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Nature Cell Biology, ISSN 1465-7392, 04/2013, Volume 15, Issue 4, pp. 373 - 384
Polycomb group (PcG) proteins play important roles in repressing lineage-specific genes and maintaining the undifferentiated state of mouse embryonic stem... 
MOUSE DEVELOPMENT | TARGET GENES | METHYLATION | HUMAN GENOME | HISTONE H3 | CXXC DOMAIN | INK4A/ARF LOCUS | PROTEINS | POLYCOMB REPRESSIVE COMPLEX-2 | PLURIPOTENT | CELL BIOLOGY | RNA, Small Interfering - genetics | Cell Proliferation | Embryonic Stem Cells - cytology | Immunoprecipitation | Luciferases - metabolism | Oligonucleotide Array Sequence Analysis | Polycomb Repressive Complex 1 - metabolism | Molecular Sequence Data | F-Box Proteins - antagonists & inhibitors | Gene Expression Profiling | Promoter Regions, Genetic - genetics | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | Polycomb Repressive Complex 1 - genetics | DNA Methylation | Transfection | Chromatin Immunoprecipitation | SOXB1 Transcription Factors - genetics | Gene Expression Regulation, Developmental | Base Sequence | Jumonji Domain-Containing Histone Demethylases - antagonists & inhibitors | Biomarkers, Tumor - metabolism | Cell Differentiation | Electrophoretic Mobility Shift Assay | Real-Time Polymerase Chain Reaction | Repressor Proteins - metabolism | Pluripotent Stem Cells - cytology | F-Box Proteins - metabolism | Pluripotent Stem Cells - physiology | RNA, Messenger - genetics | Cells, Cultured | Repressor Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Jumonji Domain-Containing Histone Demethylases - genetics | Sequence Homology, Nucleic Acid | Blotting, Western | Embryonic Stem Cells - physiology | Cell Lineage | Animals | Octamer Transcription Factor-3 - metabolism | Fluorescent Antibody Technique | CpG Islands - genetics | Biomarkers, Tumor - genetics | Mice | Genome | F-Box Proteins - genetics | Jumonji Domain-Containing Histone Demethylases - metabolism | Genes, Developmental | Enzymes | Genes | Physiological aspects | Hydrolases | Genetic aspects | Research | Embryonic stem cells
Journal Article
Journal Article
by Fitzgerald, T.W and Gerety, S.S and Jones, W.D and Van Kogelenberg, M and King, D.A and McRae, J and Morley, K.I and Parthiban, V and Al-Turki, S and Ambridge, K and Barrett, D.M and Bayzetinova, T and Clayton, S and Coomber, E.L and Gribble, S and Jones, P and Krishnappa, N and Mason, L.E and Middleton, A and Miller, R and Prigmore, E and Rajan, D and Sifrim, A and Tivey, A.R and Ahmed, M and Akawi, N and Andrews, R and Anjum, U and Archer, H and Armstrong, R and Balasubramanian, M and Banerjee, R and Baralle, D and Batstone, P and Baty, D and Bennett, C and Berg, J and Bernhard, B and Bevan, A.P and Blair, E and Blyth, M and Bohanna, D and Bourdon, L and Bourn, D and Brady, A and Bragin, E and Brewer, C and Brueton, L and Brunstrom, K and Bumpstead, S.J and Bunyan, D.J and Burn, J and Burton, J and Canham, N and Castle, B and Chandler, K and Clasper, S and Clayton-Smith, J and Cole, T and Collins, A and Collinson, M.N and Connell, F and Cooper, N and Cox, H and Cresswell, L and Cross, G and Crow, Y and D'Alessandro, M and Dabir, T and Davidson, R and Davies, S and Dean, J and Deshpande, C and Devlin, G and Dixit, A and Dominiczak, A and Donnelly, C and Donnelly, D and Douglas, A and Duncan, A and Eason, J and Edkins, S and Ellard, S and Ellis, P and Elmslie, F and Evans, K and Everest, S and Fendick, T and Fisher, R and Flinter, F and Foulds, N and Fryer, A and Fu, B and Gardiner, C and Gaunt, L and Ghali, N and Gibbons, R and Gomes Pereira, S.L and Goodship, J and Goudie, D and ... and Deciphering Dev Disorders Study and Deciphering Developmental Disorders Study and The Deciphering Developmental Disorders Study
Nature, ISSN 0028-0836, 03/2015, Volume 519, Issue 7542, pp. 223 - 228
Despite three decades of successful, predominantly phenotype-driven discovery of the genetic causes of monogenic disorders(1), up to half of children with... 
