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by Li, Jun and Woods, Susan L and Healey, Sue and Beesley, Jonathan and Chen, Xiaoqing and Lee, Jason S and Sivakumaran, Haran and Wayte, Nicci and Nones, Katia and Waterfall, Joshua J and Pearson, John and Patch, Anne-Marie and Senz, Janine and Ferreira, Manuel A and Kaurah, Pardeep and Mackenzie, Robertson and Heravi-Moussavi, Alireza and Hansford, Samantha and Lannagan, Tamsin R.M and Spurdle, Amanda B and Simpson, Peter T and da Silva, Leonard and Lakhani, Sunil R and Clouston, Andrew D and Bettington, Mark and Grimpen, Florian and Busuttil, Rita A and Di Costanzo, Natasha and Boussioutas, Alex and Jeanjean, Marie and Chong, George and Fabre, Aurélie and Olschwang, Sylviane and Faulkner, Geoffrey J and Bellos, Evangelos and Coin, Lachlan and Rioux, Kevin and Bathe, Oliver F and Wen, Xiaogang and Martin, Hilary C and Neklason, Deborah W and Davis, Sean R and Walker, Robert L and Calzone, Kathleen A and Avital, Itzhak and Heller, Theo and Koh, Christopher and Pineda, Marbin and Rudloff, Udo and Quezado, Martha and Pichurin, Pavel N and Hulick, Peter J and Weissman, Scott M and Newlin, Anna and Rubinstein, Wendy S and Sampson, Jone E and Hamman, Kelly and Goldgar, David and Poplawski, Nicola and Phillips, Kerry and Schofield, Lyn and Armstrong, Jacqueline and Kiraly-Borri, Cathy and Suthers, Graeme K and Huntsman, David G and Foulkes, William D and Carneiro, Fatima and Lindor, Noralane M and Edwards, Stacey L and French, Juliet D and Waddell, Nicola and Meltzer, Paul S and Worthley, Daniel L and Schrader, Kasmintan A and Chenevix-Trench, Georgia
The American Journal of Human Genetics, ISSN 0002-9297, 05/2016, Volume 98, Issue 5, pp. 830 - 842
Journal Article
Gut, ISSN 0017-5749, 01/2017, Volume 66, Issue 1, pp. 97 - 106
Objective Sessile serrated adenomas (SSAs) are the precursors of at least 15% of colorectal carcinomas, but their biology is incompletely understood. We... 
COLONIC POLYPS | COLON CARCINOGENESIS | COLORECTAL CANCER GENES | COLONIC NEOPLASMS | ISLAND METHYLATOR PHENOTYPE | COLON-CANCER | COLONOSCOPY | HYPERPLASTIC POLYPS | EARLY NEOPLASTIC PROGRESSION | BRAF MUTATION | PATHWAY | COLORECTAL-CANCER | MICROSATELLITE INSTABILITY | POOR SURVIVAL | GASTROENTEROLOGY & HEPATOLOGY | Adenoma - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Colorectal Neoplasms - genetics | Humans | Middle Aged | Male | Tumor Suppressor Protein p14ARF - analysis | Carcinoma - chemistry | Young Adult | Colonic Polyps - genetics | Cell Transformation, Neoplastic - genetics | Aged, 80 and over | Neoplastic Syndromes, Hereditary - genetics | Wnt Signaling Pathway | Brain Neoplasms - genetics | Tumor Burden | DNA Repair Enzymes - analysis | Phenotype | MutL Protein Homolog 1 - genetics | CpG Islands | Carcinoma - genetics | Mutation | Age Factors | beta Catenin - analysis | Colonic Polyps - pathology | DNA Repair Enzymes - genetics | Colorectal Neoplasms - chemistry | Tumor Suppressor Protein p53 - genetics | Tumor Suppressor Protein p53 - analysis | Tumor Suppressor Proteins - genetics | Adult | Female | Carcinoma - pathology | DNA Modification Methylases - analysis | Cross-Sectional Studies | Gene Silencing | Adenoma - chemistry | Colonic Polyps - chemistry | beta Catenin - genetics | MutL Protein Homolog 1 - analysis | DNA Modification Methylases - genetics | Proto-Oncogene Proteins B-raf - genetics | Sex Factors | Adenoma - pathology | Aged | Tumor Suppressor Protein p14ARF - genetics | Cell Transformation, Neoplastic - pathology | Colorectal Neoplasms - pathology | Tumor Suppressor Proteins - analysis | Dysplasia | Usage | Carcinoma | Diagnosis | Clinical pathology | Cancer | DNA methylation | Bias | Deoxyribonucleic acid--DNA | Tumors | Index Medicus | Abridged Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 2, pp. 520 - 529.e6
Background & Aims Little is known about the genetic factors that contribute to the development of sessile serrated adenomas (SSAs). SSAs contain somatic... 
