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The EMBO Journal, ISSN 0261-4189, 12/2007, Volume 26, Issue 24, pp. 5020 - 5032
The signaling lipid molecule 15‐deoxy‐delta 12,14‐prostaglandin J2 (15d‐PGJ2) has multiple cellular functions, including anti‐inflammatory and antineoplastic... 
15d‐PGJ2 | translation | proliferation | inflammation | eIF4A | Translation | Inflammation | Proliferation | 15d-PGJ2 | TRANSFORMATION SUPPRESSOR PDCD4 | KAPPA-B ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | 15-DEOXY-DELTA(12,14)-PROSTAGLANDIN J | BINDING-PROTEIN | INITIATION CODON | CELL BIOLOGY | HEPATITIS-C VIRUS | PATHWAY | STRESS GRANULE FORMATION | PRODUCT PATEAMINE-A | RNA HELICASE EIF4A | Tumor Necrosis Factor-alpha - metabolism | Arachidonic Acid - metabolism | Protein Biosynthesis | Sodium Compounds - metabolism | Hypoglycemic Agents - metabolism | Humans | TNF Receptor-Associated Factor 2 - genetics | Cytoplasmic Granules - chemistry | PPAR gamma - metabolism | Anti-Inflammatory Agents - metabolism | Chromans - metabolism | Poly(A)-Binding Proteins - metabolism | Prostaglandin D2 - metabolism | Cytoplasmic Granules - metabolism | Eukaryotic Initiation Factor-2 - metabolism | Prostaglandin D2 - analogs & derivatives | T-Cell Intracellular Antigen-1 | Eukaryotic Initiation Factor-4A - genetics | Emetine - metabolism | Enzyme Inhibitors - metabolism | Poly(A)-Binding Proteins - genetics | Gene Expression Regulation | TNF Receptor-Associated Factor 2 - metabolism | Protein Synthesis Inhibitors - metabolism | Thiazolidinediones - metabolism | Arsenites - metabolism | Dinoprostone - metabolism | Eukaryotic Initiation Factor-2 - genetics | Inflammation - genetics | Cyclopentanes - metabolism | Signal Transduction - physiology | Prostaglandins A - metabolism | HeLa Cells | Eukaryotic Initiation Factor-4A - metabolism | Lipids | Cellular biology | Molecular biology | Binding sites | Inflammatory diseases
Journal Article
Journal Article
Molecular Cell, ISSN 1097-2765, 06/2018, Volume 70, Issue 6, pp. 1089 - 1100.e8
Journal Article
The FEBS Journal, ISSN 1742-464X, 03/2015, Volume 282, Issue 5, pp. 874 - 890
Journal Article
Molecular Cell, ISSN 1097-2765, 11/2016, Volume 64, Issue 3, pp. 520 - 533
The RNA exosome is fundamental for the degradation of RNA in eukaryotic nuclei. Substrate targeting is facilitated by its co-factor Mtr4p/hMTR4, which links to... 
RNA exosome | PAXT connection | nuclear RNA decay | poly(A) tail | NEXT complex | TELOMERASE RNA | MESSENGER-RNA | CRYPTIC UNSTABLE TRANSCRIPTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RNA-QUALITY-CONTROL | MOLECULAR ARCHITECTURE | TARGETING COMPLEX | POLY(A)-BINDING PROTEIN | 3'-END MATURATION | POLY(A) TAIL LENGTH | BINDING-PROTEINS | CELL BIOLOGY | RNA Helicases - metabolism | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | Humans | RNA, Messenger - metabolism | Cell Nucleus - metabolism | HEK293 Cells | Exosome Multienzyme Ribonuclease Complex - metabolism | RNA Helicases - genetics | Exosome Multienzyme Ribonuclease Complex - genetics | Nuclear Proteins - genetics | Poly(A)-Binding Protein I - genetics | Binding Sites | RNA-Binding Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Carrier Proteins - antagonists & inhibitors | Poly(A)-Binding Protein I - metabolism | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Poly A - genetics | Transcription Factors - genetics | RNA Stability - genetics | Transcription Factors - metabolism | Carrier Proteins - genetics | Poly A - metabolism | Carrier Proteins - metabolism | Cell Nucleus - genetics | Nuclear Proteins - antagonists & inhibitors | Poly(A)-Binding Protein I - antagonists & inhibitors | Protein Binding | HeLa Cells | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Zinc compounds | RNA | Molecular biology | Protein binding
Journal Article
Molecular Cell, ISSN 1097-2765, 11/2009, Volume 36, Issue 4, pp. 571 - 582
Journal Article
Nucleic Acids Research, ISSN 0305-1048, 07/2016, Volume 44, Issue 12, pp. 5743 - 5757
DNA double-strand breaks (DSBs) are toxic lesions, which if improperly repaired can result in cell death or genomic instability. DSB repair is usually... 
