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Cancer Research, ISSN 0008-5472, 09/2015, Volume 75, Issue 18, pp. 3720 - 3727
Journal Article
Nature, ISSN 0028-0836, 07/2012, Volume 487, Issue 7408, pp. 491 - 495
Journal Article
CELL, ISSN 0092-8674, 06/2003, Volume 113, Issue 7, pp. 881 - 889
Telomerase expression is repressed in most somatic cells but is observed in stem cells and a high percentage of human cancers and has been hypothesized to... 
CATALYTIC SUBUNIT | LIFE-SPAN | CELLULAR IMMORTALIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | REVERSE-TRANSCRIPTASE | DYSKERATOSIS-CONGENITA | DOWN-REGULATION | MENIN INTERACTS | HUMAN CANCER-CELLS | MAMMALIAN-CELLS | HTERT GENE | CELL BIOLOGY | ras Proteins - genetics | Genetic Testing | Homeodomain Proteins - metabolism | Humans | ras Proteins - metabolism | Phosphoproteins - metabolism | Zinc Finger E-box Binding Homeobox 2 | Telomerase - genetics | RNA Interference | Cell Transformation, Neoplastic - genetics | Genes, Tumor Suppressor - physiology | Tumor Suppressor Proteins - genetics | Telomerase - metabolism | Proto-Oncogene Proteins | Telomere - metabolism | Neoplasm Proteins - genetics | Gene Expression Regulation, Neoplastic - genetics | Repressor Proteins - metabolism | DNA-Binding Proteins | Telomere - genetics | Cell Division - genetics | Eukaryotic Cells - enzymology | Cell Line | Antigens, Polyomavirus Transforming - genetics | Antigens, Polyomavirus Transforming - metabolism | Repressor Proteins - genetics | Phosphoproteins - genetics | Cell Transformation, Neoplastic - metabolism | Homeodomain Proteins - genetics | Nuclear Proteins | Animals | Cell Line, Transformed - metabolism | Neoplasm Proteins - deficiency | Cell Cycle Proteins | Proteins | Cell research | Gene mutations | Analysis | Physiological aspects | Causes of | Tumor suppressor genes | Genetic aspects | Gene expression | Genetic regulation | Telomerase | Tumors | Index Medicus
Journal Article
Journal Article
The Journal of Pathology, ISSN 0022-3417, 07/2014, Volume 233, Issue 3, pp. 228 - 237
Recent evidence suggests that ovarian high‐grade serous carcinoma ( HGSC ) originates from the epithelium of the fallopian tube. However, most mouse models are... 
transgenic mouse model | ovarian cancer | fallopian tube | intraepithelial carcinoma | Top2a | p53 | CONDITIONAL INACTIVATION | CANDIDATE PRECURSOR | TOPOISOMERASE-II-ALPHA | P53 SIGNATURE | TP53 MUTATIONS | PATHOLOGY | COMPREHENSIVE ANALYSIS | BREAST-CANCER | EPITHELIUM | ONCOLOGY | EXPRESSION | Neoplasms, Cystic, Mucinous, and Serous - metabolism | Adenocarcinoma - pathology | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Fallopian Tubes - pathology | Neoplasms, Cystic, Mucinous, and Serous - genetics | Ovarian Neoplasms - genetics | Poly-ADP-Ribose Binding Proteins | Adenocarcinoma - metabolism | Neoplasm Grading | Cell Transformation, Neoplastic - genetics | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Female | Adenocarcinoma - genetics | Ovarian Neoplasms - metabolism | Disease Models, Animal | Glycoproteins - genetics | Antigens, Neoplasm - genetics | DNA Topoisomerases, Type II - metabolism | Promoter Regions, Genetic | Neoplasms, Cystic, Mucinous, and Serous - pathology | Antigens, Polyomavirus Transforming - genetics | Antigens, Polyomavirus Transforming - metabolism | Neoplasm Invasiveness | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Mice, Transgenic | DNA-Binding Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Disease Progression | Fallopian Tubes - metabolism | Animals | Genetic Engineering | DNA Topoisomerases, Type II - genetics | Biomarkers, Tumor - genetics | Mice | Cell Transformation, Neoplastic - pathology | Immunohistochemistry | DNA microarrays | Carcinoma | Analysis | Genetically modified organisms | Genetic engineering | Tumor proteins | Ovarian cancer | Cancer | Medical research | Rodents | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 04/2004, Volume 6, Issue 4, pp. 308 - 318
Journal Article
International Journal of Cancer, ISSN 0020-7136, 08/2013, Volume 133, Issue 3, pp. 604 - 611
Polyomaviruses such as BK virus (BKV), JC virus (JCV) and Merkel cell polyomavirus (MCPyV) are typically nononcogenic, although they have been detected in a... 