Gastroenterology and Hepatology | Hereditary Colon Cancer | RNF43 | Sessile Serrated Polyp | Serrated Polyposis Syndrome | PROTEIN | TUMOR PROGRESSION | OF-FUNCTION VARIANTS | RISK | BRAF | CANCER | RARE VARIANTS | XAF1 | GASTROENTEROLOGY & HEPATOLOGY | HYPERPLASTIC POLYPOSIS | REVEALS | Oncogene Proteins - genetics | ras Proteins - genetics | Colonic Neoplasms - genetics | Proto-Oncogene Proteins p21(ras) | Prospective Studies | Proto-Oncogene Proteins c-ets - genetics | Adenomatous Polyps - genetics | Adenomatous Polyps - pathology | Genomics | Humans | Middle Aged | Male | Retinoblastoma-Like Protein p107 - genetics | Case-Control Studies | DNA-Binding Proteins - metabolism | Exome | Germ-Line Mutation | Adult | Female | Precancerous Conditions - pathology | Neoplasm Proteins - genetics | DNA Helicases - genetics | Intracellular Signaling Peptides and Proteins - genetics | Cellular Senescence - genetics | Genetic Predisposition to Disease | Genetic Association Studies | Oncogene Proteins - metabolism | Proto-Oncogene Proteins - genetics | Codon, Nonsense | Genetic Markers | DNA-Binding Proteins - genetics | Precancerous Conditions - genetics | Sequence Analysis, DNA | Proto-Oncogene Proteins B-raf - genetics | Colonic Neoplasms - pathology | Aged | Ataxia Telangiectasia Mutated Proteins - genetics | Medical colleges | Genetic aspects | Gene mutations | Analysis | Index Medicus | Abridged Index Medicus | hereditary colon cancer | serrated polyposis syndrome | sessile serrated polyp
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 09/2011, Volume 35, Issue 9, pp. 1274 - 1286
Abundant recent data suggest that sessile serrated adenoma/polyp (SSA/P) is an early precursor lesion in the serrated pathway of carcinogenesis. It is believed... 
dysplasia | serrated adenoma | cancer | polyp | pathology | SURGERY | CPG ISLAND METHYLATION | MICROSATELLITE INSTABILITY | PHENOTYPE | PREVALENCE | KRAS MUTATIONS | BRAF MUTATION | PATHWAY | EXPRESSION | ADENOMAS | MutL Protein Homolog 1 | United States - epidemiology | Proto-Oncogene Proteins p21(ras) | Adenoma - genetics | Colorectal Neoplasms - genetics | Humans | Hyperplasia | Middle Aged | Male | Young Adult | Colonic Polyps - genetics | Cadherins - genetics | Intestinal Polyps - ethnology | Genes, p16 | Intestinal Polyps - pathology | Mutation | Adenoma - ethnology | ras Proteins - genetics | Microsatellite Instability | Colonic Polyps - pathology | Asian Continental Ancestry Group - genetics | DNA Repair Enzymes - genetics | Gene Expression Regulation, Neoplastic | Rectal Diseases - pathology | Colonic Polyps - ethnology | Genes, APC | Rectal Diseases - ethnology | DNA Methylation | Intestinal Polyps - genetics | Chromatin Immunoprecipitation | Tumor Suppressor Proteins - genetics | Polymerase Chain Reaction | Adult | Female | Nuclear Proteins - genetics | Rectal Diseases - genetics | Republic of Korea - epidemiology | Colorectal Neoplasms - ethnology | Linear Models | Proto-Oncogene Proteins - genetics | Chi-Square Distribution | Nerve Tissue Proteins - genetics | Carrier Proteins - genetics | DNA Modification Methylases - genetics | Proto-Oncogene Proteins B-raf - genetics | Adaptor Proteins, Signal Transducing - genetics | Adenoma - pathology | Aged | Colorectal Neoplasms - pathology | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2010, Volume 207, Issue 8, pp. 1625 - 1636
Signaling through the adaptor protein myeloid differentiation factor 88 (MyD88) promotes carcinogenesis in several cancer models. In contrast, MyD88 signaling... 
