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Science (American Association for the Advancement of Science), ISSN 1095-9203, 2018, Volume 362, Issue 6414, pp. eaat8407 - 557
The pathologic accumulation and aggregation of α-synuclein (α-syn) underlies Parkinson’s disease (PD). The molecular mechanisms by which pathologic α-syn... 
STRAINS | MECHANISMS | PROTEIN | MULTIDISCIPLINARY SCIENCES | AGGREGATION | CELL-DEATH | Recombinant Proteins - metabolism | Parkinson Disease - pathology | Dopaminergic Neurons - pathology | Mice, Inbred C57BL | Poly (ADP-Ribose) Polymerase-1 - metabolism | Recombinant Proteins - genetics | Gene Knockout Techniques | Poly (ADP-Ribose) Polymerase-1 - antagonists & inhibitors | Mice, Knockout | Brain - metabolism | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Poly Adenosine Diphosphate Ribose - metabolism | Animals | Dopaminergic Neurons - metabolism | Cell Death | Brain - pathology | Benzimidazoles - pharmacology | Mice | Parkinson Disease - metabolism | alpha-Synuclein - genetics | Enzyme Activation | Nitric Oxide - metabolism | alpha-Synuclein - metabolism | Poly (ADP-Ribose) Polymerase-1 - genetics | Therapy | Parkinson's disease | Pathogenesis | Disorders | Cognitive ability | Nervous system | Cerebrospinal fluid | Rigidity | Mental depression | Proteins | Fibrillogenesis | Cell activation | Neurodegeneration | Autonomic nervous system | Neostriatum | Biocompatibility | Anxiety | Inhibition | Toxic diseases | Constipation | Movement disorders | Deoxyribonucleic acid--DNA | Neurodegenerative diseases | Mortality | Adenosine diphosphate | Pathology | Cell injury | Molecular modelling | Inhibitors | Nitric oxide | Biomarkers | Toxicity | DNA damage | Activation | Accumulation | Neurotoxicity | Clonal deletion | Feedforward | Phenotypes | Adenosine | Dopamine | Neurons | Poly(ADP-ribose) polymerase | Caspase | Axons | Sleep | Cell death | In vivo methods and tests | Apoptosis | Index Medicus
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