FIDELITY | REPAIR PATHWAY | RESECTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CTIP | LIGASE-III | DOUBLE-STRAND BREAKS | DIRECTED REPAIR | BRCA1 | DNA-POLYMERASE-THETA | DAMAGE | RNA, Small Interfering - genetics | Epithelial Cells - metabolism | Humans | DNA Breaks, Double-Stranded | DNA-Directed DNA Polymerase - genetics | DNA-Binding Proteins - metabolism | MRE11 Homologue Protein | BRCA2 Protein - antagonists & inhibitors | BRCA1 Protein - metabolism | Base Sequence | DNA End-Joining Repair | Replication Protein A - genetics | Replication Protein A - antagonists & inhibitors | Biological Assay | Osteoblasts - cytology | Epithelial Cells - cytology | Nuclear Proteins - genetics | Fibroblasts - metabolism | Recombinational DNA Repair | Nuclear Proteins - metabolism | Replication Protein A - metabolism | DNA - metabolism | DNA-Binding Proteins - genetics | Sequence Homology, Nucleic Acid | BRCA1 Protein - genetics | Carrier Proteins - genetics | Poly(ADP-ribose) Polymerases - metabolism | Sequence Alignment | BRCA2 Protein - metabolism | Carrier Proteins - metabolism | Poly(ADP-ribose) Polymerases - genetics | BRCA1 Protein - antagonists & inhibitors | Cell Line, Tumor | Fibroblasts - cytology | DNA-Directed DNA Polymerase - metabolism | Mutation | Osteoblasts - metabolism | BRCA2 Protein - genetics | RNA, Small Interfering - metabolism | Index Medicus | Genome Integrity, Repair and
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 05/2005, Volume 20, Issue 5, pp. 848 - 857
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2015, Volume 112, Issue 27, pp. E3495 - E3504
Damage repair mechanisms at transcriptionally active sites during the G0/G1 phase are largely unknown. To elucidate these mechanisms, we introduced genome... 
Dna damage | Recombination | CSB | Transcription | RNA polymerase II | COMPLEX | recombination | PROTEIN | transcription | DNA damage | HUMAN-CELLS | MULTIDISCIPLINARY SCIENCES | DOUBLE-STRAND BREAKS | PREFERENTIAL REPAIR | CHROMOSOMES | 8-OXOGUANINE | GENE | DEGRADATION | Cell Cycle - genetics | Antigens, Nuclear - metabolism | Humans | DNA Repair Enzymes - genetics | Homologous Recombination | RNA - genetics | Cockayne Syndrome - pathology | DNA-Binding Proteins - metabolism | Poly-ADP-Ribose Binding Proteins | RNA Interference | Rad52 DNA Repair and Recombination Protein - metabolism | Replication Protein A - genetics | DNA Repair Enzymes - metabolism | HEK293 Cells | Cell Cycle Proteins - genetics | Transcription, Genetic | G1 Phase - genetics | Nuclear Proteins - genetics | DNA Helicases - genetics | RNA - metabolism | Rad51 Recombinase - metabolism | Rad51 Recombinase - genetics | Cockayne Syndrome - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Nuclear Proteins - metabolism | Replication Protein A - metabolism | DNA-Binding Proteins - genetics | Blotting, Western | Rad52 DNA Repair and Recombination Protein - genetics | DNA Helicases - metabolism | Microscopy, Confocal | Antigens, Nuclear - genetics | Resting Phase, Cell Cycle - genetics | Ku Autoantigen | DNA Repair | Cockayne Syndrome - genetics | Cell Line, Tumor | Models, Genetic | DNA Damage | HeLa Cells | Physiological aspects | Observations | Genetic recombination | RNA polymerases | Biological Sciences | PNAS Plus
Journal Article
Journal Article