EXPRESSION PATTERNS | MEDICINE, RESEARCH & EXPERIMENTAL | CHRONIC INTESTINAL INFLAMMATION | MUTANT MICE | SODIUM-SULFATE COLITIS | CELL LINES | COLITIS-ASSOCIATED CANCER | IMMUNOLOGY | BETA-CATENIN | TUMORIGENESIS | INNATE IMMUNITY | BOWEL-DISEASE | Colonic Neoplasms - genetics | Intestinal Mucosa - metabolism | Adenocarcinoma - pathology | Azoxymethane - pharmacology | Epithelial Cells - metabolism | Gene Expression - drug effects | Apoptosis - drug effects | Gene Expression - genetics | Adenocarcinoma - chemically induced | Colonic Polyps - pathology | Epithelial Cells - drug effects | Colon - drug effects | DNA Repair Enzymes - genetics | Apoptosis - genetics | Colonic Neoplasms - chemically induced | Gene Expression Profiling | Inflammatory Bowel Diseases - metabolism | Colonic Neoplasms - metabolism | Intestinal Mucosa - drug effects | Adenocarcinoma - metabolism | Cyclooxygenase 2 - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Interleukin-18 Receptor alpha Subunit - genetics | Inflammatory Bowel Diseases - chemically induced | Adenocarcinoma - genetics | Phosphorylation - drug effects | STAT3 Transcription Factor - genetics | Dextran Sulfate - pharmacology | Genetic Predisposition to Disease - genetics | Specific Pathogen-Free Organisms | Colon - pathology | Mice, Inbred C57BL | Epithelial Cells - pathology | Mutation - genetics | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Animals | Colonic Neoplasms - pathology | Receptors, Interleukin-1 Type I - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | Interleukin-18 - genetics | Myeloid Differentiation Factor 88 - metabolism | Interleukin-18 - metabolism | Intestinal Mucosa - pathology | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 2, pp. 248 - 256
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 7, pp. e11450 - e11450
Background: Asthmatic chronic rhinosinusitis with nasal polyps (aCRSwNP) is a common disruptive eosinophilic disease without effective medical treatment.... 
RECRUITMENT | RESPONSES | INCREASED EXPRESSION | MICROARRAY | INFLAMMATION | EOSINOPHILS | BIOLOGY | ALLERGIC RHINITIS | CYTOKINE | DIFFERENTIATION | T-CELLS | Immunohistochemistry | Interleukin-1 Receptor-Like 1 Protein | Dermatitis, Atopic - genetics | Salivary Proteins and Peptides - genetics | Humans | Middle Aged | Receptors, Peptide - genetics | Young Adult | Sinusitis - genetics | Adult | Nasal Polyps - metabolism | Glycoproteins - genetics | Nasal Polyps - pathology | Chemokine CCL11 - genetics | Chemokine CCL2 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Radioimmunoprecipitation Assay | Asthma - genetics | Membrane Glycoproteins - genetics | Monocyte Chemoattractant Proteins - genetics | Chemokine CCL22 - genetics | Rhinitis - genetics | Adolescent | Chemokines, CC - genetics | Chemokine CCL8 - genetics | Receptors, G-Protein-Coupled - genetics | Seminal Plasma Proteins - genetics | Transcription, Genetic - genetics | Mucins - genetics | Receptors, Cell Surface - genetics | Advertising executives | Messenger RNA | Platelet-derived growth factor | Atopic dermatitis | Analysis | Genes | Genetic aspects | Inflammation | Genetic transcription | Nasal polyps | Asthma | Immunoassay | Rhinitis | Transcription | Disease | Laboratories | Rhinosinusitis | Mucosa | Medical services | CXCL13 protein | Otolaryngology | Smooth muscle | Lymphocytes T | Genomes | Dermatitis | CCL22 protein | Anaphylaxis | Transcription activation | Nose | Interleukin 1 | CCL18 protein | Endoscopy | Informatics | Chromosome 17 | Chronic illnesses | Cytokines | Dendritic cells | Leukocytes (eosinophilic) | Medical treatment | RNA polymerase | Gene expression | Chemotaxis | Allergies | Monocytes | Diagnostic systems | Arrays | Polyps | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 04/2014, Volume 370, Issue 14, pp. 1287 - 1297
Journal Article
Genes and Development, ISSN 0890-9369, 11/2010, Volume 24, Issue 21, pp. 2383 - 2388
Although a developmental role for Hippo signaling in organ size control is well appreciated, how this pathway functions in tissue regeneration is largely... 
Regeneration | Hippo signaling | Mouse | Growth control | Cancer | regeneration | ORGAN SIZE CONTROL | STAT3 | YAP | CELL-PROLIFERATION | DEVELOPMENTAL BIOLOGY | DROSOPHILA | CELL BIOLOGY | mouse | EPITHELIAL-CELLS | COLITIS | LIVER | GENETICS & HEREDITY | growth control | cancer | TUMORIGENESIS | Immunohistochemistry | Dextran Sulfate | Humans | Male | Intestines - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Drosophila Proteins - metabolism | RNA, Messenger - metabolism | Colonic Polyps - physiopathology | Colonic Polyps - genetics | Regeneration - genetics | Cell Cycle Proteins - genetics | Female | Phosphoproteins - physiology | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Gene Expression | Intestines - pathology | Mice, Inbred C57BL | RNA, Messenger - genetics | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Signal Transduction - genetics | Phosphoproteins - genetics | Regeneration - physiology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Colonic Polyps - chemically induced | Mice, Knockout | Adaptor Proteins, Signal Transducing - physiology | Animals | Adaptor Proteins, Signal Transducing - deficiency | Phosphoproteins - deficiency | Adaptor Proteins, Signal Transducing - genetics | Signal Transduction - physiology | Mice | Drosophila Proteins - genetics | Intestines - physiopathology | Dextran | Usage | Analysis | Genetic aspects | Chemical properties | Research | Gene expression | Index Medicus | Research Communication
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9061 - e9061
Journal Article