INTELLECTUAL DISABILITY | HUMAN-DISEASE | DE-NOVO MUTATIONS | MULTIDISCIPLINARY SCIENCES | FRAMEWORK | MODEL | AUTISM SPECTRUM DISORDERS | COPY-NUMBER VARIATION | CHILDREN | Rare Diseases - genetics | Humans | Parents | Child, Preschool | Infant | Male | Developmental Disabilities - genetics | Dynamin I - genetics | Mutation, Missense - genetics | Polycomb Repressive Complex 1 - genetics | Gene Expression Regulation, Developmental | Transposases - genetics | Female | Nuclear Proteins - genetics | Child | Developmental Disabilities - diagnosis | Infant, Newborn | Guanine Nucleotide Exchange Factors - genetics | Protein Phosphatase 2 - genetics | Protein-Serine-Threonine Kinases - genetics | United Kingdom | Phosphoproteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Genome, Human - genetics | Homeodomain Proteins - genetics | Zebrafish - genetics | Chromosomal Proteins, Non-Histone - genetics | Carrier Proteins - genetics | DEAD-box RNA Helicases - genetics | Exome - genetics | Animals | Adolescent | Chromosome Aberrations | Genes, Dominant - genetics | Usage | Genomics | Child development deviations | Genetic aspects | Research | Risk factors | Developmental disabilities | Studies | Hypotheses | Genes | Developmental psychology | Families & family life | Genomes | Mutation | Children & youth
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2016, Volume 291, Issue 9, pp. 4523 - 4536
Inhibiting class I histone deacetylases (HDACs) increases energy expenditure, reduces adiposity, and improves insulin sensitivity in obese mice. However, the... 
WHITE FAT | PROTEIN | GENE | BEIGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | EPIGENETIC REGULATION | UPSTREAM ENHANCER | INSULIN SENSITIVITY | EXPRESSION | ADIPOSE-TISSUE | Polycomb Repressive Complex 2 - antagonists & inhibitors | Polycomb Repressive Complex 2 - agonists | Polycomb Repressive Complex 2 - genetics | Adipocytes, Brown - metabolism | Polycomb Repressive Complex 1 - metabolism | Humans | Ion Channels - genetics | Ubiquitin-Protein Ligases - antagonists & inhibitors | Mitochondrial Proteins - genetics | Mitochondrial Proteins - agonists | Adrenergic beta-3 Receptor Agonists - pharmacology | Promoter Regions, Genetic - drug effects | Polycomb Repressive Complex 1 - genetics | Adipocytes, Brown - enzymology | Protein Processing, Post-Translational - drug effects | RNA Interference | Mitochondrial Proteins - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Polycomb Repressive Complex 1 - antagonists & inhibitors | Histone Deacetylase 1 - antagonists & inhibitors | Lysine - metabolism | Histone Deacetylase 1 - genetics | Recombinant Proteins - metabolism | Adipocytes, Brown - drug effects | Mitochondrial Proteins - antagonists & inhibitors | Ubiquitin-Protein Ligases - metabolism | Recombinant Proteins - chemistry | Transcription Factors - antagonists & inhibitors | Transcription Factors - genetics | Mice, Inbred Strains | Enhancer of Zeste Homolog 2 Protein | Ion Channels - antagonists & inhibitors | Gene Expression Regulation - drug effects | Polycomb Repressive Complex 1 - agonists | Transcription Factors - metabolism | Acetylation - drug effects | Animals | Ion Channels - metabolism | Methylation - drug effects | Ion Channels - agonists | Histones - metabolism | Thermogenesis - drug effects | Transcription Factors - agonists | Uncoupling Protein 1 | Polycomb Repressive Complex 2 - metabolism | Ubiquitin-Protein Ligases - genetics | Cell Line, Transformed | Histone Deacetylase 1 - metabolism | histone methylation | thermogenesis | brown adipocytes | UCP1 | histone deacetylation | H3K27 | Metabolism | epigenetics | obesity | histone deacetylase 1 (HDAC1) | uncoupling protein
Journal Article
Journal Article
PLoS Genetics, ISSN 1553-7390, 2014, Volume 10, Issue 7, p. e1004495
Journal Article