Bcl‐2 | BRAF | Merkel cell polyomavirus | non‐small cell lung cancer | Bcl-2 | non-small cell lung cancer | SURVIVAL | APOPTOSIS | VIRUS | PROTEIN | P53 | HUMAN CARCINOGENS | ONCOLOGY | INFLAMMATION | MOLECULAR PATHOGENESIS | INFECTION | MUTATIONS | Tumor Virus Infections - genetics | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Humans | Middle Aged | Male | RNA, Messenger - metabolism | Tumor Suppressor Protein p53 - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Female | Carcinoma, Non-Small-Cell Lung - virology | Merkel cell polyomavirus - isolation & purification | Proto-Oncogene Proteins B-raf - metabolism | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | RNA, Messenger - genetics | Proto-Oncogene Proteins - genetics | Lung Neoplasms - virology | Merkel cell polyomavirus - immunology | Polyomavirus Infections - virology | Proto-Oncogene Proteins B-raf - genetics | Phosphatidylethanolamine Binding Protein - genetics | Polyomavirus Infections - genetics | Aged | DNA, Viral - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Smoking | RNA | Analysis | Genes | Genetic research | Lung cancer, Small cell | Genetic aspects | Tumor proteins | Lung cancer, Non-small cell | Gene expression | Cancer | Polymerase chain reaction | Viruses | Lung cancer | Index Medicus
Journal Article
PLoS Pathogens, ISSN 1553-7366, 06/2013, Volume 9, Issue 6, pp. e1003429 - e1003429
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2008, Volume 118, Issue 1, pp. 64 - 78
overexpression of the receptor tyrosine kinase EPH receptor A2 (EphA2) is commonly observed in aggressive breast cancer and correlates with a poor prognosis.... 
MEDICINE, RESEARCH & EXPERIMENTAL | LIGAND-BINDING | PRIMARY BREAST-CANCER | EPITHELIAL-CELLS | EGF-R | EGFRVIII EXPRESSION | IN-VIVO | GROWTH-FACTOR | PANCREATIC ADENOCARCINOMA | PROGNOSTIC VALUE | TUMOR MICROENVIRONMENT | Adenocarcinoma - pathology | Cell Proliferation | Receptor, ErbB-2 - genetics | Humans | Receptor, EphA2 - metabolism | Receptor, ErbB-2 - metabolism | Male | Mammary Glands, Human - metabolism | Mammary Neoplasms, Experimental - metabolism | Cell Movement - genetics | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Breast Neoplasms - therapy | Neoplasm Metastasis | Adenocarcinoma - metabolism | MAP Kinase Signaling System - genetics | Cell Transformation, Neoplastic - genetics | Mammary Neoplasms, Experimental - pathology | Female | Adenocarcinoma - genetics | Antigens, Polyomavirus Transforming - genetics | Antigens, Polyomavirus Transforming - metabolism | Mammary Glands, Animal - pathology | rho GTP-Binding Proteins - genetics | Mammary Glands, Human - pathology | Mice, Transgenic | Cell Transformation, Neoplastic - metabolism | Mammary Neoplasms, Experimental - therapy | Animals | Breast Neoplasms - genetics | Adenocarcinoma - therapy | Mammary Glands, Animal - metabolism | Breast Neoplasms - pathology | rho GTP-Binding Proteins - metabolism | Receptor, EphA2 - genetics | Mice | Cell Transformation, Neoplastic - pathology | Adenocarcinoma | Polymerase chain reaction | Cell receptors | Genetic aspects | Research | Health aspects | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article