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Journal Article
PLoS ONE, ISSN 1932-6203, 10/2011, Volume 6, Issue 10, pp. e25611 - e25611
Activation-induced cytidine deaminase (AID) is expressed in B cells within germinal centers and is critically involved in class switch recombination and... 
CENTER B-CELLS | CLASS SWITCH RECOMBINATION | NASAL POLYP TISSUE | SOMATIC HYPERMUTATION | IGE ANTIBODIES | BIOLOGY | GENE-EXPRESSION | MONOCLONAL-ANTIBODY | T-CELLS | DNA RECOMBINATION | CHRONIC RHINOSINUSITIS | Nasal Polyps - pathology | Gene Regulatory Networks - genetics | Alternative Splicing - genetics | Cytidine Deaminase - genetics | Humans | RNA, Messenger - genetics | Gene Expression Regulation | Databases, Genetic | Gene Expression Profiling | Nasal Polyps - enzymology | RNA, Messenger - metabolism | Inflammation - etiology | Inflammation - complications | Nasal Polyps - complications | Sinusitis - genetics | Cytidine Deaminase - metabolism | Sinusitis - enzymology | Nasal Polyps - genetics | Sinusitis - pathology | Sinusitis - etiology | Chronic Disease | Inflammation - enzymology | Cluster Analysis | Autoimmunity | Medical research | Genes | Analysis | Immunoglobulin G | Immunoglobulin E | Medicine, Experimental | B cells | Health aspects | Causes and theories of causation | Diseases | Recombination | Disease | Pathogenesis | Rhinosinusitis | Leukemia | Activation-induced cytidine deaminase | Otolaryngology | Activation | Tissues | Immunity | Respiratory tract | Cell activation | CD23 antigen | Interleukin 5 | Immunology | Germinal centers | Lymphocytes | Etiology | Interleukin 1 | Respiratory tract diseases | Deoxyribonucleic acid--DNA | Antigens | Hypersensitivity | Interleukin 13 | Gene expression | Clustering | Polyposis | Biological activity | Allergies | Class switching | Asthma | Somatic hypermutation | Lymphocytes B | Diagnostic software | Diagnostic systems | Aberration | Autoimmune diseases | Polyps | Cytidine deaminase | Cancer | Inflammation | somatic hypermutation | rhinosinusitis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Medicine (United States), ISSN 0025-7974, 09/2017, Volume 96, Issue 38, pp. e7466 - e7466
Journal Article
Cancer Epidemiology Biomarkers and Prevention, ISSN 1055-9965, 10/2011, Volume 20, Issue 10, pp. 2288 - 2292
Journal Article
Journal of the American Academy of Dermatology, ISSN 0190-9622, 2010, Volume 65, Issue 5, pp. 1054 - 1058.e1
Muir-Torre syndrome (MTS) is an autosomal dominant genodermatosis caused by mutations in the DNA mismatch repair genes MLH1 and MSH2. This case describes a... 
Dermatology | microsatellite instability | immunosuppression | Muir-Torre syndrome, sebaceous gland neoplasm | mismatch repair genes | transplantation | ACQUIRED-IMMUNODEFICIENCY-SYNDROME | NEOPLASMS | MISMATCH REPAIR PROTEINS | DERMATOLOGY | Muir-Torre syndrome | SKIN-LESIONS | MSH2 | sebaceous gland neoplasm | GERMLINE MUTATIONS | PATIENT | GLAND TUMORS | ASSOCIATION | Colonic Neoplasms - genetics | Microsatellite Instability | Adenoma - genetics | Carcinoma, Squamous Cell - genetics | Humans | Immunosuppressive Agents - therapeutic use | Middle Aged | Colonic Neoplasms - diagnosis | Adenocarcinoma, Sebaceous - etiology | Male | Facial Neoplasms - genetics | Colonic Polyps - genetics | Colonic Polyps - diagnosis | DNA Mutational Analysis | DNA Mismatch Repair | Colonic Neoplasms - etiology | Neoplasms, Multiple Primary - etiology | Neoplasms, Multiple Primary - genetics | Muir-Torre Syndrome - diagnosis | Colonic Polyps - etiology | Adenoma - etiology | Kidney Transplantation | MutS Homolog 2 Protein - genetics | Codon, Nonsense | Hypertension, Malignant - surgery | Carcinoma, Squamous Cell - etiology | Immunosuppression | Phenotype | Skin Neoplasms - genetics | Graft vs Host Disease - prevention & control | Immunosuppressive Agents - adverse effects | Skin Neoplasms - etiology | Adenocarcinoma, Sebaceous - genetics | Facial Neoplasms - etiology | Colon cancer | Immunotherapy | Analysis | Genes | Transplantation of organs, tissues, etc | Organ transplant recipients | Cancer | Index Medicus
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