMANN NEPHRITIS | PUROMYCIN AMINONUCLEOSIDE NEPHROSIS | EXPERIMENTAL MEMBRANOUS NEPHROPATHY | EXPERIMENTAL DIABETIC-NEPHROPATHY | CYCLE REGULATORY PROTEINS | HIV-ASSOCIATED NEPHROPATHY | UROLOGY & NEPHROLOGY | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | ENDOTHELIAL GROWTH-FACTOR | Cell Proliferation | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Cell Count | Humans | Diabetic Nephropathies - drug therapy | Proteinuria - pathology | Adrenal Cortex Hormones - therapeutic use | Hypertrophy - pathology | Angiotensin II Type 1 Receptor Blockers - pharmacology | Hypertrophy - physiopathology | Diabetic Nephropathies - physiopathology | Glomerular Basement Membrane - pathology | Glomerulosclerosis, Focal Segmental - drug therapy | Glomerular Basement Membrane - physiopathology | Glomerular Basement Membrane - ultrastructure | Glomerulosclerosis, Focal Segmental - physiopathology | Tretinoin - pharmacology | Endothelium - pathology | Diabetic Nephropathies - pathology | Cytoskeleton - pathology | Adrenal Cortex Hormones - pharmacology | Cytoskeleton - ultrastructure | Podocytes - pathology | Endothelium - physiopathology | Proteinuria - physiopathology | Proteinuria - drug therapy | Tretinoin - therapeutic use | Podocytes - drug effects | Podocytes - physiology | Cell Adhesion - physiology | Cell Cycle - physiology | Cytoskeleton - physiology | Apoptosis - physiology | DNA Damage | Glomerulosclerosis, Focal Segmental - pathology
Journal Article
Kidney International, ISSN 0085-2538, 06/2018, Volume 93, Issue 6, pp. 1356 - 1366
Immunoglobulin A (IgA) nephropathy (IgAN), the most common glomerulonephritis worldwide, is characterized by IgA depositions in the kidney. Deficiency of CD37,... 
CD37 | IgA nephropathy | glomerulonephritis | IL-6 | CD151 | RECEPTOR | TETRASPANIN CD37 PROTECTS | B-CELL LYMPHOMA | DISEASE | MESANGIAL CELLS | UROLOGY & NEPHROLOGY | GROWTH-FACTOR | KIDNEY | Cell Proliferation | Humans | Glomerulonephritis, IGA - immunology | Kidney Glomerulus - physiopathology | Neutrophil Infiltration | Tetraspanins - blood | Antigens, CD - genetics | Case-Control Studies | Tetraspanins - deficiency | Antigens, Neoplasm - blood | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Tetraspanins - genetics | Interleukin-6 - metabolism | B-Lymphocytes - metabolism | Glomerulonephritis, IGA - pathology | Disease Models, Animal | Antigens, Neoplasm - genetics | Genetic Predisposition to Disease | Immunoglobulin A - metabolism | Podocytes - metabolism | Interleukin-6 - genetics | Mice, Inbred C57BL | Interleukin-6 - deficiency | Kidney Glomerulus - pathology | Podocytes - pathology | Albuminuria - prevention & control | Mice, Knockout | Glomerulonephritis, IGA - prevention & control | Phenotype | Animals | B-Lymphocytes - immunology | Immunoglobulin A - immunology | Podocytes - immunology | Albuminuria - immunology | Glomerulonephritis, IGA - metabolism | Kidney Glomerulus - immunology | Glomerulonephritis | Flow cytometry | Renal function | Kidneys | Cytokines | Neutrophils | Tumor necrosis factor-α | Nitrogen | Immunoglobulin A | Experiments | Endothelial cells | Lipopolysaccharides | Interleukin 6 | Proteins | Pathology | Cell activation | Microscopy | Lymphocytes B | Rodents | Interleukin 10 | Renal failure | Tumor necrosis factor-TNF | Immune system
Journal Article
Nature Medicine, ISSN 1078-8956, 06/2017, Volume 23, Issue 6, pp. 753 - 762
Journal Article
Frontiers in endocrinology, ISSN 1664-2392, 2019, Volume 10, p. 528
Journal Article
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2009, Volume 53, Issue 5, pp. S35 - S42
Hyperglycemia can promote vascular complications by multiple mechanisms, with formation of advanced glycation end products and increased oxidative stress... 
Cardiovascular | Internal Medicine | atherosclerosis | glucose | inflammation | advanced glycation end products | oxidative stress | PROTEIN-KINASE-C | CARDIAC & CARDIOVASCULAR SYSTEMS | INCREASED EXPRESSION | SUPEROXIDE-PRODUCTION | INSULIN-RESISTANCE | HIGH GLUCOSE | ADHESION MOLECULE-1 ICAM-1 | PHENOTYPIC HETEROGENEITY | CORONARY-ARTERY-DISEASE | ENDOTHELIAL GROWTH-FACTOR | GLYCATION END-PRODUCTS | Cardiovascular Diseases - etiology | Inflammation - pathology | Reactive Oxygen Species | Oxidative Stress | Humans | Microvessels - pathology | Podocytes - pathology | Cardiovascular Diseases - pathology | Diabetes Complications - blood | Diabetes Complications - etiology | Neovascularization, Pathologic - complications | Inflammation - complications | Diabetes Mellitus, Type 2 - blood | Pericytes - pathology | Cardiovascular Diseases - blood | Endothelium, Vascular - pathology | Glycation End Products, Advanced - blood | Diabetes Complications - pathology | Diabetes Mellitus, Type 2 - pathology | Diabetes Mellitus, Type 2 - complications | Type 2 diabetes | Blood sugar | Lipids | Protein C | Endothelium | Animal pigments | Hyperglycemia | Synthesis | Atherosclerosis | Armored personnel carriers | Neovascularization | Kidney diseases | Cardiovascular diseases | Vascular endothelial growth factor | Blood circulation disorders | Wound healing | Muscular system | Kinases | Nitric oxide | Rodents
Journal Article